ライフサイエンスコーパス: hair germ

コーパス検索結果 (１語後でソート)

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1 nner root sheaths and later in the secondary hair germ.

2 al and suprabasal cells above and around the hair germ.

3 found in the hair canal, sebaceous gland, or hair germ.

4 between early superficial BCCs and embryonic hair germs.

5 y negatively regulates Lgr5 in the secondary hair germ and inhibits HF cycling.

6 press keratin 79 (K79) and stream out of the hair germ and into the epidermis prior to lumen formatio

7 ed epithelial compartments (bulge, secondary hair germ) and co-express selected stem cell markers (So

8 Lgr5 and cell proliferation in the secondary hair germ are increased.

9 Keratin 16 initially localizes within early hair germs, but rapidly shifts to a subset of cells at t

10 ulation in the dermal papilla, the secondary hair germ cells, and the epidermis.

11 de novo epithelial buds resembling embryonic hair germs, collections of epidermal cells whose develop

12 se skin, lacking laminin-10, contained fewer hair germs compared with controls, and after transplanta

13 et of cells in the lower bulge and secondary hair germ compartments.

14 Hair germs comprising epidermal placodes and associated

15 lls of the hair-follicle bulge and secondary hair germ does not induce robust Hh signaling or produce

16 antation, Lama5 -/- skin showed a failure of hair germ elongation followed by complete hair follicle

17 Notably, however, developing hair germs evaginate rather than invaginate, thereby per

18 Wnt signaling tilts each stem cell toward a hair germ fate and, vice versa, based on a continuous sc

19 From the "infundibular cysts", hair germs form centrifugally followed by follicular bud

20 Similar to embryonic hair germs, human BCC buds showed increased levels of cy

21 o proliferate and become activated to form a hair germ is reduced.

22 cytes during catagen suggests that secondary hair germ of late catagen HF is most likely repopulated

23 appaB activity was detected in the secondary hair germ of late telogen and early anagen HFs, suggesti

24 lls migrate out of the reactivated secondary hair germ prior to formation of a new hair canal.

25 Cs) in the hair follicle bulge and secondary hair germ (sHG).

26 s required for normal advancement beyond the hair germ stage of development.

27 RA, resulting in arrest of hf growth at the hair germ stage.

28 f aPKClambda altered the fate of lower bulge/hair germ stem cells.

29 majority of cells in the bulge and secondary hair germ that proliferate in response to skin injury.

30 heir labeled daughters were not found in the hair germs through 48 h following induction of anagen by

31 til the early dermal papilla stage for guard hair germs to make follicles, but is dispensable for the

32 As in embryologically initiated hair germs, transgenic follicles induce Lef-1, but folli

33 esent in the basement membrane of elongating hair germs, when other laminins were downregulated, sugg

34 hibit molecular profiles resembling those of hair germs, yet still possess multipotentiality in vivo.

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