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1 lung tumorigenesis between light smokers and heavy smokers.
2 g (P < 0.05), with higher risks reported for heavy smokers.
3 r moderate smokers, and 1.57 (1.47-1.66) for heavy smokers.
4 25, 9.17, and 11.89 for light, moderate, and heavy smokers.
5 increased lung cancer risk, especially among heavy smokers.
6 phrenic, 26 depressed, and 26 nonpsychiatric heavy smokers.
7 beta-carotene supplements except in current heavy smokers.
10 in nonsmokers and 0.69 (95% CI, 0.4-1.2) in heavy smokers (80 pack-years; P < 0.01 for the interacti
11 pmol/mmol, mean +/- SEM) was 176.5+/-30.6 in heavy smokers, 92.7+/-4.8 (P<.05) in moderate smokers, a
12 erformed for lung cancer screening in older, heavy smokers, a simple visual assessment of CAC can be
14 ngs of this pilot study demonstrated that in heavy smokers, adjunctive systemic AZM in combination wi
18 h smoking reduction was mostly evident among heavy smokers and for cardiovascular disease mortality.
19 here were no significant differences between heavy smokers and light smokers in EDV (p = 0.52), basal
20 haring of genetic causes of low FEV1 between heavy smokers and never smokers (p=2.29 x 10(-16)) and b
21 soriasis risk was particularly augmented for heavy smokers and persons with longer durations of smoki
23 can be safely performed in healthy subjects, heavy smokers, and those with severe obstructive airflow
25 iod, assuming a 50% stage shift, the current heavy smoker cohort had 553 fewer lung cancer deaths (13
26 ark-colored irises (P < 0.009), and lower in heavy smokers compared to light (P < 0.0045) and never (
27 or lung cancer subtypes, the excess risk for heavy smokers compared with never smokers was higher for
29 w-dose CT data were evaluated in a cohort of heavy smokers consecutively recruited by the Multicentri
30 s from CARET participants (current or former heavy smokers), consisting of 100 patients who subsequen
33 er-smokers from 43 studies, we extracted the heavy smokers (CPD >20) and light smokers (CPD </=10) wi
34 ed to vary by age, with the odds ratio among heavy smokers decreasing from 2.8 among 32- to 44-year-o
37 osed to more nicotine per cigarette than are heavy smokers due to more frequent, intensive puffing.
39 in the screening of older current and former heavy smokers for early detection of lung cancer, which
40 actions of TCF4 genotype and smoking status; heavy smokers (FTND score >/= 4) showed stronger gene ef
41 ntrols, with smoking exposure categorized as heavy smoker (>/=10 pack-years) versus never smoker/<10
43 The estimated odds ratio among the 350 heavy smokers (> or = 50 pack-years) was 1.41 (95% confi
44 light smokers (< or =1 pack/week) and eight heavy smokers (> or =1 pack/day), and their sera were ad
45 ancer, respectively, and only 39% and 49% of heavy smokers (> or =40 cigarettes per day) acknowledged
46 ons were defined: never smokers (0 pack-yr), heavy smokers (>/= 13 pack-yr) and light smokers (< 13 p
48 not change above 20 CPD and was 36% lower in heavy smokers (>/=20 CPD) than in lighter smokers (<20 C
49 mortality similar to that of quitters among heavy smokers (>/=21 cigarettes/day) who reduced their s
50 r, former, light (</=10 cigarettes/day), and heavy smokers (>10 cigarettes/day) according to self-rep
51 in plasma (V(T)/f(P)) in 10 nonsmokers and 6 heavy smokers (>14 cigarettes/d; abstinent for >36 h).
52 48.3%, P<0.05), but recipients of lungs from heavy smokers (>40 pack-years smoking history) exhibited
55 [RRR] = 2.1) and severe CAL (RRR = 3.4), and heavy smokers had a higher risk for moderate (RRR = 3.0)
56 ls measured within 5 years before diagnosis, heavy smokers had a multivariable-adjusted HR for death
57 From the neutral to the cigarette cue scan, heavy smokers had greater increases than nonsmoking cont
58 y (aHR, 1.08; 95% CI, 0.96-1.20), but former heavy smokers had higher risk for both HF (aHR, 1.45; 95
59 , when compared with current smokers, former heavy smokers had lower risk of death (aHR, 0.64; 95% CI
61 y cigarette consumption at the initial exam: heavy smokers (HS) > or = 20 cigarettes/day (n = 31); li
62 smokers were significantly more likely to be heavy smokers in adulthood (odds ratio [OR] = 1.45; 95%
65 l for heavy smokers can allow us to stratify heavy smokers into subgroups with distinct risks, which,
66 tory volume in 1 s (FEV1) distribution among heavy smokers (mean 35 pack-years) and never smokers.
68 .96, 1.93) when compared with that for CARET heavy smokers not exposed to asbestos, after adjusting f
69 0.7-0.9]), with an adjusted hazard ratio for heavy smokers of 0.5 (95% CI=0.3-0.9) compared to that o
70 eriodontal health and reducing tooth loss in heavy smokers of cigarettes, cigars, and pipes with peri
71 ment therapy may be particularly helpful for heavy smokers or smokers who have experienced multiple f
74 Similarly, this risk was more evident in heavy smokers (OR, 2.55; 95% CI, 1.61-4.03) than in ligh
75 okers, OR = 1.84 (95% CI: 1.2, 2.9); and for heavy smokers, OR = 1.85 (95% CI: 1.0, 3.5), relative to
76 icantly lower in light smokers compared with heavy smokers (p < 0.006 and p < 0.004, respectively).
79 n low-dose computed tomography scans in male heavy smokers participating in a lung cancer screening s
81 n (healthy volunteers, patients with asthma, heavy smokers, patients undergoing lung volume reduction
82 moderate smokers (RR = 0.92; 0.72-1.16) and heavy smokers (RR = 0.95; 0.74-1.24), and did not change
85 s were most pronounced in non-smokers, while heavy smokers showed reduced levels of IL-1alpha protein
86 udies, especially for adenocarcinoma and for heavy smokers, suggesting that more emphasis should be p
87 ioeconomic status (OR = 4.5), in moderate or heavy smokers than nonsmokers (OR = 3.1), and in subject
89 less likely than younger, more educated, and heavy smokers to perceive an increased personal risk of
90 ST randomized 53,454 older current or former heavy smokers to receive LDCT or chest radiography (CXR)
92 5.18, respectively, for light, moderate, or heavy smokers, whereas among the individuals from famili
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