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1 induced by right ventricular pacing (25% of heart beats).
2 red with vitamin C (+10.0 +/- 6.9 bursts/100 heart beats).
3 sponsible for originating and regulating the heart beat.
4 are evident even before the first embryonic heart beat.
5 the complex electrical system that makes the heart beat.
6 approximately one third of the period of the heart beat.
7 ly and rejection was defined by cessation of heart beat.
8 red electrocardiogram (ECG) gating over many heart beats.
9 l pressure waveform despite multiple ectopic heart beats.
10 elax as an integrated functional unit as the heart beats.
11 We move our eyes more often than our heart beats.
12 single volume acquisition was achieved in 24 heart beats.
13 and was embryonic lethal within days of the heart beating.
14 Why does the heart beat?
17 striking consistency in the total number of heart beats accrued over a lifetime across a range of an
18 ted MSNA indices (%(Delta)bursts, bursts/100 heart beats and signal averaged MSNA), attenuated H(+) a
20 but the molecular underpinnings of the first heart beat are not known, nor whether function determine
24 ience validating a novel, noninvasive, whole heart, beat-by-beat, 3-dimensional mapping technology wi
25 diastolic LV/RV function in the brain-dead, heart-beating cadaver, which may contribute to early pos
26 rent from recipients of livers procured from heart-beating cadaveric donors (P=0.74, log-rank test).
27 , cells were isolated from the livers of non-heart-beating cadaveric mice long after death and transp
28 ), von Willebrand factor (12%), next-morning heart beat cycle length (6%), next-morning heart rate va
29 characterized by a progressive heart-beat-to-heart-beat decrease in the lag of SND relative to the AP
30 gnals, as measured by, for example, tests of heart beat detection, perform better in laboratory studi
31 oved from 30 kg Yorkshire pigs in a model of heart-beating donation and either preserved in cold hist
32 ified to carry out the tasks involved in non-heart-beating donation, and may even potentially comprom
33 as older or sicker donors and so-called non-heart-beating donation, now referred to as donation afte
34 influence transplant outcome after deceased heart beating donor kidney transplant in the United King
38 on of NHBD recipients compared to a group of heart-beating donor (HBD) recipients from a single insti
42 to resuscitate and maintain viability of non-heart-beating donor (NHBD) livers that have undergone si
43 iorates warm ischemic lung injury in the non-heart-beating donor (NHBD), thereby improving function w
51 n of kidneys not subjected to warm ischemia (heart-beating donor model), but there was no evidence th
52 ility, which will advance the utility of non-heart-beating donor organs for cell therapy or other app
54 r transplant unit, and compared with matched heart-beating donor transplants as a case control analys
58 long-waiting patients with kidneys from non-heart beating donors has reduced waiting times without c
62 f delayed graft function (DGF) compared with heart-beating donors (42.4% vs. 23.3%, respectively).
63 ibility of using livers from controlled, non-heart-beating donors (CNHBD) with good immediate graft f
64 tors from the donors were suspected from non-heart-beating donors (n=1) and cardiac-arrest donors (n=
69 ticularly the case for organs taken from non-heart-beating donors (NHBD), but there is still a lack o
71 oeficient (Kfc), in lungs retrieved from non-heart-beating donors (NHBDs) and reperfused with the add
73 r transplantation (LT) using grafts from non-heart-beating donors (NHBDs) has been shown to be a succ
80 5 years), obese (weight >or=200 lb), and non-heart-beating donors and donors with an elevated amylase
82 ecember 1998, 150 kidneys were procured from heart-beating donors and preserved in our laboratory by
85 Similarly, although hepatocytes from non-heart-beating donors engrafted and proliferated after tr
87 to molecular changes in hepatocytes from non-heart-beating donors offer opportunities for improving d
89 n profiling in hepatocytes isolated from non-heart-beating donors showed far greater perturbations co
90 allocation scheme for kidneys from deceased heart-beating donors was required in the United Kingdom
91 , open-label, monocenter trial, 160 deceased heart-beating donors were allowed to perform 236 renal t
93 hemia time, prolonged donor hypotension, non-heart-beating donors) who received thymoglobulin inducti
95 ct over prolonged periods after death in non-heart-beating donors, but extensive molecular perturbati
96 ssfully isolated viable hepatocytes from non-heart-beating donors, especially up to 4 hours after dea
99 e hundred kidneys were locally procured from heart-beating donors, preserved in our laboratory, and t
100 nvolved in withdrawal of life support in non-heart-beating donors, unless specialty trained in end-of
108 ntries, to measure whether the potential for heart-beating (HB) deceased donors was adequately conver
118 the oxygen pulse (ie, the O2 utilization per heart beat) mirrored that of peak VO2, whereas the longi
119 ing gap between organ supply and demand, non-heart-beating (NHB) donors will become increasingly impo
121 y in the transplantation of kidneys from non-heart-beating or marginal donors or organs experiencing
125 sthesiologists should not be involved in non-heart-beating organ donations; potential exceptions incl
126 All non-brain-dead patients who became non-heart-beating organ donors at the University of Pittsbur
130 ts frequently become involved with such 'non-heart-beating' organ donations when they are asked to wi
131 om 53.3 +/- 12.2 to 47.4 +/- 11.5 bursts/100 heart beats; P < 0.01) but not in healthy subjects (from
132 tients receiving transplants from obese, non-heart-beating, pediatric, or hyperamylasemic donors comp
133 ovascular defects, including an abnormal low heart beat rate (bradycardia) and severely hypoplastic S
135 oth the Phe-Met-Arg-Phe-amide maturation and heart beat rate defects observed in Ctr1A mutant larvae
138 hase walk was characterized by a progressive heart-beat-to-heart-beat decrease in the lag of SND rela
139 ess of defibrillation, return of spontaneous heart beat, weanability from extracorporeal cardiopulmon
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