ライフサイエンスコーパス: heart block

1 ardia and atrioventricular (AV) dysfunction (heart block).

2 insight into the pathogenesis of congenital heart block.

3 rial fibrillation without producing complete heart block.

4 led him to discover the mechanism of partial heart block.

5 mportant risk factor for having a child with heart block.

6 RS complex, 4 resulted in transient complete heart block.

7 ite their strong association with congenital heart block.

8 larly 52Ro, in the development of congenital heart block.

9 similar time course to that of postsurgical heart block.

10 rothers of another family had arrhythmia and heart block.

11 adverse events on all cases was queried for heart block.

12 icular conduction in a rat model of complete heart block.

13 PM4 have been linked to progressive familial heart block.

14 lar nodal cells, accounting for the observed heart block.

15 r contractions, tachycardia, and high-degree heart block.

16 sistent with bradyarrhythmia and progressive heart block.

17 lower success rate and a higher incidence of heart block.

18 a may be characteristic of an acute stage of heart block.

19 ns to ventricular arrhythmias and congenital heart block.

20 was frequently followed by atrioventricular heart block.

21 s; and 6) the introduction of the concept of heart block.

22 chronically instrumented dogs with complete heart block.

23 ne of the most common pathologies underlying heart blocks.

24 r arrhythmia (9 patients, or 14 percent), or heart block (3 patients, or 5 percent), although in some

25 portantly, DMPK+/- mice develop first-degree heart block, a conduction defect strikingly similar to t

26 lar conduction with transition into complete heart block after 160 Gy.

27 help identify patients at risk for complete heart block after septal reduction procedures for HCM.

28 onatal lupus (cardiac NL) include congenital heart block and cardiomyopathy.

29 bradyarrhythmia characterized by progressive heart block and impaired ventricular depolarization.

30 others with a previous child with congenital heart block and in 3 of 74 pregnancies (4%) in mothers w

31 the near universal association of congenital heart block and maternal Abs to SSA/Ro and SSB/La, the i

32 e birth to children with complete congenital heart block and photosensitive skin lesions.

33 egment depression, complete atrioventricular heart block and right precordial ST segment elevation.

34 nt mice develop progressive atrioventricular heart block and significant ultrastructural changes in b

35 nother patient with syncope had intermittent heart block and survived as the result of pacing feature

36 cts with progressive development of complete heart block and various arrhythmias, all of whom died su

37 into the pathogenesis of cardiac conduction, heart block and ventricular fibrillation.

38 ular anterior wall of adult dogs in complete heart block and with backup electronic pacemakers.

39 with small hearts, bradycardia, intermittent heart block, and a markedly reduced cardiac output.

40 irth, developed significant bradycardia with heart block, and died within the first weeks after birth

41 ctural defects but have progressive complete heart block, and massive trabecular muscle overgrowth fo

42 e ventricular contractions, atrioventricular heart block, and ventricular tachycardia.

43 diomyopathy are at risk for acute high-grade heart block, and, in this cohort, bradycardic events rep

44 ailure, cardiac arrest, arrhythmia, complete heart block, and/or stroke requiring hospitalization <1

45 o deaths were caused by development of acute heart block; another patient with syncope had intermitte

46 sociated cardiac defects, operative history, heart block, arrhythmias and tricuspid (i.e., systemic a

47 s conduction system abnormalities that cause heart block, arrhythmias, and sudden death are more comm

48 with neonatal lupus erythematosus (NLE) and heart block, as well as patients with Sjogren's syndrome

49 t groups more likely to develop third-degree heart block associated with Lyme carditis is essential t

50 risk for atrial and ventricular arrhythmias, heart block, asystole, development of pulmonary congesti

51 ing the DN allele displayed slow conduction, heart block, atrial fibrillation, ventricular tachycardi

52 ate cardiac erosions, thrombus formation and heart block; (b) the transcatheter closure of muscular v

53 evelop ventricular arrhythmias or high-grade heart block because the treatment is different and drama

54 e elinogrel arms, but there were no cases of heart block, bradycardia, hypotension, or liver failure.

55 or until a significant arrhythmia (asystole, heart block, bradycardia, supraventricular or ventricula

56 e molecular basis of atrial fibrillation and heart block but also may suggest targets for the develop

57 eading to sudden cardiac death from complete heart block, but no longer developed DCM or the other ph

58 a significantly higher incidence of complete heart block, but the risk was reduced with contrast echo

59 dence and determinants of permanent complete heart block (CHB) after nonsurgical septal reduction the

60 cardiac tissue from a fetus with congenital heart block (CHB) and an age-matched control.

61 description and understanding of congenital heart block (CHB) came in the 1970s with the observation

62 ce rate of anti-SSA/Ro-associated congenital heart block (CHB) is 17%.

63 Congenital heart block (CHB) is a conduction abnormality characteri

64 Congenital heart block (CHB) is a disease that affects the offsprin

65 Congenital heart block (CHB) is an autoimmune disease that affects

66 It is a widely held view that congenital heart block (CHB) is caused by the transplacental transf

67 lesion of autoantibody-associated congenital heart block (CHB) is fibrosis of the conducting tissue.

68 Congenital heart block (CHB) is presumed to be caused by transplace

69 Autoimmune associated congenital heart block (CHB) may result from pathogenic cross-talk

70 Identification of isolated congenital heart block (CHB) predicts, with near certainty, the pre

71 ce rates of autoimmune-associated congenital heart block (CHB) using information from the Research Re

72 relevant in autoimmune-associated congenital heart block (CHB) where the obligate factor is a materna

73 e report 16 infants with complete congenital heart block (CHB) who developed late-onset dilated cardi

74 Congenital heart block (CHB), associated with antibodies to SS-A/Ro

75 In congenital heart block (CHB), binding of maternal anti-SSA/Ro Abs t

76 necessary for the development of congenital heart block (CHB), the low frequency suggests that fetal

77 atrioventricular node leading to congenital heart block (CHB).

78 3 children (22 with rash, 35 with congenital heart block [CHB], 26 unaffected siblings) and 58 mother

79 of the literature on catheterization-induced heart block (CIHB) was published >30 years ago.

80 imary cardiac arrest, and sustained complete heart block, classified by a reviewer blinded to preoper

81 is associated with cardioskeletal myopathy, heart block, delayed growth and early postnatal death.

82 Complete heart block developed in 11 patients, who then required

83 A young woman with complete heart block died suddenly.

84 Destructive counterparts include familial heart block ending in fatal arrhythmias, similar results

85 after in utero identification of congenital heart block, especially in fetuses with associated myoca

86 utoantibodies with the genesis of congenital heart block, female BALB/c mice were immunized with huma

87 systemic lupus erythematosus, and congenital heart block from anti-Ro52 Ab-positive mother.

88 plays progressive muscle wasting, cataracts, heart block, gonadal atrophy, insulin resistance and neu

89 ce developing cardiomyopathy associated with heart block, impaired repolarization, and ventricular ar

90 ications included tamponade in 1 patient and heart block in 2 patients.

91 We describe the occurrence of complete heart block in a patient with Sjogren's syndrome.

92 anisms resulting in immune-mediated complete heart block in a small subset of 'at-risk' fetuses is un

93 ed K(+) channel (IKACh) could rescue SSS and heart block in Cav1.3(-/-) mice.

94 There was a trend toward more congenital heart block in fetuses of women with previously affected

95 ink between ETV1 and bundle branch block and heart block in humans.

96 entricular contractions and atrioventricular heart block in junctin-null mice.

97 t, second, and third-degree atrioventricular heart block in pediatric patients.

98 at hydroxychloroquine may prevent congenital heart block in pregnancies exposed to SSA/Ro antibodies.

99 Incidence of third-degree heart block in the STZ+RH group was 5.6%, 7.8-fold less

100 radually progressive development of complete heart block in young people often is associated with car

101 Complete heart block is a common complication after NSRT.

102 Heart block is a potential complication of both catheter

103 Heart block is associated with substantial morbidity and

104 Congenital heart block is considered a model of passively acquired

105 Childhood morbidity with heart block is high, with 63% eventually requiring pacem

106 ti-SSA/Ro-associated third-degree congenital heart block is irreversible, prompting a search for earl

107 The timing of heart block is not random; bradycardia is most often ide

108 ant to the development of anti-Ro-associated heart block, it can nevertheless be affected.

109 ssociated with an increased risk of complete heart block, it should not be performed routinely.

110 ta suggest that patients affected by SSS and heart block may benefit from IKACh suppression achieved

111 ded tamponade (n = 4), pericarditis (n = 3), heart block (n = 1, prior to radiofrequency application)

112 Other adverse events included complete heart block (n=2) and increased atrioventricular valve r

113 ses fulminant heart failure, arrhythmias, or heart block, necessitating aggressive immunosuppression,

114 Transient second- and third-degree heart block occurred at higher doses (300/600, 450/900,

115 Heart block occurred in 3 of 16 pregnancies (19%) in mot

116 nt heart rate or during pacing if Wenckebach heart block occurred.

117 Heart block occurs with increasing age.

118 ugs or other environmental stressors such as heart block or heart failure.

119 ailure predicted IGCM, and presentation with heart block or more than nine weeks of symptoms predicte

120 ailure predicted IGCM, and presentation with heart block or more than nine weeks of symptoms predicte

121 ers without a previous child with congenital heart block or rash (P=0.067).

122 mber of deaths (4 versus 1), final degree of heart block, or requirement for a pacemaker (14 versus 1

123 s6795970 is associated with a higher risk of heart block (P < 0.05) and a lower risk of ventricular f

124 sence of arrhythmias (P > 0.2), first-degree heart block (P = 0.12), bundle-branch block (P > 0.2), a

125 8.6%) had serious adverse events of complete heart block, peri-hepatic bleeding, and rupture of tricu

126 of having a subsequent child with congenital heart block ranges between 12-16%.

127 autoantibodies only after identification of heart block/rash in a child.

128 Other complications included heart block requiring a pacemaker (13%) and thrombosis (

129 Complications after initial MVR included heart block requiring pacemaker (16%), endocarditis (6%)

130 gnificantly increased the risk of a complete heart block requiring pacemaker implantation (8.1% versu

131 Heart block requiring permanent pacemaker implantation w

132 Four patients (3%) developed heart block requiring permanent pacing after the procedu

133 Six patients (12%) developed complete heart block requiring permanent pacing, three of whom ha

134 is of neonatal lupus syndrome and congenital heart block reveals important information about prospect

135 gene transfer of SEK-1(KR) to the adult rat heart blocks SAPK activation by pressure overload, demon

136 One survivor developed complete heart block shortly after the study visit.

137 hol ablation, but with a higher incidence of heart block than in cases where only surgery is performe

138 d by heart failure, ventricular arrhythmias, heart blocks, thromboembolic phenomena, and sudden death

139 nt patients who exhibit evidence of neonatal heart block, treatment with dexamethasone is preferred o

140 bundle-branch disorder progressive familial heart block type I (PFHBI) and isolated cardiac conducti

141 -in a large family with progressive familial heart block type I and showed that these mutations preve

142 mon adverse effects associated with TAVI are heart block, vascular complications, and renal failure.

143 Isolated heart block was associated with a 7.8% case fatality rat

144 e antibodies to have a child with congenital heart block was at or below one in 50.

145 Congenital heart block was diagnosed in utero in 12 patients and at

146 dence of pacemaker implantation for complete heart block was higher (22% vs. 2% in surgery; p = 0.02)

147 Second-degree heart block was increased 12- and 6.8-fold in the STZ an

148 A high rate of complete heart block was noted within the MVR group (37.5%).

149 Transient complete heart block was seen in 4 patients; the duration was <5

150 s cause rhythm disturbances such as complete heart block, which can be fatal.