ライフサイエンスコーパス: hematogenously

1 pread occurs via both lymphatic channels and hematogenously.

2 her opportunistic pathogens that disseminate hematogenously.

3 l adhesion molecule A (JAM-A) to disseminate hematogenously.

4 As it disseminates hematogenously and invades a wide range of tissues, T. p

5 r propensity for the bacteria to disseminate hematogenously, and a strong splenic T cell cytokine res

6 that extrapulmonary tissues may be infected hematogenously by RSV and harbor this virus allowing the

7 sity for treponemes to migrate through skin, hematogenously disseminate, and invade targeted tissues.

8 IFN-gamma augments the host defense against hematogenously disseminated candidal infections.

9 sphosphate, which was shown to contribute to hematogenously disseminated candidiasis (DC) after sever

10 Using RNA-seq analysis of a mouse model of hematogenously disseminated candidiasis (HDC) and episod

11 t Hyr1p significantly protected mice against hematogenously disseminated candidiasis (P = .001).

12 e findings indicate that the murine model of hematogenously disseminated candidiasis accurately recap

13 n (SSK21) was avirulent in a murine model of hematogenously disseminated candidiasis and less able to

14 required for virulence in a murine model of hematogenously disseminated candidiasis and that strains

15 Mice with hematogenously disseminated candidiasis died of progress

16 The murine model of hematogenously disseminated candidiasis is the standard

17 l adults, but its role in host resistance to hematogenously disseminated candidiasis is unknown.

18 During hematogenously disseminated candidiasis, bloodborne Cand

19 or caPLB1, as assessed in a murine model for hematogenously disseminated candidiasis, was significant

20 The incidence of life-threatening, hematogenously disseminated candidiasis, which is predom

21 significant attenuation in a mouse model of hematogenously disseminated candidiasis, while the doubl

22 t system is important for host resistance to hematogenously disseminated candidiasis.

23 ation of MBL increased resistance of mice to hematogenously disseminated candidiasis.

24 ion (HR), was evaluated in a murine model of hematogenously disseminated candidiasis.

25 identify some virulence factors relevant to hematogenously disseminated candidiasis.

26 e of infections, from superficial mucosal to hematogenously disseminated candidiasis.

27 plenic immune responses in a murine model of hematogenously disseminated candidiasis.

28 aplb1-2 mutant were indistinguishable during hematogenously disseminated candidiasis.

29 a successful infection in a murine model of hematogenously disseminated candidiasis.

30 irulence of C. albicans in a murine model of hematogenously disseminated candidiasis.

31 During hematogenously disseminated infection, blood-borne Candi

32 culature to invade the deep tissues during a hematogenously disseminated infection.

33 rmation is critical for C. albicans to cause hematogenously disseminated infections.

34 Hematogenously disseminated M. tuberculosis infection wa

35 uction in virulence using the mouse model of hematogenously disseminated systemic candidiasis.

36 ugh the skin, the organism causes disease by hematogenously disseminating to multiple organs.

37 e been speculated that platelets may protect hematogenously disseminating tumor cells from NK-depende

38 -borne disease Lyme borreliosis, disseminate hematogenously from the tick bite site to the joints, th

39 Animals hematogenously infected with the C. albicans clinical is

40 The ability of tumor cells to metastasize hematogenously is regulated by their interactions with p

41 4T1 hematogenously metastasizes to the lung, liver, bone, an

42 gy of ovarian carcinoma differs from that of hematogenously metastasizing tumors because ovarian canc

43 may represent TNF-alpha protein released by hematogenously recruited macrophages.

44 o form locomotory organelles and spread, and hematogenously seed distant organs.

45 ens gaining access to surgical wounds either hematogenously, through drains, or through slowly healin

46 g it to cause disease locally or disseminate hematogenously throughout the body.

47 vidence that F. nucleatum may be transmitted hematogenously to the placenta and cause adverse pregnan

48 ection, only the tPMP(r) strain disseminated hematogenously to the spleen (15 of 15 rabbits) versus 0