ライフサイエンスコーパス: harmless

1 ade or moderately low-grade tumors (possibly harmless).

2 any proponents think, not all treatments are harmless.

3 Mn down-regulates GPP130, rendering STx/STx1 harmless.

4 ecific tobacco etch virus protease (TEVP) is harmless.

5 sitivity to calcium ions and is biologically harmless, aequorin is widely used as a probe to monitor

6 induce allergic sensitization to a normally harmless Ag, and they may explain why secondhand smoke i

7 ne and inflammatory responses to an inhaled, harmless Ag, whereas endogenous IL-13 was necessary for

8 r the priming of naive mice with an inhaled, harmless Ag.

9 bility to AAI after mild exposure to inhaled harmless Ags that otherwise induce Ag tolerance.

10 -state conditions and that their sampling of harmless airborne antigen and presentation to antigen-sp

11 likely to represent an abnormal response to harmless airborne particles.

12 so mount maladaptive Th2 immune responses to harmless allergens and, in this way, contribute to immun

13 Commensal bacteria are largely seen as harmless and can survive symbiotically (in many cases) i

14 Most human papillomavirus infection is harmless and clears spontaneously but persistent infecti

15 We live and die surrounded by harmless and harmful organisms that affect us in a multi

16 mplex with Hsp90, the oligomers are rendered harmless and nontoxic to cells.

17 ed by asymptomatic carriers finding, that if harmless and pathogenic strains of the infected agent co

18 ice concerning 'stress steroids' is probably harmless and possibly beneficial, similar benefits might

19 either harmful (loss-of-function) alleles or harmless and thus inconsequential polymorphisms.

20 nd adolescents increasingly view cannabis as harmless, and some can use cannabis without harm.

21 n tolerance/ignorance of the large volume of harmless antigens and bacterial commensals encountered a

22 le balancing this immunity with tolerance to harmless antigens and bacterial commensals.

23 mune homeostasis by suppressing responses to harmless antigens and by enforcing the integrity of the

24 normalities in mucosal responses to normally harmless antigens in the mucosal microflora and therefor

25 Unresponsiveness to harmless antigens is established through central and per

26 Aberrant antibody production to otherwise harmless antigens may result in autoimmunity.

27 for inducing immunological tolerance toward harmless antigens to maintain immune system homeostasis.

28 against any of a broad variety of apparently harmless antigens.

29 ich helps establish tolerance to foreign yet harmless antigens.

30 ed against any of a broad range of seemingly harmless antigens.

31 erance is the usual outcome of inhalation of harmless antigens.

32 discriminate between potentially harmful and harmless antigens.

33 e bacterial species Vibrio cholerae includes harmless aquatic strains as well as strains capable of c

34 or where changes in the tract are apparently harmless are uncertain.

35 The concept of a harmless artificial spore can be applied to developing i

36 solid tumour growth, linking the relatively harmless avascular growth phase and the potentially fata

37 arbon nanotube (MWCNT) films using cultured, harmless bacteria as porogens, and demonstrate substanti

38 necessary immune responses against otherwise harmless bacteria.

39 Yet, cells fused by viruses are considered harmless because they die.

40 The normally harmless behavior of bacteria in the intestinal tract is

41 The most prominent hot spot was a harmless chromosomal niche in the rRNA gene repeats, whe

42 The transition from harmless colonization to disease is typically preceded b

43 Although independently harmless, combined treatment of 5-fluoro-1-propargyl-ura

44 reased inflammatory response to the normally harmless commensal flora.

45 accidental pathogens as mutants of otherwise harmless commensal organisms is the challenging new feat

46 to defend against pathogens while tolerating harmless commensal organisms remains an important puzzle

47 ious infections, as well as tolerance toward harmless commensals.

48 that metabolically healthy obesity is not a harmless condition and that the obese phenotype, regardl

49 the mobbing of cuckoos but not of parrots (a harmless control) on their own territory.

50 e susceptible to false-positive signals from harmless dead pathogens.

51 ce the greenhouse gas nitrous oxide (N2O) to harmless dinitrogen gas are receiving increased attentio

52 the photosynthetic membrane, facilitate the harmless dissipation of excess excitation energy directl

53 s composed of pathogenic E. coli O157:H7 and harmless E. coli DH5alpha using flow cytometry.

54 ombined with an aerobic on site treatment to harmless end products.

55 to understand the biology of how this mostly harmless endemic virus gives rise to lethal disease.

56 ecific immune responses against an otherwise harmless enteric microbiota are key factors in the patho

57 unting inappropriate inflammatory attacks on harmless environmental antigens (leading to asthma), ben

58 peptides derived from the microbe as well as harmless environmental antigens.

59 n predisposition to infection with otherwise harmless environmental or microbiota-derived species.

60 ignorance or tolerance not to overrespond to harmless environmental stimuli.

61 ted with neuronal deterioration, rather than harmless epiphenomena, during the process of neurodegene

62 ly distinguishing pathogenic infections from harmless exposures.

63 exists both as functional toxic pores and as harmless fibrils suggests that protein aggregation into

64 the development of sensitization to normally harmless food allergens, as well as delineating the crit

65 es "false alarms," causing animals to reject harmless foods.

66 rable ("sensitizing drive") in a way that is harmless for a wild-type allele.

67 Although most avian influenza viruses are harmless for humans, some (such as highly pathogenic H5N

68 onsecutive reactions converting toxic TCP to harmless glycerol.

69 uce an attacking O(2) molecule completely to harmless H(2)O.

70 the therapeutic use of a defined mixture of harmless, health-associated bacteria, holds promise for

71 need precise molecular thermometers to sense harmless increments in the ambient temperature to induce

72 ishes a systemic infection with a robust yet harmless infection of the brain, seemingly analogous to

73 ible-infected-recovered system (SIR) for the harmless infective agent, acting as a background to a mu

74 nd in the priming of an immune response to a harmless, inhaled Ag.

75 rprise, the same Th2 T cells that produced a harmless insulitis in neonatal NOD mice produced intense

76 e by selectively habituating to the calls of harmless killer whales.

77 used to run virtual experiments, in which a harmless labeling mutation is bestowed upon a single cel

78 ores was suggested as the most efficient and harmless Lf loading method.

79 sensitization, an exaggerated sensitivity to harmless light, occurs genetically in rare diseases, suc

80 al molecular imaging makes use of relatively harmless, low-energy light and technically straightforwa

81 rategies that convert female mosquitoes into harmless males.

82 in the process by which CTXvarphi converts a harmless marine microbe into a deadly human pathogen.

83 ovides a new model in which a common, mostly harmless member of the human viral flora can initiate ca

84 ra, highlighting the complex interplay among harmless microbes, pathogens, and host immunity in the r

85 tion between natural stimulation coming from harmless microbiota and food, and pathogens that need to

86 ptures a pathogenic mutagenic mechanism as a harmless mistake which, when it occurs in other genetic

87 ortant role in the balance between tolerance harmless mucosal Ags and commensal bacteria and immunity

88 mutations are assumed to be deleterious but harmless (neutral) in appropriate combinations.

89 apting their aversive behavioral response to harmless noxious compounds.

90 This often leads to the misidentification of harmless objects as weapons held by a Black individual.

91 oth Gram-negative and positive bacteria were harmless on SsoPox-W263I activity and could reactivate h

92 arly and distinguish vulnerable plaques from harmless ones.

93 cancer, a complex disease, can be relatively harmless or extremely aggressive.

94 atoris' remarkable virulence amid a clade of harmless or much less virulent species.

95 e the virus or suppress it indefinitely as a harmless, persisting infection.

96 ferentiate between deleterious mutations and harmless polymorphic variants.

97 t to distinguish inactivating mutations from harmless polymorphisms.

98 uild learning to associate that pattern with harmless prey.

99 and hydrogen peroxide, are converted to the harmless product water.

100 ulatory signal through OX40 allows otherwise harmless, proliferating, autoreactive T cells to acquire

101 folded or toxic misfolded polypeptides into harmless protease-degradable or biologically active nati

102 n catabolism is widespread (70 to 75%) among harmless respiratory isolates but is nearly universal (9

103 triphosphate (ATP) synthesis, this initially harmless response might enhance the vulnerability of hep

104 step intoxication process, transforming from harmless, robust crystals, to soluble protoxin heterodim

105 agreeable experiences, (ii) the more or less harmless, sessile, and largely edible mimics vastly outn

106 Bacillus subtilis spores, harmless simulants for Bacillus anthracis, were studied

107 be highly maladaptive when individuals elude harmless situations in anxiety disorders, but the neural

108 The otherwise harmless skin inhabitant Staphylococcus epidermidis is a

109 icultural pest that is able to switch from a harmless solitarious stage, during recession periods, to

110 nature, and while most isolates appear to be harmless, some are associated with food-borne illnesses,

111 Batesian mimicry, in which harmless species (mimics) deter predators by deceitfully

112 not, these species are mislabelled as other harmless species, causing severe disruptions to consumer

113 Harmless stimuli are treated as potentially harmful, res

114 ritically, the overgeneralization of fear to harmless stimuli or situations is a burden to daily life

115 te of immune ignorance or tolerance to these harmless stimuli to retain pulmonary homeostasis and to

116 animals to reduce their reaction to repeated harmless stimuli.

117 eneralization of threat-related behaviors to harmless stimuli.

118 one of these devices alone, some common and harmless substances show the same response as the hazard

119 95% of the symbiotic pairs are economically harmless, there are also three types of pest damage: tre

120 in the developing brain but is comparatively harmless to adults.

121 incompetent L1s, previously considered to be harmless to genomic integrity, may have the potential to

122 sterile males or genes rendering mosquitoes harmless to humans will depend on competitive mating beh

123 n a way that is toxic to its competitors but harmless to itself.

124 onsible for the production is encoded by the HARMLESS TO OZONE LAYER (HOL) gene.

125 Most exanthems are virtually harmless to the healthy child, but others can be signs o

126 and the nonangiogenic dormant tumor remains harmless to the host.

127 il bacterium Bacillus thuringiensis (Bt) are harmless to vertebrates, but they are highly toxic to in

128 icles can be achieved remotely by applying a harmless trigger such as visible-light irradiation.

129 Here, we show that an otherwise harmless virus rapidly causes massive chromosomal instab

130 ng approach to treating cancer that combines harmless visible and near-infrared light with a nontoxic

131 tion of non-toxic photosensitizing dyes with harmless visible light that together produce abundant de

132 excitation of nontoxic photosensitizers with harmless visible light-producing cytotoxic reactive oxyg

133 V region, preventing their excitation in the harmless visible/red region, as well as the strong quenc

134 ng paradigm allows for reliable, stable, and harmless visualization of cellular trafficking, persiste

135 e O(2) rapidly, converting the dioxygen into harmless water before it can damage the protein.

136 ur-electron reduction of molecular oxygen to harmless water ensures that the active site survives to

137 completely reduce O2 with four electrons to harmless water.

138 y toxic when free in solution but completely harmless when bound to GSTs.

139 ilder RTT phenotypes, yet both mutations are harmless when expressed with endogenous Mecp2.

140 ature; several species have been shown to be harmless with more stringent scientific evidence involvi