ライフサイエンスコーパス: heart rate

1 he first evidence of miR control of HCN4 and heart rate.

2 ympathetic nerve stimulation to increase the heart rate.

3 revent the high altitude-induced increase in heart rate.

4 n decreased temperature, blood pressure, and heart rate.

5 es (CamNtech) to assess resting and stressed heart rate.

6 plus; LiDCO, Cambridge, United Kingdom), and heart rate.

7 ause of a temporarily or permanently reduced heart rate.

8 ed during handgrip exercise due to increased heart rate.

9 varied according to compensatory increase in heart rate.

10 ng inside the body, such as hunger, pain and heart rate.

11 ractile dysfunction, arrhythmia, and reduced heart rate.

12 ed heart wall thickness as well as increased heart rate.

13 lished the high altitude-induced increase in heart rate.

14 the small size of the mouse embryo and rapid heart rate.

15 of these changes are abnormally low resting heart rates.

16 ure different cardiac phenomena at different heart rates.

17 nt genetic correlation is found for HRV with heart rate (-0.74<rg<-0.55) and blood pressure (-0.35<rg

18 20.6+/-4.5 minutes; P<0.001) and lower peak heart rates (159+/-20 versus 184+/-9 beats/min; P<0.001)

19 tasks (METs; 11.6 vs 11.7; P = .80), maximum heart rate (174 vs 175 beats/min; P = .41), and heart ra

20 on for systolic blood pressure (213 [8.8%]), heart rate (223 [9.2%]), and anterior MI location (279 [

21 c diameter -1.3 mm, (-2.3 to 0.3, p=0.0128), heart rate -3.0 beats per min (-5.1 to -0.8, p=0.0070),

22 (-7 mm Hg; 95% CI, -12 to -1; p = 0.02), and heart rate (-6 beats/min; 95% CI, -10 to -1; p = 0.03).

23 or >/=460 ms in women (1.72, 1.19-2.49); and heart rate 60-90 beats per minute (1.21, 0.89-1.63) and

24 often male (82.0% versus 75.4%), had higher heart rate (80 versus 72), had higher Global Registry of

25 stolic depolarization and, consequently, the heart rate, a mechanism that is distinct from those of o

26 onotonically increasing relationship between heart rate, a physiological index of arousal, and within

27 .3 [0.6] years at conscription) with resting heart rates above 82 beats per minute had a 69% (95% CI,

28 s were fitted for 7 days to measure sleeping heart rate, activity levels, and resting, active and tot

29 Heart rate alerts were too few for model development.

30 in A1c, weight, systolic blood pressure, and heart rate; all-cause mortality; cardiovascular and cere

31 The blinking rate, eye closure, heart rate, alpha and beta band power were used to ident

32 ), whereas suboptimal heating led to a lower heart rate and a higher SD (470 +/- 84 beats/min).

33 ), whereas suboptimal heating led to a lower heart rate and a higher SD (470 +/- 84 beats/min).

34 Unadjusted and adjusted associations between heart rate and all-cause mortality and heart failure hos

35 ), which is expressed partly by increases in heart rate and arterial pressure.

36 ratrial septum is followed by an increase in heart rate and atrioventricular nodal conduction propert

37 ch the present study suggests that increased heart rate and autonomic changes are prevalent in behavi

38 ous Cx36 KO mice revealed higher variance in heart rate and blood pressure during rest and activity c

39 results suggest that for men, differences in heart rate and blood pressure in late adolescence are as

40 Physiologic regulation of heart rate and blood pressure involves connexin 36-conta

41 system testing revealed no abnormalities in heart rate and blood pressure variability however the sy

42 or second eye, pain control, intra-operative heart rate and blood pressure, age, and case complexity.

43 consisting of periodic episodes of increased heart rate and blood pressure, sweating, hyperthermia, a

44 Sympathetic NE increases heart rate and contractility through activation of beta

45 poxia, as indicated by reduced elevations in heart rate and exaggerated changes in femoral vascular c

46 rosclerosis Risk In Communities) cohort with heart rate and HRV measures obtained from 2-min electroc

47 In contrast, lower resting heart rate and lower systolic blood pressure were associ

48 Heart rate and mean blood pressure were monitored.

49 e, cutaneous vasodilatation, blood pressure, heart rate and middle cerebral artery velocity (MCAv) we

50 riptional end points but additive effects in heart rate and pepck1 upregulation, which indicates that

51 Hemorrhage increased heart rate and plasma renin activity in intact sheep, bu

52 and emotionality of the memories as well as heart rate and skin conductance level during memory retr

53 es evoke reflex increases in blood pressure, heart rate and sympathetic nerve activity.

54 We observed that a 10% reduction in both heart rate and temperature sustained for greater than or

55 Heart rate and temperature were significantly elevated i

56 Changes in heart rate and the peak high frequency were correlated w

57 tion in heart cell ion channels leads to low heart rates and arrhythmia by an unknown route.

58 of ACh and NE may impair adaptation to high heart rates and increase arrhythmia susceptibility.

59 of ACh and NE may impair adaptation to high heart rates and increase arrhythmia susceptibility.

60 y mass index than controls; however, resting heart rates and QT/QTc intervals were similar at baselin

61 decrease in muscle contraction, increase in heart rate, and accelerated hatching.

62 nd blood pressure at different increments of heart rate, and atrioventricular and interventricular de

63 Homeostatic control of breathing, heart rate, and body temperature relies on circuits with

64 ng the expected variation in blood pressure, heart rate, and cortisol.

65 MAp44 caused impaired cardiogenesis, lowered heart rate, and decreased cardiac output.

66 rmalization of mean arterial blood pressure, heart rate, and increased survival were observed in 4-am

67 the knowledge of the genetic architecture of heart rate, and indicate new candidate genes for follow-

68 infections, and changes in body temperature, heart rate, and minute ventilation.

69 induces a reciprocal phenotype of increased heart rate, and prevents the adaptive intrinsic bradycar

70 arameters (arterial pressure, cardiac index, heart rate, and pulse pressure variations) was observed.

71 tion (APD), and variations in APD at a fixed heart rate are both reliable biomarkers of electrophysio

72 Heart rate, arrhythmia, patient discomfort, and adverse

73 ange of cardiovascular parameters, including heart rate, arterial pressures, and body temperature.

74 evidence behind using beta-blocker dose and heart rate as therapeutic targets.

75 energetic expenditure by 10% and counteracts heart rates as high as 900 bpm during flight.

76 We meta-analysed heart rate association results from 104 452 European-anc

77 rt rate (174 vs 175 beats/min; P = .41), and heart rate at 2 minutes recovery (44 vs 43 beats/min; P

78 A higher heart rate at baseline was associated with greater all-c

79 yses have found loci associated with resting heart rate, at the time of our study these loci explaine

80 and observed that, in addition to decreased heart rate, atrial conduction velocity is persistently s

81 ety disorders compared with men with resting heart rates below 62 beats per minute.

82 Regardless of pre-treatment heart rate, beta-blockers reduce mortality in patients w

83 Heart rate, blood pressure, and heart rate variability w

84 omes included adjusted time-weighted average heart rate, blood pressure, and respiratory rate, along

85 l small fiber evaluation, such as autonomic (heart rate, blood pressure, and sweat testing) and subje

86 binding of TRalpha and TRbeta, most notably heart rate, body temperature, blood glucose, and triglyc

87 We report health outcomes (blood pressure, heart rate, body weight, lung function, respiratory symp

88 a phenotype including unexplained slowing of heart rate (bradycardia).

89 ted with a lower blood pressure and a higher heart rate but also with the hemoglobin phenotype.

90 The maximum reduction in mean heart rate by Holter monitoring during the first 6 h in

91 in humans including various ways to measure heart rate, catecholamines, and sympathetic neural activ

92 Blood pressure, ECG, oxygen levels, heart rate, CBC, and metabolic panel were obtained befor

93 Independent of this heart rate change, CCh caused a significant increase in

94 esterol and blood pressure, without altering heart rate; changes in the number and size of adipocytes

95 significantly correlated with the intrinsic heart rate, consistent with HCN repression in athletes.

96 and 20 (38%) had abnormal blood pressure or heart rate control.

97 dition, the best initial treatment strategy--heart-rate control or rhythm control--remains controvers

98 enabled continuous, real-time measurement of heart rate, core temperature, and mobility.

99 ndividual subject's drug-induced increase in heart rate-corrected QT (QTc) versus drug concentration.

100 SNP haplotypes were neither associated with heart rate-corrected QT interval duration (QTc) nor card

101 Increased resting heart rate correlated with behavioural (Cambridge Behavi

102 =0.03 and 0.01), respectively; and decreased heart rate correlated with lower valvuloarterial impedan

103 Heart rate decreased by 15% during saline infusion (P=0.

104 Heart Failure) registry, 6,286 had a stable heart rate, defined as </=20 beats/min variation between

105 ith less heat discomfort, but did not affect heart rate differently compared with iopromide 300.

106 t mutations in GNB5 that are associated with heart-rate disturbance, eye disease, intellectual disabi

107 All participants with baseline heart rate documented by a 12-lead electrocardiogram wit

108 dren's exposure to neighborhood violence and heart rate during a stressful task were assessed.

109 rate, optimal animal heating led to a stable heart rate during acquisition (515 +/- 35 [mean +/- SD]

110 rate, optimal animal heating led to a stable heart rate during acquisition (515 +/- 35 [mean +/- SD]

111 The heart rate during scanning ranged from 50 to 133/min.

112 Results Mean heart rate during scanning was 83 beats per minute +/- 2

113 stratified by target temperature and minimum heart rate during targeted temperature management (< 50,

114 ted analysis of continuous electrocardiogram heart rate dynamics detects new-onset atrial fibrillatio

115 s associated with significant alterations in heart rate dynamics, including HRV and complexity.

116 lood pressure (finger photoplethysmography), heart rate (electrocardiogram), oxygen saturation (pulse

117 latory response, reduced the hypoxia-induced heart rate elevation and exaggerated the blood pressure

118 The heart rate elevation was 23.8+/-12.5%, and the Wenckebac

119 Beat-to-beat variation in heart rate (f H ) has been used as a tool for elucidatin

120 rmance including cardiorespiratory function (heart rate [fH ] and ventilation rate [fV ]), metabolic

121 ional MRI data and continuously assessed the heart rate from 120 healthy human adults as they viewed

122 1) , n = 9) significantly (P < 0.05) reduced heart rate from 292 +/- 8 to 224 +/- 6 b.p.m.

123 The primary endpoint was >/=20% reduction in heart rate from baseline without inducing bradycardia or

124 to discover new genetic loci associated with heart rate from Exome Chip meta-analyses.Heart rate was

125 rmed at the clinician set RR interval and at heart rates from 70 to 110 beats/min, in increments of 1

126 olus injection (0.04 mg/kg) did not increase heart rate further.

127 ic blood pressure of less than 90 mm Hg or a heart rate greater than 120 beats per minute.

128 t Association class II-IV, sinus rhythm, and heart rate >/=70 beats per minute) and non-SHIFT type.

129 curred during the recovery period, after the heart rate had largely returned to baseline levels, with

130 fitness were forced vital capacity, resting heart rate, hand grip strength, sit and reach distance,

131 role that M3R plays in the regulation of the heart rate, has led to the assumption that cardiovagal d

132 th BMI, waist circumference, blood pressure, heart rate, HbA1c, blood glucose, LDL-to-HDL cholesterol

133 y comparing the hypoxia-induced elevation in heart rate (HR [bpm]), ventilation (VE [L min(-1)]) with

134 We recorded heart rate (HR) and corresponding activity of an adult f

135 ucose, body mass index, waist circumference, heart rate (HR) and diabetes, but were not associated wi

136 Trigger sounds elicited heightened heart rate (HR) and galvanic skin response (GSR) in miso

137 st-, 24 h-post-exposure, we measured resting heart rate (HR) and heart rate variability (HRV) with el

138 computed autonomic cardiac markers, such as heart rate (HR) and HR variability (HRV), and cardiac cy

139 Heart rate (HR) and oxygen saturation were monitored.

140 e objective of this work was to characterize heart rate (HR) responses (HRRs) during the active phase

141 low (RBF), mean arterial pressure (MAP), and heart rate (HR) were continuously measured and urine was

142 MSNA, blood pressure and heart rate (HR) were recorded in age- and sex-matched RA

143 cts first received the sample infusions, and heart rate (HR), blood pressure, and subjective stimulat

144 Mean arterial pressure (MAP), heart rate (HR), BT, motor activity (MA), and oxygen con

145 ABSTRACT: Chronic hypoxia increases resting heart rate (HR), but the underlying mechanism remains in

146 ial application of bradykinin (BK) increased heart rate (HR), left ventricular systolic pressure (LVS

147 Systolic blood pressure (SBP), heart rate (HR), pathology, and left ventricular mass in

148 eters [pulsatile arterial pressure (PAP) and heart rate (HR)].

149 after multivariable adjustment for age, sex, heart rate, hypertension, systolic blood pressure, left

150 To study the prognostic utility of heart rate in a community-based African American cohort

151 Increased resting heart rate in behavioural variant frontotemporal dementi

152 s the main mechanism underlying the elevated heart rate in chronic hypoxia.

153 n core body temperature, blood pressure, and heart rate in febrile critically ill patients.

154 alysis explored the prognostic importance of heart rate in patients with heart failure with reduced e

155 inoatrial cell pacemaker mechanisms to lower heart rate, including sarcolemmal hyperpolarization-acti

156 vivo, the atropine-dependent prolongation of heart rate increase was blunted in PDE4D but not in wild

157 ring the peak period of sympathetic arousal, heart rate increase, and cardiorespiratory sensation.

158 AI, corrected for heart rate, increased more rapidly with age in SCD patie

159 Heart rate initially increased in participants with T2DM

160 to 0.79; p < 0.001), regardless of baseline heart rate (interaction p = 0.35).

161 0.96, 95% CI: 0.81 to 1.12; p = 0.58) at any heart rate (interaction p = 0.48).

162 A lower achieved resting heart rate, irrespective of treatment, was associated wi

163 Resting heart rate is a heritable trait correlated with life spa

164 Resting heart rate is a heritable trait, and an increase in hear

165 ur results reveal that in mammals, for which heart rate is a key determinant of cardiac energy demand

166 A lower heart rate is associated with better outcomes in patient

167 Achieving a lower heart rate is associated with better prognosis, but only

168 ate is a heritable trait, and an increase in heart rate is associated with increased mortality risk.

169 Increased resting heart rate is associated with worse outcomes in studies

170 KEY POINTS: Heart rate is increased in chronic hypoxia and we tested

171 ent physiological monitors (e.g. actigraphy, heart rate) largely lack in cross-modal ability, are inc

172 ympathetic neurotransmitter acetylcholine on heart rate leading to tachycardia.

173 ine, with no patient having a minimum hourly heart rate less than 45 bpm.

174 informatics methods.We discovered five novel heart rate loci, and one new independent low-frequency n

175 ncy non-synonymous variant in an established heart rate locus (KIAA1755).

176 same hemodynamic mechanism, pharmacological heart rate lowering also engenders an increase in centra

177 or coronary heart disease, or even athletes, heart rate lowering consistently increases central systo

178 erapeutic agent now exists to target further heart rate lowering in patients who have been stable on

179 ar down-regulation (extreme bradycardia with heart rate </=4 beats per minute) superimposed on exerci

180 A baseline heart rate <60 beats/min was associated modestly with an

181 A heart rate <70 beats/min was also associated with a lowe

182 Propensity scores for discharge heart rate <70 beats/min, estimated for each of the 6,28

183 ble a cohort of 2,031 pairs of patients with heart rate <70 versus >/=70 beats/min, balanced on 58 ba

184 us 70% for matched patients with a discharge heart rate <70 versus >/=70 beats/min, respectively (haz

185 s, and implications of relative bradycardia (heart rate, < 80 beats/min) in septic shock are unknown.

186 A low heart rate may be associated with higher AF risk.

187 ly assigned to HIIT at 90% to 95% of maximal heart rate, MCT at 60% to 70% of maximal heart rate, or

188 lacebo, metoprolol (100+/-53 mg/d) decreased heart rate; mean difference (95% confidence interval) -8

189 Cox proportional hazard ratios (HR) modeling heart rate measured at baseline and approximately 6 mont

190 Aerobic fitness was assessed by means of heart rate measurement following a standardized submaxim

191 ing them unsuitable for applications such as heart-rate monitoring, which require a much lower pressu

192 4 (L4), which included oxygen saturation and heart rate (n = 135).

193 w plasma norepinephrine levels, slow resting heart rate, no REM sleep behavior disorder, and preserve

194 Of these, 2,369 (38%) had a discharge heart rate of <70 beats/min.

195 (r = 0.61, P < 0.001) or AI c normalized for heart rate of 75 bpm (r = 0.65, P < 0.001).

196 Ivabradine safely reduced the resting heart rate of children with chronic HF and dilated cardi

197 c death was associated with a higher resting heart rate, older age, elevated creatinine, larger left

198 We sought to determine the acute impact of heart rate on cardiac electromechanics, cardiac output,

199 length, body weight, body surface area, and heart rate on PAAT were investigated.

200 Echocardiography-guided heart rate optimization results in a significant increas

201 and oxygen consumption, (3) methods based on heart rate or (4) breathing rate, and (5) methods that c

202 However, with increased heart rate or beta-adrenergic stimulation, cTnIS200D mic

203 However, whether low heart rate or heart rate variability (HRV), which are no

204 95% CI, -0.63 to -0.13; I = 84.0%), but not heart rate or minute ventilation.

205 , respectively; P < .001), no differences in heart rate or rhythm were observed.

206 five 25-min sessions/wk at 75-85% of maximum heart rate) or no training.

207 mal heart rate, MCT at 60% to 70% of maximal heart rate, or RRE.

208 s-over-time temperature, blood pressure, and heart rate outcomes were also significantly lower at 2 h

209 e of violence (p = 0.004) and with decreased heart rate (p = 0.002).

210 ed compared to control subjects, and resting heart rate (P = 0.020) compared to Alzheimer disease pat

211 = 0.001), stressed (P = 0.037) and sleeping heart rate (P = 0.038) were increased compared to contro

212 ith AI r (age, P < 0.001; gender, P < 0.001; heart rate, P < 0.001; diastolic blood pressure, P < 0.0

213 rience during the first technique had higher heart rate (partial-eta(2) = 0.09, p < 0.05).

214 h the composite outcome included: lower peak heart rate predicted, lower blood pressure response, low

215 es were collected, and standard vital signs (heart rate, pulse oximetry, and body temperature) were m

216 The highest heart rate quintile (73-118 bpm) had higher rates of dia

217 changes in the pulse pressure (r = 0.18) and heart rate (r = 0.09).

218 nd body weight (r=0.825) and negatively with heart rate (r=-0.906).

219 Control of the heart rate (rate control) is central to atrial fibrillat

220 We developed a non-invasive visible optical heart rate recording system especially suitable for long

221 Heart rate recovery after a maximal cardiopulmonary exer

222 fraction (<41%) had a significantly reduced heart rate recovery in comparison with patients with hig

223 ve (AV) gradient, indexed AV area, METs, and heart rate recovery were 2.9+/-3%, 58+/-4%, 35+/-11 mm H

224 Heart rate reduction as a therapeutic target has been in

225 echanisms include neurohormonal blockade and heart rate reduction.

226 viduals benefit from interventions targeting heart rate reduction.

227 ndogenous phosphodiesterase 2 contributes to heart rate regulation.

228 nnabis cigarette (eg, strength, liking), and heart rate relative to inactive cannabis.

229 Blood pressure and heart rate remained stable in all cohorts.

230 The relationship between mortality and heart rate remains unclear for patients with heart failu

231 atropine led to increased contractility and heart rates, respectively.

232 did not differ from the controls in terms of heart rate, respiratory rate, or rectal temperature.

233 Noninvasive vital sign monitoring data (heart rate, respiratory rate, peripheral oximetry) recor

234 requency-amplitude-pulse width, where a null heart rate response is reproducibly evoked during the on

235 In addition, heart rate response to mental stress was diminished over

236 By contrast, opposing heart rate responses were evoked by adenosine (bradycard

237 The optimal heart rate should be individualized for each patient, an

238 Heart rate, sinus node recovery time, Wenckebach cycle l

239 8) showed more eye-blink reflexes and larger heart rate, skin conductance, and pupil area responses t

240 Significant heart rate slowing and frequent sinus pauses are observe

241 x, race, physical activity, body mass index, heart rate, smoking status, systolic blood pressure, fas

242 lla, positively correlated with increases in heart rate, suggesting that the intention to willfully m

243 tinal bleeding, rectal examination findings, heart rate, systolic blood pressure, and haemoglobin con

244 Heart rate, systolic blood pressure, rate x pressure pro

245 Miniaturized heart rate telemetry shows that they use a novel, cyclic

246 a significant fall in arterial pressure and heart rate that was similar in magnitude between normote

247 Neither genotype nor IR affected BP, heart rate, urine volume, or albumin excretion.

248 Conventional heart rate variability (HRV) analysis, and complexity in

249 Frequency domain measures of heart rate variability (HRV) are associated with adverse

250 duced cardiac vagal control reflected in low heart rate variability (HRV) is associated with greater

251 ure, we measured resting heart rate (HR) and heart rate variability (HRV) with electrocardiogram, and

252 influence (cardiorespiratory interaction) on heart rate variability (HRV) with heartbeats increasing

253 uence of disinhibited eating and vagal tone (heart rate variability (HRV)) on hunger and the postpran

254 n ANS activity during waking, as measured by heart rate variability (HRV), have been correlated with

255 However, whether low heart rate or heart rate variability (HRV), which are noninvasive meas

256 polycyclic aromatic hydrocarbons (PAHs) and heart rate variability (HRV).

257 6 +/- 0.2 vs. 0.2 +/- 0.1 low/high frequency heart rate variability (LF/HFHRV )] and cardiac arrhythm

258 vary cortisol (hypothalamic pituitary axis), heart rate variability (sympathetic adrenal medullary sy

259 leus predicted the magnitude of reduction in heart rate variability after induction.

260 Heart rate variability analysis revealed sympathetic pre

261 Heart rate variability components were analyzed for the

262 we investigate a possible mechanism by which heart rate variability could protect against cardiac arr

263 data, consisting of an individual's labeled heart rate variability epochs from the preceding 24 hour

264 Depressed heart rate variability is a well-established risk factor

265 ognostic tool, it is unclear whether reduced heart rate variability itself is proarrhythmic or if it

266 the probability of each sedation level from heart rate variability measures derived from the electro

267 Although measurements of heart rate variability provide a valuable prognostic too

268 Heart rate, blood pressure, and heart rate variability were assessed every 30 min and co

269 Blood pressure and heart rate variability were registered at day 5 post-MI.

270 panied by an increase in peak high frequency heart rate variability, indicating an increase in parasy

271 vation (noradrenaline, rho = 0.26, P = 0.05; heart rate variability, rho = -0.43, P = 0.003), and tre

272 he asymmetric barrier induced a reduction in heart rate variability, suggesting that this is a more d

273 een sedation levels in ICU patients based on heart rate variability.

274 ocardiography measure of PNS (high frequency heart rate variability; HF-HRV).

275 ink to health outcomes has been reported for heart-rate variability (HRV), a marker of physiological

276 We tested whether resting heart-rate variability (HRV), a physiological indicator

277 eased cardiovascular mortality and decreased heart-rate variability (HRV).

278 respiratory distress syndrome, temperature, heart rate, vasopressor use, Sequential Organ Failure As

279 planchnic SNA (SSNA), arterial pressure, and heart rate via projections to the paraventricular nucleu

280 Median (25th-75th percentile) baseline heart rate was 63 beats per minute (bpm) (57-71 bpm).

281 At first CPET, the median peak heart rate was 80% (IQR, 70-88%) of predicted, median pe

282 Baseline heart rate was assessed by quintiles and as a continuous

283 Each increase of 5 beats/min in initial heart rate was associated with a 22% increase in the odd

284 Higher baseline heart rate was associated with increased mortality and h

285 As a continuous variable, elevated heart rate was associated with increased mortality and h

286 athetic activity and thus blood pressure and heart rate was determined using a mouse with specific ge

287 lized patients with HFpEF, a lower discharge heart rate was independently associated with a lower ris

288 In seven lowlanders, heart rate was measured at sea level and after 2 weeks a

289 ith heart rate from Exome Chip meta-analyses.Heart rate was measured from either elecrtrocardiograms

290 items after a 12-hour interval during which heart rate was recorded continuously.

291 METHODS AND As in rodents, the intrinsic heart rate was significantly lower in human athletes tha

292 iolence exposure as other children and their heart rate was similar to that of adults.

293 Ivabradine's effect on heart rate was variable, highlighting the importance of

294 Although the difference in heart rates was slight, optimal heating yielded signific

295 g the neural correlates of increased resting heart rate were investigated including cortical thicknes

296 olumes were unchanged but blood pressure and heart rate were reduced in both groups (P < 0.001).

297 a marked reduction in resting and in maximal heart rate, whereas cardiac output was completely preser

298 cause pronounced beta1-mediated reduction of heart rate while showing no effect on beta2-mediated hin

299 dy were to examine associations of discharge heart rate with outcomes in hospitalized patients with H

300 ssociated with LV end-systolic dimension and heart rate (z-score difference per year= -0.11; p = 0.05