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1 ty contractile exercise (25% and 80% maximal handgrip).
2 reased within 1 to 2 seconds of the onset of handgrip.
3 d all of the vasodilatation to contralateral handgrip.
4 ed by damage in brain areas activated during handgrip.
5 one (-54 +/- 11 ms), and also with PS during handgrip (+10 +/- 10 ms) compared with PS alone (-74 +/-
6  vascular resistance increases (basal versus handgrip, 17 +/- 1 versus 26 +/- 2 U; P = .01) in respon
7 l cortical vascular resistance (basal versus handgrip, 18 +/- 1 versus 25 +/- 3 U; P = .002); and (4)
8 e (BP; Finapres) were measured during static handgrip (20 s) at 10% and 70% of maximum voluntary cont
9                      During mild to moderate handgrips (20-33% MVC) that do not evoke reflex-sympathe
10 l cortical vascular resistance (basal versus handgrip, 20 +/- 1 versus 25 +/- 2 U; P = .04); (3) the
11 er post-flight in all subjects before static handgrip (26 +/- 4 post- vs. 15 +/- 4 bursts min(-1) pre
12 pared with metoprolol and placebo (isometric handgrip -3.5 U for carvedilol versus -1.2 U for metopro
13                        Likewise, when static handgrip (30 % MVC) was performed to fatigue, MSNA incre
14                             During sustained handgrip (30% maximum voluntary contraction), peak renal
15 y decrease in renal blood flow (basal versus handgrip, 4.2 +/- 0.2 versus 3.5 +/- 0.3 mL.min-1.g-1; P
16  cortical blood flow decreases (basal versus handgrip, 4.4 +/- 0.1 versus 3.5 +/- 0.1 mL.min-1.g-1; P
17                        During high intensity handgrip, (45% MVC), contraction-induced decreases in mu
18                         During contralateral handgripping after stellate block, blood flow in the res
19  unexpectedly increased by 54 +/- 11% during handgrip alone.
20  peripheral afferent stimulation produced by handgrip and a cold pressor test in humans.
21 e subjects, vasomotor responses to isometric handgrip and cold pressor test did not differ between tr
22 m vascular resistance responses to isometric handgrip and cold pressor test were determined by plethy
23 55.3%), we compared the responses to dynamic handgrip and during a 3-minute period of posthandgrip re
24                                              Handgrip and forearm vasodilation yielded no haplotype d
25 i-Mental State Examination (MMSE) score, and handgrip and handheld dynamometer strength.
26                       In addition, simulated handgrip and intermittent forearm compression produced b
27 e is greater in men than women during static handgrip and LBNP.
28 limb function and increased activations with handgrip and median nerve stimulation, but reduced activ
29 ty and blood pressure responses to fatiguing handgrip and post-exercise circulatory occlusion were si
30 he left primary motor cortex leg area during handgrip and the left primary sensory cortex face area d
31 al activation in response to right-sided (i) handgrip; and (ii) median and tibial nerve stimulation w
32  patient (I.G.) was quite unable to open her handgrip appropriately when directly reaching out to pic
33 unctional composite scores: muscle strength (handgrip, arm, and leg) and mobility (timed-up-and-go, c
34 d at rest and during right and left rhythmic handgrip before and 4 to 7 weeks after right TST.
35 thy volunteers performed fatiguing isometric handgrip before and after a local infusion of pyridoxine
36 e (FVC) during infusion of ADO to FVC during handgripping before and after infusion of dipyridamole (
37 ed exercise hyperaemia during heavy rhythmic handgripping, but vasodilator responses to exogenous ADO
38                             MSNA during post-handgrip circulatory arrest was higher post- than pre- o
39                     In contrast, during post-handgrip circulatory arrest, which isolates muscle metab
40 s during a 3-min sustained maximal voluntary handgrip contraction.
41 lthy subjects in two tasks: (1) intermittent handgrip contractions at 20, 40, 60, and 80% of maximal
42 thy subjects performed repetitive unilateral handgrip contractions that induced significant muscle fa
43 ndgrip, whereas the pressor response to left handgrip did not change.
44          There was no effect at 6 months for handgrip (difference, 2.0 kg [95% CI, -1.3 to 5.4], P =
45 X-ray absorptiometry, muscle strength with a handgrip dynamometer, and blood biochemical indexes of n
46 sment (cognition), manual muscle testing and handgrip dynamometry (muscle and/or nerve function), and
47 ater reliability of handheld dynamometry and handgrip dynamometry was assessed in one study, and resu
48                    Interrater reliability of handgrip dynamometry was very good in two studies (intra
49 subjective global assessment, anthropometry, handgrip dynamometry, biochemical and amino acid profile
50 arm (-12 +/- 1%) that were attenuated during handgrip exercise (-3 +/- 1%; P < 0.05).
51 2 agonist) in 10 healthy men during rhythmic handgrip exercise (10-15 % of maximum) and during a cont
52 energic receptor stimulation during rhythmic handgrip exercise (15% maximum voluntary contraction) an
53  (alpha(2)-agonist) during moderate rhythmic handgrip exercise (15% maximum voluntary contraction), d
54 etomidine (alpha(2)-agonist) during rhythmic handgrip exercise (15% MVC), a control non-exercise vaso
55  Seven subjects performed 2 min of isometric handgrip exercise (35% of maximal voluntary contraction)
56 cle (forearm) blood flow (FBF) during graded handgrip exercise (5, 15, 25% maximal voluntary contract
57 alsalva manoeuvre (by approximately 45%) and handgrip exercise (by approximately 27%) with unaffected
58 sed by vasomotor reactivity during isometric handgrip exercise (IHE), was recently quantified noninva
59  in contracting muscle during heavy rhythmic handgrip exercise (n = 4).
60 d when forearm ischemia was maintained after handgrip exercise (posthandgrip circulatory arrest).
61  forearm vascular resistance at rest, during handgrip exercise and after transient arterial occlusion
62  (ml/min/100 ml) at rest and during rhythmic handgrip exercise and after transient arterial occlusion
63 energic receptor stimulation during rhythmic handgrip exercise and during a control non-exercise vaso
64                                       Graded handgrip exercise and post-handgrip ischemic arrest were
65 r transduction was evaluated during ischemic handgrip exercise and postexercise ischemia, and it was
66 nce (FVC) responses during 5 min of rhythmic handgrip exercise at 20% maximal voluntary contraction i
67                      In protocol 2, rhythmic handgrip exercise at 35% maximum voluntary contraction w
68 .5 tesla before, during, and after isometric handgrip exercise at a level that was 30 percent of the
69                                              Handgrip exercise changes ocular perfusion pressure free
70            Wave energy also decreased during handgrip exercise due to increased heart rate.
71 vidence of an abnormal metabolic response to handgrip exercise in at least some women with chest pain
72                             During sustained handgrip exercise in heart failure, both the magnitude a
73 conductance (CVC) decreases during isometric handgrip exercise in heat stressed individuals, and we h
74 the influence of blood flow occlusion during handgrip exercise on neuromuscular fatigue development a
75 (15% maximal voluntary contraction) rhythmic handgrip exercise or adenosine infusion.
76 tic traffic to the resting forearm, rhythmic handgrip exercise to fatigue followed by post-exercise i
77 rther aim was to examine the effect of local handgrip exercise training on radial artery L-FMC and fl
78 ted and randomly assigned to either a 6-week handgrip exercise training program (N=9) or a nonexercis
79 ng a sympatho-excitatory stimulus (isometric handgrip exercise) after either exercise (60 min cycling
80 e deoxygenation (systemic hypoxia and graded handgrip exercise) with age, which was caused by reduced
81 ed brachial artery blood flow during maximal handgrip exercise, 6-minute walk test, maximal oxidative
82 RI was performed before and during isometric handgrip exercise, an endothelial-dependent stressor, an
83  were quantified before and during isometric handgrip exercise, an endothelial-dependent stressor.
84 (2)) in cubital venous blood at rest, during handgrip exercise, and during recovery in 13 patients wi
85 forearm vascular conductance during ischemic handgrip exercise, despite a normal pressor response, su
86     Retina/choroid BF increases during brief handgrip exercise, paralleling increases in mean arteria
87                                       Graded handgrip exercise, posthandgrip circulatory arrest, and
88 r data indicate that during graded intensity handgrip exercise, the reduced FVC and subsequently lowe
89 ow during mild (10% MVC) continuous rhythmic handgrip exercise.
90 r of the muscle metaboreflex during ischemic handgrip exercise.
91 alva manoeuvre but remained unaltered during handgrip exercise.
92 uctance catheter during basal conditions and handgrip exercise.
93  also obtained during Valsalva manoeuvre and handgrip exercise.
94 nce imaging (MRI) at rest and during maximal handgrip exercise.
95 alva manoeuvre but remained unchanged during handgrip exercise.
96  rest and during moderate-intensity rhythmic handgrip exercise.
97 s 26 +/- 2 U; P = .01) in response to static handgrip exercise; (2) central command and/or the mechan
98 KCl) at rest; (2) mild or moderate intensity handgrip exercise; and (3) combined mild exercise + ACh,
99 adrenergic stimulation achieved through post-handgrip-exercise ischaemia (PEI) and beta1 -adrenergic
100  The ergoreceptor test involved two 5-minute handgrip exercises.
101                                       During handgrip, femoral vascular conductance was reduced 47.2
102 ollowing leg cycling exercise, (3) isometric handgrip followed by PEI.
103                                          The handgrip force and electromyograms (EMG) of the finger f
104                    The task involved trading handgrip force production against monetary benefits.
105 ings of scalp electroencephalographic (EEG), handgrip force, and finger flexor surface electromyograp
106                                         Both handgrip (HG) and disengagement of baroreflexes with low
107  (BMI) and lean body mass (LBM) depletion on handgrip (HG) force and inspiratory muscle function (IMF
108                         During PEI following handgrip, HR was similarly elevated from rest under cont
109                          Interestingly, post-handgrip hyperaemia is greater in women than men and is,
110 ate were measured during fatiguing isometric handgrip (IHG) at 30% maximum voluntary contraction and
111                                    Isometric handgrip (IHG) exercise increases sweat rate and arteria
112 arm flow and autonomic responses to ischemic handgrip in young and older subjects.
113 as they executed their fastest and strongest handgrips in response to a visual cue, which was accompa
114            Graded handgrip exercise and post-handgrip ischemic arrest were used to clarify the reflex
115 um and phentolamine prior to another bout of handgripping, little or no vasodilatation was seen eithe
116             Eight participants performed 200 handgrip maximal voluntary contractions (MVCs) with simu
117 ry responses to stressful stimuli (sustained handgrip, maximal forearm ischemia, mental stress, and t
118         Measurements included quadriceps and handgrip maximum voluntary isometric force and the relax
119 lly from those obtained in a 2-min sustained handgrip MVC published in a recent report, in which the
120 icant fatigue were induced; (2) intermittent handgrip MVCs (100 trials) that resulted in significant
121 man participants performed approximately 100 handgrip MVCs (each 2-s contraction was followed by a 1-
122  The fMRI data from the task of intermittent handgrip MVCs differed dramatically from those obtained
123  CI, -4.07 to -1.30; P < .001) and decreased handgrip myotonia on clinical examination (mexiletine, 0
124                                              Handgrip myotonia was seen in three-quarters of particip
125 myotonia assessment; quantitative measure of handgrip myotonia; and Individualized Neuromuscular Qual
126 h partial flow restriction and PEI following handgrip (P > 0.05 vs. rest).
127 line leg power (p trend = 0.046), and poorer handgrip (p trend = 0.005) were associated with higher a
128 sing post-exercise ischaemia (PEI) following handgrip partially maintains exercise-induced increases
129 (PEI-M) and high (PEI-H) intensity isometric handgrip performed at 25% and 40% maximum voluntary cont
130                 In controls, 2 min of static handgrip performed at 33 % or 45 % of maximal voluntary
131  rested for 1 minute followed by 1 minute of handgrip, repeating three times, while maintaining stabl
132 uding the cold pressor test (CPT), sustained handgrip (SHG), and mental stress (MS).
133 e activity (SSNA) during intermittent static handgrip (SHG; at 45% of maximal voluntary contraction;
134                                              Handgrip significantly increased retina/choroid BF by 25
135 gh muscle volume (3.6%; 95% CI: 0.2%, 7.0%), handgrip strength (2.3 kg; 95% CI: 0.8, 3.7 kg), and 1-R
136                        Athletes had superior handgrip strength (Athletes: 55.92 +/- 17.06 vs. CONTROL
137 idarm and calf circumference, serum albumin, handgrip strength (HGS), and patient-self assessment of
138 9), android distribution of fat (P = 0.021), handgrip strength (P = 0.001), standardized summary scor
139    Longer ICU stay was associated with lower handgrip strength (P<0.01) and lower aromatic amino acid
140 th a few additional tests that often include handgrip strength and arm-muscle circumference.
141                                              Handgrip strength and body mass index (BMI; in kg/m(2))
142                           Lower preoperative handgrip strength and branched chain amino acid levels a
143                                              Handgrip strength and phase angle improved after LTx (P<
144 cant increase of about 11% in quadriceps and handgrip strength at mid-cycle compared with both the fo
145 tified analysis, LTL was not associated with handgrip strength in either men or women.
146                    We noted no difference in handgrip strength in ROMANA 1 (-1.10 kg [-1.69 to -0.40]
147 were the median change in lean body mass and handgrip strength over 12 weeks and were measured in all
148 ombined resting energy expenditure (REE) and handgrip strength provided a valuable assessment in data
149 was assessed by means of a maximal isometric handgrip strength test and a test of lower-back trunk mu
150                                              Handgrip strength test results and discomfort ratings di
151 patency of the ulnopalmar arches, as well as handgrip strength tests to examine the isometric strengt
152       We designed an effort measure based on handgrip strength to assess the motivation to earn money
153  were at least 30% lower than predicted, but handgrip strength was relatively preserved.
154 , systolic and diastolic blood pressure, and handgrip strength), behavioural (smoking, alcohol consum
155  by anthropometric assessment, bioimpedance, handgrip strength, and dietary intake (before and 30, 90
156 mpedance, quadriceps, respiratory muscle and handgrip strength, and physical performance with the Sho
157 he secondary outcomes were body composition, handgrip strength, and physical performance.
158                Subjective Global Assessment, handgrip strength, multifrequency bioelectrical impedanc
159                         Thigh muscle volume, handgrip strength, one-repetition maximum (1-RM) lower-
160         Physical performance was assessed by handgrip strength, the Short Physical Performance Batter
161                                  We compared handgrip strength, usual gait speed, timed up and go (TU
162            We find unique evidence of ID for handgrip strength, waist/hip ratio, and visual and audit
163 tional indices, anthropometric variables and handgrip strength.
164  Timed Up and Go (TUG) test, and dynamometer handgrip strength.
165 d isokinetic strength of the lower limbs and handgrip strength.
166                             In children, the handgrip strength/body mass levels for a low metabolic r
167  from Colombia, using MS relative to weight (handgrip strength/body mass).
168 nificantly increased lean body mass, but not handgrip, strength in patients with advanced non-small-c
169  suspected myocardial ischemia underwent MRS handgrip stress testing and follow-up evaluation.
170 MSNA) were recorded before and during static handgrip sustained to fatigue at 40 % of maximum volunta
171 ography during a visually cued, incentivized handgrip task in subjects with Parkinson's disease (n =
172 sure by 25%+/-6% (averaged across the entire handgrip task) (P<0.01), but did not change intraocular
173 e by 22%+/-5% (measured at the middle of the handgrip task), and ocular perfusion pressure by 25%+/-6
174 forming an isometric, dynamic visually paced handgrip task.
175  the reference group (normal BMI and highest handgrip tertile), the risk of all-cause mortality incre
176               For participants in the lowest handgrip tertile, there was little difference in the ris
177 ot affected by melatonin during an isometric handgrip test (30% maximum voluntary contraction) and a
178            Basic fitness measures included a handgrip test and 3-minute step test.
179                                    Isometric handgrip testing (IHGT), a well-established laboratory-b
180  179 +/- 4 cm) completed four constant power handgrip tests to exhaustion under conditions of control
181  Most patients had markedly impaired TUG and handgrip tests, and 21% recalled having fallen more than
182                  Subjects performed rhythmic handgrip (thirty 1-sec contractions/min) at 30% maximal
183  more avidly an action involving squeezing a handgrip to earn money.
184 ubjects (3 women, 7 men) performed ischaemic handgripping to fatigue before and after acute local ana
185   Manoeuvres such as contralateral ischaemic handgripping to fatigue that cause vasoconstriction in t
186 excitation evoked by contralateral ischaemic handgripping to fatigue.
187 orearm vasoconstriction during contralateral handgripping to fatigue.
188 trations may not rise enough during rhythmic handgripping to have a major impact on these responses.
189                    cBRS was unchanged during handgrip under free-flow conditions, handgrip with parti
190                  Pressor responses to static handgrip were also attenuated in patients compared to co
191                        Increases in CVR with handgrip were greater in men vs. women (1.25 +/- 0.49 vs
192                          Changes in CBV with handgrip were linked to the myocardial oxygen consumptio
193    In the patients, MSNA responses to static handgrip were markedly attenuated (33 +/- 14 % at 33 % M
194 ure and mean arterial pressure during static handgrip were not different before, during and after spa
195 fter practice, I.G. became able to scale her handgrip when grasping a real target object that she had
196 ced mean arterial pressure response to right handgrip, whereas the pressor response to left handgrip
197 coronary vasoconstriction occurs with static handgrip with a time course that suggests a sympathetic
198  during handgrip under free-flow conditions, handgrip with partial flow restriction and PEI following
199                                        After handgrip, women had a greater rise in conductance than m
200  unilateral thigh-cuff release and isometric handgrip) would be greater after the administration of t

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