ライフサイエンスコーパス: helplessness

1 and inescapable stress (i.e., struggling and helplessness).

2 inescapable electric shock in rats (learned helplessness).

3 ing (NSF), social defeat stress, and learned helplessness.

4 cient to convert the resilient behavior into helplessness.

5 ell function, reduced Th17-dependent learned helplessness.

6 le urine sniffing in mice that had developed helplessness.

7 aradigms for anhedonia, despair, and learned helplessness.

8 associated with greater levels of perceived helplessness.

9 ronic unpredictable stress (CUS) and learned helplessness.

10 oping resources, resulting in depression and helplessness.

11 ion of escape deficits in a model of learned helplessness.

12 res assessing disability, pain, fatigue, and helplessness.

13 iving, marked pain and fatigue, and moderate helplessness.

14 mental functioning, poor social support, and helplessness.

15 these neurons was decreased in mice showing helplessness, a behavioral state that is thought to rese

16 synaptic potentiation was linked to learned helplessness, a depression-like behavior, synaptic weake

17 ely bred line of rats susceptible to learned helplessness, a model of depression, presents an opportu

18 he transcription factor DeltaFosB on learned helplessness, an animal model of affective disorder wher

19 to social aversion, "anhedonia," and learned helplessness and causes impaired glucocorticoid-mediated

20 se brain Th17 cells were elevated by learned helplessness and chronic restraint stress, two common de

21 gulation as a proximate mechanism in learned helplessness and conservation-withdrawal.

22 Attention to the patient's level of pain and helplessness and duration of the visit may limit reports

23 wishes and values, counteracting surrogates' helplessness and ending the uncertainty and suffering.

24 ed in the hippocampus after both the learned helplessness and forced swim test (FST) paradigms.

25 at 5 years in women with high scores on the helplessness and hopelessness category of the MAC scale

26 such as a fighting spirit or an attitude of helplessness and hopelessness toward the disease, has be

27 ication of pain-related stimuli, feelings of helplessness, and a generally pessimistic orientation, t

28 , absence of HLA-DRB1*0301, higher levels of helplessness, and abnormal illness-related behaviors.

29 Pain, helplessness, and depression were the strongest predicto

30 es to regain control, counteract feelings of helplessness, and end their empathic suffering.

31 items, as well as scales for pain, fatigue, helplessness, and global health status on a 2-page quest

32 of interest, and feelings of worthlessness, helplessness, and hopelessness, in the desipramine-mazin

33 re no vehicle-treated mice developed learned helplessness, and impaired novelty suppressed feeding an

34 Animals underwent learned helplessness, and subsequently immobility time was score

35 ion of SGK1 in the rat medial PFC results in helplessness- and anhedonic-like behaviors in rodent mod

36 cial support, illness-related behaviors, and helplessness), as obtained at enrollment into the study,

37 After helplessness assessment, we performed in vivo electrophy

38 contrast, fewer BAG1+/- mice recovered from helplessness behavior compared with their WT controls.

39 d had higher spontaneous recovery rates from helplessness behavior compared with WT mice.

40 To evaluate learned helplessness behavior, we used an active avoidance task

41 , but did not affect stress sensitization or helplessness behavior.

42 d unpredictable stress and increased learned helplessness behavior.

43 SCN circadian rhythms is sufficient to cause helplessness, behavioral despair, and anxiety-like behav

44 -like behaviors induced by CUS, and reversed helplessness behaviors.

45 lizing symptoms (e.g., depression), and more helplessness behaviors.

46 areas that control the expression of learned helplessness behaviors.

47 tic potentiation correlates with an animal's helplessness behaviour and is due to an enhanced presyna

48 slices and can significantly reduce learned helplessness behaviour in rats.

49 l fatigue was correlated with depression and helplessness, but the model predicted only 54% of the va

50 oups were studied: (1) rats bred for learned helplessness (cLH); (2) rats resistant to learned helple

51 essness (cLH); (2) rats resistant to learned helplessness (cNLH); and (3) control Sprague Dawley rats

52 , lack of control, anxiety, feeling blocked, helplessness, concern about "supplies," depression, and

53 ptor activation in the frontal cortex in the helplessness effect.

54 n agreement with previously reported learned-helplessness effects.

55 owed antidepressant-like behavior in learned helplessness, forced-swim (FST) and tail suspension para

56 For 5-year event-free survival a high helplessness/hopelessness score has a moderate but detri

57 l production exhibited resistance to learned helplessness, identifying modulation of RORgammaT as a p

58 Th17 cell administration promoted learned helplessness in 89% of mice in a paradigm where no vehic

59 le anagrams have been used to induce learned helplessness in humans, this finding may provide an init

60 Inescapable stress can induce learned helplessness in many species of animals.

61 factors contributed to surrogates' sense of helplessness in the ICU.

62 Additionally, Arthritis Helplessness Index (P = 0.02), Arthritis Impact Measurem

63 sively activated by designer drug), promoted helplessness, indicating that activation of these neuron

64 pecifically: struggling, aggression, learned helplessness, inhibitory avoidance, and escape behavior.

65 Learned helplessness is a phenomenon which has some behavioral a

66 behavioral models of depression, the learned helplessness (LH) and forced swim test (FST) paradigms.

67 quences of uncontrollable stress, or learned helplessness (LH) behaviors, are thought to involve hype

68 us (DRN) are implicated in mediating learned helplessness (LH) behaviors, such as poor escape respond

69 the response of brain FoxO3a in the learned helplessness (LH) paradigm and tested signaling pathways

70 moter, showed protection against the learned helplessness (LH) paradigm, an animal model to test stre

71 tail suspension test (TST), and the learned helplessness (LH) paradigm-as well as in the female urin

72 nic social defeat (SD) stress model, learned helplessness (LH), and a chronic corticosterone (CORT) m

73 hereas yoked rats failed to learn (a learned helplessness-like effect).

74 In learned helplessness, mice were exposed to a shuttle box for 4 d

75 -dynorphin influence behavior in the learned helplessness model and suggest that this signaling casca

76 Hb neurons contributes to the rodent learned helplessness model of depression.

77 Here we show that in two learned helplessness models of depression, excitatory synapses o

78 common among patients with greater baseline helplessness (odds ratio [OR] 1.9, 95% confidence interv

79 nt by showing across primate genera that the helplessness of infants is a particularly strong predict

80 rom prior beliefs about the loss of agency ('helplessness'), or from an inability to inhibit the ment

81 sant-like behavioural effects in the learned helplessness paradigm and regulates molecular events imp

82 of this transcription factor in the learned helplessness paradigm, a behavioral model of depression.

83 e mediator of escape deficits in the learned helplessness paradigm, suggesting that neuronal overacti

84 In the learned helplessness paradigm, the SCN-Bmal1-KD mice were slower

85 e following inescapable shock in the learned helplessness paradigm.

86 ike immunoreactivity (FLI) using the learned helplessness paradigm.

87 Here we used the learned helplessness procedure in mice to examine the role of th

88 l responses to stress induced by the learned helplessness procedure, in which animals are subjected t

89 s on health status (functional status, pain, helplessness, psychological status) and visit duration.

90 l (illness behavior, social support, learned helplessness, smoking, drinking), clinical, serologic (a

91 essive ratio responding to food, and learned helplessness task were normal, such avolition-like behav

92 olt, novelty induced hypophagia, and learned helplessness tests in rats without exhibiting substance

93 velty-evoked hyperactivity and stress-evoked helplessness, to map regional brain metabolic effects ca

94 y of the effects of fluoxetine after learned helplessness training.

95 is pattern of brain metabolism suggests that helplessness vulnerability is linked to altered function

96 In foot-shock groups, learned helplessness was more robust in heterozygotes than in WT

97 on to be explored, 2) explore the problem of helplessness while monitoring their own countertransfere

98 Evidence of hopelessness, helplessness, worthlessness, guilt, and suicidal ideatio