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   1 if germinants are removed, and loss of spore heat resistance.                                        
     2 ore core dehydration and a decrease in spore heat resistance.                                        
     3 important to accurately determine bacteria's heat resistances.                                       
     4 osa primarily in stationary phase and boosts heat resistance 100-fold when expressed in Escherichia c
  
  
     7  similar to endospores in ultrastructure, in heat resistance and in the presence of dipicolinic acid.
  
  
  
    11 ores of Clostridium perfringens possess high heat resistance, and when these spores germinate and ret
    12 cribed to intracellular trehalose, including heat resistance, are not due to the presence of trehalos
    13 and wild-type cells, suggesting induction of heat resistance at low growth rates is independent of re
    14 ified as type F because of their exceptional heat resistance but later identified as type C strains. 
    15  a 1 degrees C warming scenario as increased heat resistance cannot be achieved without a reduction i
    16 rocess-like and operating conditions such as heat resistance, contact with organic solvents, steriliz
    17 , and dacC and wild-type spores had the same heat resistance, cortex structure, and germination and o
    18  and sporulation, and ywhE spores had normal heat-resistance, cortex structure, and germination and o
    19 s of two Bacillus species, the early loss in heat resistance during germination is most likely due to
    20 scopic morphology, production of extrolites, heat-resistance fungi, and sequencing of DNA regions.   
    21  and IB by a more acidic pH optimum, greater heat resistance, greater sensitivity to alkylating agent
  
  
    24 tation and maximum temperature, showing that heat resistance is an important determinant of Drosophil
    25    The latter event is puzzling, since spore heat resistance is due largely to core water content, wh
  
  
    28 cause human food-borne illness share a spore heat resistance mechanism that likely favors their survi
  
  
  
    32 germination, heat activation optima, and wet-heat resistance of superdormant spores and the heterogen
  
  
    35 alyses we showed that phylogenetic signal in heat resistance reflects phylogenetic inertia rather tha
    36 iated TAG accumulation was found to increase heat resistance, since nonacclimated pdat1 mutant seedli
  
  
  
  
  
  
  
    44 his protein did not restore UV radiation and heat resistance to spores lacking the majority of their 
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