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1 referably point to CHCT/MRI in patients with head and neck squamous cell carcinoma.
2 Mediterranean Diet Score (aMED)] and risk of head and neck squamous cell carcinoma.
3 r the start of radiotherapy in patients with head and neck squamous cell carcinoma.
4 not in mucoepidermoid salivary carcinomas or head and neck squamous cell carcinoma.
5 vival (HR, 3.98; P = 0.001) in patients with head and neck squamous cell carcinoma.
6 genesis in multiple human cancers, including head and neck squamous cell carcinoma.
7 and growth in non-small cell lung cancer and head and neck squamous cell carcinoma.
8 ost commonly used agents in the treatment of head and neck squamous cell carcinoma.
9 been undertaken to improve classification of head and neck squamous cell carcinoma.
10 -L1 immunohistochemistry in 96 patients with head and neck squamous cell carcinoma.
11 us new understanding of the pathogenesis of head and neck squamous cell carcinoma.
12 ng concurrent chemoradiotherapy for advanced head and neck squamous cell carcinoma.
13 g metabolic and malignant phenotype in human head and neck squamous cell carcinoma.
14 rrelates with poor prognosis in melanoma and head and neck squamous cell carcinoma.
15 is down-regulated in glioblastoma cells and head and neck squamous cell carcinoma.
16 ut with lost or down-regulated expression in head and neck squamous cell carcinoma.
17 in patient survival of five cancers, such as head and neck squamous cell carcinoma.
18 d with esophageal squamous cell carcinoma or head and neck squamous cell carcinoma.
19 urine models of melanoma, neuroblastoma, and head and neck squamous cell carcinoma.
20 oor outcome in an independent data set of 71 head and neck squamous cell carcinomas.
21 s, other anogenital cancers, and a subset of head and neck squamous cell carcinomas.
22 agents of a subset of anogenital cancers and head and neck squamous cell carcinomas.
23 quenced the whole mitochondrial genome in 83 head and neck squamous cell carcinomas.
24 ositol 3-kinase (PI3Kalpha), are frequent in head and neck squamous cell carcinomas.
25 ting dose-limiting toxicity in patients with head and neck squamous cell carcinomas.
26 cted after antiangiogenic therapy, except in head and neck squamous cell carcinoma 19 d after the sta
28 approved for the treatment of patients with head and neck squamous cell carcinoma and metastatic col
29 powerful immunotherapeutic tool to eradicate head and neck squamous cell carcinoma and other poorly i
30 ALR1) maps to a common region of 18q loss in head and neck squamous cell carcinomas and is frequently
31 y methylated and silenced in the majority of head and neck squamous cell carcinomas and non-small-cel
32 uding non-small cell lung carcinoma (NSCLC), head and neck squamous cell carcinoma, and ovarian cance
33 o different cancer cell lines, FaDu, a human head and neck squamous cell carcinoma, and RKO, a human
34 and management of HPV-related oropharyngeal head and neck squamous cell carcinoma, and to describe u
35 cer, multiple myeloma, neuroendocrine tumor, head and neck squamous cell carcinoma, and unknown prima
36 at the gene is mutated in 6% of GBMs, 13% of head and neck squamous cell carcinomas, and in 9% of lun
37 26 with non-small-cell lung cancer, 55 with head and neck squamous-cell carcinoma, and 55 with gastr
38 ng cancer, four (9%, 2-21) of 45 people with head and neck squamous-cell carcinoma, and four (9%, 2-2
39 greater enrichment of previously identified head and neck squamous cell carcinoma associated gene se
40 derwent upfront nodal dissection for mucosal head and neck squamous cell carcinoma between 2004 and 2
41 rlotinib are currently used in patients with head and neck squamous cell carcinoma, but, in this dise
43 pithelial cell lines, was present in several head and neck squamous cell carcinoma cell lines and was
44 ing radiation and delays the growth of human head and neck squamous cell carcinoma cell lines in 3D c
46 nts described here compared the responses of head and neck squamous cell carcinoma cell lines to two
48 mote cancer stem cell-like properties in two head and neck squamous cell carcinoma cell lines, UMSCC-
49 correlates with loss of CXCL14 secretion by head and neck squamous cell carcinoma cell lines, wherea
52 The selective action of MT-DADMe-ImmA on head and neck squamous cell carcinoma cells suggests pot
53 s were tested against naturally HPV-infected head and neck squamous cell carcinoma cells using IFN-ga
54 he molecular effects of SCH66336, an FTI, in head and neck squamous cell carcinoma cells using proteo
56 epidemic of human papillomavirus-associated head and neck squamous cell carcinomas characterized by
58 r, other anogenital cancers, and a subset of head and neck squamous cell carcinomas, encode three onc
59 n of NANOG in cell lines from oesophageal or head and neck squamous cell carcinomas (ESCCs or HNSCCs,
61 idence of human papillomavirus (HPV)-related head and neck squamous cell carcinoma has increased in r
62 cancers, including chronic myeloid leukemia, head and neck squamous cell carcinoma, hepatocellular ca
64 nsferase NSD1 define an intrinsic subtype of head and neck squamous cell carcinoma (HNSC) that featur
66 ng chain ceramides were measured in 32 human head and neck squamous cell carcinoma (HNSCC) and 10 non
67 diotherapy and cetuximab in locally advanced head and neck squamous cell carcinoma (HNSCC) and biomar
68 ear survival rate is 40-50% in patients with head and neck squamous cell carcinoma (HNSCC) and curren
69 EphB4 receptor is ubiquitously expressed in head and neck squamous cell carcinoma (HNSCC) and has be
71 Given the potential relationship between head and neck squamous cell carcinoma (HNSCC) and microb
72 mechanism strongly inhibited cell growth in head and neck squamous cell carcinoma (HNSCC) and non-sm
73 channel that is frequently overexpressed in head and neck squamous cell carcinoma (HNSCC) and other
74 DNA amplification is frequently observed in head and neck squamous cell carcinoma (HNSCC) and potent
76 We investigated how frequent this occurs in head and neck squamous cell carcinoma (HNSCC) and whethe
78 rates of local recurrence in tobacco-related head and neck squamous cell carcinoma (HNSCC) are common
80 ur purpose was to examine whether staging of head and neck squamous cell carcinoma (HNSCC) by upfront
83 iously shown that some gefitinib-insensitive head and neck squamous cell carcinoma (HNSCC) cell lines
84 89) inhibits cell cycle progression of human head and neck squamous cell carcinoma (HNSCC) cell lines
85 We screened a panel of 49 established human head and neck squamous cell carcinoma (HNSCC) cell lines
86 NOK strains (n = 8), dysplasias (n = 5) and head and neck squamous cell carcinoma (HNSCC) cell lines
88 DeltaNp63alpha in the nucleus of a subset of head and neck squamous cell carcinoma (HNSCC) cell lines
89 elial growth factor (VEGF) autocrine loop in head and neck squamous cell carcinoma (HNSCC) cell lines
90 ed in Non-Small Cell Lung Cancer (NSCLC) and Head and Neck Squamous Cell Carcinoma (HNSCC) cell lines
91 levels of the oncogenic DeltaNp73 isoform in head and neck squamous cell carcinoma (HNSCC) cell lines
94 he effects of the retinoid MX3350-1 on human head and neck squamous cell carcinoma (HNSCC) cell lines
95 gainst EGFR-expressing cell lines, including head and neck squamous cell carcinoma (HNSCC) cell lines
98 rx metabolism enhanced cell killing of human head and neck squamous cell carcinoma (HNSCC) cells by a
99 we report studies of the in vivo invasion of head and neck squamous cell carcinoma (HNSCC) cells in r
100 e and TIC-like phenotypes in patient-derived head and neck squamous cell carcinoma (HNSCC) cells in v
101 Sema4D, the product of which is elevated in head and neck squamous cell carcinoma (HNSCC) cells, con
102 as been developed for metabolic profiling of head and neck squamous cell carcinoma (HNSCC) cells.
103 nvasion, migration and the CIC population in head and neck squamous cell carcinoma (HNSCC) cells.
105 ologic studies suggest that the incidence of head and neck squamous cell carcinoma (HNSCC) correlates
106 s epigenetically upregulated in experimental head and neck squamous cell carcinoma (HNSCC) dormancy m
109 therapies, approximate 50% survival rate of head and neck squamous cell carcinoma (HNSCC) has had ma
110 reated with EPO, the role of EPO and EPOR in head and neck squamous cell carcinoma (HNSCC) has not be
111 ar survival rate in patients with late-stage head and neck squamous cell carcinoma (HNSCC) has not im
112 dence of human papillomavirus (HPV)-positive head and neck squamous cell carcinoma (HNSCC) has rapidl
113 nts with human papillomavirus (HPV)-positive head and neck squamous cell carcinoma (HNSCC) have bette
114 tribute to the initiation and progression of head and neck squamous cell carcinoma (HNSCC) have not b
137 re compared for activity in protecting human head and neck squamous cell carcinoma (HNSCC) lines from
138 s frequently downregulated not only in human head and neck squamous cell carcinoma (HNSCC) malignant
139 ies have implicated a role for SDF-1alpha in head and neck squamous cell carcinoma (HNSCC) metastasis
140 To investigate how STAT3 activity in human head and neck squamous cell carcinoma (HNSCC) might alte
141 of therapy for patients with advanced stage head and neck squamous cell carcinoma (HNSCC) or recurre
142 h factor receptor (EGFR)-targeted therapy in head and neck squamous cell carcinoma (HNSCC) patients f
145 of approximately 6,000 single cells from 18 head and neck squamous cell carcinoma (HNSCC) patients,
146 ance to chemotherapy and reduced survival of head and neck squamous cell carcinoma (HNSCC) patients.
147 get of rapamycin (mTOR) signaling network in head and neck squamous cell carcinoma (HNSCC) progressio
150 critical mediator of this pathway, Smad4, in head and neck squamous cell carcinoma (HNSCC) remains un
152 isons of global miRNA expression profiles of head and neck squamous cell carcinoma (HNSCC) samples an
153 nt study, microarray expression profiling on head and neck squamous cell carcinoma (HNSCC) samples id
155 s from human papillomavirus-related (HPV(+)) head and neck squamous cell carcinoma (HNSCC) samples.
156 from several studies, including data from a Head and Neck Squamous Cell Carcinoma (HNSCC) study and
157 In this study, miRNA expression profiles of head and neck squamous cell carcinoma (HNSCC) tumor and
158 l cell line and expression data from primary head and neck squamous cell carcinoma (HNSCC) tumor tiss
159 we analyzed integrated data obtained from 19 head and neck squamous cell carcinoma (HNSCC) tumors and
160 es the invasive cell growth of p53-deficient head and neck squamous cell carcinoma (HNSCC) UM-SCC-1 c
161 e SELP analogs in a xenograft mouse model of head and neck squamous cell carcinoma (HNSCC) was shown
162 specific immune and metabolic phenotypes in head and neck squamous cell carcinoma (HNSCC) were asses
163 ceptor (EGFR) is frequently overexpressed in head and neck squamous cell carcinoma (HNSCC) where aber
164 Major weight loss is common in patients with head and neck squamous cell carcinoma (HNSCC) who underg
165 on factor RUNX2 is widely upregulated in the head and neck squamous cell carcinoma (HNSCC) with lymph
166 trategies are needed to improve treatment of head and neck squamous cell carcinoma (HNSCC), an aggres
167 papillomavirus (HPV) is an accepted cause of head and neck squamous cell carcinoma (HNSCC), and patie
168 is occurs in more than half of patients with head and neck squamous cell carcinoma (HNSCC), and these
169 For patients with locoregional advanced head and neck squamous cell carcinoma (HNSCC), concurren
171 Atlas in October 2013 for 305 patients with head and neck squamous cell carcinoma (HNSCC), from 14 i
172 or gene is involved in the carcinogenesis of head and neck squamous cell carcinoma (HNSCC), the entir
173 e been the standard treatment modalities for head and neck squamous cell carcinoma (HNSCC), the integ
174 er exploited this synergistic combination in head and neck squamous cell carcinoma (HNSCC), up to 90%
175 ls of non-small cell lung cancer (NSCLC) and head and neck squamous cell carcinoma (HNSCC), where AXL
176 r (EGFR) is a clinically validated target in head and neck squamous cell carcinoma (HNSCC), where EGF
177 is a common event in many cancers, including head and neck squamous cell carcinoma (HNSCC), where STA
178 n several epithelial malignancies, including head and neck squamous cell carcinoma (HNSCC), which exh
179 TP53 is the most frequently altered gene in head and neck squamous cell carcinoma (HNSCC), with muta
180 present study, we examined the role of human head and neck squamous cell carcinoma (HNSCC)-secreted S
211 variety of aggressive tumor types, including head and neck squamous cell carcinoma (HNSCC); however,
212 ctor receptor (EGFR) is found in over 80% of head and neck squamous cell carcinomas (HNSCC) and assoc
213 e ability of celecoxib to induce toxicity in head and neck squamous cell carcinomas (HNSCC) and explo
214 ssociated endothelial cells of patients with head and neck squamous cell carcinomas (HNSCC) as compar
215 cells are located in the invasive fronts of head and neck squamous cell carcinomas (HNSCC) close to
217 The Cancer Genome Atlas (TCGA) reveals that head and neck squamous cell carcinomas (HNSCC) harbor th
218 nt and the appearance of secondary tumors in head and neck squamous cell carcinomas (HNSCC) have been
219 production of prostaglandin E(2) (PGE(2)) by head and neck squamous cell carcinomas (HNSCC) induce ty
222 ding of cancer, the 5-year survival rate for head and neck squamous cell carcinomas (HNSCC) patients
224 sifiers of CIMP) in 344 bladder cancers, 346 head and neck squamous cell carcinomas (HNSCC), 146 non-
225 rtactin is associated with poor prognosis in head and neck squamous cell carcinomas (HNSCC), possibly
226 ns in serum or tumor tissue of patients with head and neck squamous cell carcinomas (HNSCC), suggesti
236 beta1) was frequently overexpressed in human head and neck squamous cell carcinomas (HNSCCs) and adja
237 Bcl-2 family member, occurs in a majority of head and neck squamous cell carcinomas (HNSCCs) and corr
241 highly expressed in cell lines derived from head and neck squamous cell carcinomas (HNSCCs) at both
243 16, are associated with approximately 20% of head and neck squamous cell carcinomas (HNSCCs) in the g
245 ay a role in the pathogenesis of a subset of head and neck squamous cell carcinomas (HNSCCs), particu
256 esophageal squamous cell carcinomas, and 270 head and neck squamous cell carcinomas (identified from
257 established as a risk factor for developing head and neck squamous cell carcinoma, independent of to
259 nt of lymph node metastasis in patients with head and neck squamous cell carcinoma is not accurate en
260 scan of the neck in locoregionally advanced head-and-neck squamous cell carcinoma (LAHNSCC) after co
262 ending toxin; CSC, cancer stem cells; HNSCC, head and neck squamous cell carcinoma; MAb, monoclonal a
263 in 1 trial of patients with locally advanced head and neck squamous cell carcinoma (median survival,
264 ated in multiple aggressive cancers, such as head and neck squamous cell carcinoma, melanoma and panc
265 nal cell carcinoma, glioblastoma multiforme, head and neck squamous cell carcinoma, melanoma, thyroid
266 of cancer stem cells in the pathobiology of head and neck squamous cell carcinomas might have a prof
267 ancer models driven by MMTV-Wnt1 and a human head and neck squamous cell carcinoma model (HN30).
268 ith OPSCC and patients with nonoropharyngeal head and neck squamous cell carcinoma (non-OP HNSCC).
269 ith OPSCC and patients with nonoropharyngeal head and neck squamous cell carcinoma (non-OP HNSCC).
270 ell lung cancer, non-small-cell lung cancer, head and neck squamous-cell carcinoma, or gastro-oesopha
271 present in tumors or the peripheral blood of head and neck squamous cell carcinoma patients co-expres
273 table safety profile in recurrent/metastatic head and neck squamous cell carcinoma previously treated
275 approved treatments for recurrent/metastatic head and neck squamous cell carcinoma refractory to plat
276 y (ICT) in the treatment of locally advanced head and neck squamous cell carcinoma remains controvers
277 gFuge to RNA-seq cohorts of 177 lung and 279 head and neck squamous cell carcinoma samples from the C
278 gene sets relative to training data sets of head and neck squamous cell carcinoma samples, one inclu
280 ts of Rap1GAP on invasion and progression of head and neck squamous cell carcinoma (SCC) and the role
282 orming growth factor (TGF)-beta signaling in head and neck squamous cell carcinoma (SCC) is not yet f
283 he associations between dietary patterns and head and neck squamous cell carcinoma (SCC) or whether t
284 Tumor RF ablation therapy was performed on head and neck squamous cell carcinoma (SCC) xenograft tu
286 We found TMEM16A overexpression in 80% of head and neck squamous cell carcinoma (SCCHN), which cor
289 is the first report of elevated PKC iota in head and neck squamous cell carcinoma that may have sign
291 nd DUSP1 mRNA and protein decreased in human head and neck squamous cell carcinoma tissues compared w
293 gnant biopsy specimens from 23 patients with head and neck squamous cell carcinoma to characterize th
294 rived from autologous primary and metastatic head and neck squamous cell carcinoma, to investigate th
295 and diagnostic CT images in 20 patients with head and neck squamous cell carcinoma treated with curat
296 In the first experiment, a single human head-and-neck squamous cell carcinoma tumor was grown in
297 ipts were detected using RNA-Seq analysis in head-and-neck squamous cell carcinoma, uterine endometri
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