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1 tment to the bone marrow, a key location for hematopoietic tumors.
2 the highly sensitive and readily accessible hematopoietic tumors.
3 helial carcinomas, soft tissue sarcomas, and hematopoietic tumors.
4 g pathway play an important role in lymphoid/hematopoietic tumors.
5 elial cancers, but has not been described in hematopoietic tumors.
6 in the cell lines derived from solid as from hematopoietic tumors.
7 earrangements are found in a large number of hematopoietic tumors.
8 tatic tumor growth in a variety of solid and hematopoietic tumors.
9 ted equivalent immunity against A20 and P815 hematopoietic tumors.
10 ontaneous aneuploidy and a predisposition to hematopoietic tumors.
11 pectral karyotypic analysis that MYC-induced hematopoietic tumors are highly genetically complex and
13 three genes are almost universal in lymphoid/hematopoietic tumors but the patterns of gene methylated
17 ion of mRNA and protein for VEGF in 12 human hematopoietic tumor cell lines, representing multiple li
19 f TME-mediated drug resistance that protects hematopoietic tumor cells from the initial effect of div
23 tions reveal that Cdk4 and Cdk6 cooperate in hematopoietic tumor development and suggest a role for C
25 d, or constitutively activated in many human hematopoietic tumors; however, the molecular mechanisms
27 sufficient to induce sustained regression of hematopoietic tumors in transgenic mice, except in tumor
28 ene is aberrantly methylated in 86% of human hematopoietic tumors, including 8 of 9 pediatric acute l
29 Whereas H-Ras(G12V) elicited papillomas and hematopoietic tumors, K-Ras(G12V) induced lung tumors an
30 We show here in a Drosophila melanogaster hematopoietic tumor model, however, that JAK overactivat
31 ccine approach was assessed in several other hematopoietic tumor models and was also therapeutically
33 Retroviral insertional mutagenesis in mouse hematopoietic tumors provides a potent cancer gene disco
34 Retroviral insertional mutagenesis in mouse hematopoietic tumors provides a powerful cancer gene dis
36 ubiquitin ligase Fbw7, a recently identified hematopoietic tumor suppressor that can target for degra
38 itutive activation of Rel/NF-kappaB in human hematopoietic tumors, the v-Rel oncoprotein induces aggr
39 because bfl-1 is up-regulated in many human hematopoietic tumors, this finding suggests that strateg
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