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1 referably point to CHCT/MRI in patients with head and neck squamous cell carcinoma.
2 Mediterranean Diet Score (aMED)] and risk of head and neck squamous cell carcinoma.
3 r the start of radiotherapy in patients with head and neck squamous cell carcinoma.
4 not in mucoepidermoid salivary carcinomas or head and neck squamous cell carcinoma.
5 vival (HR, 3.98; P = 0.001) in patients with head and neck squamous cell carcinoma.
6 genesis in multiple human cancers, including head and neck squamous cell carcinoma.
7 and growth in non-small cell lung cancer and head and neck squamous cell carcinoma.
8 ost commonly used agents in the treatment of head and neck squamous cell carcinoma.
9 been undertaken to improve classification of head and neck squamous cell carcinoma.
10 -L1 immunohistochemistry in 96 patients with head and neck squamous cell carcinoma.
11  us new understanding of the pathogenesis of head and neck squamous cell carcinoma.
12 ng concurrent chemoradiotherapy for advanced head and neck squamous cell carcinoma.
13 g metabolic and malignant phenotype in human head and neck squamous cell carcinoma.
14 rrelates with poor prognosis in melanoma and head and neck squamous cell carcinoma.
15  is down-regulated in glioblastoma cells and head and neck squamous cell carcinoma.
16 ut with lost or down-regulated expression in head and neck squamous cell carcinoma.
17 in patient survival of five cancers, such as head and neck squamous cell carcinoma.
18 d with esophageal squamous cell carcinoma or head and neck squamous cell carcinoma.
19 urine models of melanoma, neuroblastoma, and head and neck squamous cell carcinoma.
20 oor outcome in an independent data set of 71 head and neck squamous cell carcinomas.
21 s, other anogenital cancers, and a subset of head and neck squamous cell carcinomas.
22 agents of a subset of anogenital cancers and head and neck squamous cell carcinomas.
23 quenced the whole mitochondrial genome in 83 head and neck squamous cell carcinomas.
24 ositol 3-kinase (PI3Kalpha), are frequent in head and neck squamous cell carcinomas.
25 ting dose-limiting toxicity in patients with head and neck squamous cell carcinomas.
26 cted after antiangiogenic therapy, except in head and neck squamous cell carcinoma 19 d after the sta
27          The most frequent solid tumors were head and neck squamous cell carcinomas (40% of patients
28  approved for the treatment of patients with head and neck squamous cell carcinoma and metastatic col
29 powerful immunotherapeutic tool to eradicate head and neck squamous cell carcinoma and other poorly i
30 ALR1) maps to a common region of 18q loss in head and neck squamous cell carcinomas and is frequently
31 y methylated and silenced in the majority of head and neck squamous cell carcinomas and non-small-cel
32 uding non-small cell lung carcinoma (NSCLC), head and neck squamous cell carcinoma, and ovarian cance
33 o different cancer cell lines, FaDu, a human head and neck squamous cell carcinoma, and RKO, a human
34  and management of HPV-related oropharyngeal head and neck squamous cell carcinoma, and to describe u
35 cer, multiple myeloma, neuroendocrine tumor, head and neck squamous cell carcinoma, and unknown prima
36 at the gene is mutated in 6% of GBMs, 13% of head and neck squamous cell carcinomas, and in 9% of lun
37  26 with non-small-cell lung cancer, 55 with head and neck squamous-cell carcinoma, and 55 with gastr
38 ng cancer, four (9%, 2-21) of 45 people with head and neck squamous-cell carcinoma, and four (9%, 2-2
39  greater enrichment of previously identified head and neck squamous cell carcinoma associated gene se
40 derwent upfront nodal dissection for mucosal head and neck squamous cell carcinoma between 2004 and 2
41 rlotinib are currently used in patients with head and neck squamous cell carcinoma, but, in this dise
42 c bioconjugate was examined in the HN5 human head and neck squamous cell carcinoma cell line.
43 pithelial cell lines, was present in several head and neck squamous cell carcinoma cell lines and was
44 ing radiation and delays the growth of human head and neck squamous cell carcinoma cell lines in 3D c
45                          PpFur expression in head and neck squamous cell carcinoma cell lines showed
46 nts described here compared the responses of head and neck squamous cell carcinoma cell lines to two
47                   Transfection of four human head and neck squamous cell carcinoma cell lines with th
48 mote cancer stem cell-like properties in two head and neck squamous cell carcinoma cell lines, UMSCC-
49  correlates with loss of CXCL14 secretion by head and neck squamous cell carcinoma cell lines, wherea
50 and induced apoptosis in FaDu and Cal27, two head and neck squamous cell carcinoma cell lines.
51                                              Head and neck squamous cell carcinoma cells exposed to c
52     The selective action of MT-DADMe-ImmA on head and neck squamous cell carcinoma cells suggests pot
53 s were tested against naturally HPV-infected head and neck squamous cell carcinoma cells using IFN-ga
54 he molecular effects of SCH66336, an FTI, in head and neck squamous cell carcinoma cells using proteo
55  and undergoes caspase-dependent cleavage in head and neck squamous cell carcinoma cells.
56  epidemic of human papillomavirus-associated head and neck squamous cell carcinomas characterized by
57      In this study, we determined that human head and neck squamous cell carcinoma-derived HSC-3 cell
58 r, other anogenital cancers, and a subset of head and neck squamous cell carcinomas, encode three onc
59 n of NANOG in cell lines from oesophageal or head and neck squamous cell carcinomas (ESCCs or HNSCCs,
60                               Critically, in head and neck squamous cell carcinoma, Fxr1 overexpressi
61 idence of human papillomavirus (HPV)-related head and neck squamous cell carcinoma has increased in r
62 cancers, including chronic myeloid leukemia, head and neck squamous cell carcinoma, hepatocellular ca
63 are putative tumor-initiating cells (TIC) in head and neck squamous cell carcinomas (HNC).
64 nsferase NSD1 define an intrinsic subtype of head and neck squamous cell carcinoma (HNSC) that featur
65                                              Head and neck squamous cell carcinoma (HNSCC) accounts f
66 ng chain ceramides were measured in 32 human head and neck squamous cell carcinoma (HNSCC) and 10 non
67 diotherapy and cetuximab in locally advanced head and neck squamous cell carcinoma (HNSCC) and biomar
68 ear survival rate is 40-50% in patients with head and neck squamous cell carcinoma (HNSCC) and curren
69  EphB4 receptor is ubiquitously expressed in head and neck squamous cell carcinoma (HNSCC) and has be
70                                              Head and neck squamous cell carcinoma (HNSCC) and many o
71     Given the potential relationship between head and neck squamous cell carcinoma (HNSCC) and microb
72  mechanism strongly inhibited cell growth in head and neck squamous cell carcinoma (HNSCC) and non-sm
73  channel that is frequently overexpressed in head and neck squamous cell carcinoma (HNSCC) and other
74  DNA amplification is frequently observed in head and neck squamous cell carcinoma (HNSCC) and potent
75                              The majority of head and neck squamous cell carcinoma (HNSCC) and some c
76  We investigated how frequent this occurs in head and neck squamous cell carcinoma (HNSCC) and whethe
77                                Patients with head and neck squamous cell carcinoma (HNSCC) are at ele
78 rates of local recurrence in tobacco-related head and neck squamous cell carcinoma (HNSCC) are common
79                               The prototypic head and neck squamous cell carcinoma (HNSCC) arises fro
80 ur purpose was to examine whether staging of head and neck squamous cell carcinoma (HNSCC) by upfront
81                                              Head and neck squamous cell carcinoma (HNSCC) can be div
82                Human papillomavirus-negative head and neck squamous cell carcinoma (HNSCC) carries a
83 iously shown that some gefitinib-insensitive head and neck squamous cell carcinoma (HNSCC) cell lines
84 89) inhibits cell cycle progression of human head and neck squamous cell carcinoma (HNSCC) cell lines
85  We screened a panel of 49 established human head and neck squamous cell carcinoma (HNSCC) cell lines
86  NOK strains (n = 8), dysplasias (n = 5) and head and neck squamous cell carcinoma (HNSCC) cell lines
87                          Our work shows that head and neck squamous cell carcinoma (HNSCC) cell lines
88 DeltaNp63alpha in the nucleus of a subset of head and neck squamous cell carcinoma (HNSCC) cell lines
89 elial growth factor (VEGF) autocrine loop in head and neck squamous cell carcinoma (HNSCC) cell lines
90 ed in Non-Small Cell Lung Cancer (NSCLC) and Head and Neck Squamous Cell Carcinoma (HNSCC) cell lines
91 levels of the oncogenic DeltaNp73 isoform in head and neck squamous cell carcinoma (HNSCC) cell lines
92         We modulated cortactin expression in head and neck squamous cell carcinoma (HNSCC) cell lines
93              We found that OPN is induced in head and neck squamous cell carcinoma (HNSCC) cell lines
94 he effects of the retinoid MX3350-1 on human head and neck squamous cell carcinoma (HNSCC) cell lines
95 gainst EGFR-expressing cell lines, including head and neck squamous cell carcinoma (HNSCC) cell lines
96       We examined NKG2D ligand expression in head and neck squamous cell carcinoma (HNSCC) cells and
97                   Rac was activated in human head and neck squamous cell carcinoma (HNSCC) cells as e
98 rx metabolism enhanced cell killing of human head and neck squamous cell carcinoma (HNSCC) cells by a
99 we report studies of the in vivo invasion of head and neck squamous cell carcinoma (HNSCC) cells in r
100 e and TIC-like phenotypes in patient-derived head and neck squamous cell carcinoma (HNSCC) cells in v
101  Sema4D, the product of which is elevated in head and neck squamous cell carcinoma (HNSCC) cells, con
102 as been developed for metabolic profiling of head and neck squamous cell carcinoma (HNSCC) cells.
103 nvasion, migration and the CIC population in head and neck squamous cell carcinoma (HNSCC) cells.
104                 Like many epithelial tumors, head and neck squamous cell carcinoma (HNSCC) contains a
105 ologic studies suggest that the incidence of head and neck squamous cell carcinoma (HNSCC) correlates
106 s epigenetically upregulated in experimental head and neck squamous cell carcinoma (HNSCC) dormancy m
107                                              Head and neck squamous cell carcinoma (HNSCC) has a proc
108                                              Head and neck squamous cell carcinoma (HNSCC) has a vari
109  therapies, approximate 50% survival rate of head and neck squamous cell carcinoma (HNSCC) has had ma
110 reated with EPO, the role of EPO and EPOR in head and neck squamous cell carcinoma (HNSCC) has not be
111 ar survival rate in patients with late-stage head and neck squamous cell carcinoma (HNSCC) has not im
112 dence of human papillomavirus (HPV)-positive head and neck squamous cell carcinoma (HNSCC) has rapidl
113 nts with human papillomavirus (HPV)-positive head and neck squamous cell carcinoma (HNSCC) have bette
114 tribute to the initiation and progression of head and neck squamous cell carcinoma (HNSCC) have not b
115                    Although the incidence of head and neck squamous cell carcinoma (HNSCC) in the Uni
116                                              Head and neck squamous cell carcinoma (HNSCC) includes e
117                  Both PGE2 and BK stimulated head and neck squamous cell carcinoma (HNSCC) invasion v
118                                              Head and neck squamous cell carcinoma (HNSCC) is a commo
119                                        Human head and neck squamous cell carcinoma (HNSCC) is a highl
120                                              Head and neck squamous cell carcinoma (HNSCC) is a highl
121                                              Head and neck squamous cell carcinoma (HNSCC) is a leadi
122                                              Head and neck squamous cell carcinoma (HNSCC) is a leadi
123                                              Head and neck squamous cell carcinoma (HNSCC) is a very
124                                              Head and neck squamous cell carcinoma (HNSCC) is an aggr
125                                              Head and neck squamous cell carcinoma (HNSCC) is an aggr
126                                              Head and neck squamous cell carcinoma (HNSCC) is charact
127                                              Head and neck squamous cell carcinoma (HNSCC) is general
128                                              Head and neck squamous cell carcinoma (HNSCC) is one of
129                 The unique characteristic of head and neck squamous cell carcinoma (HNSCC) is that lo
130                                              Head and neck squamous cell carcinoma (HNSCC) is the eig
131                                              Head and neck squamous cell carcinoma (HNSCC) is the mos
132                                              Head and neck squamous cell carcinoma (HNSCC) is the six
133                                              Head and neck squamous cell carcinoma (HNSCC) is the six
134                                              Head and neck squamous cell carcinoma (HNSCC) is the six
135                                              Head and neck squamous cell carcinoma (HNSCC) is the six
136                                              Head and neck squamous cell carcinoma (HNSCC) is the six
137 re compared for activity in protecting human head and neck squamous cell carcinoma (HNSCC) lines from
138 s frequently downregulated not only in human head and neck squamous cell carcinoma (HNSCC) malignant
139 ies have implicated a role for SDF-1alpha in head and neck squamous cell carcinoma (HNSCC) metastasis
140   To investigate how STAT3 activity in human head and neck squamous cell carcinoma (HNSCC) might alte
141  of therapy for patients with advanced stage head and neck squamous cell carcinoma (HNSCC) or recurre
142 h factor receptor (EGFR)-targeted therapy in head and neck squamous cell carcinoma (HNSCC) patients f
143                                              Head and neck squamous cell carcinoma (HNSCC) patients h
144                                         Most head and neck squamous cell carcinoma (HNSCC) patients p
145  of approximately 6,000 single cells from 18 head and neck squamous cell carcinoma (HNSCC) patients,
146 ance to chemotherapy and reduced survival of head and neck squamous cell carcinoma (HNSCC) patients.
147 get of rapamycin (mTOR) signaling network in head and neck squamous cell carcinoma (HNSCC) progressio
148                                              Head and neck squamous cell carcinoma (HNSCC) progressio
149                                              Head and neck squamous cell carcinoma (HNSCC) remains a
150 critical mediator of this pathway, Smad4, in head and neck squamous cell carcinoma (HNSCC) remains un
151                                              Head and neck squamous cell carcinoma (HNSCC) represents
152 isons of global miRNA expression profiles of head and neck squamous cell carcinoma (HNSCC) samples an
153 nt study, microarray expression profiling on head and neck squamous cell carcinoma (HNSCC) samples id
154                                 By comparing head and neck squamous cell carcinoma (HNSCC) samples wi
155 s from human papillomavirus-related (HPV(+)) head and neck squamous cell carcinoma (HNSCC) samples.
156  from several studies, including data from a Head and Neck Squamous Cell Carcinoma (HNSCC) study and
157  In this study, miRNA expression profiles of head and neck squamous cell carcinoma (HNSCC) tumor and
158 l cell line and expression data from primary head and neck squamous cell carcinoma (HNSCC) tumor tiss
159 we analyzed integrated data obtained from 19 head and neck squamous cell carcinoma (HNSCC) tumors and
160 es the invasive cell growth of p53-deficient head and neck squamous cell carcinoma (HNSCC) UM-SCC-1 c
161 e SELP analogs in a xenograft mouse model of head and neck squamous cell carcinoma (HNSCC) was shown
162  specific immune and metabolic phenotypes in head and neck squamous cell carcinoma (HNSCC) were asses
163 ceptor (EGFR) is frequently overexpressed in head and neck squamous cell carcinoma (HNSCC) where aber
164 Major weight loss is common in patients with head and neck squamous cell carcinoma (HNSCC) who underg
165 on factor RUNX2 is widely upregulated in the head and neck squamous cell carcinoma (HNSCC) with lymph
166 trategies are needed to improve treatment of head and neck squamous cell carcinoma (HNSCC), an aggres
167 papillomavirus (HPV) is an accepted cause of head and neck squamous cell carcinoma (HNSCC), and patie
168 is occurs in more than half of patients with head and neck squamous cell carcinoma (HNSCC), and these
169      For patients with locoregional advanced head and neck squamous cell carcinoma (HNSCC), concurren
170                        Here, we show that in head and neck squamous cell carcinoma (HNSCC), CTGF prom
171  Atlas in October 2013 for 305 patients with head and neck squamous cell carcinoma (HNSCC), from 14 i
172 or gene is involved in the carcinogenesis of head and neck squamous cell carcinoma (HNSCC), the entir
173 e been the standard treatment modalities for head and neck squamous cell carcinoma (HNSCC), the integ
174 er exploited this synergistic combination in head and neck squamous cell carcinoma (HNSCC), up to 90%
175 ls of non-small cell lung cancer (NSCLC) and head and neck squamous cell carcinoma (HNSCC), where AXL
176 r (EGFR) is a clinically validated target in head and neck squamous cell carcinoma (HNSCC), where EGF
177 is a common event in many cancers, including head and neck squamous cell carcinoma (HNSCC), where STA
178 n several epithelial malignancies, including head and neck squamous cell carcinoma (HNSCC), which exh
179  TP53 is the most frequently altered gene in head and neck squamous cell carcinoma (HNSCC), with muta
180 present study, we examined the role of human head and neck squamous cell carcinoma (HNSCC)-secreted S
181 tment efficacy, of anticancer drugs in human head and neck squamous cell carcinoma (HNSCC).
182 l role in epithelial malignancies, including head and neck squamous cell carcinoma (HNSCC).
183 e role in various types of cancer, including head and neck squamous cell carcinoma (HNSCC).
184 ial to the development of novel treatment of head and neck squamous cell carcinoma (HNSCC).
185 e, we reveal a defective molecular switch in head and neck squamous cell carcinoma (HNSCC).
186  the pathogenesis of many cancers, including head and neck squamous cell carcinoma (HNSCC).
187 the three most commonly amplified regions in head and neck squamous cell carcinoma (HNSCC).
188 potential role of hedgehog (Hh) signaling in head and neck squamous cell carcinoma (HNSCC).
189 ng is common in many malignancies, including head and neck squamous cell carcinoma (HNSCC).
190 and downstream targets, and TP53 genotype in head and neck squamous cell carcinoma (HNSCC).
191 enografts derived from 3 cell lines of human head and neck squamous cell carcinoma (HNSCC).
192 and usually fatal for patients with advanced head and neck squamous cell carcinoma (HNSCC).
193 inase as a novel target for the treatment of head and neck squamous cell carcinoma (HNSCC).
194 tion could effectively inhibit the growth of head and neck squamous cell carcinoma (HNSCC).
195 investigated TLR4 signaling effects on human head and neck squamous cell carcinoma (HNSCC).
196 ttle change in the survival of patients with head and neck squamous cell carcinoma (HNSCC).
197 l growth factor receptor (EGFR) is common in head and neck squamous cell carcinoma (HNSCC).
198 rk was to identify new molecular markers for head and neck squamous cell carcinoma (HNSCC).
199  CXCL5, in the proliferation and invasion of head and neck squamous cell carcinoma (HNSCC).
200 f the p16INK4A gene, is commonly observed in head and neck squamous cell carcinoma (HNSCC).
201 d together with the p53 family member p63 in head and neck squamous cell carcinoma (HNSCC).
202 rtant role in various cancers, including the head and neck squamous cell carcinoma (HNSCC).
203 n papillomavirus (HPV) detection and risk of head and neck squamous cell carcinoma (HNSCC).
204 adical chemoradiotherapy in locally advanced head and neck squamous cell carcinoma (HNSCC).
205           Invasion is a hallmark of advanced head and neck squamous cell carcinoma (HNSCC).
206 tive chemotherapy of solid tumors, including head and neck squamous cell carcinoma (HNSCC).
207 tegies have been successfully implemented in head and neck squamous cell carcinoma (HNSCC).
208 velopment, establishment, and progression of head and neck squamous cell carcinoma (HNSCC).
209 inase inhibitors in unselected patients with head and neck squamous cell carcinoma (HNSCC).
210 ients with recurrent and/or metastatic (R/M) head and neck squamous cell carcinoma (HNSCC).
211 variety of aggressive tumor types, including head and neck squamous cell carcinoma (HNSCC); however,
212 ctor receptor (EGFR) is found in over 80% of head and neck squamous cell carcinomas (HNSCC) and assoc
213 e ability of celecoxib to induce toxicity in head and neck squamous cell carcinomas (HNSCC) and explo
214 ssociated endothelial cells of patients with head and neck squamous cell carcinomas (HNSCC) as compar
215  cells are located in the invasive fronts of head and neck squamous cell carcinomas (HNSCC) close to
216                                              Head and neck squamous cell carcinomas (HNSCC) exhibitin
217  The Cancer Genome Atlas (TCGA) reveals that head and neck squamous cell carcinomas (HNSCC) harbor th
218 nt and the appearance of secondary tumors in head and neck squamous cell carcinomas (HNSCC) have been
219 production of prostaglandin E(2) (PGE(2)) by head and neck squamous cell carcinomas (HNSCC) induce ty
220                                              Head and neck squamous cell carcinomas (HNSCC) is a comm
221                                              Head and neck squamous cell carcinomas (HNSCC) often met
222 ding of cancer, the 5-year survival rate for head and neck squamous cell carcinomas (HNSCC) patients
223               Combined treatment of advanced head and neck squamous cell carcinomas (HNSCC) with radi
224 sifiers of CIMP) in 344 bladder cancers, 346 head and neck squamous cell carcinomas (HNSCC), 146 non-
225 rtactin is associated with poor prognosis in head and neck squamous cell carcinomas (HNSCC), possibly
226 ns in serum or tumor tissue of patients with head and neck squamous cell carcinomas (HNSCC), suggesti
227                                              Head and neck squamous cell carcinomas (HNSCC), the majo
228 inhibitors of IKKbeta or EGFR is observed in head and neck squamous cell carcinomas (HNSCC).
229 have been reported to occur in 10% to 15% of head and neck squamous cell carcinomas (HNSCC).
230 erved in human papillomavirus (HPV)-positive head and neck squamous cell carcinomas (HNSCC).
231 matrix assembled by stromal fibroblasts from head and neck squamous cell carcinomas (HNSCC).
232 lay an important role in the pathobiology of head and neck squamous cell carcinomas (HNSCC).
233 ces cell death and inhibits tumorigenesis in head and neck squamous cell carcinomas (HNSCC).
234       EGFR is upregulated in the majority of head and neck squamous cell carcinomas (HNSCC).
235                             The prognosis of head-and-neck squamous cell carcinoma (HNSCC) has not be
236 beta1) was frequently overexpressed in human head and neck squamous cell carcinomas (HNSCCs) and adja
237 Bcl-2 family member, occurs in a majority of head and neck squamous cell carcinomas (HNSCCs) and corr
238          Human papillomavirus (HPV)-negative head and neck squamous cell carcinomas (HNSCCs) are dead
239                                              Head and neck squamous cell carcinomas (HNSCCs) are high
240                                              Head and neck squamous cell carcinomas (HNSCCs) are refr
241  highly expressed in cell lines derived from head and neck squamous cell carcinomas (HNSCCs) at both
242               Only a subset of patients with head and neck squamous cell carcinomas (HNSCCs) benefit
243 16, are associated with approximately 20% of head and neck squamous cell carcinomas (HNSCCs) in the g
244              However, more recent studies of head and neck squamous cell carcinomas (HNSCCs) suggest
245 ay a role in the pathogenesis of a subset of head and neck squamous cell carcinomas (HNSCCs), particu
246           To identify miRNAs associated with head and neck squamous cell carcinomas (HNSCCs), we anal
247 erse-phase protein array data (n = 212) from head and neck squamous cell carcinomas (HNSCCs).
248 ation of Brca1 begins at the early stages of head and neck squamous cell carcinomas (HNSCCs).
249 ve been independently detected in subsets of head and neck squamous cell carcinomas (HNSCCs).
250 ve factor in over 90% of cervical and 25% of head and neck squamous cell carcinomas (HNSCCs).
251 nd prosurvival roles, respectively, in human head and neck squamous cell carcinomas (HNSCCs).
252 , more than 90% of head and neck cancers are head and neck squamous cell carcinomas (HNSCCs).
253 /TAp73 transcription factors we uncovered in head and neck squamous cell carcinomas (HNSCCs).
254              Human papillomavirus-associated head and neck squamous cell carcinomas (HPV-HNSCC) origi
255                      Analysis of 47 lung and head and neck squamous cell carcinomas identified a case
256 esophageal squamous cell carcinomas, and 270 head and neck squamous cell carcinomas (identified from
257  established as a risk factor for developing head and neck squamous cell carcinoma, independent of to
258                                        Human head and neck squamous cell carcinoma is a solid tumor m
259 nt of lymph node metastasis in patients with head and neck squamous cell carcinoma is not accurate en
260  scan of the neck in locoregionally advanced head-and-neck squamous cell carcinoma (LAHNSCC) after co
261                                           In head and neck squamous cell carcinoma, local failure aft
262 ending toxin; CSC, cancer stem cells; HNSCC, head and neck squamous cell carcinoma; MAb, monoclonal a
263 in 1 trial of patients with locally advanced head and neck squamous cell carcinoma (median survival,
264 ated in multiple aggressive cancers, such as head and neck squamous cell carcinoma, melanoma and panc
265 nal cell carcinoma, glioblastoma multiforme, head and neck squamous cell carcinoma, melanoma, thyroid
266  of cancer stem cells in the pathobiology of head and neck squamous cell carcinomas might have a prof
267 ancer models driven by MMTV-Wnt1 and a human head and neck squamous cell carcinoma model (HN30).
268 ith OPSCC and patients with nonoropharyngeal head and neck squamous cell carcinoma (non-OP HNSCC).
269 ith OPSCC and patients with nonoropharyngeal head and neck squamous cell carcinoma (non-OP HNSCC).
270 ell lung cancer, non-small-cell lung cancer, head and neck squamous-cell carcinoma, or gastro-oesopha
271 present in tumors or the peripheral blood of head and neck squamous cell carcinoma patients co-expres
272 e in primary human tumor tissue samples from head and neck squamous cell carcinoma patients.
273 table safety profile in recurrent/metastatic head and neck squamous cell carcinoma previously treated
274 CaMKII-mediated cytoskeleton function and in head and neck squamous cell carcinoma progression.
275 approved treatments for recurrent/metastatic head and neck squamous cell carcinoma refractory to plat
276 y (ICT) in the treatment of locally advanced head and neck squamous cell carcinoma remains controvers
277 gFuge to RNA-seq cohorts of 177 lung and 279 head and neck squamous cell carcinoma samples from the C
278  gene sets relative to training data sets of head and neck squamous cell carcinoma samples, one inclu
279             In an orthotopic murine model of head and neck squamous cell carcinoma (SCC VII/SF) we st
280 ts of Rap1GAP on invasion and progression of head and neck squamous cell carcinoma (SCC) and the role
281                  Carcinogens associated with head and neck squamous cell carcinoma (SCC) genesis shou
282 orming growth factor (TGF)-beta signaling in head and neck squamous cell carcinoma (SCC) is not yet f
283 he associations between dietary patterns and head and neck squamous cell carcinoma (SCC) or whether t
284   Tumor RF ablation therapy was performed on head and neck squamous cell carcinoma (SCC) xenograft tu
285         Here, we show that PKCalpha promotes head and neck squamous cell carcinoma (SCCHN) by a feed-
286    We found TMEM16A overexpression in 80% of head and neck squamous cell carcinoma (SCCHN), which cor
287 mal differentiation and cause anogenital and head and neck squamous cell carcinomas (SCCs).
288                A retrospective evaluation of head and neck squamous cell carcinomas showed heterogene
289  is the first report of elevated PKC iota in head and neck squamous cell carcinoma that may have sign
290 t that GSPs may be a promising candidate for head and neck squamous cell carcinoma therapy.
291 nd DUSP1 mRNA and protein decreased in human head and neck squamous cell carcinoma tissues compared w
292 ignificant promoter demethylation in primary head and neck squamous cell carcinoma tissues.
293 gnant biopsy specimens from 23 patients with head and neck squamous cell carcinoma to characterize th
294 rived from autologous primary and metastatic head and neck squamous cell carcinoma, to investigate th
295 and diagnostic CT images in 20 patients with head and neck squamous cell carcinoma treated with curat
296      In the first experiment, a single human head-and-neck squamous cell carcinoma tumor was grown in
297 ipts were detected using RNA-Seq analysis in head-and-neck squamous cell carcinoma, uterine endometri
298              In this study, 16 patients with head and neck squamous cell carcinoma were enrolled (med
299                                 Treatment of head and neck squamous cell carcinomas with radiotherapy
300             Eighty mice with patient-derived head and neck squamous cell carcinomas xenografts, SCCNi

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