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1 While alpha5-null/B6 embryos develop normal headfolds, alpha5-null/129 embryos have headfold defects
5 ver, FLRT3 mutant embryos display defects in headfold fusion, definitive endoderm migration and a fai
7 ptor mRNA is detected in the mesoderm of the headfold stage and persists in myocardial precursors of
8 udal levels of the head process derived from headfold stage embryos can induce discrete regions of th
9 hed independently of each other at the early headfold stage, and then their expression rapidly become
16 and cultured in isolation, neural plate and headfold-stage allantoises formed a conspicuous vascular
17 ntaining blood islands into blood islands of headfold-stage host conceptuses provided no evidence tha
19 e posterior primitive streak at neural plate/headfold stages (NP/HF, ~7.5 dpc-8 dpc) represents an op
20 hat discrete regions of axial mesendoderm at headfold stages are not normally responsible for the est
22 are coexpressed in the embryo, except in the headfold state (when only the HGF receptor can be detect
23 d in all three germ layers of the developing headfold, suggesting roles in both the establishment and
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