ライフサイエンスコーパス: hard exudate

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1 east in some cases it resolves with residual hard exudate.

2 ated with increased risk of CSME and retinal hard exudate.

3 al pigment epithelium alterations with dense hard exudates.

4 e associated with the development of retinal hard exudates.

5 loss is likely to be associated with retinal hard exudate and serum lipid abnormalities.

6 Hard exudates and CME regressed completely in 15 of 15 a

7 id not show significant risk in worsening of hard exudates and severity of DME in the lipid-lowering

8 clinically significant macular edema (CSME), hard exudates, and other diabetic retinopathy (DR) end p

9 Hard exudate area was assessed from color fundus stereop

10 es from autopsy specimens have characterized hard exudates as a composition of lipid-laden macrophage

11 d exudate scores, and ETDRS scores minus the hard exudate component.

12 and triglycerides) with development of CSME, hard exudate, DR progression, and development of prolife

13 ological outcomes between eyes with baseline hard exudates (HE) and all other eyes among patients wit

14 inopathy Study protocol) for the presence of hard exudates (HE), retinal thickening (RT), clinically

15 Hard exudates (HEs) are the classical sign of diabetic r

16 ith higher risk of incident CSME and macular hard exudate in the DCCT cohort.

17 they were mild increase in CFT, presence of hard exudates in center subfield, and absence of renal d

18 However, the characteristics of hard exudates in living patients have not been examined

19 This differentiation of retinal hard exudates in patients by AO-SLO may help in understa

20 y (AO-SLO) to examine the characteristics of hard exudates in patients with retinal vascular diseases

21 acy of radially arranged sectors affected by hard exudates in the detection of CSME.

22 on-wool spots, intraretinal hemorrhages, and hard exudates in the macula were observed by ophthalmosc

23 bles morphological classification of retinal hard exudates into two types, which could not be disting

24 Clinically, this appeared as hard exudate on funduscopic images.

25 than the thickness in regions with irregular hard exudates (P = 0.01 -->0.02).

26 tinal mass, and vitreous seeds resolved, the hard exudates persisted for more than 2 years after the

27 Hard exudates regressed completely in 14 of 15 eyes and

28 he macular edema had completely resolved and hard exudates regressed slowly in 100% of patients.

29 lipid-lowering group compared with placebo (hard exudates: relative risk, 1.00; 95% CI, 0.47-2.11; P

30 ed visual acuity (BCVA), presence of retinal hard exudates, retinal detachment (RD), cystoid macular

31 and RR 1.95, P for trend = 0.03 for LDL) and hard exudate (RR 2.44, P for trend = 0.0004 for total-to

32 t Diabetic Retinopathy Study (ETDRS) scores, hard exudate scores, and ETDRS scores minus the hard exu

33 Similarly, for the development of retinal hard exudates, the RR for the top vs bottom quintile of

34 The retinal thickness in regions with round hard exudates was significantly greater than the thickne

35 Retinal hard exudate, which is accompanied by macular edema, is

36 The cases of hard exudates within 1 disc diameter (DD) of the fovea (

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