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1 was significantly longer in the BRCA mutant (hazard ratio 0.27, 95% CI 0.16-0.44, p<0.0001) and LOH h
2 elevated LMR was associated with better OS (hazard ratio 0.569, 95% confidence interval: 0.478-0.677
3 n colorectal cancer incidence rate (adjusted hazard ratio 0.57, 95% CI 0.40-0.80 for one visit; 0.51,
4 5 months (1.5-1.7) in the dacarbazine group (hazard ratio 0.62 [95% CI 0.47-0.80]; one-sided p<0.001)
7 sociated with graded reduction in mortality (hazard ratio 0.80, 95% CI 0.74-0.87 and 0.65, 0.60-0.71;
9 istory (in patients with blood group A or O: hazard ratio 0.94, 95% CI 0.73-1.20, p=0.60; in all 0.91
12 her graft failure risks than males (adjusted hazard ratios 0-14 years: 1.51 [95% confidence intervals
13 s longer across symptom frequency subgroups (hazard ratios 0.54 [95% CI 0.34-0.86] for 0-1 symptom da
15 mokers had significantly lower risks of BCC (hazard ratio = 0.6; 95% confidence interval = 0.4-0.9) b
16 ders indicated no increase in risk (adjusted hazard ratio = 0.83, 95% confidence interval: 0.63, 1.08
17 at menarche, risk of MS was reduced by 13% (hazard ratio = 0.87, 95% confidence interval: 0.79, 0.96
18 red with those aged 18 to 24 years (adjusted hazard ratio, 0-5 years = 0.36; 6-11 = 0.29; 12-17 = 0.4
19 as compared with 535 in the ADT-alone group (hazard ratio, 0.29; 95% CI, 0.25 to 0.34; P<0.001); the
20 nd 21 events per 100 patients, respectively (hazard ratio, 0.35; 95% confidence interval [CI], 0.22 t
21 her associated with a lower intubation rate (hazard ratio, 0.42; 95% CI, 0.11-1.61; p = 0.2) nor day
22 earlier-generation fluoroquinolone (adjusted hazard ratio, 0.46 [95% confidence interval, .26-.80]) d
26 idence than lifestyle modification patients (hazard ratio, 0.54; 95% confidence interval, 0.36-0.82).
28 atively similar in the unmatched population (hazard ratio, 0.57; 95% confidence interval, 0.33-0.98;
30 associated with no bystander resuscitation (hazard ratio, 0.62; 95% confidence interval [CI], 0.47 t
31 ed with a lower likelihood of ICU discharge (hazard ratio, 0.65 [0.42-1.00]; p = 0.01), longer deliri
34 ell as a lower risk of death from any cause (hazard ratio, 0.70; 95% CI, 0.50 to 0.99) and a lower ri
35 in the hazard of death after LVAD (adjusted hazard ratio, 0.73; 95% confidence interval, 0.58-0.92;
36 as associated with a significant OS benefit (hazard ratio, 0.77 [95% CI, 0.68 to 0.88]; P < .001).
43 h PCI, CABG still showed a survival benefit (hazard ratio, 0.82; 95% confidence interval, 0.75-0.90;
44 262 of 9151 (2.9%) randomized to PCI alone (hazard ratio, 0.84; 95% confidence interval, 0.70-1.01;
45 4 patients [9.8%] vs. 1563 patients [11.3%]; hazard ratio, 0.85; 95% confidence interval [CI], 0.79 t
46 per 100 person-years) in the placebo group (hazard ratio, 0.91; 95% confidence interval [CI], 0.83 t
47 oup and in 356 (9.3%) in the glargine group (hazard ratio, 0.91; 95% confidence interval, 0.78 to 1.0
48 rtality rates compared with whites (adjusted hazard ratio, 0.92; 95% CI, 0.76-1.11 and adjusted hazar
49 ratio, 0.92; 95% CI, 0.76-1.11 and adjusted hazard ratio, 0.92; 95% CI, 0.76-1.12, respectively).
50 interval, 0.94-1.15; P=0.46) or amlodipine (hazard ratio, 0.93; 95% confidence interval, 0.84-1.03;
51 mortality after propensity score adjustment (hazard ratio, 0.94; 95% CI, 0.89-0.99), and the benefici
53 nths for nivolumab versus 14 months for ICC (hazard ratio, 0.95; 95.54% CI, 0.73 to 1.24); median pro
54 .58) or the propensity score-matched cohort (hazard ratio, 0.97; 95% confidence interval, 0.64-1.47).
55 59; P=0.0006), but not of bleeding (adjusted hazard ratio, 0.99; 95% confidence interval, 0.68-1.46;
56 confidence interval, 1.06-1.24; P=0.001, and hazard ratio, 0.99; 95% confidence interval, 0.92-1.07;
57 onger activated partial thromboplastin time (hazards ratio, 0.98; p = 0.002) and decreased platelet c
58 red for 77 DCS and 136 BMS patients (12.0%) (hazard ratio: 0.54; 95% confidence interval: 0.41 to 0.7
59 s with DCM, no such difference was observed (hazard ratio: 0.92; 95% CI: 0.73 to 1.16; p = 0.49).
61 confidence interval, 0.61-0.78) and sisters hazard ratios=0.65 (95% confidence interval, 0.52-0.80),
62 bling): >/=10 cm difference between brothers hazard ratios=0.69 (95% confidence interval, 0.61-0.78)
63 n observed BMI and AF (age- and sex-adjusted hazard ratio 1.05 [1.04-1.06] per kg/m(2), P<0.001).
64 cardiovascular disease events (standardised hazard ratio 1.59, 95% CI 1.27-1.98; p<0.0001), a findin
65 n incidence of offspring lymphoid neoplasms (hazard ratio = 1.03, 95% confidence interval: 1.02, 1.04
66 ach additional experience of discrimination, hazard ratio = 1.09, 95% confidence interval: 1.01, 1.17
67 an in the antiinflammatory-stable DII group (hazard ratio = 1.32, 95% confidence interval: 1.01, 1.74
68 r categorically (for >/=2 experiences vs. 0, hazard ratio = 1.34, 95% confidence interval: 1.08, 1.66
69 erval: 1.4, 1.6) and cardiovascular disease (hazard ratio = 1.4, 95% confidence interval: 1.2, 1.6),
70 5 million/mL and all-cause hospitalizations (hazard ratio = 1.5, 95% confidence interval: 1.4, 1.6) a
71 atus was confirmed on multivariate analysis (hazard ratio = 1.694, 95% confidence interval 1.175-2.44
73 al MBF and clinical covariates; and adjusted hazard ratio, 1.03; 95% CI, 0.84-1.27; P=0.8 per unit de
74 alidone, randomization to either lisinopril (hazard ratio, 1.04; 95% confidence interval, 0.94-1.15;
75 ts was similar to standard ICD RPM patients (hazard ratio, 1.06; 95% confidence interval, 0.94-1.19;
76 1-U increase, P<0.001) and incident AF (FTO, hazard ratio, 1.07 [1.02-1.11] per A-allele, P=0.004; BM
78 idence interval: 1.11, 1.33) and MELD score (hazard ratio, 1.08; 95% confidence interval: 1.06, 1.11)
80 1.11] per A-allele, P=0.004; BMI gene score, hazard ratio, 1.11 [1.05-1.18] per 1-U increase, P<0.001
81 I], -1.5 to 2.8; P=0.007 for noninferiority; hazard ratio, 1.12; 95% CI, 0.79 to 1.58; P=0.53); the u
82 for health status and psychosocial factors (hazard ratio, 1.14; 95% confidence interval, 0.96-1.35),
83 edictor of outcomes in men but not in women (hazard ratio, 1.14; 95% confidence interval, 1.06-1.24;
84 ed with post-LT survival among all patients (hazard ratio, 1.15; 95% confidence interval, 0.84-1.58)
85 he chronic phase, the effect was attenuated (hazard ratio, 1.16; 95% confidence interval, 1.08-1.25;
87 r adjustment for competing risks, LSN score (hazard ratio, 1.22; 95% confidence interval: 1.11, 1.33)
90 wever, the risk of hospitalization (adjusted hazard ratio, 1.34; 95% confidence interval, 1.11-1.60;
92 eting risks including MELD score, LSN score (hazard ratio, 1.38; 95% confidence interval: 1.06, 1.79)
93 e (hazard ratio, 1.03), a medical diagnosis (hazard ratio, 1.43), inotrope use (hazard ratio, 3.47),
94 ed with children without asthma at baseline (hazard ratio, 1.51; 95% confidence interval, 1.08-2.10)
95 fter adjustment for all confounding factors (hazard ratio, 1.69; 95% confidence interval, 1.10-2.61;
97 , and revascularization after scan (adjusted hazard ratio, 1.79; 95% confidence interval [CI], 1.38-2
98 en target vessel revascularization (adjusted hazard ratio, 1.82; 95% confidence interval, 1.37-2.42;
99 predicted risk for subsequent MACE (adjusted hazard ratio, 1.90; 95% confidence interval, 1.46-2.48;
100 and to have reduced rejection-free survival (hazard ratio, 1.953; 95% confidence interval, 1.234-3.09
101 was the strongest univariable PFS predictor (hazard ratio, 1.97; 95% CI, 1.44 to 2.71; P < .001).
103 98; p = 0.002) and decreased platelet count (hazards ratio, 1.19; p = 0.03) were associated with a re
105 ity (p < 0.0005) and 1-year death and/or HF (hazard ratio: 1.27; 95% CI: 1.10 to 1.45; p = 0.001).
106 d a higher risk of bleeding hospitalization (hazard ratio=1.21; P=0.021) but a similar risk of death,
107 /=5 m/s had greater mortality risk (adjusted hazard ratio=1.86 [1.55-2.54]; P<0.001), even in the sub
108 , and the occurrence of early complications (hazard ratio, 11.34; 95% confidence interval: 1.82, 70.6
110 rd ratio, 4.6; 95% CI, 1.2-17.2; P = .02 and hazard ratio, 13.7; 95% CI, 2.0-91.9; P = .007, respecti
111 ns at a rate >200 beats per minute (adjusted hazard ratio, 15.63; 95% confidence interval, 4.01-60.89
112 atio, 3.47), and treatment limitation order (hazard ratio, 17.8) were all independently associated wi
115 -0.9) but significantly higher risks of SCC (hazard ratio = 2.3; 95% confidence interval = 1.5-3.6).
116 vels had 2.72 times the risk of miscarriage (hazard ratio = 2.72, 95% CI: 1.42-5.19) than those with
117 nts with gallstone disease vs 0.96% without; hazard ratio, 2.04; 95% confidence interval, 1.10-3.78).
118 highly associated with recurrent stroke/TIA (hazard ratio, 2.0; 95% confidence interval, 1.9-2.1).
119 nce interval, 1.11-1.60; P=0.002) and death (hazard ratio, 2.10; 95% confidence interval, 1.60-2.75;
120 sed mortality at 4 months both in allo-HSCT (hazard ratio, 2.13; 95% confidence interval, 1.45-3.13;
121 P<0.001), and cardiovascular death (adjusted hazard ratio, 2.16; 95% confidence interval, 1.36-3.43;
122 lin therapy had greater hazard of infection (hazard ratio, 2.18; 95% CI, 1.90-2.51) but no increased
123 2; P<0.0001), and stent thrombosis (adjusted hazard ratio, 2.26; 95% confidence interval, 1.42-3.59;
124 ry were because of noncardiovascular causes (hazard ratio, 2.32; 95% confidence interval, 1.92-2.81 v
125 sk of major adverse cardiac events (adjusted hazard ratio, 2.34; 95% confidence interval, 1.69-3.23;
126 creased rate of ILD-specific death (adjusted hazard ratio, 2.3; 95% confidence interval, 1.7-3.0; P <
127 dent prognostic factor for overall survival (hazard ratio, 2.480; 95% confidence interval [CI], 1.386
128 apy with TP53/KRAS comutations versus WT/WT (hazard ratio, 2.49 [95% CI, 1.10 to 5.64]; P = .03).
129 46-2.48; P<0.001), all-cause death (adjusted hazard ratio, 2.96; 95% confidence interval, 2.01-4.36;
131 ciated with a higher risk of major bleeding (hazard ratio: 2.19; 95% confidence interval: 1.07 to 4.4
133 [DDKT] without delayed graft function [DGF] hazard ratio: 24.634.447.9, P < 0.001; with DGF: 10.815.
134 er first-generation antipsychotics (adjusted hazard ratio, 3.06; 95% CI, 1.32-7.05) or second-generat
137 r second-generation antipsychotics (adjusted hazard ratio, 3.44; 95% CI, 1.73-6.83) increased the ris
138 iagnosis (hazard ratio, 1.43), inotrope use (hazard ratio, 3.47), and treatment limitation order (haz
139 -year cumulative event rates, 3.5% vs. 0.9%; hazard ratio, 3.87; 95% CI, 1.78 to 8.42; P<0.001).
140 18; P = .017), more than two initial stents (hazard ratio, 3.90; 95% confidence interval:1.16, 13.10;
141 C <25 mg/dl (2.6%) versus >/=25 mg/dl (0.8%; hazard ratio: 3.40; 95% confidence interval: 1.58 to 7.3
142 re, widowhood increased risk for AUD in men (hazard ratio=3.85, 95% CI=2.81-5.28) and women (hazard r
143 ling with AS had an exceptionally high risk (hazard ratio, 32.84) but were uncommon (34 siblings from
144 roke if warfarin treatment was discontinued (hazard ratio, 4.6; 95% CI, 1.2-17.2; P = .02 and hazard
145 with ultralow-risk patients by Cox analysis (hazard ratios, 4.73 [95% CI, 1.38-16.22] and 4.54 [95% C
149 ater was independently associated with CAMR (hazard ratio, 5.2; P = 0.03) even when DSA was undetecte
150 4.01-60.89; P<0.0001) and >7 beats (adjusted hazard ratio, 6.26; 95% confidence interval, 2.02-19.41;
152 es within 3 months of medication initiation (hazard ratio=6.7, 95% CI=2.0-22.4), with similar results
153 sociated with a myeloproliferative neoplasm (hazard ratio, 8.18; 95% confidence interval: 1.45, 46.18
154 associated with ICD-treated VT/VF (adjusted hazard ratio, 9.22; 95% confidence interval, 2.53-33.60;
155 o those with no increase or increases <0.25 (hazard ratio, 9.766; 95% confidence interval [CI], 4.356
156 harge was associated with improved survival (hazard ratio [95% CI], 0.96 [0.94-0.98]), with similar f
157 c survival, with an effect-allele-attributed hazards ratio (adjusted hazards ratio) of 1.99 (95% conf
158 os were not associated with 30-day mortality hazard ratios after controlling for baseline characteris
159 re treatment was associated with an adjusted hazard ratio (aHR) for treatment failure of 20.4 (95% CI
160 with a 28% higher risk of dementia (adjusted hazard ratio [aHR], 1.28; 95% CI, 1.13-1.46) adjusted fo
161 in persons with moderate fibrosis (adjusted hazard ratio [aHR], 1.42 [95% confidence interval {CI},
162 r patients with metastatic disease (adjusted hazard ratio [AHR], 2.3; 95% CI, 1.0 to 5.1; P = .04) an
163 s had an increased hazard of death (adjusted hazard ratio [aHR], 3.13; 95% confidence interval [CI],
164 en CIT and all-cause graft failure (adjusted hazard ratio [aHR]: 1.01, 95% CI: 0.98-1.04, P = .4), de
166 rd models to calculate multivariate-adjusted hazard ratios and 95% confidence intervals (CIs) for FI
167 incident cardiac events were estimated with hazard ratios and 95% confidence intervals computed in C
168 on to examining melanoma incidence, melanoma hazard ratios and 95% confidence intervals for lithium e
174 ars of age in ARIC and REGARDS, age-adjusted hazard ratios comparing blacks versus whites were 2.61 (
175 between readmissions and hospital survival (hazard ratios: first readmission 0.88, second readmissio
178 s 25 months (21-31) in the once-daily group (hazard ratio for death in the once daily group 1.18 [95%
179 nusinersen group than in the control group (hazard ratio for death or the use of permanent assisted
180 -therapy group and 77% in the placebo group (hazard ratio for death, 0.57; 95% CI, 0.42 to 0.79; P=0.
181 RVD alone (50 months vs. 36 months; adjusted hazard ratio for disease progression or death, 0.65; P<0
182 independently with a higher risk of AF; the hazard ratio for each 1 SD lower SD of normal-to-normal
183 e, the three higher quartiles combined had a hazard ratio for ESRD of 1.24 (95% confidence interval [
184 ed weight loss after 1 year was related to a hazard ratio for heart failure of 0.77 (95% confidence i
185 ger varied by acuity of presentation, with a hazard ratio for MACCE in ACS patients of 0.67 (95% CI:
186 se who received CRT without a defibrillator (hazard ratio for mortality adjusted on propensity score
190 ients of 0.67 (95% CI: 0.55 to 0.81) and the hazard ratio for SIHD patients of 0.55 (95% CI: 0.40 to
191 ose in the lowest, the multivariate-adjusted hazard ratio for T2D was 1.23 (95% confidence interval (
192 nalysis to estimate a cohort-specific causal hazard ratio for the association between BMI and AF.
193 ced AKI Network stage 2 or 3 had an adjusted hazard ratio for the primary composite outcome of 3.52 (
197 uations), we compared multivariable-adjusted hazard ratios for the composite outcome of incident ASCV
198 Health, Masters et al. reported age-specific hazard ratios for the contrasts in mortality rates betwe
199 sistent remission associated with unadjusted hazard ratios for the primary outcome that ranged by lan
200 r disease-specific survival [(+)transfusion: hazard ratio (HR) 1.19, 95% confidence interval (CI) 1.0
201 treatment to significantly improve survival [hazard ratio (HR) = 0.77, 95% confidence interval (CI):
202 did not have a higher risk of breast cancer (hazard ratio (HR) = 0.95, 95% confidence interval (95% C
203 breast cancer among those with AF (adjusted hazard ratio (HR) = 1.19, 95% confidence interval (CI):
205 CHOP) of patients having had PFS events, the hazard ratio (HR) for PFS was 0.73 (90% CI, 0.43 to 1.24
206 ional biopsy was associated with an adjusted hazard ratio (HR) of 3.0 (95% CI, 2-4.5) and, via core n
208 netics subgroups: in high-risk patients, the hazard ratio (HR) was 0.543 (95% confidence interval [CI
211 t [first quintile, referent; second quintile hazard ratio (HR)=1.03 [95% confidence interval (CI): 0.
215 ardiovascular events (multivariable adjusted hazard ratio [HR(adj)]=0.75, 95% CI 0.66-0.85, p<0.0001)
216 hs [95% CI 2.8-4.2] vs 1.8 months [1.5-2.8]; hazard ratio [HR] 0.67, 95% CI 0.53-0.84, one-sided p=0.
217 ths vs 30 months in the Rd group; stratified hazard ratio [HR] 0.712, 96% CI 0.56-0.906; one-sided p
218 [95% CI 11.8-15.7] vs 9.6 months [8.6-11.2]; hazard ratio [HR] 0.73 [95% CI 0.62-0.87], p=0.0003).
219 5% CI 2.96-4.47] vs 2.76 months [2.60-2.96]; hazard ratio [HR] 0.757, 95% CI 0.607-0.943; p=0.0118).
220 end point consistently in patients with PAD (hazard ratio [HR] 0.79; 95% confidence interval [CI], 0.
223 ] of 283) than for BUP-NX (163 [57%] of 287; hazard ratio [HR] 1.36, 95% CI 1.10-1.68), most or all o
224 from an African countries other than Egypt (hazard ratio [HR] 1.59 [95% CI 1.13-2.20]; p=0.01), hepa
225 th increased hazards of virological failure (hazard ratio [HR] 2.6, 95% CI 2.5-2.8; p<0.0001) and swi
226 nitive status (subjective cognitive decline: hazard ratio [HR] = 0.57, p < 0.05; mild cognitive impai
227 (belatacept more-intensive vs cyclosporine: hazard ratio [HR] = 0.95; 95% confidence interval [CI] 0
228 ARDs) had the highest risk of HBVr (adjusted hazard ratio [HR] = 5.14; 95% confidence interval [CI] =
229 eased incidence of hepatocellular carcinoma (hazard ratio [HR] compared with patients without an SVR,
230 and DBP, jointly suggested that, at Y0, SBP (hazard ratio [HR] per 1-SD increase, 1.32; 95% CI, 1.09-
232 eline predictors of SCAF were increased age (hazard ratio [HR] per decade, 1.55; 95% CI, 1.11-2.15),
233 of 90 people with a Mars1 endotype had died (hazard ratio [HR] vs all other endotypes 1.86 [95% CI 1.
234 f the two-sided 95% CI for the local relapse hazard ratio [HR] was less than 2.03), analysed by inten
235 ion analysis, treatment with either regimen (hazard ratio [HR], 0.43; 95% confidence interval [CI], .
236 oved survival were treatment with sorafenib (hazard ratio [HR], 0.66; 95% confidence interval [CI], 0
237 ons between low IOP and time to reoperation (hazard ratio [HR], 0.73; 95% CI, 0.32-1.68), vision loss
238 icantly improved with PAG treatment overall (hazard ratio [HR], 0.73; 95% CI, 0.53 to 1.00; P = .049)
241 other potential confounders (1 cup per day: hazard ratio [HR], 0.88 [95% CI, 0.85 to 0.91]; 2 to 3 c
242 placebo + docetaxel (difference, 1.1 months; hazard ratio [HR], 0.93 [95% CI, 0.77-1.12]; P = .44).
243 ted with the appearance of any autoantibody (hazard ratio [HR], 0.98; 95% CI, 0.95-1.01), multiple is
244 therapy group vs 27.6% in the placebo group (hazard ratio [HR], 0.99 [95% CI, 0.94-1.03]) in the over
245 ficant difference in the risk of DFS events (hazard ratio [HR], 1.00; 95% CI, 0.78 to 1.28; P = 1.00)
246 ssociated with higher CSF neutrophil counts (hazard ratio [HR], 1.10 per 10% increase; 95% confidence
247 lically unhealthy non-obese (MUNO) subjects (hazard ratio [HR], 1.29; 95% confidence interval [CI], 1
248 rtality was higher for nonadherent patients (hazard ratio [HR], 1.57; 95% CI, 1.07-2.30; P < .001), a
249 sarcopenia independently predicted overall (hazard ratio [HR], 1.64; 95% CI, 1.40-1.91 and HR, 1.28;
250 95% CI, 1.06-1.25]; autism spectrum disorder hazard ratio [HR], 2.02 [95% CI, 1.80-2.26]; attention-d
251 spital Pneumonia Severity Index (PSI) class (hazard ratio [HR], 2.45, P = .027; HR, 4.23, P < .001; H
252 nalysis (MVA), pre-existing cardiac disease (hazard ratio [HR], 2.96; 95% CI, 1.07 to 8.21; P = .04)
253 inferior OS on multivariable analysis: age (hazard ratio [HR], 3.299; P < .001), white blood cell co
254 analysis showed that macrovascular invasion (hazard ratio [HR], 4.8; P < 0.001), pre-LT waiting time
255 of the composite of all-cause death and MI (hazard ratio [HR]: 0.65; 95% confidence interval [CI]: 0
256 ith reduced risks of >/=30% decline in eGFR (hazard ratio [HR]: 0.77; 95% confidence interval [CI]: 0
257 nsistent at each subsequent landmark (year 1 hazard ratio [HR]: 0.82; 95% confidence interval [CI]: 0
258 ly reduced the risk of MACE by 14% in women (hazard ratio [HR]: 0.86; 95% confidence interval [CI]: 0
259 re was associated with reductions in stroke (hazard ratio [HR]: 0.91; 95% confidence interval [CI]: 0
260 ween PCSK9 levels and the risk of mortality (hazard ratio [HR]: 1.24; 95% confidence interval [CI]: 1
263 onal hazards regression was used to estimate hazard ratios (HRs) and 95% CIs for overall survival (OS
266 Cox regression models were used to calculate hazard ratios (HRs) and 95% confidence intervals (CIs) f
268 Cox regression models were used to calculate hazard ratios (HRs) and C statistics to determine predic
269 e used Cox models to estimate cause-specific hazard ratios (HRs) for 12 CVDs, adjusted for cardiovasc
270 h model was applied to estimate the adjusted hazard ratios (HRs) for 3 health transitions (healthy to
275 s was inversely associated with incident HF: hazard ratios (HRs) of 0.99 per year (95% confidence int
277 e and time-varying confounders, we estimated hazard ratios (HRs) of death and of AIDS-defining illnes
280 rd analysis was used to extract the adjusted hazard ratios (HRs) with adjustments for baseline age, s
281 s (RRs) for number of deaths/recurrences and hazard ratios (HRs), with 95% confidence intervals (CI),
283 events) independently of each other, with a hazard ratio of 1.8 and 95% confidence interval of 1.1 t
284 95% confidence interval of 1.1 to 2.0, and a hazard ratio of 2.1 and 95% confidence interval of 1.3 t
285 estimate (as a continuous variable) showed a hazard ratio of 2.25 (95% confidence interval, 1.70-2.99
286 relatives of patients with AVNRT presented a hazard ratio of at least 3.6 for exhibiting AVNRT compar
287 ate Cox proportional hazards regression, the hazard ratio of HD in the first 30 days was 2.17 (95% CI
289 ngle food frequency questionnaire, displayed hazard ratios of 1.31 (95% CI: 1.14, 1.51) and 1.07 (95%
291 ct-allele-attributed hazards ratio (adjusted hazards ratio) of 1.99 (95% confidence interval = 1.41-2
292 and incident cardiovascular disease events (hazard ratio per SD increment, 1.38; 95% confidence inte
294 higher mortality throughout 1-year (adjusted hazard ratio range, 1.30-1.92; p < 0.001), which was att
295 o, 0.29; 95% CI, 0.25 to 0.34; P<0.001); the hazard ratio was 0.21 in patients with nonmetastatic dis
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