ライフサイエンスコーパス: hamster

1 ecies, including cow, dog, cat, chicken, and hamster.

2 ural animal model of adiposity, the Siberian hamster.

3 or specific diagnosis of allergy to Siberian hamster.

4 and cloning of the major allergen from this hamster.

5 mal species, namely, ferret, guinea pig, and hamster.

6 feration in amoebic liver abscess induced in hamster.

7 from three species: human, mouse, and Syrian hamster.

8 high-titer AFIA-positive donations infected hamsters.

9 quality RSV-neutralizing serum antibodies in hamsters.

10 ncreased the F-specific antibody response in hamsters.

11 lowering and antioxidant effects on HFCD fed hamsters.

12 ble to torcetrapib (1) in moderately-fat fed hamsters.

13 levels of anti-GFP antibodies in permissive hamsters.

14 g both PRV152 and H129 into IBAT of Siberian hamsters.

15 severe inflammatory lung disease in infected hamsters.

16 HPS disease in humans and ANDV infection of hamsters.

17 diameter of 83 nm were administered i.v. in hamsters.

18 e spleen and liver samples of SFTSV-infected hamsters.

19 a and early atherosclerosis in Golden Syrian hamsters.

20 lA in transcytosis in vitro and virulence in hamsters.

21 without causing a disseminated infection in hamsters.

22 d no cross-reactivity with common and golden hamsters.

23 ageni strain Fiocruz L1-130 in Golden Syrian hamsters.

24 dose and milder histopathological injury in hamsters.

25 the lack of genetic resources available for hamsters.

26 inhibition (HAI) titers than RD-Ad in Syrian hamsters.

27 d packaging was substantially attenuating in hamsters (10- to 100-fold) and rhesus monkeys (100- to 1

28 Therefore, we tested this role in Siberian hamsters, a model of human energy balance, and C57BL/6 m

29 studies have been conducted in golden Syrian hamsters, a species particularly sensitive to acute dise

30 l Syrian golden hamster model of MHF using a hamster-adapted MARV variant Angola.

31 d scrapie, deer chronic wasting disease, and hamster-adapted scrapie prions.

32 d a high titer of neutralizing antibodies in hamsters against RSV and HPIV3.

33 The major Siberian hamster allergen was cloned, and allergenic properties w

34 Thirteen Siberian hamster-allergic patients were recruited at the outpatie

35 ove the specific diagnosis and treatment for hamster-allergic patients.

36 models, including the mouse, guinea pig, and hamster, also exist for Ebola, Marburg, and Ravn viruses

37 ns on reconfiguration dynamics of the Syrian hamster and rabbit prion proteins.

38 (PrP) amyloids from human, mouse and Syrian hamster and show that their structural differences are m

39 s of some of the viruses are lethal in mice, hamsters and guinea pigs.

40 we detected EV proteins in bile of infected hamsters and humans.

41 iverrini and reacted with sera from infected hamsters and humans.

42 HT1a agonist stimulated aggression in female hamsters and inhibited aggression in males, whereas inje

43 FD hamsters and mice increased AgRP within the arcuate hypo

44 ansport was seen in experimental and control hamsters and mice, indicating that it was not species sp

45 P) and PPARgamma mRNA expression in Siberian hamsters and mice; (3) whether intraperitoneally adminis

46 nd quality of RSV-neutralizing antibodies in hamsters and nonhuman primates.

47 mmune responses than RD-Ad vectors in Syrian hamsters and rhesus macaques.

48 d infectivity by means of the inoculation of hamsters and the subsequent examination for parasitemia.

49 ral DPP4 orthologs, including mouse, ferret, hamster, and guinea pig DPP4, do not.

50 as a determinant of permissivity for ferret, hamster, and guinea pig DPP4.

51 In mouse, human, hamster, and macaque cells, SC-Ad6 still replicated its

52 duction of Th1 type immune response in mice, hamsters, and dogs.

53 igher titers of pre-F specific antibodies in hamsters, and improved the quality of RSV-neutralizing s

54 rs were induced in zebrafish, rainbow trout, hamsters, and mice by carcinogenic agents (methylcholant

55 ated male monkeys, guinea pigs, rabbits, and hamsters; and female dogs.

56 tant biomedical research questions for which hamsters are an excellent model.

57 Hamsters are an ideal animal model for a variety of biom

58 us burdens in the livers of HAdV-C6-infected hamsters are higher than the virus burdens in HAdV-C5-in

59 Furthermore, when hamsters are infected intravenously with HAdV-C6, live,

60 Syrian hamsters are permissive for the replication of species C

61 Syrian hamsters are susceptible to infection with certain human

62 We have studied mice and hamsters as potential small-animal models of SFTSV infec

63 s more attractive than the odour of the same hamsters, before they were infected.

64 To address this, male Siberian hamsters born and maintained outdoors were sampled every

65 When inoculated into hamsters, both of these types of synthetic prions initia

66 or mucosal intranasal immunization of Syrian hamsters, both SC-Ad and RC-Ad expressed transgenes at l

67 cephalopathy-associated forms from mouse and hamster brain homogenates were used to seed RT-QuIC-indu

68 HSI examples acquired from cryo-sections of hamster brain tissue using Fourier-transform infrared (F

69 ranscript expression in both female and male hamster brains and offers invaluable information to prom

70 r endothelial growth factor (VEGF) levels in hamster bronchoalveolar lavage (BAL) fluid early after i

71 amount of virus in the lung of ANDV-infected hamsters but altered neutrophil recruitment, MIP-1alpha

72 tes the efficacy of a novel FGF21 mimetic in hamsters, but reveals attenuated effects in the animal m

73 cs of leptospiral infection in Golden Syrian hamsters by a sensitive quantitative real-time PCR (TaqM

74 hich proved efficacious in murine sepsis and hamster C. difficile models of disease.

75 tantly, we also show that SFTSV infection in hamsters can be effectively treated with a broad-spectru

76 nd as well as the soft-shell clam and Syrian hamster, can advance studies of tumor biology.

77 ir complementing defective repair in Chinese hamster cells 5 (XRCC5).

78 e demonstrate that FANCJ deficient human and hamster cells exhibit reduction in the overall gene conv

79 ,12-Dimethylbenz[a]anthracene (DMBA)-induced hamster cheek pouch model of oral squamous cell carcinom

80 the treatment of head and neck cancer in the hamster cheek pouch oral cancer model is presented.

81 2 receptor (B2R) inhibited plasma leakage in hamster cheek pouch topically exposed to tissue culture

82 chemically-induced epithelial tumors in the hamster cheek pouch were treated.

83 to the loxP site of the alphoid(tetO)-HAC in hamster CHO cells from where the HAC may be MMCT-transfe

84 donorleucine (Anl) to elongator tRNA(Met) in hamster (CHO), monkey (COS7), and human (HeLa) cell line

85 dependent escalation of aggression in female hamsters depends on activation of mGluR5 receptors.

86 Male European hamsters, deprived of seasonal time cues by pinealectomy

87 Here, we demonstrate that hamsters devoid of functional STAT2 are highly susceptib

88 We found that immunocompetent adult mice and hamsters did not become ill after SFTSV infection.

89 erformance liquid chromatography analyses of hamster ear extracts showed that OG treatment increased

90 ecyloxy)-2-furoic acid significantly reduced hamster ear sebaceous gland size, indicating that this p

91 In hamster efficacy studies, surotomycin protected animals

92 We created browsers for new species (Chinese hamster, elephant shark, minke whale), 'mined the web' f

93 xerts profound metabolic effects in Siberian hamsters exposed to long day (LD) photoperiods that incr

94 the study (HFD-FBX4w group) to Golden Syrian hamsters fed a high-fat diet (HFD) for 8 weeks were inve

95 xamined ACSL1 expression in liver tissues of hamsters fed a normal diet, a high fat diet, or a high c

96 acids found in the faeces of both groups of hamsters fed the FIBEROX suggests that this mechanism is

97 o the third ventricle (3V) affected Siberian hamster feeding behaviors; (2) whether food deprivation

98 origin were permissive of MERS-CoV, whereas hamster, ferret, or mouse cell lines were not, despite t

99 l studies identified rhesus macaques but not hamsters, ferrets, or mice to be susceptible for MERS-Co

100 icantly upregulated the percentage of PFs in hamster fetal ovaries in vitro compared with either of t

101 t of inhaled prions between cells of mice or hamsters following extranasal inoculation with mock-infe

102 Here, we describe a novel model of SFTS in hamsters genetically engineered to be deficient in a pro

103 ed to investigate viral pathogenesis (mouse, hamster, guinea-pig and ferret) are naturally resistant

104 antibody higher than class 1 to mice, rats, hamsters, guinea pigs, and/or rabbits in the examinees w

105 riment, high-fat/cholesterol-diet (HFCD) fed hamsters had higher (p<0.05) weight gains, relative visc

106 Similarly to mouse models, in hamsters, HAdV-C6 is sequestered by macrophages to a les

107 The hamster has been shown to share a variety of metabolic s

108 The use of hamsters has declined, however, most likely due to the d

109 ding individuals, and those that are 13-week hamsters have breeding phenomena.

110 Feeding hamsters HCHFD markedly reduced hepatic Acsl1 mRNA and p

111 LTR-mediated gene remodeling also extends to hamster, human, and bovine oocytes.

112 we cloned 643 bp RFRP cDNA from the striped hamster hypothalamus, which contained an ORF of 570 bp e

113 th either the stimulatory mAb to LAIR-1 or a hamster IgG control.

114 We show that in hamsters immunized with Bacillus subtilis spores express

115 the attractiveness of the odour of the same hamster in a Y-tube olfactometer bioassay, at a late sta

116 effect on the male Syrian and male Siberian hamster in short days.

117 To investigate this, we inoculated hamsters in the sciatic nerve with long-incubation-perio

118 ne expression in entrained versus arrhythmic hamsters in the suprachiasmatic nucleus (SCN) that paral

119 in all organs of intraperitoneally infected hamsters, including the eye and brain, within 1 h after

120 ely, administration of rosuvastatin (RSV) to hamsters increased hepatic Acsl1 expression.

121 ts which are similar to what is exhibited by hamsters infected with ANDV.

122 These results strongly indicate that hamsters infected with Le. infantum become significantly

123 Immunosuppressed hamsters infected with SNV have a mean number of days to

124 this study was to determine if the odour of hamsters, infected with Le. infantum, was more attractiv

125 d a marked, patchy bronchopneumonia, whereas hamster infection with wt Ad14 caused minimal peribronch

126 on the reproductive activity for the striped hamster is less understood.

127 emonstrate that the nasal mucosa of mice and hamsters is not an absolute anatomical barrier to inhale

128 that SFTSV infection in STAT2 knockout (KO) hamsters is responsive to favipiravir treatment, which p

129 heir replication cycles was examined in baby hamster kidney (BHK) cells.

130 (DENV) infection of surface-immobilized baby hamster kidney (BHK-21) fibroblast cells.

131 assembly of Sindbis virus particles in baby hamster kidney cells.

132 thesized in vitro were transfected into baby hamster kidney cells.

133 sage of foot-and-mouth disease virus in baby hamster kidney-21 cells, the virus accumulated multiple

134 nistration of BBR to hyperlipidemic mice and hamsters lowered circulating PCSK9 concentrations and he

135 early onset- (BRCA2-) deficient V79 Chinese hamster lung fibroblast cell line derivative (VC8), and

136 Chinese hamster lung V79 cells and its mutant cell lines, V-C8 (

137 was also observed in calorically restricted hamsters maintained in long days, suggesting that it is

138 The golden hamster (Mesocricetus auratus) is a susceptible model to

139 ons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricetus auratus) of either sex to study t

140 infect commonly used animal models, such as hamster, mice, and ferrets, indicates the presence of a

141 c against the RSV F protein and HPIV1 in the hamster model and provided substantial protection agains

142 PCR1 was confirmed by mortality rates in the hamster model and strong inflammatory responses induced

143 port the use of the newly described STAT2 KO hamster model for evaluation of promising antiviral ther

144 f the vaccine was evaluated in a susceptible hamster model in terms of the humoral immune response an

145 owed enhanced virulence in the Golden Syrian hamster model of acute C. difficile infection.

146 We next used a hamster model of amebic liver abscess to determine the e

147 re severely attenuated for pathogenesis in a hamster model of C. difficile infection.

148 mounts of spores recovered from feces in the hamster model of C. difficile infection.

149 Optimal efficacy in the hamster model of C. difficile was achieved with compound

150 n in vitro and in decreased virulence in the hamster model of CDI.

151 lowers LDL (-74%) and raises HDL (+73%) in a hamster model of dyslipidemia.

152 te to hantavirus disease pathogenesis in the hamster model of HPS.

153 bited increased toxin gene expression in the hamster model of infection.

154 eir ability to cause disease in the standard hamster model of leptospirosis.

155 t of a novel, uniformly lethal Syrian golden hamster model of MHF using a hamster-adapted MARV varian

156 Using the hamster model of visceral disease, we demonstrate that p

157 This MHF hamster model represents a powerful tool for further dis

158 Using the recently described hamster model, along with T-cell depletion strategies, w

159 In a hamster model, prefusion F induced increased quantity an

160 le these studies reflect pathogenesis in the hamster model, they should help us rule out specific cel

161 however, by directly manipulating the Syrian hamster model, we continue to eliminate individual immun

162 ic antibodies than RD in a permissive Syrian hamster model.

163 (b.i.d., orally) in the Leishmania infantum hamster model.

164 the advantages in lipid metabolism features, hamster models are ideally suited for the studies of pan

165 zlotoxumab-mediated protection in murine and hamster models of CDI, suggesting that Fc-dependent tran

166 the therapeutic and toxic effects in Syrian hamster models of pancreatic cancer (PaCa).

167 s) or liver microsomes from male guinea pig, hamster, monkey, mouse, and rabbit or female dog and det

168 ctiveness of odour collected from individual hamsters (n = 13), before they were infected, was compar

169 tinal ganglion cells, and with data from the hamster, Nile grass rat, and laboratory rat.

170 rotein RBD to DPP4 of human (susceptible) or hamster (nonsusceptible) identified five amino acid resi

171 MERS-CoV replication, whereas expression of hamster or ferret DPP4 did not.

172 re injected with eggs purified from infected hamsters or vehicle prepared from uninfected hamster tis

173 Moreover, in YFV-infected hamsters, oral administration of BDAA protected 90% of t

174 and acLDL to CR1 on CR1-transfected Chinese Hamster Ovarian cells (CHO-CR1) was tested by flow cytom

175 n demonstrated from both E. coli and Chinese hamster ovaries (CHO) cell expression platforms; however

176 (E2) upregulates PF formation in developing hamster ovaries.

177 rfusion culture of an IgG1-producing Chinese hamster ovary (CHO) cell line for 18-25 days.

178 nd glycosaminoglycan (GAG)-deficient Chinese hamster ovary (CHO) cell lines and soluble GAGs, we foun

179 For Chinese Hamster ovary (CHO) cell lines, key indicators of metabo

180 platform to image endogenous H2S in Chinese hamster ovary (CHO) cells and use the developed construc

181 While parental Chinese hamster ovary (CHO) cells are permissive for both strain

182 Chinese hamster ovary (CHO) cells are widely used for the produc

183 te any current when transfected into Chinese hamster ovary (CHO) cells but, surprisingly, exerted "ch

184 n neonatal mouse cardiac myocytes or Chinese hamster ovary (CHO) cells expressing the mouse or human

185 dy, we evaluated the genotoxicity to Chinese hamster ovary (CHO) cells induced by municipal secondary

186 Recombinant antibodies produced in Chinese hamster ovary (CHO) cells often exhibit a slight yellow-

187 Chinese hamster ovary (CHO) cells stably expressing a Tet-on ROR

188 NAc on the complex N-glycans of Lec8 Chinese hamster ovary (CHO) cells that lack UDP-Gal transporter

189 totoxicity and acute genotoxicity in Chinese hamster ovary (CHO) cells to compare the toxicity of ana

190 We show that Chinese hamster ovary (CHO) cells used to express recombinant gp

191 Treatment of fibroblasts or Chinese hamster ovary (CHO) cells with 25OH caused a 50-70% redu

192 eased cytosolic G protein by 350% in Chinese hamster ovary (CHO) cells with genetically induced expre

193 ish glycomic survey of bioengineered Chinese Hamster Ovary (CHO) cells with knock-in/out enzymes invo

194 Treating Chinese hamster ovary (CHO) cells with monoHANs followed by the

195 ed as luciferase reporter fusions in Chinese hamster ovary (CHO) cells, where the putative cis elemen

196 Therefore, we generated rhC7 from Chinese hamster ovary (CHO) cells.

197 olate receptor (FR) alpha-expressing Chinese hamster ovary (CHO) cells.

198 stribution in the plasma membrane of Chinese hamster ovary (CHO) cells.

199 human embryonic kidney (HEK 293) and Chinese hamster ovary (CHO) cells.

200 cells used in bioreactors, including Chinese Hamster Ovary (CHO) cells.

201 We show in a Chinese hamster ovary (CHO) disease model cell line and mouse em

202 analogues inhibited proliferation of Chinese hamster ovary (CHO) sublines expressing folate receptors

203 as performed on mouse myeloma SP2/0, Chinese hamster ovary (CHO), and human embryonic kidney (HEK) ce

204 InvD1L expressed in Chinese hamster ovary (CHO)-K1 cells was activated by dopamine w

205 ed pathway inhibition in recombinant Chinese hamster ovary (CHO)-S1P2 cells as well as human lung myo

206 king the cytotoxicity of the HBQs in Chinese hamster ovary (CHO-K1) cells.

207 icles isolated from AQP1-transfected Chinese hamster ovary cell cultures.

208 at its endogenous locus in isogenic Chinese hamster ovary cell lines.

209 tencies based on the comet assay for Chinese hamster ovary cells (assesses the level of DNA strand br

210 (ILs) on zebrafish (Danio rerio) and Chinese hamster ovary cells (CHO) was investigated with specific

211 three homologs that are expressed in Chinese hamster ovary cells (DPY19L1, DPY19L3, and DPY19L4) and

212 ecordings from channels expressed in Chinese Hamster Ovary Cells at different temperatures (32, 37, a

213 ADAP was heterologously expressed in Chinese hamster ovary cells co-expressing alphaIIbbeta3, talin,

214 hat use human receptors expressed in Chinese hamster ovary cells demonstrate that NDD-713 and -825 ha

215 nds from a bioreactor cultivation of Chinese hamster ovary cells expressing a recombinant antibody.

216 m, using RNA interference to deplete Chinese hamster ovary cells of NPC1 alone or in combination with

217 uman beta1-adrenoceptor expressed in Chinese hamster ovary cells revealed negative cooperative intera

218 In Chinese hamster ovary cells that express the channels, Kir2.1 cu

219 Second, in Chinese hamster ovary cells that heterologously express the chan

220 Transport was measured in Chinese hamster ovary cells that stably expressed the human orth

221 31 pentameric complex and gH/gL from Chinese hamster ovary cells to >95% purity.

222 crease in the luciferase activity in Chinese Hamster Ovary cells transfected with CACNA1C and CACNB2

223 m cell-derived cardiomyocytes and in Chinese hamster ovary cells transfected with SCN5A, encoding sod

224 expressed mutant forms of GPIHBP1 in Chinese hamster ovary cells, rat and human endothelial cells, an

225 ity of our setup on fixed and living Chinese hamster ovary cells, showing the cytoskeleton dynamics i

226 channel heterologously expressed in Chinese hamster ovary cells, supporting our findings in DRG neur

227 ptor (1.14 +/- 0.44 nM) expressed in Chinese hamster ovary cells, the binding being reversible and co

228 ypes 1-5 heterologously expressed in Chinese Hamster Ovary cells.

229 nvasion/gentamicin-survival assay in Chinese hamster ovary cells.

230 mary human alveolar macrophages, and Chinese hamster ovary cells.

231 al cultures and human APP-expressing Chinese hamster ovary cells.

232 iotics) to inhibit OAT1 expressed in Chinese hamster ovary cells.

233 cancer cell lines (OVCAR3 and A2780), normal hamster ovary control cells (CHOK1) and alphavbeta3-defi

234 odify protein glycosylation, we used Chinese hamster ovary ldl-D cells defective in UDP-Gal/UDP-GalNA

235 ndogenously expresses DRD2) and CHO (Chinese hamster ovary) cell lines, decreasing luciferase activit

236 in the arrestin recruitment assay in Chinese hamster ovary-K(1) cells expressing the long isoform of

237 binding assays using hB1R-expressing Chinese hamster ovary-K1 cell membranes.

238 upregulation; however, in HEK293T or Chinese hamster ovary-K1 cells overexpressing M3R, pilocarpine i

239 oint mutations were transfected into Chinese hamster ovary-K1 cells, and affinity and function were s

240 These findings indicate that in hamsters, pathology resulting from intravenous infection

241 Here, we show that, in the Siberian hamster (Phodopus sungorus), the programming effect of m

242 Circadian-arrhythmic Siberian hamsters (Phodopus sungorus) exhibit substantial deficit

243 mediated immune responses in adult Siberian hamsters (Phodopus sungorus).

244 eptibilities to aggregation: the susceptible hamster prion (GHaPrP) and its less susceptible rabbit h

245 We conclude that the hamster prion is prone to aggregation at pH 4.4 because

246 -infected transgenic mice overexpressing the hamster prion protein (Tg7 mice) suffer from mitochondri

247 linically ill transgenic mice overexpressing hamster prion protein (Tg7) infected with the hamster pr

248 previous HDX studies on the human and Syrian hamster prion proteins at a higher pH, various segments

249 amster prion protein (Tg7) infected with the hamster prion strain 263K suffer from a severe deficit i

250 Tg7 mice infected with the 263K hamster prion strain have little or no signs of mitochon

251 ts to separate PrP(Sc) aggregates from three hamster prion strains (Hyper, Drowsy, SSLOW) subjected t

252 Here we show that hamster prion-infected transgenic mice overexpressing th

253 similar IgE reactivity in vitro to Siberian hamster protein extracts.

254 cofactors, the P102L mutation in recombinant hamster PrP promoted prion formation when seeded by minu

255 und that in transgenic mice that overexpress hamster PrP(C), PrP(C) overexpression accelerated recomb

256 ng assay using the GeneBridge4 human/Chinese hamster radiation hybrid panel and found to be the MPZL1

257 Furthermore, EhMSP-1 immunization of hamsters reduced development of severe amebic liver absc

258 the 13-week male (r=0.998, P=0.029) striped hamsters respectively, which suggest that RFRP-3 has inh

259 hat repeated aggressive experience in female hamsters resulted in an escalated response to future agg

260 ma within the alveolar septa of SNV-infected hamsters, results which are similar to what is exhibited

261 is of infection and colonization in mice and hamsters revealed that the 50% infectious dose (ID50 ) o

262 on by reconfiguring 10 times faster than the hamster sequence.

263 ansgenic mice that express the Syrian golden hamster (SGH) Prnp.

264 44 of human (Hu), bank vole (BV), and Syrian hamster (SHa) prion protein, from disordered monomers to

265 ansport of prions across the nasal mucosa of hamsters, some of which occurs rapidly in relatively lar

266 the hair, urine, and salivary glands of four hamster species (European, golden, Siberian, and Roborov

267 11 human pathogens, and mitogenic effect on hamster spleen lymphocytes were also tested.

268 two human agents (sCJDMM2 and vCJD) and one hamster strain (263K).

269 he attenuated phenotype observed in previous hamster studies utilizing rNiVs void of C expression.

270 Tissue distribution studies in hamsters suggest, rather, that the transport of antibodi

271 Here, we present a cryo-EM structure of a hamster SUR1/rat Kir6.2 channel bound to a high-affinity

272 The striped hamsters that are 7 weeks and 1.5 years old are non-bree

273 previous LigA vaccine studies, all immunized hamsters that survived infection had renal leptospiral c

274 In hamsters, the presence of the RSV F gene, and in particu

275 In hamsters, the rHPIV1-C(Delta170)-F1, -F2, and -F3 vector

276 in the upper and lower respiratory tracts of hamsters: this was not influenced by the level of RSV F

277 dy, we demonstrate that immunosuppression of hamsters through the administration of a combination of

278 hamsters or vehicle prepared from uninfected hamster tissues (xenoantigen and injection trauma contro

279 The transmission of L. donovani from sick hamsters to flies was surprisingly low (mean, 24% of fed

280 rain transcriptome of male and female Syrian hamsters to generate the necessary resources to continue

281 Our laboratory uses hamsters to study acute social stress, and we are beginn

282 tudy of the thermal activation of a Siberian hamster ultraviolet (SHUV) pigment.

283 L. donovani-infected hamsters underwent xenodiagnoses with Lutzomyia longipal

284 In nature Siberian hamsters utilize the decrement in day length following t

285 -protein-specific serum antibody response in hamsters was increased for the mutants compared to wt HP

286 The odour of six of the golden hamsters was significantly more attractive to 50% of the

287 nfection and the odour of four of the golden hamsters was significantly more attractive to 75% of the

288 To replicate human acute pancreatitis with hamsters, we comparatively studied the efficacy of commo

289 Adult male and female Siberian hamsters were exposed to either dark nights (DARK) or dL

290 These hamsters were found to be susceptible to SFTSV and share

291 Female hamsters were given five daily aggression tests with or

292 Syrian hamsters were inoculated intraperitoneally with brain-de

293 TNF-alpha and IL-1beta contents in HFCD-fed hamsters were reduced (p<0.05) by supplementing BV due t

294 ed for at least 3 weeks after the arrhythmic hamsters were switched back to ad libitum feeding.

295 Syrian hamsters were treated, intratracheally, with clodronate-

296 A radiation hybrid panel of chicken-hamster Wg3hCl2 cells was used to map the genome locatio

297 ed by investigating effects of FGF21 in aged hamsters, which is associated with reduced adiposity.

298 Intratracheal infection of Syrian hamsters with Ad14p1 caused a marked, patchy bronchopneu

299 LDL cholesterol levels were investigated in hamsters with diet-induced dyslipidemia.

300 In LD hamsters with increased adiposity, FGF21 lowered body we