ライフサイエンスコーパス: half-time

1 vitro, showed an approximately 3-fold longer half time.

2 K viruses reconstituted the capsid-uncoating half-time.

3 ion of various severities did not affect the half-time.

4 mean liver half-time exceeded the mean tumor half-time.

5 d rapidly from the blood pool, with a 12-min half-time.

6 es in [Ca(2+) ]m occurred more slowly (decay half time = 1.17 +/- 0.07 s).

7 sustained depolarization slowly inactivated (half-time = 10-15 min) ROMK channels that had been engin

8 ompared with that from wild-type xenografts (half-time, 12 +/- 1 h) and other organs (half-time, 2.6-

9 change was found in gastric emptying (median half-time 137 vs. 132 vs. 125 min, p = 0.52) or nutritio

10 heart 2.6 times more slowly than PHEN (mean half-time 155+/-52 versus 55+/-10 min, respectively; P <

11 ts (half-time, 12 +/- 1 h) and other organs (half-time, 2.6-21 h).

12 nvolvement exhibited a faster mean clearance half-time (28.6 minutes) than did children without evide

13 f at least two species of free radicals with half-times: 3.3 and 125.4 h.

14 iving IVIG had a significantly shorter serum half-time (41.6 +/- 14.0 h) than those without (65.2 +/-

15 2 children had a mean radioaerosol clearance half-time (58.1 minutes; 162 studies in 108 children) si

16 4)I and to the retention and slow clearance (half-time, 63 +/- 6 h) of MIBG from transduced xenograft

17 its activation rate was surprisingly faster (half time about 20 s).

18 nstant of about 40 s, and it refills slowly (half-time about 8 min) as nascent vesicles bud from rand

19 meter volume, generator output rate, wash-in half-time, administration time).

20 s suggested by a 2-fold increase in recovery half-time after fluorescence photobleaching.

21 D52 and RAD54 leaves unchanged the rejoining half times and the contribution of the slow component, a

22 se-deficient mutant displayed 4.5-s recovery half-time and a reduction in the mobile fraction to 30%.

23 g and therapy, respectively, a biocompatible half-time and absence of pathways contributing to myelot

24 linkage to ankyrin in resealed cells with a half-time and first order rate constant of 12 +/- 3.6 mi

25 isplaces endogenous ankyrin from IOVs with a half-time and first order rate constant of 42 +/- 14 min

26 Proximal colonic emptying half-time and geometric center at 48 hours were also acc

27 in a dual-phase solid-liquid study (emptying half-time and percentage emptying at 1, 2, 3, and 4 h),

28 abeled antibody formats showed shorter blood half-times and higher liver uptake than the radiolabeled

29 The half-time appears to vary inversely with the anatomic or

30 ) is reduced, flash excitation induces fast (half-time approximately 0.1 ms) oxidation of heme b(H),

31 E3BD folded fairly rapidly at 298K (folding half-time approximately 25 micros) and BBL and POB folde

32 and BBL and POB folded even faster (folding half-times approximately 3-5 micros).

33 hibits equally rapid dynamics of the Z ring (half time, approximately 8 s).

34 in the identification of more than two and a half times as many cases of severe CAD compared with Can

35 at elevated temperatures, we infer that the half-time at 25 degrees C for spontaneous hydrolytic dec

36 ol transfer (% s(-1)) was unprecedented, the half-time at 37 degrees C being ~1 s.

37 The half-time at the nonstenotic mitral valve was less than

38 isoform 4b was found to be very slow, with a half-time (at 235 nM calmodulin and 0.5 microM free Ca(2

39 ent of variation (15.2%, 5.8%, and 19.0% for half-time, cell volume [V(o)], and hydraulic conductivit

40 d exponentially, with the dose-rate decrease half-time corresponding to the effective clearance half-

41 ing detection limits, extending dissociation half-times, decreasing drug dosages and increasing drug

42 The half-time disappearance rate of I-gp120 from brain was 1

43 The pressure half-time Doppler echocardiography method is a practical

44 ose of normal tissues, except the mean liver half-time exceeded the mean tumor half-time.

45 Half-dose and half-time FA-guided PDT were both effective and safe in

46 n in muscles from wild-type mice (relaxation half-time fell from approximately 150 to approximately 9

47 e ADF/cofilin protein actophorin reduced the half time for both dissociation of gamma-phosphate from

48 erimental conditions, we have found that the half time for fluorescence recovery of E. coli Z rings i

49 The half time for modification (t0.5) for the wild-type prot

50 The half time for the slow component of the loss with age wa

51 l: 2.45, 5.89) and approximately two and one-half times for stage IIA or stage IIB disease (odds rati

52 steine 822 may be responsible for the longer half-time for acid recovery observed with pantoprazole.

53 The half-time for activation by LSS was approximately 5 s.

54 At 0 degree C, the half-time for addition of the first G residue is about 3

55 ngle exponential process to obtain a washout half-time for all left ventricular regions.

56 however, mts1 had only a small affect on the half-time for assembly and predominately affected the ex

57 The half-time for dispersal of proregion-EGFP following WPB

58 The half-time for dispersal of VWF-EGFP from extracellular p

59 ilibrium binding occurred within 2 h and the half-time for dissociation was approximately 4 h.

60 The half-time for dissociation was approximately 9.6 h with

61 The apparent half-time for fluorescence decay (t(1/2)) in PSII(-Mn) i

62 This is comparable with the 9-s half-time for FtsZ turnover in vivo and the 8-s turnover

63 ence of this negative regulatory domain, the half-time for fusion of an entire population of lipid ve

64 The half-time for gastric emptying was determined by scintig

65 The half-time for kinetic exchange of the 3HT_6M complex was

66 In addition, the half-time for MT turnover was decreased in cells treated

67 rom monomer, our results agree with the 16-h half-time for N-site GTP in vitro and 33 h half-life for

68 Moreover, a small increase in the half-time for photoactivation of the oxygen-evolving com

69 trated an approximate 7-fold increase in the half-time for plasma membrane translocation at 23 degree

70 g depolarizations, with 3 muM BBG increasing half-time for recovery (measured at -120 mV) from 24 to

71 caffeine application and also increased the half-time for recovery of [Ca2+]i to resting values.

72 The half-time for recovery of function from desensitization

73 es Mn(II) --> Mn(III) photooxidation, with a half-time for regeneration of the Mn(II) complex of ca.

74 rol myocardium, and a 2-fold increase in the half-time for relaxation from steady-state submaximal fo

75 elaxation, shorter time to peak pressure and half-time for relaxation than wild-type hearts.

76 ctin nucleating factor (ANF), decreasing the half-time for spontaneous actin polymerization.

77 Specifically, the half-time for subunit turnover in vitro at pH 7.7 was 42

78 The half-time for the accumulation of GLUT4-eGFP (enhanced g

79 The overall half-time for the conversion of proinsulin to insulin is

80 The inferred half-time for the decay of this productive conformation

81 e is placed by a deoxyguanylate residue, the half-time for the first addition increases from about 3

82 The half-time for the liquid crystalline phase to switch is

83 nges in the glycosylation state of SUR1, the half-time for the maturation of K(ATP) channels, includi

84 Binding was rapid, with a half-time for the on-rate <1.8 min and for the off-rate

85 (t(12) = approximately 100 micros), but the half-time for the reduction of cytochrome f is increased

86 In 1.0 M PO4(3-), the calculated half-time for water oxidation is approximately 7 mus.

87 Because 20 h corresponds to eight half-times for dissociation, only monomer would be prese

88 . actinomycetemcomitans and achieved shorter half-times for killing than control PMNs when assayed at

89 Based on comparatively slow half-times for phosphorylation and dephosphorylation, ph

90 y of the guest Gly-Xaa-Yaa triplet, with the half-times for refolding varying from 6 to 110 min (conc

91 After phosphorylation of RLC, the half-times for relaxation from 0.36 +/- 0.03 Po, 0.59 +/

92 ars, several orders of magnitude longer than half-times for spontaneous decomposition of other enviro

93 Half-times for the fluorescence increase are consistent

94 als as dictated by their effective clearance half-times from the mouse femurs.

95 Overall, the gastric emptying half-time (GE t50) was 54.7 +/- 3.8 min.

96 nd liquids by scintigraphy (gastric emptying half-time [GE t(1/2)]); gastric volumes by single-photon

97 umber of synapses was three to three and one-half times greater onto the somata of dorsal striatal sp

98 the half-dose group and 2 of 26 eyes in the half-time group had recurrence of CSC during follow-up;

99 d) = 48 fM and slower dissociation kinetics (half-time > 5 days) than those for the streptavidin-biot

100 dmission within 72 hrs were nearly two and a half times higher on days when > or =9 patients were adm

101 an extraordinarily fast recovery of KRas WT [half-time (HT), approximately 1.3 s] compared to HRas, R

102 state but with a much longer (undetectable) half-time in the reduced state.

103 The biologic half-time in tumors was greater than in lungs.

104 This half time is 20 times slower than that for control dye-s

105 This half-time is substantially longer than that of exocytic

106 romagnetic exchange interaction is one and a half times larger than the ferromagnetic one, a magnetic

107 0 s), liquid-only gastric emptying (emptying half-time), liquid and solid emptying in a dual-phase so

108 age, it takes a human expert about one and a half times longer to decide an image is normal than to r

109 n <10 s after stimulation and decayed with a half time <20 s, indicating that the time course of bulk

110 iated endocytosis from clathrin-coated pits (half-time <10 min).

111 tide dissociates rapidly from alphaIIbbeta3 (half-time <2 min), consistent with the priming window de

112 ension after quick recovery) was analysed by half-time measurement and also by exponential curve fitt

113 acquired for MVA assessment by the pressure half-time method and by two 3D quantification methods (m

114 ntervention, the median MVAs by the pressure half-time method, mitral valve quantification software,

115 percentage gastric emptying (%GE) at 90 min, half-time (min) based on an exponential fit, and clearan

116 The Pd-Pt nanodendrites were two and a half times more active on the basis of equivalent Pt mas

117 CI, 6.46-10.73); cancer decedents, one and a half times more likely (OR, 1.57 [95% CI, 1.25-1.96]); a

118 , although equilibration is very slow with a half-time near 6 h.

119 The determination of the half-time obviates the need for the commonly used exerci

120 cycles, and an exceptionally low adsorption half time of 10.6 min were achieved.

121 rm is a primary event, which proceeds with a half time of 14 min, and is followed by authentic proces

122 iency, electron injection kinetics exhibit a half time of 150 ps.

123 ls, but the slow component, operating with a half time of 18.4 h, is still able to rejoin the majorit

124 ynamic, continually remodeling itself with a half time of 30 s, similar to microtubules in the mitoti

125 dissociating from the mother filament with a half time of 500 seconds.

126 which peaked at 8+/-3 pA and declined with a half time of 7+/-2 ms.

127 CEF appeared with a half time of about 20 min after exposure to H2O2, sugges

128 y fast turnover, decaying in the dark with a half time of about 6 h.

129 om temperature at high concentrations with a half time of approximately 1 h.

130 ctal epithelial cells are turned over with a half time of approximately 1month in adult virgin mice.

131 unced slow relaxation component with a decay half time of more than 50 s was measured for deltat > 10

132 h of protein-polyphenol interactions and (a) half time of protein decay in gastric conditions (R(2)=0

133 lathrin- and K(+)-independent process with a half time of several seconds.

134 t DT40 cells rejoin IR-induced DNA DSBs with half times of 13 min and 4.5 h and contributions by the

135 e to a change of energy intake is slow, with half times of about 1 year.

136 The half times of the slow component of the cell loss after

137 The half times of the slow component were only 26 years afte

138 proceeded faster than its exit, i.e., with a half-time of <1 s at 37 degrees C.

139 s, hydrolytic dechlorination proceeds with a half-time of 0.18 s.

140 into two phases; the first occurring with a half-time of 0.8 s, the second with a half-time of 14 s,

141 -type HELLS is very dynamic, with a recovery half-time of 0.8s and a mobile protein fraction of 61%.

142 (1/2)) approximately 26 h) with an estimated half-time of 1.2 h.

143 ure to an odor, and recovery occurred with a half-time of 1.5 min, under a set of test conditions.

144 f-time, Td, equal to the effective clearance half-time of 117mSn(4+)DTPA from the femur (222 h).

145 The concentration (%ID/g) and biologic half-time of 131-Lym-1 in tumor were about twice those i

146 The effective half-time of 131I measured in athyrotic patients was use

147 with a half-time of 0.8 s, the second with a half-time of 14 s, possibly reflecting the differential

148 oth a slower response to AMP-PNP (activation half-time of 140 +/- 20 s vs. 21 +/- 4 s for wild type)

149 d on repolarization and decays slowly with a half-time of 150-300 ms.

150 This expression was reversible with a half-time of 17.2 +/- 0.5 h.

151 tion constant of 87 pM, an inhibitor release half-time of 17.2 min, and a Km/Kd ratio of 70,100.

152 ODCase turns its substrate over with a half-time of 18 ms, in a reaction that proceeds in its a

153 rted from the cell surface to the TGN with a half-time of 20-25 min, six to seven times faster than L

154 Ca2+] amplitude of 289 +/- 3 nM with a decay half-time of 20.8 +/- 0.2 ms and a full width at half-ma

155 o proceed from the N719 triplet state with a half-time of 200 +/- 60 ps and quantum yield of 84 +/- 5

156 ynamic, continually remodeling itself with a half-time of 30 s.

157 caused disassembly of protofilaments with a half-time of 5 s.

158 that disulfide reduction: (i) occurs with a half-time of 6 h after folate-FRET endocytosis, (ii) beg

159 0.8% ID/femur) and cleared with an effective half-time of 62 hr.

160 tion constant of 30 pM, an inhibitor release half-time of 64 min, and a Km/Kd ratio of 203,300.

161 The uptake and biological half-time of 67Cu-21T-BAT-Lym-1 in tumors were greater t

162 secreted from transfected HeLa cells with a half-time of 7 h and the rate-limiting step was exported

163 s were mixed, a FRET signal developed with a half-time of 7 s, demonstrating a rapid and continuous d

164 tion is rapid and profound, occurring with a half-time of 70 s.

165 a conversion occurred at 37 degrees C with a half-time of 73 h.

166 reaction that proceeds in its absence with a half-time of 78 million years in neutral solution.

167 increased back to pre-stimulus values with a half-time of 8-10 min.

168 eady state) was similar between groups; mean half-time of 92 plus minus 4 min (NS between groups).

169 on recovered to 83% of control values with a half-time of about 10.5 min.

170 rature and racemizes at 199 degrees C with a half-time of about 11 h.

171 cluster) that exchanged with solvent with a half-time of about 2 days in the oxidized state but with

172 lmodulin was removed was even slower, with a half-time of about 20 min.

173 ence of insulin, IRAP was endocytosed with a half-time of about 3-5 min.

174 ls of similar morphology as Abeta42, but the half-time of aggregation (t1/2) increases exponentially

175 flet of liposomes and proteoliposomes with a half-time of approximately 0.1 min.

176 (125)I-MCP-1 has a blood clearance half-time of approximately 10 minutes and circulates in

177 SP rapidly - with a half-time of approximately 10 s with intracellular GTPga

178 ster than those determined by the LS method (half-time of approximately 120 micros vs half-time of ap

179 HPV31 entry was remarkably slow, with a half-time of approximately 14 h, whereas the entry half-

180 as does the truncation mutant CaSRDelta868 (half-time of approximately 16 h).

181 desensitization was initially rapid (with a half-time of approximately 230 ms), but dramatically slo

182 od (half-time of approximately 120 micros vs half-time of approximately 235 micros, in the presence o

183 BC) approximated a monoexponential response (half-time of approximately 26 s).

184 III3-FRET fit a first-order reaction with a half-time of approximately 30 s; the kinetics with desta

185 eformed complexes, and the other half with a half-time of approximately 40 micros attributed to cyt c

186 of less than 10 mum(2), with a concentration half-time of approximately 40 s.

187 Of these 30%, about half were reduced with a half-time of approximately 5 micros attributed to prefor

188 PIP(2) was resynthesized with a half-time of approximately 50 s.

189 ach the PVC via early endosomes (EEs) with a half-time of approximately 60 min.

190 r GTP association, both with PLD2), it has a half-time of approximately 7 min, is enhanced with incre

191 1alpha carboxyl terminus matures completely (half-time of approximately 8 h) and without a lag period

192 elationship between Ca2+ wave period and the half-time of basal [Ca2+]SR recovery following Ca2+ rele

193 First, the half-time of conversion for both de novo and seeded kine

194 e concentration of mCAH3 increased, with the half-time of CP at the barbed end decreasing from approx

195 l (V(H)) of -30 mV, the enzyme decreased the half-time of decay and charge transfer of EPSCs by 32 an

196 tized in the continuous presence of agonist (half-time of desensitization, t(1/2), 2.13 s).

197 The decay half-time of each mutant correlated with the yield of th

198 igher maximal concentration and has a longer half-time of elimination than 3F-2.

199 he K(d) for ethylene binding to ETR1 and the half-time of ethylene dissociation from ETR1 do not unde

200 -daily relamorelin significantly reduced the half-time of gastric emptying and vomiting, as well as n

201 The primary end point was the half-time of gastric emptying.

202 (210)Pb from foliage operate with a maximum half-time of greater than 370 days (P > 99%), which is a

203 (v) The half-time of HbNO in vivo is approximately 40 min.

204 ime of approximately 14 h, whereas the entry half-time of HPV16 was 4 h.

205 rast to these relatively modest effects, the half-time of inactivation of the mutant was reduced by m

206 HR) are approximately 89% identical, but the half-time of internalization of agonist mediated by the

207 However, the half-time of intracellular [Ca2+] decay was significantl

208 hat Pex18p is constitutively degraded with a half-time of less than 10 min in wild-type Saccharomyces

209 duced receptor destabilization and increased half-time of ligand dissociation.

210 antidepressant treatment both increases the half-time of maximum recovery of GFP-Galphas and decreas

211 -170-decorated MT plus ends could reduce the half-time of melanosome aggregation by ~50%.

212 f P(700)(+)/P(700) is 447 mV and the in vivo half-time of P(700)(+) reduction by its natural donor, p

213 The half-time of recovery of acid secretion in rats followin

214 Over the course of 20 contractions the half-time of relaxation increased from 36.5 +/- 0.09 ms

215 are wortmannin-insensitive and occur with a half-time of roughly 5 min.

216 unexpectedly stable state with an unfolding half-time of several days.

217 y after peptide addition with a deactivation half-time of several minutes.

218 l transfer by 2.9-fold and by decreasing the half-time of sterol transfer by 10-fold (from 11.6 to 1.

219 ance gene) through the BDP, the evolutionary half-time of the gene of interest increases 4-10 times,

220 e experimental results, showing an increased half-time of the recorded events, are complemented by an

221 , 6-minute walk), muscle aerobic metabolism (half-time of VO2 and near infrared recovery), and forear

222 relatively rapid reaction, with an apparent half-time of ~1 s.

223 MCA4b or C28 is much slower, with an overall half-time of ~10 min.

224 issociation of the enzyme-substrate complex (half-time of ~30 s) is rate-limiting for the investigate

225 , suggesting that they were endocytosed with half-times of 1-2 h.

226 rin and GGA1 cycle on and off membranes with half-times of 10-20 s, independently of vesicle budding.

227 293 to 15.8 and 18.1 nM, respectively, with half-times of 2-3 minutes.

228 ference in the rate of GDP dissociation with half-times of 2.3 +/- 0.2 and 10.4 +/- 1.3 min, respecti

229 n thus hydrolyze bound GTP with deactivation half-times of 25-75 ms at 30 degrees C, commensurate wit

230 as significantly faster than that of WT with half-times of 25.1 +/- 1.8 and 32.2 +/- 2.5 min (mean +/

231 ation rate and leaf water potential (typical half-times of 30 min versus 1-2 h).

232 eclined from the peak in 1965 with effective half-times of 6-8 years, only interrupted by a temporary

233 0.88, and 0.79 cGy/h for dose rate decrease half-times of 62, 255, and 425 h and infinity, respectiv

234 gh iron-sulfur cluster F(X) is biphasic with half-times of approximately 5-30 ns ("fast" phase) and a

235 domain (450 nm) recovers spontaneously with half-times of approximately 50 s.

236 7.4, 20 degrees C, and in 0.3% SDS (w/v) the half-times of bR denaturation were 19.2 min for the wild

237 (WT) mice, nor were the delays in onset and half-times of Ca2+ sensitization, initiated by flash pho

238 ocessing in PC1/3 nulls with prolongation of half-times of conversion by 7- and 10-fold for proinsuli

239 The half-times of cyclization have been determined for both

240 We derived approximate expressions for the half-times of degradation and aggregation and show that

241 80Y and alpha7-P180F were studied and showed half-times of desensitization of 650 and 160 ms, respect

242 iciency in DNA-PK activity did not alter the half-times of either of these components of rejoining bu

243 e of dithiothreitol (DTT), and shows typical half-times of equilibration in the 10 h range.

244 smic tails are significantly different, with half-times of ligand internalization ranging from less t

245 is suppressed in HeLa cervical cancer cells, half-times of procaspase-9 processing, as well as the mo

246 The half-times of response are 8.8+/-0.2 seconds at 15 mM HC

247 ent exhibited two similar kinetic pools, the half-times of which were consistent with protein (t(b)(1

248 onset of desensitization dramatically, with half-times of ~930 and 700 ms, respectively, compared to

249 A plot of the phase 2b rate (or 1/half-time) of tension decay after stretch versus [P(i)]

250 fect gastric emptying ( approximately 30-min half time) or pH (mean +/- SD: 4.4 +/- 0.8) at 90 min.

251 tic pools of NBD-cholesterol efflux but with half-times over 200% shorter than those of the cytoplasm

252 treatment effect on ascending colon emptying half-time (P = .015) and overall colonic transit at 48 h

253 All eyes that received half-time PDT showed complete resolution of SRF within 6

254 received half-dose PDT and 26 eyes received half-time PDT.

255 to evaluate the reliability of the pressure half-time (PHT) method for estimating mitral valve areas

256 Glycoside cleavage proceeds with half-times ranging from 4 years for 2'-deoxyinosine to 4

257 alysis of the size-dependent energy transfer half times revealed that the distinct relaxation rate co

258 LNCaP cells was unstable with a degradation half-time (t(1/2)) of 3 h at 37 degrees C.

259 bunits returned to the outer segments with a half-time (t(1/2)) of approximately 24 and 29 minutes, r

260 The penetration index (P.I.) and clearance half-time (T(1/2)) of DTPA were measured in each lung.

261 The half-time (t(1/2)) of release of retinal from VCOP was 7

262 occurs from two distinct kinetic pools: the half-times (t(1/2)) for the two pools in CHO-K1 cells ar

263 /- 0.5 mm/min, and K(+) release and reuptake half-times (t(1/2)) of 12 +/- 2 and 32 +/- 4 s, respecti

264 apparatuses was rapid and exponential, with half-times (t(1/2)) of 5 min (WP), less than 4 min (MB),

265 The half time, t(1/2), for the conversion to normal islet am

266 hly dynamic, which are evaluated by recovery half time (t1/2) in seconds, determined by fluorescence

267 The clearance half-time (T1/2) of 99mTc-DTPA from each lung was measur

268 The half-time (t1/2) to peak for the increase in [Ca2+]m was

269 Dose-rate decrease half-times (Td) (time required for 137Cs gamma ray dose

270 f 137Cs gamma-rays with a dose-rate decrease half-time, Td, equal to the effective clearance half-tim

271 ime corresponding to the effective clearance half-time (Te) of the radioactivity from the femoral bon

272 n rate annual woody cover increase two and a half times that of Australian savannas.

273 The HGNs exhibited energy transfer half times that ranged from 105 +/- 10 ps to 1010 +/- 80

274 Five nodes regressed with a half-time that was shorter than their observed effective

275 ferase concentrations of more than two and a half times the upper limit of normal, history of diabete

276 ore, the authors calculated gastric-emptying half-time, the time for half of the ingested solids or l

277 nation thresholds were on average five and a half times those of controls and were proportional to th

278 le mutant Ku70(-/-)/RAD54(-/-) shows similar half times to Ku70(-/-) cells.

279 In contrast, the half-time to maximal constriction was significantly incr

280 to initial graviresponse and the subsequent half-time to maximum steady state bending rate increased

281 n varied by orders of magnitude, from a 14-h half-time to no regression at all.

282 e of appearance in interstitial fluid (i.e., half-time to steady state) was similar between groups; m

283 i, but both the maximum bending rate and the half-time to the maximum rate were significantly reduced

284 ents were obtained by matching the simulated half-times to those observed experimentally and reported

285 ifference to fall to half its initial value (half-time) varied with the anatomic severity of the valv

286 a-Yaa were slower and showed a wide range of half-times, varying with the identity of the residues in

287 r vesicles containing a single flippase, the half-time was 3.3 min.

288 held at -20 mV compared to -80 mV (refilling half-time was 38 +/- 10 and 20 +/- 6 s, respectively).

289 and 24% HbF in the HDE RBCs, the circulation half-time was 40, 80, and 180 hours, respectively.

290 The blood clearance was biphasic; the alpha half-time was 5 min and the beta half-time was approxima

291 Dopamine half-time was 5.4 +/- 0.7 min at normothermia, increased

292 rate, and cardiac output significantly, the half-time was affected only slightly.

293 In mild mitral stenosis, the half-time was approximately 100 msec; in moderate stenos

294 ; the alpha half-time was 5 min and the beta half-time was approximately 120 min.

295 Pulmonary clearance half-time was measured by using conventional gamma camer

296 wice those in normal tissues, although tumor half-time was similar to that of the thyroid.

297 Half times were calculated from the exponential rate con

298 Proportionate reductions in equilibration half-time were observed for preparations with a higher a

299 ocreatine and adenosine diphosphate recovery half-times were almost twice as long as in controls (P <

300 g studies in vivo showed a range of turnover half-times with an average of 25 s for MtbFtsZ as compar