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1 ithorax (Ubx) to directly repress sal in the haltere.
2 sion in the posterior (P) compartment of the haltere.
3 n the developing Drosophila wing but not the haltere.
4 t development of campaniform sensilla on the haltere.
5 r 20 bristles on the anterior margin of each haltere.
6 al features that differ between the wing and haltere.
7 ganized into five fields at the base of each haltere.
8 s on leg shape, but no detectable effects on halteres.
9  the campaniform sensilla at the base of the halteres.
10 gulatory network of the wing to generate the haltere, a modified hindwing.
11 utation was used to analyze synapses between haltere afferents and a flight motoneuron in adult Droso
12 fast monosynaptic electrical pathway between haltere afferents and mnb1 may be responsible in part fo
13  shaking-B2 flies is at the synapses between haltere afferents and the flight motoneuron.
14     In shaking-B2 flies dye coupling between haltere afferents and the motoneuron is abolished, and a
15                           In wild-type flies haltere afferents are dye-coupled to the first basalar m
16 idate for such a control involving the fly's haltere and first basalar motor neuron.
17  to be directly repressed by Ubx in the flys haltere and leg primordia, respectively, and led to the
18                  These results indicate that haltere and metathoracic segment morphology is not achie
19 r cross vein, humeral outgrowths, absence of halteres and eye pigmentation defects.
20       Although tight phase synchrony between halteres and wings is essential for providing proper tim
21 o completely repress this cis-element in the haltere, and that individual Ubx-binding sites are suffi
22 gically specialized wing derivatives such as halteres, and not the more ancestral wings, requires exa
23 everal Ubx-regulated genes in the Drosophila haltere are not repressed by Ubx in butterfly hindwings,
24 singly, there is little evidence that mutant halteres are more variable than wild-type ones, so it is
25  the campaniform sensilla at the base of the halteres are responsible for the phasic activity of b1.
26                                          The halteres are sensitive to Coriolis forces that result fr
27                                              Halteres are sophisticated equilibrium organs of flies t
28 e aerodynamically functional fore wings, the halteres beat during flight and are equipped with their
29      Ubx restricts Dpp's distribution in the haltere by increasing the amounts of the Dpp receptor, t
30             The transformation of wings into halteres by the Hox gene Ultrabithorax (Ubx) in Drosophi
31 al stimulation, we have found one identified haltere campaniform field (dF2) that provides strong syn
32  formation, can still elicit many aspects of haltere cell morphology.
33             By the end of pupal development, haltere cells are 8-fold smaller in apical surface area
34                                              Haltere cells continue to secrete cuticle after eclosion
35         Differences in the shape of wing and haltere cells reflect differences in the architecture of
36  appear to be intermediates between wing and haltere cells, contesting the view that homeotic genes a
37 opmental processes that distinguish wing and haltere cells.
38 ks the early primordia for both the wing and haltere, collectively referred to as the DP.
39 ressed, low levels of posterior dally in the haltere contribute to a reduced P compartment size and a
40 ranous forewings and the modified hindwings (halteres) depend on the Hox gene Ultrabithorax (Ubx).
41 the Hox protein Ultrabithorax (Ubx) promotes haltere development and suppresses wing development by s
42 e than wild-type ones, so it is unclear that haltere development is also canalized.
43 ese sites are critical for activation in the haltere disc.
44 fically expressed in either the wing disc or haltere disc.
45 also required for the growth of the wing and haltere discs, as mutants for these alleles have tiny do
46 d description of cell differentiation in the haltere epidermis, and of the developmental processes th
47 erved between afferents originating from the haltere fields and those from serially homologous fields
48 projections is not limited to axons from the haltere fields, but is also observed between afferents o
49   In the metathorax, Ubx expression promotes haltere formation and suppresses wing development.
50        The differentiation of the Drosophila haltere from the wing through the action of the Ultrabit
51 logy, although no Ubx-regulated genes in the haltere have been identified previously.
52 re important for wing development, promoting haltere identity.
53       Ectopic expression of DIP1 in wing and haltere imaginal discs malforms the adult structures and
54 y differentially expressed genes in wing and haltere imaginal discs.
55 ifferential development of the fore-wing and haltere in Drosophila.
56 ed wing modification is the specification of halteres in Drosophila by a Hox-dependent mechanism, in
57 induce a dramatic homeotic transformation of halteres into wings.
58 otic mutant phenotype: the transformation of halteres into wings.
59                               The Drosophila haltere is a much reduced and specialised hind wing, whi
60 ded by the complex fields on the base of the haltere is mapped onto different functional regions with
61 equence of dally repression in the posterior haltere is to reduce Dpp diffusion into and through the
62                             The evolution of haltere morphology involved changes in UBX-regulated tar
63                                          The halteres of dipteran insects are essential sensory organ
64                                          The halteres of Dipteran insects play an important role in f
65                                          The haltere-off (bare airframe) system revealed a slow (heav
66                                          The haltere-on system revealed a stabilized system with a sl
67                                              Halteres oscillate at the same frequency as and precisel
68 at passively mediate both wing-wing and wing-haltere phase synchronization.
69 ant enhancement of the haploinsufficient Ubx haltere phenotype and second for effects on the splicing
70 uding reduced eye size and abnormal wing and haltere posture.
71 tund gene is required in the wings, antenna, haltere, proboscis and legs.
72  that motoneurons innervating muscles of the haltere receive strong excitatory input from directional
73 feedback to wing motor system is provided by halteres, reduced hind wings that evolved into gyroscopi
74 cific inhibitor of B-family DNA polymerases, haltering replication and possessing a strong antimitoti
75 how that the primary afferent neurons of the haltere's mechanoreceptors respond selectively with high
76 ermined the specific cellular targets of the haltere sensory cells, the afferents of a dorsal field c
77 tic backgrounds result in enlargement of the haltere significantly beyond the normal range of haploin
78                    We find that Ubx controls haltere size by restricting both the transcription and t
79    The correlation between wild-type and Ubx haltere size is very low, indicating that interactions a
80  molecular analysis of this 358 bp wing- and haltere-specific dpp enhancer, which demonstrates a dire
81 y an electrical synapse, and thus can follow haltere stimulation at high frequencies.
82 histicated pair of equilibrium organs called halteres that evolved from hind wings.
83 x (Ubx) modulates morphogen signaling in the haltere through transcriptional regulation of the glypic
84                                         This haltere to mnb1 connection consists of a fast and a slow
85 ined with diverse phase encoding, allows the haltere to transmit information at a high rate about num
86 ounts for three-quarters of the variance for haltere to wing margin transformation in Ultrabithorax f
87 ient phenotypes, from overlap with wild-type halteres to dramatic transformations such as a 50% incre
88        Together, these results show that the haltere-to-flight motoneuron synapses comprise an electr
89 olinergic antagonist mecamylamine blocks the haltere-to-flight motoneuron synapses in shaking-B2 flie
90 erve decreased expression of Ubx and partial haltere-to-wing transformation phenotypes.
91 e Ultrabithorax (Ubx) limits the size of the haltere, which, by the end of larval development, has ap
92 re modified into club-shaped, mechanosensory halteres, which detect Coriolis forces and thereby media

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