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1 ted genes, commonly occurs among aging human hematopoietic stem cells.
2 which facilitated their direct contact with hematopoietic stem cells.
3 graft-versus-host disease after transfer of hematopoietic stem cells.
4 rgets the bone marrow compartment, including hematopoietic stem cells.
5 stim to facilitate collection of circulating hematopoietic stem cells.
6 s to impairment of long-term self-renewal of hematopoietic stem cells.
7 tions within a seemingly homogenous group of hematopoietic stem cells.
8 p-dioxin on long-term self-renewal of murine hematopoietic stem cells.
9 The platform was used to deliver single hematopoietic stem cells.
10 ent, possibly directly stemming from infused hematopoietic stem cells.
11 drift acting on a small population of active hematopoietic stem cells.
12 ogical effects of Runx1 in the generation of hematopoietic stem cells.
13 l factor transgene were engrafted with human hematopoietic stem cells.
14 ntisickling beta-globin gene into autologous hematopoietic stem cells.
17 he percentage of stromal cells, macrophages, hematopoietic stem cells and fibroadipogenic cells in th
18 ow progenitor analysis revealed depletion of hematopoietic stem cells and multipotent progenitors acr
19 nd to germ-free mice restored the numbers of hematopoietic stem cells and precursors in bone marrow a
20 tophagy on leukemic transformation of normal hematopoietic stem cells and summarizes its role on leuk
21 escence/cycling balance of murine short-term hematopoietic stem cells and their differentiation into
22 ( + ) mice, which conditionally lack ET-1 in hematopoietic stem cells and vascular endothelial cells,
23 ro, decreased expression of Hox genes in the hematopoietic stem cells, and decreased MLL occupancy at
25 o identify functional long-term repopulating hematopoietic stem cells, and has been detected in certa
26 vity of innate immune cells, mesenchymal and hematopoietic stem cells, and insulin-releasing pancreat
27 axis is involved in the interaction between hematopoietic stem cells (as well as hematologic and sol
31 -binding protein-2 (GATA-2) was expressed in hematopoietic stem cells, but not in NK-cell progenitors
35 r the first time that optimized protocols of hematopoietic stem cell collection from FA patients, fol
36 ansfer genes specifically into the primitive hematopoietic stem cell compartment through the utilizat
38 l-plastin; and disrupts the specification of hematopoietic stem cells (definitive hematopoiesis), as
39 dited CD34+ cells are long-term repopulating hematopoietic stem cells, demonstrating the potential of
40 iated with the rise of founding clones or of hematopoietic stem cells devoid of recurrent mutations.
42 oth LANCL-2 and GRP78 is up-regulated during hematopoietic stem cell differentiation into mature mega
43 se group of bone marrow disorders and clonal hematopoietic stem cell disorders characterized by abnor
45 upply in peripheral blood from Krt7-positive hematopoietic stem cells during unperturbed homeostatic
46 gut, brain, kidney and lungs of human CD34+ hematopoietic stem cell engrafted virus-infected NOD.Cg-
50 uced vascular permeability, preserved normal hematopoietic stem cell function, and improved treatment
51 in BM-derived cells precipitates defects in hematopoietic stem cell function, contributing to extram
52 lobin expression, is required for immune and hematopoietic stem cell functions and brain development.
55 factor expressed by myeloid-biased long term-hematopoietic stem cells, guides the lineage specificati
56 are present and expressed within a leukemic hematopoietic stem cell has engendered some controversy.
57 ently add new copies of the relevant gene to hematopoietic stem cells have led to safe and effective
59 oproliferative neoplasms (MPNs) arise in the hematopoietic stem cell (HSC) compartment as a result of
60 e tracked back to the phenotypically defined hematopoietic stem cell (HSC) compartment in all investi
63 (GFs) that together promote quiescent human hematopoietic stem cell (HSC) expansion ex vivo have bee
68 linically to treat leukopenia and to enforce hematopoietic stem cell (HSC) mobilization to the periph
69 ulations that express characteristics of the hematopoietic stem cell (HSC) niche contain precursors t
72 le bone marrow cells that regulate different hematopoietic stem cell (HSC) properties such as prolife
73 The role of osteolineage cells in regulating hematopoietic stem cell (HSC) regeneration following mye
75 eptor (EPCR/CD201/PROCR) when exposed to the hematopoietic stem cell (HSC) self-renewal agonist UM171
76 ne marrow microenvironment (niche) regulates hematopoietic stem cell (HSC) self-renewal and commitmen
77 em declines with age, resulting in decreased hematopoietic stem cell (HSC) self-renewal capacity, mye
78 ietic-specific loss of Tet2 induces aberrant hematopoietic stem cell (HSC) self-renewal/differentiati
80 n precedes, and is necessary for, successful hematopoietic stem cell (HSC) transplantation, the only
82 dent transformation in MLL-CSCs derived from hematopoietic stem cell (HSC)-enriched LSK population bu
83 ch, we identify ZNF521/Zfp521 as a conserved hematopoietic stem cell (HSC)-enriched transcription fac
84 itive and erythromyeloid progenitor waves of hematopoietic stem cell (HSC)-independent hematopoiesis
85 herapy and show BCR-ABL1 positivity in their hematopoietic stem cell (HSC)/progenitor/myeloid compart
88 ease in miR101c, which downregulated Tet1 in hematopoietic stem cells (HSC), resulting in reduced exp
89 ynamically drive mature cell production, and hematopoietic stem cells (HSC), which provide a quiescen
90 erved microRNAs that are highly expressed in hematopoietic stem cells (HSCs) and acute myeloid leukem
92 fects the maintenance and differentiation of hematopoietic stem cells (HSCs) and committed progenitor
93 with its receptor as a means of targeting MF hematopoietic stem cells (HSCs) and hematopoietic progen
95 revious studies have revealed an increase in hematopoietic stem cells (HSCs) and multipotent progenit
96 o investigate the role of DDT in maintaining hematopoietic stem cells (HSCs) and progenitors, we used
97 -2 protein family member highly expressed in hematopoietic stem cells (HSCs) and regulated by growth
100 ormal globin-producing genes into autologous hematopoietic stem cells (HSCs) are in clinical trials f
106 ould be greatly improved if patient-specific hematopoietic stem cells (HSCs) could be generated from
110 erve cancer but instead found that aneuploid hematopoietic stem cells (HSCs) exhibit decreased fitnes
111 nals that enhance the retention or egress of hematopoietic stem cells (HSCs) from bone marrow (BM).
123 r humanized through the engraftment of human hematopoietic stem cells (HSCs) that can lead to human h
124 rom a small population of disease-initiating hematopoietic stem cells (HSCs) that persist and expand
126 s (2016) determine the divisional history of hematopoietic stem cells (HSCs) to be a key player of re
127 we investigated its role in the response of hematopoietic stem cells (HSCs) to liver fibrosis in mic
128 e occurred in parallel with specification of hematopoietic stem cells (HSCs) to the myeloid and lymph
134 xt of hematopoiesis, and specifically within hematopoietic stem cells (HSCs), have not clearly been d
135 or and tightly controls cardinal features of hematopoietic stem cells (HSCs), including self-renewal,
136 is not known if adult LCH or ECD arises from hematopoietic stem cells (HSCs), nor which potential blo
137 ic homeostasis depends on the maintenance of hematopoietic stem cells (HSCs), which are regulated wit
140 attributed to cell-intrinsic alterations in hematopoietic stem cells (HSCs); however, the contributi
142 als evaluating the activity of gene-modified hematopoietic stem cells in conferring resistance to HIV
143 ilar to their healthy counterpart, malignant hematopoietic stem cells in myeloid malignancies, such a
144 , reduced regeneration of leukemic long-term hematopoietic stem cells in secondary transplant recipie
145 al vessels and altered population balance of hematopoietic stem cells in the bone marrow, manifested
146 transferring a GFP reporter gene into adult hematopoietic stem cells in vivo, which are predominantl
151 enance and functional integrity of long-term hematopoietic stem cells (LT-HSCs) is critical for lifel
152 ) results from transformation of a long-term hematopoietic stem cell (LTHSC) by expression of the BCR
153 bility, indicating that Erf is necessary for hematopoietic stem cell maintenance or differentiation.
156 mbination with other cell therapies (such as hematopoietic stem cells or bone marrow-derived MSC or d
157 lines can be efficiently differentiated into hematopoietic stem cell precursors, as well as APC, unde
158 e lesions, resulted in the perinatal loss of hematopoietic stem cells, progressive loss of bone marro
159 such as CD33 and CD123, is not expressed on hematopoietic stem cells providing potential hematopoiet
161 Adoptive transfer of WT bone marrow-derived hematopoietic stem cells reconstituted c-kit but not MAT
163 gents paired with cell populations including hematopoietic stem cells, regulatory T cells, and facili
165 evealed that transformed MEPs gain a partial hematopoietic stem cell signature and largely retain an
166 the blood program during development, adult hematopoietic stem cell survival and quiescence, and ter
167 mice engrafted with human thymus, liver, and hematopoietic stem cells (termed Bone marrow, Liver, Thy
168 able to engraft murine recipients with human hematopoietic stem cells that develop into functional hu
169 to the embryonic development and function of hematopoietic stem cells that form the adult hierarchy,
170 or maintaining the replicative quiescence of hematopoietic stem cells throughout life by limiting the
171 istinctive feature of neoplastic B cells and hematopoietic stem cells, thus identifying ILT3 as a sel
172 , Wu et al. use genetic barcoding of macaque hematopoietic stem cells to demonstrate that, after tran
173 to the peripheral immune system that biases hematopoietic stem cells to differentiate into a glucoco
174 iew highlights recent advances in the use of hematopoietic stem cells to facilitate genetic screening
175 immunogenicity results of MVA in allogeneic hematopoietic stem cell transplant (HCT) recipients and
176 is widely used as a surveillance method for hematopoietic stem cell transplant (HCT) recipients.
179 (CMV) retinitis in the pediatric allogeneic hematopoietic stem cell transplant (HSCT) population is
180 ratory virus detected in >/=9% of allogeneic hematopoietic stem cell transplant (HSCT) recipients, in
185 versus-host disease (cGVHD) after allogeneic hematopoietic stem cell transplant reflects a complex im
187 representing 664 admissions for induction or hematopoietic stem-cell transplant (HSCT) from 2006 to 2
188 ents receive myeloablative chemotherapy with hematopoietic stem-cell transplant followed by adjuvant
189 ses can eradicate lymphomas after allogeneic hematopoietic stem cell transplantation (AHSCT), but can
190 -versus-host disease (GVHD) after allogeneic hematopoietic stem cell transplantation (allo-HCT) by su
191 intestinal toxemia botulism in an allogeneic hematopoietic stem cell transplantation (allo-HCT) recip
192 have been successfully treated by allogeneic hematopoietic stem cell transplantation (Allo-HSCT) in c
194 ent complication in recipients of allogeneic hematopoietic stem cell transplantation (allo-HSCT), who
195 of morbidity and mortality after allogeneic hematopoietic stem cell transplantation (allo-HSCT).
196 has not been investigated during allogeneic hematopoietic stem cell transplantation (allo-HSCT).
197 t-versus-leukemia (GVL) effect in allogeneic hematopoietic stem cell transplantation (alloSCT) is pot
198 elapse remains the major cause of allogeneic hematopoietic stem cell transplantation (HCT) failure, a
200 n transplants, virome dynamics in allogeneic hematopoietic stem cell transplantation (HSCT) and enter
202 virus (EBV) infections following allogeneic hematopoietic stem cell transplantation (HSCT) are a maj
204 common and poorly recognized complication of hematopoietic stem cell transplantation (HSCT) associate
205 ry of CMV-specific T-cell immunity following hematopoietic stem cell transplantation (HSCT) could ass
207 ity conditioning has improved survival after hematopoietic stem cell transplantation (HSCT) for hemop
209 anti-HBc)-positive patients after allogeneic hematopoietic stem cell transplantation (HSCT) has not b
210 ion developed recommendations for allogeneic hematopoietic stem cell transplantation (HSCT) in myelod
219 rophylaxis has revolutionized haploidentical hematopoietic stem cell transplantation (HSCT), allowing
220 is a major cause of illness and death after hematopoietic stem cell transplantation (HSCT), and upda
221 ely on intensive chemotherapy and allogeneic hematopoietic stem cell transplantation (HSCT), at least
222 t and devastating complication of allogeneic hematopoietic stem cell transplantation (HSCT), posing a
229 in 75 patients (15.6%) undergoing allogeneic hematopoietic stem cell transplantation and 58 patients
230 D) is a notorious complication of allogeneic hematopoietic stem cell transplantation and causes disab
231 been reported after definitive therapy with hematopoietic stem cell transplantation and gene therapy
233 systemic mastocytosis, including allogeneic hematopoietic stem cell transplantation and multikinase
235 outcomes for patients undergoing allogeneic hematopoietic stem cell transplantation continue to impr
236 s in patients treated early after allogeneic hematopoietic stem cell transplantation do not support t
237 ve been successfully treated with allogeneic hematopoietic stem cell transplantation for more than 4
238 ute myeloid leukemia who received allogeneic hematopoietic stem cell transplantation from a homozygou
239 ents with naive repertoires at 9-12 mo after hematopoietic stem cell transplantation had increased di
240 amma (IFN-gamma) therapy is inefficient, and hematopoietic stem cell transplantation has a poor progn
241 The thymus plays a key role post allogeneic hematopoietic stem cell transplantation in the generatio
244 ntrol in severely affected patients for whom hematopoietic stem cell transplantation is not available
246 n of protoporphyrin in the liver, LT without hematopoietic stem cell transplantation leaves the new l
248 signaling assays of 30 primary samples from hematopoietic stem cell transplantation patients with an
251 rome but has not been assessed in allogeneic hematopoietic stem cell transplantation recipients.
253 oxygenation within 240 days after allogeneic hematopoietic stem cell transplantation survived compare
254 may lead to improved KIR-HLA mismatching in hematopoietic stem cell transplantation therapy for leuk
258 yndrome is a frequent complication following hematopoietic stem cell transplantation, dramatically in
259 t-related complications, donor selection for hematopoietic stem cell transplantation, evaluation of c
261 d graft-versus-host disease after allogeneic hematopoietic stem cell transplantation, suggesting norm
262 d with CNS disease and was reduced following hematopoietic stem cell transplantation, which is the on
279 mune suppressants, etoposide, and allogeneic hematopoietic stem cell transplantation; more than 50% o
280 e rates for patients treated with allogeneic hematopoietic stem-cell transplantation (HSCT) will requ
281 subgroups of patients undergoing allogeneic hematopoietic stem-cell transplantation for MDS may info
282 emia (AML) can only be cured when allogeneic hematopoietic stem-cell transplantation induces a graft-
283 neutropenia (FN) in children with cancer and hematopoietic stem-cell transplantation recipients.
284 ven responders (44%) proceeded to allogeneic hematopoietic stem-cell transplantation, including 55% (
285 esidual disease response, rate of allogeneic hematopoietic stem-cell transplantation, relapse-free su
290 gh-throughput integration site analysis in a hematopoietic stem cell-transplanted humanized mouse mod
295 perturbations can enhance fitness of clonal hematopoietic stem cells, which can impact outcome throu
296 Blood cells are derived from a common set of hematopoietic stem cells, which differentiate into more
297 atopoiesis results from somatic mutations in hematopoietic stem cells, which give an advantage to mut
298 e was reproducible in in vitro cultured cDKO-hematopoietic stem cells, which were significantly rescu
299 tachment could provide cancer stem cells and hematopoietic stem cells with a means to cycle from trop
300 d, we show how mRNA nanocarriers can program hematopoietic stem cells with improved self-renewal prop
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