ライフサイエンスコーパス: hallucination

1 lus when only noise is present: a perceptual hallucination.

2 f human consciousness and clinical models of hallucination.

3 drowsiness, confusion, loss of balance, and hallucination.

4 later emergence of persecutory ideation and hallucinations.

5 clinical symptoms of schizophrenia, such as hallucinations.

6 ucinations, relative to those without visual hallucinations.

7 components of a model of oscillating visual hallucinations.

8 arent-rated negative symptoms and lowest for hallucinations.

9 fluctuations and clozapine is effective for hallucinations.

10 ucinations, but not the subgroup with visual hallucinations.

11 yperactivity in sensory cortex that leads to hallucinations.

12 m underlying 5-HT2A receptor-mediated visual hallucinations.

13 ual perceptual alterations, including visual hallucinations.

14 brain circuit that mediated the conditioned hallucinations.

15 vercompensate in the case of CBS, leading to hallucinations.

16 psychotic hallucinations, and true psychotic hallucinations.

17 back mechanisms can enhance patterned visual hallucinations.

18 the observed regularity of geometric visual hallucinations.

19 These are experienced as geometric visual hallucinations.

20 function in patients with and without visual hallucinations.

21 de for reporting of persistent formed visual hallucinations.

22 cutive function, were associated with visual hallucinations.

23 linical study of whom 32% experienced visual hallucinations.

24 re added to the list of procedures provoking hallucinations.

25 of presence and passage) and complex visual hallucinations.

26 st that up to 40% of patients have long-term hallucinations.

27 ifference in mean time to onset of falls and hallucinations.

28 2ARs) are molecular targets for drug-induced hallucinations.

29 ircular patterns are the most salient biased hallucinations.

30 ularly for patients with a history of visual hallucinations.

31 ients, especially those with severe auditory hallucinations.

32 ratings are perceptually opponent in biasing hallucinations.

33 lated to positive symptoms, such as auditory hallucinations.

34 g movements, especially patients with severe hallucinations.

35 t ILF (P = .02) than patients without visual hallucinations.

36 for severe (77.8%) compared with mild (36%) hallucinations.

37 aberrant listening in patients with musical hallucinations.

38 posite effects on goal-directed behavior and hallucinations.

39 pal activity at baseline and during auditory hallucinations.

40 nia was a mediator of change in paranoia and hallucinations.

41 e psychological treatment of auditory verbal hallucinations.

42 nsomnia leads to a reduction in paranoia and hallucinations.

43 sual recognition to social vision and visual hallucinations.

44 ring current models for mechanisms of visual hallucinations.

45 tem atrophy, including a patient with visual hallucinations.

46 sence of concurrent persecutory ideation and hallucinations.

47 ng stimulus onset predisposes an observer to hallucinations.

48 eep in patients with persistent delusions or hallucinations.

49 n symptoms (fatigue, myoclonus, sedation and hallucinations, 0 = best and 40 = worst possible) betwee

50 mean difference 0.3, 95% CI -2.0 to 2.6) and hallucinations (-1.9, -6.5 to 2.7).

51 Among patients with baseline hallucinations, 13 of 17 (76.5%) who discontinued risper

52 ents for ADS-5102 vs placebo included visual hallucinations (15 [23.8%] vs 1 [1.7%]), peripheral edem

53 confusion (50%), movement disorder (38%) and hallucinations (25%) were common.

54 omnia 89.7%, confusion 65.5%, amnesia 55.6%, hallucinations 51.9%), dysautonomia (hyperhidrosis 86.2%

55 toms-71 years (66-92 years), six with visual hallucinations-72 years (64-90 years), seven with dement

56 Visual and auditory hallucinations accompany certain neuropsychiatric disord

57 an interesting perspective for understanding hallucinations accompanying sleep paralysis (SP; "night-

58 ents with schizophrenia who have experienced hallucinations against patients who have not, matched on

59 Delusion, hallucination, agitation, anxiety, apathy, motor-disturb

60 ences in ocular pathology between the visual hallucination and non-visual hallucination groups.

61 past large-sample investigations of auditory hallucination and suggest potentially important new find

62 onse (>25% reduction in scores on delusions, hallucinations and agitation domains of the Neuropsychia

63 nd reported increased sleepiness, hypnagogic hallucinations and cataplexy-like symptoms, suggesting a

64 allucinations, and the link between auditory hallucinations and characterological entities.

65 symptoms than patients with PDD and had more hallucinations and cognitive fluctuations.

66 ent group included hyper-religiosity, visual hallucinations and cross-modal sensory experiences.

67 tic of positive symptoms, including auditory hallucinations and delusions of control.

68 refers to the spectrum of illusions, formed hallucinations and delusions that occur in PD.

69 risperidone better controlled mean levels of hallucinations and delusions throughout follow-up (beta

70 Early emergence of comorbid hallucinations and delusions were observed in 57% of ind

71 arkinson's disease psychosis, which includes hallucinations and delusions, is frequent and debilitati

72 Despite their effectiveness in treating hallucinations and delusions, prolonged exposure to anti

73 ain Outcome Measure Children's self-reported hallucinations and delusions.

74 f positive symptoms in the disorder, such as hallucinations and delusions.

75 This framework may explain hallucinations and delusions.

76 rnal insomnia, REM sleep behaviour disorder, hallucinations and depression, symptoms which are freque

77 nificant differences favoring citalopram for hallucinations and favoring placebo for sleep/nighttime

78 Visual hallucinations and illusions are thought to be caused by

79 contribute to the pathophysiology of visual hallucinations and may explain their predominantly visua

80 ut also in fact driven by, top-down effects: hallucinations and mental imagery.

81 15%-59%), with modest shared environment for hallucinations and negative symptoms (17%-24%) and signi

82 dorsolateral prefrontal cortex could affect hallucinations and negative symptoms, respectively.

83 her delineate an association between musical hallucinations and neurodegenerative disease.

84 ent adverse neuropsychiatric events, such as hallucinations and night terrors, reported by HIV patien

85 acterized by paranoid delusions and auditory hallucinations and often associated with disturbances in

86 xole group had serious events, two of which (hallucinations and orthostatic hypotension) were deemed

87 The neural mechanisms that produce hallucinations and other psychotic symptoms remain uncle

88 mise for treating refractory auditory verbal hallucinations and other selected manifestations of schi

89 on selectivity provides a mechanism for both hallucinations and perception.

90 tive decline with new-onset recurrent visual hallucinations and progressive lethargy.

91 ce occurs in most patients with delusions or hallucinations and should be treated as a clinical probl

92 ew the demographics of subjects with musical hallucinations and to determine the prevalence of neurol

93 elopment of therapies specifically targeting hallucinations and visuoperceptive functions.

94 be associated with exacerbating confusion or hallucinations and with behavioral changes.

95 mptoms of schizophrenia (e.g., delusions and hallucinations) and have been approved by regulatory age

96 ith a psychotic disorder and auditory verbal hallucinations, and 50 healthy control subjects underwen

97 higher order visual areas can induce complex hallucinations, and also reaffirm direct electrical brai

98 es persecutory ideation, auditory and visual hallucinations, and diagnosis of probable psychosis.

99 ore features of DLB (dementia, parkinsonism, hallucinations, and fluctuations) with striatal dopamine

100 ible neuronal events at the origin of visual hallucinations, and further suggests that brain oscillat

101 linked to cognitive dysfunction, audiovisual hallucinations, and negative affective states akin to th

102 , cognitive impairment, motor deterioration, hallucinations, and premature death by the third to four

103 clude myoclonic seizures, auditory or visual hallucinations, and renal failure.

104 evaluable items were most often orientation, hallucinations, and speech or mood content.

105 ation, Hoehn and Yahr score, fluctuations or hallucinations, and striatal DAT availability as measure

106 atic and multisensorial features of auditory hallucinations, and the link between auditory hallucinat

107 , quasi-psychotic delusions, quasi-psychotic hallucinations, and true psychotic hallucinations.

108 neurocognitive processes in auditory verbal hallucinations, and, consequently, it is proposed that p

109 s with hallucinations, particularly auditory hallucinations, antipsychotic discontinuation should be

110 offer a rare and unique opportunity to study hallucinations apart from confounding clinical factors,

111 Auditory hallucinations are associated with abnormalities in this

112 We found that flicker-induced hallucinations are biased by the presentation of adjacen

113 Hallucinations are common in psychiatric disorders, and

114 These results suggest that perceptual hallucinations are due to an imprecise and biased state

115 Visual hallucinations are frequent, disabling complications of

116 Intrusive memories, images, and hallucinations are hallmark symptoms of psychiatric diso

117 Visual illusions and hallucinations are hallmarks of serotonergic hallucinoge

118 ngs suggest early onset illusions and formed hallucinations are linked to amyloid pathology in PD and

119 forts for the treatment of disorders wherein hallucinations are part of the etiology, such as schizop

120 kinson's disease and, with respect to visual hallucinations, are an important predictor of cognitive

121 s, defined as the occurrence of delusions or hallucinations, are frequent in Alzheimer disease (AD),

122 s or no hallucinations, patients with severe hallucinations as a presenting symptom at baseline had a

123 in reducing the severity of auditory verbal hallucinations as well as negative symptoms.

124 omnia Severity Index (ISI) and delusions and hallucinations assessed by the Psychotic Symptoms Rating

125 ary outcome was reduction in auditory verbal hallucinations at 12 weeks, measured by total score on t

126 inant effects induced by LSD included visual hallucinations, audiovisual synesthesia, and positively

127 (rTMS) for the treatment of auditory verbal hallucinations (AVH), an update is provided on the effic

128 ients with pharmacoresistant auditory verbal hallucinations (AVH).

129 hizophrenia who report daily auditory verbal hallucinations (AVHs) and sociodemographically matched h

130 Auditory/verbal hallucinations (AVHs) are accompanied by activation in W

131 nic patients with or without auditory verbal hallucinations (AVHs) to that of normal controls remain

132 they included a reduction in the severity of hallucinations but an increase in the severity of sleep/

133 t was present for the subgroup with auditory hallucinations, but not the subgroup with visual halluci

134 n sulcal length increasing the likelihood of hallucinations by 19.9%, regardless of the sensory modal

135 tural markers that predict the occurrence of hallucinations by comparing patients with schizophrenia

136 eceptors may lead to the formation of visual hallucinations by increasing cortical excitability and a

137 Visual hallucinations can be a major hallmark of late stage dis

138 Here we show that visual hallucinations can be induced in the normal population b

139 els have revealed that the geometry of these hallucinations can be related to functional neuro-anatom

140 Our results demonstrate that hallucinations can be studied objectively, and they shar

141 This case series shows that musical hallucinations can occur in association with a wide vari

142 The primary outcome measure was the Hallucination Change Score (HCS).

143 of advanced disease (frequent falls, visual hallucinations, cognitive disability and need for reside

144 as quantitative traits (self-rated paranoia, hallucinations, cognitive disorganization, grandiosity,

145 and had an extremely low frequency of visual hallucinations compared with Parkinson's disease.

146 effect of AVATAR therapy on auditory verbal hallucinations, compared with a supportive counselling c

147 tter atrophy patterns associated with visual hallucinations, comparing Parkinson's disease hallucinat

148 between abnormal visual function and visual hallucinations, considering current models for mechanism

149 behaviorally as positive symptoms (including hallucinations, delusions and thought disorder), negativ

150 ween-group d = -0.66) and positive symptoms (hallucinations, delusions, disorganization) (adjusted me

151 With its hallucinations, delusions, thought disorder, and cogniti

152 of advanced disease (frequent falls, visual hallucinations, dementia and need for residential care)

153 itions experience persistent auditory verbal hallucinations, despite treatment.

154 Delusion and hallucination did not.

155 actors include the often-reported delusions, hallucinations, disorganization, negative, and affective

156 a second experiment, we find that simulated hallucinations do not evoke the temporal distortion comm

157 y several different authors as formed visual hallucinations due to disturbances of the visual system

158 and maintenance of persecutory ideation and hallucinations during 18 months of follow-up in the 2000

159 symptoms), and had enduring auditory verbal hallucinations during the previous 12 months, despite co

160 allucinators, all of whom experienced visual hallucinations, exhibited grey matter atrophy with signi

161 eduncular hallucinosis, a syndrome of visual hallucinations following subcortical lesions long hypoth

162 from three centres in the UK who had command hallucinations for at least 6 months leading to major ep

163 mprovements relative to sham stimulation for hallucination frequency and clinical global improvement

164 Hallucination frequency, total auditory hallucination ra

165 Area Spt acts as a gateway to the hallucination-generating cerebral network.

166 Patients in the visual hallucinations group had similar disease duration but ha

167 ween the visual hallucination and non-visual hallucination groups.

168 ealed that patients with a history of visual hallucinations had lower fractional anisotropy in the le

169 r multiple comparisons, patients with visual hallucinations had significantly greater disease duratio

170 ese that the uniform nature of many of these hallucinations has been demonstrated.

171 Auditory verbal hallucinations have attracted a great deal of scientific

172 Musical hallucinations have been linked to multiple associated c

173 agonists, including sedation, dysphoria, and hallucinations, have limited their clinical use.

174 Auditory verbal hallucinations (hearing voices) are typically associated

175 he absence of a sensory signal, also termed "hallucinations." Here, we investigated whether spontaneo

176 oral gyrus of either hemisphere and auditory hallucination; (ii) left superior-/middle-temporal gyri

177 Visual hallucinations, illusions and extrapyramidal tract signs

178 e correlated strongly with ratings of visual hallucinations, implying that intrinsic brain activity e

179 motor impairment correlated positively with hallucination improvement following rW rTMS.

180 continence in 50% and dementia in 39%.Visual hallucinations in 0%.

181 early onset of the disease, the presence of hallucinations in 45% (14/31) and symptoms of cognitive

182 induced radial (<10 Hz) or spiral (10-20 Hz) hallucinations in a behavioral experiment involving full

183 is has implications for the understanding of hallucinations in clinical and non-clinical populations,

184 lovian learning task, we induced conditioned hallucinations in four groups of people who differed ort

185 ve high affinity at 5-HT(2A)R and can induce hallucinations in humans.

186 Induced hallucinations in normal individuals provide a model for

187 The literature related to visual hallucinations in ophthalmological settings from 2007 to

188 dementia, which are common co-morbidities of hallucinations in Parkinson's disease and whose neuroana

189 The exact pathogenesis of visual hallucinations in Parkinson's disease is not known but a

190 t structural MRI differences associated with hallucinations in Parkinson's disease may permit earlier

191 ic and ophthalmological correlates of visual hallucinations in Parkinson's disease, the combined data

192 pathways have all been posited as causes of hallucinations in Parkinson's disease.

193 y contributing to the pathogenesis of visual hallucinations in Parkinson's disease.

194 ehension during movements and drive auditory hallucinations in pathological states, the synaptic orga

195 tome, and lead to the hypothesis that visual hallucinations in patients with COS may be because of de

196 Acting on command hallucinations in psychosis can have serious consequence

197 Auditory hallucinations in schizophrenia are alleviated by antips

198 rs) with persistent distressing delusions or hallucinations in the context of insomnia and a schizoph

199 although it fails to explain the absence of hallucinations in the majority of patients with eye dise

200 rm repertoire of processes that generate the hallucinations in the occipital cortex.

201 The resulting paranoia and hallucinations in turn cause further stress, and eventua

202 The phenomenon of musical hallucinations, in which individuals perceive music in t

203 Structural lesions associated with musical hallucinations involved both hemispheres with a preferen

204 f the variation in subjective experiences of hallucination is central to psychiatry, yet systematic e

205 Current research into visual hallucination is predominantly ophthalmology-led, with i

206 The presence of hallucinations is a key diagnostic symptom of schizophre

207 r regimes where activity that corresponds to hallucinations is induced by both spatially homogeneous

208 which plays the role in the pathogenesis of hallucinations, is activated by DOI in MIA offspring onl

209 The beneficial effect on hallucinations lasted for up to 3 months.

210 zures, motor impairments, cognitive decline, hallucinations, loss of circadian rhythm, and premature

211 Overall, the Hallucination Machine offers a valuable new technique fo

212 nts illustrate potential applications of the Hallucination Machine.

213 e we describe such a tool, which we call the Hallucination Machine.

214 These results suggest that perceptual hallucinations may be due to an imprecise and biased sta

215 ndividuals with non-clinical auditory verbal hallucinations (most of them also experienced other non-

216 A subset of patients with early onset formed hallucinations (n=21) had reduced higher visual function

217 tions that have been associated with musical hallucinations: neurological, psychiatric, structural, d

218 Further, auditory hallucinations occur across a range of healthy and disea

219 Hallucinations occur in both normal and clinical populat

220 d the wider clinical context in which visual hallucinations occur.

221 Psychotic symptoms, delusions and hallucinations, occur in approximately 50% of individual

222 with the occurrence of sensory illusions or hallucinations (odds ratio: 8.68, P < 0.001) and eyeball

223 Hallucinations of musical notation may occur in a variet

224 oss of vision leads to complex, vivid visual hallucinations of objects, people, and whole scenes.

225 e highly distinctive 'presence' or 'passage' hallucinations of Parkinson's disease and can help to ex

226 Unique aspects of SP hallucinations offer additional prospects for investigat

227 Compared with patients with mild hallucinations or no hallucinations, patients with sever

228 basimglurant, including three patients with hallucinations or psychosis.

229 Auditory hallucinations--or voices--are a common feature of many

230 us activity that predisposed participants to hallucinations: overall lower prestimulus activity and a

231 us activity that predisposed participants to hallucinations: overall lower prestimulus activity and a

232 lestones of dementia (P < 0.0005) and visual hallucinations (P = 0.02) as well as the accumulation of

233 There were more postoperative hallucinations (p=0.01) and nightmares (p=0.03) with inc

234 sions), P2 (conceptual disorganisation), P3 (hallucinations), P5 (grandiosity), P6 (suspiciousness/pe

235 For patients with hallucinations, particularly auditory hallucinations, an

236 with patients with mild hallucinations or no hallucinations, patients with severe hallucinations as a

237 lnesses are associated with psychosis, i.e., hallucinations (perceptions in the absence of causative

238 gest mixed-methods investigation of auditory hallucination phenomenology so far.

239 iated derealisation, olfactory and gustatory hallucinations, physical symptoms such as headaches, abd

240 hought mentioned above except true psychotic hallucinations plus marked superstitiousness, sixth sens

241 included a small visual field defect, visual hallucinations, prosopagnosia, topographical disorientat

242 ch as psychotic or pharmacologically-induced hallucinations, provide a unique opportunity to examine

243 using severe neurological symptoms including hallucinations, psychosis, and seizures, and may result

244 he Psychotic Symptoms Rating Scales Auditory Hallucinations (PSYRATS-AH).

245 Hallucination frequency, total auditory hallucination rating scale score, and clinical global im

246 ubjects with Parkinson's disease with visual hallucinations, relative to those without visual halluci

247 ty and complexity, the mechanisms underlying hallucinations remain largely untested.

248 ed with presence of persecutory ideation and hallucinations, remaining so after adjustment for sociod

249 ion between brain oscillations and geometric hallucinations remains a mystery.

250 al research on the phenomenology of auditory hallucinations remains scarce.

251 Whether hallucinations require treatment and, if so, what that t

252 Recent theories propose that hallucinations result from an imbalance of prior expecta

253 and specific eye pathology do not influence hallucination risk.

254 eriencing subsequent subclinical paranoia or hallucinations rose by 133% and 92%, respectively.

255 Level of hallucination salience may usefully guide selection of W

256 lated prespeech neural synchrony to auditory hallucination severity in patients.

257 ve daytime sleepiness, cataplexy, hypnagonic hallucinations, sleep paralysis, and disturbed nocturnal

258 Hallucination strength peaked at ~11 Hz flicker and was

259 sual stimulation produces regular, geometric hallucinations such as radial or spiral patterns.

260 ents with schizophrenia have auditory verbal hallucinations that are refractory to antipsychotic drug

261 1] years; P = .03), had more frequent visual hallucinations that did not achieve significance (9 of 1

262 ent in schizophrenia; (2) evidence regarding hallucinations that suggest they are not due to excessiv

263 nd sometimes curious, if not bizarre, visual hallucinations, the forms of which suggest that extrastr

264 fMRI and iEEG, induced a topographic visual hallucination: the patient described seeing indoor and o

265 ducing positive symptoms (i.e. delusions and hallucinations), they do little to improve the disabling

266 Symptoms of psychosis, including hallucinations, thought insertion, strange experiences,

267 icant and consistent risk factors for visual hallucinations, together with new evidence to suggest th

268 re assigned to cognitive therapy for command hallucinations + treatment as usual and 99 (50%) to trea

269 ) of 79 in the cognitive therapy for command hallucinations + treatment as usual group (odds ratio 0.

270 1: 1 ratio to cognitive therapy for command hallucinations + treatment as usual versus just treatmen

271 rles Bonnet syndrome is a disorder of visual hallucinations typically occurring in older persons with

272 For many years, visual hallucinations (VH) in idiopathic Parkinson's disease (P

273 the pathophysiological mechanisms of visual hallucinations (VHs) in patients with Parkinson disease

274 Visual hallucinations (VHs) occur in macular degeneration patie

275 onsequences, including anxiety, tachycardia, hallucinations, violent behavior, and psychosis.

276 The mean age at onset of the hallucinations was 56 years, ranging from 18 to 98 years

277 randomization, and the presence of baseline hallucinations was associated with a higher risk of rela

278 group, the propensity to experience auditory hallucinations was associated with relatively increased

279 al vision, diminished tactile sensation, and hallucinations was persistently Bartonella koehlerae ser

280 which is hypothesized to account for visual hallucinations, we found connectivity with this region t

281 chniques and automated, data-driven methods, hallucinations were associated with specific brain morph

282 th 75.5 +/- 8.0 years) and persistent visual hallucinations were documented in 63%.

283 yo medical record, 393 subjects with musical hallucinations were identified and divided into five cat

284 Visual hallucinations were more common in the group with neurol

285 ifiable groups (P < 0.001), whereas auditory hallucinations were more common in the psychiatric group

286 Myoclonus, rigidity and hallucinations were more frequent in patients with sCJD

287 Patients with severe baseline hallucinations were more likely to relapse after randomi

288 ia and medication-refractory auditory verbal hallucinations were randomly allocated to receive 20 min

289 the four independent determinants of visual hallucinations were rapid eye movement sleep behavioural

290 Auditory verbal hallucinations were robustly reduced by tDCS relative to

291 s/signs, 3 (thought disorder, delusions, and hallucinations) were included in all diagnostic systems

292 nd the presence of visual and other auditory hallucinations, were evaluated independently in all five

293 l diagnosis, depicting other forms of visual hallucination which result from a variety of non-migrain

294 completion is also critical for formation of hallucinations, which constitute a severe symptom of the

295 that some 5-HT(2A)R agonists induce LSD-like hallucinations, while others lack this psychoactive prop

296 mechanisms underlying its ability to induce hallucinations with greater duration and potency than cl

297 g the severity of persistent auditory verbal hallucinations, with a large effect size.

298 siological mechanism of illusions and formed hallucinations, with implications for their respective l

299 rse event (four due to psychotic disorder or hallucination within 10 days of start of the study drug)

300 n patients who go on to develop illusions or hallucinations within 3-4 years of follow-up in the Park