1 ic of excitation-contraction coupling in the heart ventricle.

2 s in an anatomically accurate model of human heart ventricles.

3 ally (Acsl1(T-/-)) and mice lacking ACSL1 in heart ventricles (Acsl1(H-/-)) at different times.

4 ecies examined, three lack Mb polypeptide in heart ventricle, although one of these expresses the Mb

5 x9b is expressed in the developing zebrafish heart ventricle and that TCDD exposure markedly reduces

6   Image-derived input functions (IDIFs) from heart ventricles and myocardial time-activity curves wer

7 experiments from three mouse tissues, liver, heart ventricles, and primary keratinocytes, indicate th

8 isoforms that are expressed in the mammalian heart (ventricle- and atrium-specific MLC2) remain undef

9  I and Ang II was increased in failing human heart ventricles but was mediated by at least two differ

10       Results indicate that Mb is present in heart ventricles from five of these species of icefish.

11 s, extensor digitorum longus, diaphragm, and heart ventricle proteins were oxidatively damaged after