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1  induced by right ventricular pacing (25% of heart beats).
2 red with vitamin C (+10.0 +/- 6.9 bursts/100 heart beats).
3 sponsible for originating and regulating the heart beat.
4  are evident even before the first embryonic heart beat.
5 the complex electrical system that makes the heart beat.
6 approximately one third of the period of the heart beat.
7 ly and rejection was defined by cessation of heart beat.
8 red electrocardiogram (ECG) gating over many heart beats.
9 l pressure waveform despite multiple ectopic heart beats.
10 elax as an integrated functional unit as the heart beats.
11         We move our eyes more often than our heart beats.
12 single volume acquisition was achieved in 24 heart beats.
13  and was embryonic lethal within days of the heart beating.
14                                 Why does the heart beat?
15  0.54 baseline vs. 5.64 +/- 0.67 bursts (100 heart beats)(1) mmHg(1) at 120 min; P < 0.05).
16  0.29 baseline vs. 4.74 +/- 0.71 bursts (100 heart beats)(1) mmHg(1) at 120 min; P < 0.05).
17  striking consistency in the total number of heart beats accrued over a lifetime across a range of an
18 ted MSNA indices (%(Delta)bursts, bursts/100 heart beats and signal averaged MSNA), attenuated H(+) a
19                  Data collected over >100 ms/heart beat are also susceptible to bulk cardiac and resp
20 but the molecular underpinnings of the first heart beat are not known, nor whether function determine
21 sing the speed of XB recruitment so that the heart beats at a rate commensurate with f(min).
22 o the arterial pulse at the frequency of the heart beat became maximal.
23                               The decline in heart-beating brainstem dead organ donors has necessitat
24 ience validating a novel, noninvasive, whole heart, beat-by-beat, 3-dimensional mapping technology wi
25  diastolic LV/RV function in the brain-dead, heart-beating cadaver, which may contribute to early pos
26 rent from recipients of livers procured from heart-beating cadaveric donors (P=0.74, log-rank test).
27 , cells were isolated from the livers of non-heart-beating cadaveric mice long after death and transp
28 ), von Willebrand factor (12%), next-morning heart beat cycle length (6%), next-morning heart rate va
29 characterized by a progressive heart-beat-to-heart-beat decrease in the lag of SND relative to the AP
30 gnals, as measured by, for example, tests of heart beat detection, perform better in laboratory studi
31 oved from 30 kg Yorkshire pigs in a model of heart-beating donation and either preserved in cold hist
32 ified to carry out the tasks involved in non-heart-beating donation, and may even potentially comprom
33  as older or sicker donors and so-called non-heart-beating donation, now referred to as donation afte
34  influence transplant outcome after deceased heart beating donor kidney transplant in the United King
35                      Outcomes after deceased heart beating donor kidney transplantation are good, but
36 r novel Bristol and Region Allocation by Non-heart beating Donor Score (BRANDS).
37 articularly suited to retrieval from the non-heart beating donor.
38 on of NHBD recipients compared to a group of heart-beating donor (HBD) recipients from a single insti
39  solution (MPS) improves the function of non-heart-beating donor (NHBD) canine kidneys.
40 be a successful experience of controlled non-heart-beating donor (NHBD) liver transplantation.
41                                          Non-heart-beating donor (NHBD) livers are an untapped source
42 to resuscitate and maintain viability of non-heart-beating donor (NHBD) livers that have undergone si
43 iorates warm ischemic lung injury in the non-heart-beating donor (NHBD), thereby improving function w
44                             Uncontrolled non-heart-beating donor (UNHBD) transplantation offers a maj
45                   After removing one kidney (heart-beating donor [HBD]), the dog was exsanguinated.
46 riovenous extracorporeal removal of CO2 in a heart-beating donor awaiting organ harvest.
47 ygenation will be highly significant for non-heart-beating donor cells.
48 d NEVKP is feasible and safe in good quality heart-beating donor kidney grafts.
49 othermic ex vivo kidney perfusion (NEVKP) in heart-beating donor kidney transplantation.
50  impact of this technology in preserving non-heart-beating donor livers.
51 n of kidneys not subjected to warm ischemia (heart-beating donor model), but there was no evidence th
52 ility, which will advance the utility of non-heart-beating donor organs for cell therapy or other app
53                               We studied non-heart-beating donor rats for global gene expression with
54 r transplant unit, and compared with matched heart-beating donor transplants as a case control analys
55                                    A control heart-beating-donor (HBD) group was taken as the next co
56                                          Non-heart beating donors (NHBD) can provide an alternative s
57                 Donation of kidneys from non-heart beating donors (NHBD) is increasingly being used t
58  long-waiting patients with kidneys from non-heart beating donors has reduced waiting times without c
59                       One kidney from 40 non-heart beating donors was allocated to the highest BRANDS
60 ctive oxygenation during HMP of kidneys from heart beating donors.
61 llografts procured in standard fashion, from heart-beating donors
62 f delayed graft function (DGF) compared with heart-beating donors (42.4% vs. 23.3%, respectively).
63 ibility of using livers from controlled, non-heart-beating donors (CNHBD) with good immediate graft f
64 tors from the donors were suspected from non-heart-beating donors (n=1) and cardiac-arrest donors (n=
65                      Wide recruitment of non-heart-beating donors (NHBD) could significantly increase
66                  Hepatic allografts from non-heart-beating donors (NHBD) have been cited as a means t
67                                          Non-heart-beating donors (NHBD) offer a potential source of
68                                          Non-heart-beating donors (NHBD) offer a promising potential
69 ticularly the case for organs taken from non-heart-beating donors (NHBD), but there is still a lack o
70 but little used, is that of kidneys from non-heart-beating donors (NHBD).
71 oeficient (Kfc), in lungs retrieved from non-heart-beating donors (NHBDs) and reperfused with the add
72                                          Non-heart-beating donors (NHBDs) are generally not deemed su
73 r transplantation (LT) using grafts from non-heart-beating donors (NHBDs) has been shown to be a succ
74                                          Non-heart-beating donors (NHBDs) have the potential to reduc
75                 Use of liver grafts from non-heart-beating donors (NHBDs) warrants consideration so t
76 lated interest in the use of organs from non-heart-beating donors (NHBDs).
77 in the use of extended donors, including non-heart-beating donors (NHBDs).
78 y were inferior to those of hepatocytes from heart-beating donors (P < 0.05).
79 imals, this was inferior to hepatocytes from heart-beating donors (P < 0.05).
80 5 years), obese (weight >or=200 lb), and non-heart-beating donors and donors with an elevated amylase
81                   Liver grafts procured from heart-beating donors and preserved by SCS served as cont
82 ecember 1998, 150 kidneys were procured from heart-beating donors and preserved in our laboratory by
83                              Organs from non-heart-beating donors are attractive for use in cell ther
84         These data provide evidence that non-heart-beating donors could be a suitable source of hepat
85     Similarly, although hepatocytes from non-heart-beating donors engrafted and proliferated after tr
86         Consent for cornea donation from non-heart-beating donors is often requested by means of tele
87 to molecular changes in hepatocytes from non-heart-beating donors offer opportunities for improving d
88      Ethical controversies in the use of non-heart-beating donors remain despite thorough review.
89 n profiling in hepatocytes isolated from non-heart-beating donors showed far greater perturbations co
90  allocation scheme for kidneys from deceased heart-beating donors was required in the United Kingdom
91 , open-label, monocenter trial, 160 deceased heart-beating donors were allowed to perform 236 renal t
92 organ transplantations, split grafts, or non-heart-beating donors were not included.
93 hemia time, prolonged donor hypotension, non-heart-beating donors) who received thymoglobulin inducti
94 veral strategies, such as live donation, non-heart-beating donors, and expanded criteria donors.
95 ct over prolonged periods after death in non-heart-beating donors, but extensive molecular perturbati
96 ssfully isolated viable hepatocytes from non-heart-beating donors, especially up to 4 hours after dea
97                                   Use of non-heart-beating donors, international organ sharing, and p
98                                       In non-heart-beating donors, liver tissue was morphologically i
99 e hundred kidneys were locally procured from heart-beating donors, preserved in our laboratory, and t
100 nvolved in withdrawal of life support in non-heart-beating donors, unless specialty trained in end-of
101 donor organs including those from viable non-heart-beating donors.
102 urs after death, respectively, compared with heart-beating donors.
103 e similar to 242 synchronous recipients from heart-beating donors.
104 ntroversies remain concerning the use of non-heart-beating donors.
105  treated with water survived (i.e., palpable heart beat) for 16.0+/-0.5 days (n=6).
106                     The relationship between heart beat frequency and the rate of oxygen consumption
107                        At the 1st to the 3rd heart beat from the onset of stretch, MSNA (199 +/- 30%,
108 ntries, to measure whether the potential for heart-beating (HB) deceased donors was adequately conver
109  protein-labeled nerve terminals, to monitor heart beat in intact and semi-intact preparations.
110                The null hypotheses were that heart beat interval fluctuations at usual breathing freq
111                 The SERCA group shows longer heart beat intervals (Mean +/- SD: 1009.7 +/- 151.6 ms)
112                               A synchronized heart beat is controlled by pacemaking impulses conducte
113                                   A rhythmic heart beat is coordinated by conduction of pacemaking im
114                                 The rhythmic heart beat is coordinated by electrical impulses transmi
115                                          The heart beat is coordinated by the integrated activities o
116                                    A regular heart beat is dependent on a specialized network of pace
117 atisfactorily assesses and preserves the non-heart-beating lung.
118 the oxygen pulse (ie, the O2 utilization per heart beat) mirrored that of peak VO2, whereas the longi
119 ing gap between organ supply and demand, non-heart-beating (NHB) donors will become increasingly impo
120 om 42.0 +/- 10.1 to 42.7 +/- 11.8 bursts/100 heart beats; NS).
121 y in the transplantation of kidneys from non-heart-beating or marginal donors or organs experiencing
122                                          Non-heart-beating organ donation for transplantation is incr
123                                 Further, non-heart-beating organ donation has failed to produce the w
124                      In a small study of non-heart-beating organ donation, circulation never resumed
125 sthesiologists should not be involved in non-heart-beating organ donations; potential exceptions incl
126   All non-brain-dead patients who became non-heart-beating organ donors at the University of Pittsbur
127 g the arm and hand) was performed in 9 human heart-beating organ donors.
128 tation and deaths of patients who became non-heart-beating organ donors.
129 ic (CAD) organs and compare the results with heart-beating organ sources.
130 ts frequently become involved with such 'non-heart-beating' organ donations when they are asked to wi
131 om 53.3 +/- 12.2 to 47.4 +/- 11.5 bursts/100 heart beats; P < 0.01) but not in healthy subjects (from
132 tients receiving transplants from obese, non-heart-beating, pediatric, or hyperamylasemic donors comp
133 ovascular defects, including an abnormal low heart beat rate (bradycardia) and severely hypoplastic S
134                                              Heart beat rate analysis and metabolome analysis indicat
135 oth the Phe-Met-Arg-Phe-amide maturation and heart beat rate defects observed in Ctr1A mutant larvae
136              In sinoatrial node cells of the heart, beating rate is controlled, in part, by local Ca(
137                   Voluntary movement and the heart beat require this calcium flow to be massive and f
138 hase walk was characterized by a progressive heart-beat-to-heart-beat decrease in the lag of SND rela
139 ess of defibrillation, return of spontaneous heart beat, weanability from extracorporeal cardiopulmon

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