ライフサイエンスコーパス: hair dye

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1 manent, semipermanent, temporary, or gradual hair dyes.

2 Using hair dye and rhodamine labeling we established that the

3 igated associations between use of synthetic hair dyes and risk of brain tumors in a hospital-based c

4 is are chemicals and agricultural exposures, hair dyes, and blood transfusions.

5 s, medical conditions, pesticides, solvents, hair dyes, and diet.

6 history of hematopoietic cancer, and use of hair dyes are associated with t(14;18)-defined subsets o

7 Certain chemicals in hair dyes are known animal carcinogens.

8 ed as never-users or ever-users of permanent hair dye, based on self-report.

9 For women who started using hair dye before 1980 as compared with never users, a sta

10 use was observed among women who began using hair dye before 1980.

11 ainly observed among women who started using hair dyes before 1980.

12 Although use of permanent brown hair dye for 20 or more years was associated with glioma

13 observed association reflects the change in hair dye formula contents during the past two decades or

14 Personal use of hair dye has been inconsistently linked to risk of non-H

15 For women who began using hair dye in 1980 or afterward, increased FL risk was lim

16 mbranous ventricular septal defect, 5.5%; 7) hair dye in multiple/multiplex membranous ventricular se

17 tions were found for women who started using hair dyes in 1980 or afterward.

18 ommon allergens include fragrance chemicals, hair dyes, metals, rubber accelerators and preservatives

19 es (pOR=2.2, 95% CI=1.0-4.7), have ever used hair dyes (pOR=4.5, 95% CI=1.3-15.4), and have had a gre

20 drogen peroxide concentrations in commercial hair dye products, which is a significant issue in the h

21 Compared with never-users of permanent hair dye, the age-adjusted relative risks (RR) for the d

22 ecause of the complexity of chemicals in the hair dye, the consecutive reaction monitoring of PhIP at

23 odds ratio was for 15 or more years of using hair dyes up to six times per year (odds ratio = 2.4, 95

24 pathway genes modifies the relation between hair dye use and risk of non-Hodgkin lymphoma.

25 tly increased risk was observed for starting hair dye use before 1980 (relative to never use) among w

26 obiotic metabolism in the carcinogenicity of hair dye use needs to be confirmed in larger studies.

27 Hair dye use was not associated with either subtype.

28 Duration of hair dye use was not related to risk of SLE.

29 es have suggested an association of personal hair dye use with bladder and hematopoietic cancers.

30 ent evidence for an association of synthetic hair dye use with glioma, meningioma, or acoustic neurom

31 We found no evidence that permanent hair dye use, age at first use, frequency of use, or dur

32 d nonpermanent (semipermanent and temporary) hair dye use.

33 r a lack of detailed information on personal hair-dye use in previous studies.

34 These results indicate that personal hair-dye use may play a role in risks of FL and CLL/SLL

35 nce interval (CI): 1.1, 1.4) associated with hair-dye use was observed among women who began using ha

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