ライフサイエンスコーパス: hairless mouse

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1 During chronologic aging in the hairless mouse, baseline epidermal DNA synthesis rates r

2 We used the outbred, immune-competent Skh-1 hairless mouse model of UVB-induced inflammation and non

3 citation spectra (emission at 380 nm) of SKH hairless mouse model skin are characterized by two bands

4 Using a hairless mouse model, we have demonstrated that testoste

5 findings to the in vivo situations in SKH-1 hairless mouse model, which is regarded to have relevanc

6 les and a solar ultraviolet radiation-driven Hairless mouse model.

7 ion-mediated skin tumorigenesis in the SKH-1 hairless mouse model.

8 The molecular basis of the hairless mouse phenotype was previously found to be the

9 .5 uM EGFR siRNA (50 nM SNA-NCs) for 3 wk to hairless mouse skin almost completely abolishes EGFR exp

10 -7-ene, in propylene glycol:ethanol (7:3) to hairless mouse skin and assessed whether discrete pH cha

11 Changes in hairless mouse skin as a function of age and chronic UVB

12 elphinidin (1 mg/0.1 ml DMSO/mouse) to SKH-1 hairless mouse skin inhibited UVB-mediated apoptosis and

13 hemistry to examine IL-1 alpha expression in hairless mouse skin under basal conditions and following

14 e polymer Nafion) and a biological membrane (hairless mouse skin) recorded during diffusive and ionto

15 In hairless mouse skin, immunohistochemical analysis and fl

16 studied the role of acid-sphingomyelinase in hairless mouse skin.

17 nsdermally delivered by iontophoresis across hairless mouse skin.

18 is factor in cultures of cells isolated from hairless mouse skin.

19 n immortalized HaCaT keratinocytes and SKH-1 hairless mouse skin.

20 MMP-3 (63%), MMP-7 (62%), and MMP-9 (60%) in hairless mouse skin.

21 days x 7 exposures) radiations in the SKH-1 hairless mouse skin.

22 enzoyl peroxide produces skin changes in the hairless mouse that qualitatively resemble those produce

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