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1 wide insertional mutagenesis screen in human haploid cells.
2 a known ubiquitin pathway substrate in alpha haploid cells.
3 tory element that represses transcription in haploid cells.
4 f IME2, a meiotic gene normally repressed in haploid cells.
5 cell-specific promoter in meiotic and early haploid cells.
6 are dominant in diploid cells and lethal in haploid cells.
7 uring fission yeast karyogamy upon mating of haploid cells.
8 the use of forward genetic screens in human haploid cells.
9 establish the physiological role of Ime4 in haploid cells.
10 s that escape sex chromosome inactivation in haploid cells.
11 and was manifested by delayed production of haploid cells.
12 -activated cell sorting (FACS) enrichment of haploid cells.
13 PCR triggers events leading to the fusion of haploid cells.
14 breaks within the chromosomes of nondividing haploid cells.
15 lpha mating or from unisexual mating between haploid cells.
16 ks being required to isolate PGCs as well as haploid cells.
17 ation, and chromosome dosage compensation in haploid cells.
18 g balanced expression of the genome in these haploid cells.
19 re susceptible to copy number fluctuation in haploid cells.
20 d cells, was localized to the distal pole of haploid cells.
21 simultaneous cytokinesis yields a tetrad of haploid cells.
22 th the effects of the tub2-T143G mutation in haploid cells.
23 y of approximately 0.1% in HR-competent Rad+ haploid cells.
24 kinase cascade that also regulates mating of haploid cells.
25 s, expansion is limited to the post-meiotic, haploid cell and therefore cannot involve mitotic replic
26 te asexually within their mammalian hosts as haploid cells and are subject to DNA damage from the imm
29 ormed a genome-wide knockout screen in human haploid cells and identified the calcium pump SPCA1.
30 f SIN3 and RPD3 in that it represses IME2 in haploid cells and is necessary for sporulation in diploi
31 e chromosome segregation defect of bub1Delta haploid cells and restores viability to bub1Delta tetrap
32 oteins were present in APC preparations from haploid cells arrested in G(1), S, and M phases and from
33 ed in both haploid and diploid cells, and in haploid cells arrested in G1 with alpha-factor or in S p
36 rting approach also enables the isolation of haploid cells at low percentages, as well as the mainten
38 that YAR1 is not an essential gene, but that haploid cells bearing a yar1 deletion grow significantly
39 ferently in haploid and diploid yeast cells: haploid cells bud in an axial manner, while diploid cell
42 scovered to influence mating-type control in haploid cells by locus-specific transcriptional silencin
49 pecifically increased in the G1 stage of the haploid cell cycle, as well as by the glucose depletion-
52 oexpression of MATa1 and MATalpha2 in Sir(+) haploid cells did not lead to lethality from the HO-indu
53 mportant in regulation of diploid as well as haploid cell differentiation in a variety of tissues.
57 rowth is determined genetically: a and alpha haploid cells exhibit an axial budding pattern, and a/al
58 ious environmental conditions, revealed that haploid cells experienced higher rates of silencing loss
66 e higher eukaryotes, the first mitosis after haploid cell fusion in budding yeast may forgo cell cycl
68 elopment of forward genetic screens in human haploid cells has the potential to transform our underst
71 stranded breaks that cannot be repaired by a haploid cell if induced before replication, does not inv
72 d 1-butanol, stimulate filamentous growth in haploid cells in which this differentiation is normally
73 functions as an activator site in vegetative haploid cells, it seemed likely that the factors binding
75 is needed for proper bud site selection, as haploid cells lacking Dbm1p bud predominantly in a bipol
78 and the development of genetic tools such as haploid cell lines, allowing high-throughput screening t
79 amental genetic feature in mammals, in which haploid cells normally arise only as post-meiotic germ c
82 re we report that simultaneous expression in haploid cells of both MATa and MATalpha information supp
84 myces cerevisiae, the pheromones that induce haploid cells of opposite cell types to mate activate th
91 yeast Schizosaccharomyces pombe normally has haploid cells of two mating types, which differ at the c
92 ump2 eliminated the filamentous phenotype of haploid cells on low ammonium, while ump1 disruption onl
94 tures, telomerase activity becomes limiting: haploid cell populations senesce and generate aneuploid
97 plex system for switching the mating type of haploid cells, requiring the genome to have three mating
99 the budding yeast Saccharomyces cerevisiae, haploid cells respond to mating pheromone through a G-pr
103 e simulations, colonies initiated by an aged haploid cell show declined mating probability at an earl
104 ase is similar in haploid and diploid cells, haploid cells spend longer in mitosis, indicative of pro
108 ls undergo meiosis to produce sperm or eggs, haploid cells that are primed to meet and propagate life
114 es are brought together by the fusion of two haploid cells, the Y and Z proteins from different matin
116 d a loss-of-function genetic screen in human haploid cells to identify host factors important in C. t
118 esults demonstrate the utility of screens in haploid cells to study interactions of human cells with
119 d cooperatively to sites in the promoters of haploid cell-type-specific genes (hsg) to repress their
120 large number of distinctly regulated genes: haploid cell-type-specific genes, G2-cell-cycle-regulate
122 and efficient transitions between alternate haploid cell types allow the organism to access the adva
124 es can also stimulate filamentation, whereas haploid cells undergo a similar invasive growth response
125 In eukaryotes, diploid cells give rise to haploid cells via meiosis, a program of two cell divisio
127 ow glucose influences budding pattern in the haploid cell, we examined the roles of bud-site-selectio
128 These include the lethality of tub1-724 haploid cells when the beta-tubulin-binding protein Rbl2
130 sive synaptonemal complexes and post-meiotic haploid cells with a similar pattern of ACROSIN staining
131 lates to a high level in mitotically growing haploid cells, yet transposition occurs at very low freq
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