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1 n the osteogenic patterning of the posterior hard palate.
2 the maxillary and mandibular gingiva and the hard palate.
3 d to a distinct adult tissue type resembling hard palate.
4 isional wounds were created in the scalp and hard palate.
5 o incomplete clefting within the presumptive hard palate.
6 rogressively enlarging ulcerated mass on the hard palate.
7 sgenesis and bilateral cleft of the soft and hard palate.
11 nail pigmentation, blue pigmentation on the hard palate, and black pigment deposits on the conjuctiv
12 trinsically coupled to the elongation of the hard palate, and is critical for modulating the growth o
14 lesions (four on the gingiva and two on the hard palate), clinically identical to the previously dis
15 terms of relative tissue composition in the hard palate, DE-harvested CTG contains much larger amoun
16 ns in all four gingival quadrants and on the hard palate (eight on the gingiva and six on the palate)
19 what is believed to be the initial report of hard-palate infection caused by Blastomyces dermatitidis
20 shortening of the snout and widening of the hard palate is common to brachycephalic dogs and is a we
21 .7 +/- 0.6 versus 6.2 +/- 0.4 cm, p < 0.05), hard palate length (3.2 +/- 0.4 versus 3.7 +/- 0.2 cm, p
24 reduction in bone formation in the posterior hard palate, resulting in the classic notch associated w
25 sitions: buccal mucosa, keratinized gingiva, hard palate; saliva, tongue, tonsils, throat; sub- and s
27 olume and fat content were assessed from the hard palate to the vocal cords using T1-weighted images.
28 ramembranous bone formation in the posterior hard palate, which underpins normal palate development a
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