ライフサイエンスコーパス: heat inactivation

1 hiotreitol, ethylenediaminetetraacetic acid, heat inactivation).

2 ss that is resistant to N-ethylmaleimide and heat inactivation.

3 to the unbound state indicates irreversible heat inactivation.

4 mplate-primer, which likely protects against heat inactivation.

5 Mos(1-198)/SH2 enzyme is also more stable to heat inactivation.

6 s of ACSL6 were more resistant than ACSL4 to heat inactivation.

7 ediated cytotoxicity, which was inhibited by heat inactivation.

8 inhibitory activity was markedly reduced by heat inactivation.

9 esent in rhHSP60-2 were equally sensitive to heat inactivation.

10 plus rhHsp70-1 were all equally sensitive to heat inactivation.

11 RNA promoter protects the polymerase against heat inactivation.

12 LV RT and therefore is better protected from heat inactivation.

13 e of the mutants were also more sensitive to heat inactivation.

14 ven when other chaperones are present during heat inactivation.

15 l2 and ATP and formed complexes resistant to heat inactivation.

16 ected in SDS-PAGE without reducing agent and heat inactivation.

17 Enhancement (i) was destroyed by heat inactivation (30 min, 56 degrees C); (ii) did not r

18 Glycodelin activity was totally reversed by heat inactivation (95 degrees C x 15 min) and neutralize

19 is important to understand how formalin and heat inactivation affected the antigenicity and immunoge

20 strain fail to form mature spliceosomes upon heat inactivation, although commitment complexes and pre

21 arvicidal activities were retained following heat inactivation and drying of the yeast into tabular f

22 the presence of heparin and was reversed by heat inactivation and the protease inhibitor leupeptin,

23 Heat inactivation and trypsin treatment of cytosol, as w

24 r PBMC AI, an effect that was increased with heat inactivation and was corrected with CVF treatment.

25 ccessory subunit, as judged by processivity, heat inactivation, and N-ethylmaleimide protection assay

26 cific ACAT inhibitor, and sensitivity toward heat inactivation, are essentially unaltered.

27 n the absence of plasma and was inhibited by heat inactivation, as well as by the terminal complement

28 ic peptide Cet1(232-265), protects Ceg1 from heat inactivation at physiological temperatures.

29 ne activity was unable to protect MKP-3 from heat inactivation but interfered with MEK1/2 activation

30 wo mutants were similar in susceptibility to heat inactivation, but one of those mutants and one othe

31 concentrations is shown to be stabilized to heat inactivation by E. coli molecular chaperones DnaK o

32 Complement heat-inactivation, C5 depletion, and C5a receptor inhibi

33 r and SLE or control plasma, with or without heat inactivation, cobra venom factor (CVF), or lipopoly

34 re vulnerable to proteolytic degradation and heat inactivation compared with the wild-type enzyme.

35 the presence of plasma and its abrogation by heat inactivation, EDTA, and eculizumab.

36 in; this proteolytic action was inhibited by heat inactivation, EGTA, or ALLN.

37 ted lysate assay reactions can be stopped by heat inactivation (enabling ready control of selection s

38 Heat inactivation experiments with the variants suggest

39 Several assay parameters such as serum heat-inactivation (HI) and dilution can alter WNV MIA se

40 However, it was protected from heat inactivation if both the 5' and 3' strands of the v

41 ncluded (i) proteinase K digestion (PKD) and heat inactivation; (ii) PKD and ethanol precipitation (E

42 tein expression, which were abrogated by Tat heat inactivation, immunodepletion, and cysteine mutatio

43 oral poliovirus vaccine seed stocks based on heat inactivation in the presence of MgCl2 did not compl

44 re vulnerable to proteolytic degradation and heat inactivation in vitro compared with the oligomeric

45 were abolished by both trypsin treatment and heat inactivation, indicating the protein nature of the

46 We conclude that heat inactivation is a simple step that eliminates false

47 men were comparable to those obtained by the heat-inactivation method (except for subjects with therm

48 This was not due to heat inactivation of A. actinomycetemcomitans AI-2 since

49 Heat inactivation of BPEx had no effect on the developme

50 Serum pretreated by either heat inactivation of complement or immunoadsorption with

51 The same tautomer shift is also induced by heat inactivation of Fe(II)CBS, or by an Arg266Met repla

52 Heat inactivation of H. somnus culture filtrates partial

53 Heat inactivation of HIV-1 blocks nuclear localization o

54 Using heat inactivation of influenza virus, we show that viral

55 Neither heat inactivation of plasma nor ultrafiltration of plasm

56 Tat from Tat-containing conditioned media or heat inactivation of recombinant Tat abrogated those eff

57 Heat inactivation of restriction enzymes followed by lig

58 This attachment is not reduced by heat inactivation of the serum, suggesting complement in

59 In vitro heat inactivation of the temperature-sensitive prp5-1 mu

60 ted genuine infection included the fact that heat inactivation of the virus eliminated it, the levels

61 taminating adenovirus infectious activity by heat inactivation or by purification.

62 tion in chicken macrophages was abrogated by heat inactivation or pre-exposure of the lysate to PMSF.

63 cell extract could be abrogated by dilution, heat inactivation, or chromatographic depletion.

64 The cytotoxicity of sCD44 was verified by heat-inactivation, pretreatment with a pan-caspase inhib

65 The cytotoxicity of sCD44 was blocked by heat-inactivation, pretreatment with a pan-caspase inhib

66 Because heat inactivation primarily denatures C1q, we used serum

67 this observation, biochemical properties and heat inactivation profiles of the genetically altered en

68 c activity, thrombin activation profile, and heat inactivation properties similar to those of wild-ty

69 r Ad5dl309 as a helper demonstrated that the heat inactivation protocol generally used does not remov

70 melting temperature and 20 degrees C in the heat-inactivation temperature.

71 bility of vRNA to protect the enzyme against heat inactivation, we established a novel assay, in conj