ライフサイエンスコーパス: heart mitochondria

1 hasome disassembly in WT, but not in CypD KO heart mitochondria.

2 sines 19 and 26 of MPC2 is enhanced in Akita heart mitochondria.

3 f complex I purified from Ovis aries (ovine) heart mitochondria.

4 ith experimental data obtained from isolated heart mitochondria.

5 s reinhardtii and cytochrome bc(1) from beef heart mitochondria.

6 brane (85:6:9) distribution of PKA in bovine heart mitochondria.

7 observed only with BM and not with liver or heart mitochondria.

8 tion on ROS production by isolated energized heart mitochondria.

9 ygenases opened the myocardial mPTP in human heart mitochondria.

10 h calcium ions (Ca(2+)) signaling in porcine heart mitochondria.

11 f of that of the enzyme purified from bovine heart mitochondria.

12 nd energetic substrate availability of mouse heart mitochondria.

13 uced a rapid increase in A520, especially in heart mitochondria.

14 ctrometric analysis of highly purified human heart mitochondria.

15 tabolite of oleic acid, was studied with rat heart mitochondria.

16 rotein and a cyclophilin D purified from rat heart mitochondria.

17 in Sod2 in heart tissue, and no increase in heart mitochondria.

18 significant decrease from 6 to 23 months in heart mitochondria.

19 e effects of TG on MPT in isolated liver and heart mitochondria.

20 TP channels purified from rat liver and beef heart mitochondria.

21 Furthermore, isolated, intact rat heart mitochondria 1) synthesize malonyl-CoA with simult

22 ATP synthesis rates in hypoxic heart mitochondria (3.92+/-0.23 micromol ATP. min(-1). m

23 lationship between complex I from Bos taurus heart mitochondria, a close model for the human enzyme,

24 ution structure of complex I from Bos taurus heart mitochondria, a close relative of the human enzyme

25 tive channel in the native inner membrane of heart mitochondria and characterized its pharmacological

26 ydroxyl radical production in isolated mouse heart mitochondria and F2-isoprostanes in brains and hea

27 In bovine heart mitochondria and in submitochondrial particles, me

28 The precursor was imported by rat heart mitochondria and processed in a single step to a 2

29 We have purified complex I from beef heart mitochondria and reconstituted the enzyme into lip

30 Purified cytochrome b-c1 complexes from beef heart mitochondria and Rhodobacter sphaeroides were reco

31 The cytochrome c oxidase content of porcine heart mitochondria and whole tissue was determined to be

32 thesis by MF(1), the F(1)-ATPase from bovine heart mitochondria; and (ii) the possibility that the be

33 cattering were evaluated in isolated porcine heart mitochondria at 37 degrees C using a variety of op

34 ty against 48 kD OGDC-E2 when probed on beef heart mitochondria (BHM) but retained reactivity toward

35 e in the rate of pyruvate transport in Akita heart mitochondria but no decrease in the mitochondrial

36 ucture of supercomplex I1III2IV1 from bovine heart mitochondria by cryo-EM at 9 A resolution.

37 structure of intact ATP synthase from bovine heart mitochondria by electron cryomicroscopy of single

38 In contrast to brain mitochondria, liver and heart mitochondria challenged with Ca(2+) experienced su

39 reased the rate of ATP synthesis in normoxic heart mitochondria consistent with mitochondrial K(ATP)

40 Here, we report that human heart mitochondria contain frataxin species of increasin

41 (NADH:ubiquinone oxidoreductase) from bovine heart mitochondria contains 45 different subunits and ni

42 inhibition of the succinate dehydrogenase in heart mitochondria, contributes to the cause of death in

43 Heart mitochondria did not display a substantial alterat

44 Under conditions of ischemia heart mitochondria expressing MLS-STAT3E exhibited modes

45 155W mutant of Escherichia coli F(1) (bovine heart mitochondria F(1) residue number) can quantitative

46 Isolated heart mitochondria from 5-day-old sod2 null animals resp

47 Analyses of purified rat heart mitochondria from normal and streptozocin-treated

48 xidation of palmitoyl CoA (PCoA) in isolated heart mitochondria from Sham and streptozotocin (STZ)-in

49 We have used heart mitochondria from sod2 null mice to better underst

50 e a set of alterations caused by diabetes in heart mitochondria from streptozotocin-treated rats.

51 Biochemically, isolated heart mitochondria from the highly sensitive 129SVEMS mi

52 Isolated heart mitochondria from wild-type (WT) and CypD knockout

53 Functionally, isolated Akita heart mitochondria have significantly impaired maximal (

54 Incubation of rat liver and heart mitochondria in KCl medium containing Mg2+ and ino

55 inone oxidoreductase (complex I) from bovine heart mitochondria is a complicated, energy-transducing,

56 inone oxidoreductase (complex I) from bovine heart mitochondria is a highly complicated, energy trans

57 ce of ryanodine, Ca(2+) uptake into isolated heart mitochondria is suppressed.

58 of OGT and complex IV observed in normal rat heart mitochondria is visibly reduced in diabetic sample

59 ndria as well as unpurified brain, liver and heart mitochondria, isolated from 2- and 10-month-old YA

60 Parallel activation of heart mitochondria NADH and ATP production by Ca(2+) has

61 With the non-bovine serum albumin brain and heart mitochondria oxidizing succinate, the addition of

62 in, and in native gels of the entire porcine heart mitochondria proteome.

63 Peroxidative treatment of rat heart mitochondria results in a gradual increase of the

64 oupling in cardiac muscle using isolated rat heart mitochondria (RHMs).

65 the "OH" state) from several sources (bovine heart mitochondria, Rhodobacter sphaeroides, and Paracoc

66 Surprisingly, heart mitochondria show a dramatically lower mitochondri

67 Immunoblotting of purified human heart mitochondria showed an intense signal of ERbeta, w

68 experimental data obtained from isolated rat heart mitochondria subjected to physiological conditions

69 spiratory inhibition of MT in liver, but not heart, mitochondria suggest a hitherto unknown biologica

70 Isolated rat heart mitochondria supplemented with complex I or II sub

71 In hypoxic heart mitochondria, the rate of ATP synthesis was not af

72 f the rate of ATP hydrolysis by F1 from beef heart mitochondria to the ATP concentration dependence o

73 ochondria as well as resilience of liver and heart mitochondria to the deleterious effect of Ca2+.

74 tyrosine-nitrated proteins generated in the heart mitochondria under different in vitro and in vivo

75 dynamically monitor DeltaPsi of isolated rat heart mitochondria using a ratio fluorescence approach.

76 Heart mitochondria utilize multiple Ca(2+) transport mec

77 mitochondrial NAD(P)H generation flux in rat heart mitochondria utilizing pyruvate and malate as subs

78 s.e.m.) and the [Ca2+]m of the injected rat heart mitochondria was 116 +/- 10 nM (n = 18, mean +/- s

79 potential (delta psi(m)) in single isolated heart mitochondria was measured by confocal microscopy w

80 hondrial Ca2+ concentration ([Ca2+]m) in rat heart mitochondria was measured quantitatively by loadin

81 found that membrane depolarization in murine heart mitochondria was sensitized to Ca(2+) by the prese

82 quinone oxidoreductase) purified from bovine heart mitochondria was treated with the detergent N, N-d

83 ld be carried out with as little as 10 ng of heart mitochondria/well and as few as 3 x 10(4) cells/we

84 tional beta-oxidation complex (TOC) from pig heart mitochondria were analyzed with the aim of elucida

85 their interplay with matrix GSH in isolated heart mitochondria were examined.

86 omponents of matrix NADH in isolated porcine heart mitochondria were investigated using time-resolved

87 re almost completely trimethylated in bovine heart mitochondria, whereas ETFalpha is not methylated.

88 mol/L) and 5-HD (300 micromol/L) in normoxic heart mitochondria, whereas glibenclamide and 5-HD alone

89 on by skeletal muscle, brown fat, brain, and heart mitochondria with an emphasis on conditions under

90 evious study, we found that treatment of rat heart mitochondria with H(2)O(2) resulted in a decline a

91 the present study, treatment of isolated rat heart mitochondria with H(2)O(2) resulted in a decline a

92 In the present study, treatment of rat heart mitochondria with HNE resulted in the selective in

93 ume, pH(m), and O2 consumption in guinea pig heart mitochondria with or without ruthenium red, carbox