ライフサイエンスコーパス: hematuria

1 ons, and urologic procedures to manage gross hematuria).

2 nephrosis requiring nephrostomy due to gross hematuria).

3 s with persistent, asymptomatic, microscopic hematuria.

4 gic procedure to investigate or manage gross hematuria.

5 rtion of patients with persistent glomerular hematuria.

6 osis and management of persistent glomerular hematuria.

7 ons typically cause isolated, nonprogressive hematuria.

8 lify the imaging evaluation of patients with hematuria.

9 l subjects and in four of five patients with hematuria.

10 the characteristic presentation of painless hematuria.

11 cluded proteinuria, renal insufficiency, and hematuria.

12 urinary tract infections, renal stones, and hematuria.

13 isodes of microscopic or gross nonglomerular hematuria.

14 hematuria and those with negative or minimal hematuria.

15 ancer detection in the initial evaluation of hematuria.

16 ions for clinicians evaluating patients with hematuria.

17 oscopic hematuria about any history of gross hematuria.

18 ant proportions and may present with massive hematuria.

19 families, respectively) and benign familial hematuria.

20 ing, or infected stone causing pain or gross hematuria.

21 department due to right-sided flank pain and hematuria.

22 pack-year history of smoking developed gross hematuria.

23 me, 68% had renal insufficiency, and 77% had hematuria.

24 nclude Alport's syndrome and benign familial hematurias.

25 =3 bleeding events in >/=2% of patients were hematuria (2%) and subdural hematoma (2%).

26 on (UTI), 8; gross hematuria, 5; microscopic hematuria, 2; dysuria without infection, 6; difficulty v

27 loped renal-related complications, including hematuria (24 patients), urinoma (15 patients), urinary

28 left abdomen for one month and an episode of hematuria 3 days earlier accompanied by an attack of acu

29 ethritis and extravasation (7%), and chronic hematuria (3%).

30 7), bacterial urinary tract infection (3/7), hematuria (3/7), cytomegalovirus infection (3/7), and im

31 turia than patients with minimal or negative hematuria (30.4% and 37.0% versus 10.6% and 15.2%, respe

32 were urinary tract infection (UTI), 8; gross hematuria, 5; microscopic hematuria, 2; dysuria without

33 redictors were 15.6% (15 of 96 patients) for hematuria, 6.0% (13 of 216) for lower rib fractures, 7.6

34 Sixty of the 73 patients with nonglomerular hematuria (82%) had cystoscopy at the NIH.

35 e gut, elevated serum IgA levels, and severe hematuria, a hallmark of IgAN.

36 ecifically ask all patients with microscopic hematuria about any history of gross hematuria.

37 adder stones, blockage, false passage, gross hematuria, accidental removal, urine leakage, or urethra

38 ric hematomas accounting for 13.4% and gross hematuria accounting for 2.7%.

39 ominal hemorrhage and one case each of gross hematuria, allograft pancreatitis, and severe pain requi

40 ography was higher in patients evaluated for hematuria alone (98%, 589 of 603).

41 ith SCI, 13.5% (CI, 3.4% to 21.9%) had gross hematuria and 1.0% (CI, 0.0% to 5.0%) developed bladder

42 cystoscopy was performed in 13 patients with hematuria and abnormal findings at conventional cystosco

43 e diagnosed by renal biopsy in patients with hematuria and adenoviruria.

44 of lymphoma was the sudden onset of painless hematuria and ARF is described.

45 no significant comorbidities presented with hematuria and dysuria.

46 Hematuria and fracture of the lower ribs, lumbar spine,

47 opathies invariably present with microscopic hematuria and frequently progress to proteinuria and CKD

48 cycline-treated C57BL/6 x SJL mice developed hematuria and glomerulonephritis on the MR and standard

49 Children with microscopic hematuria and no evidence of multiple system trauma seem

50 = 13; 32.5%), pollakiuria (n = 11; 28%), and hematuria and nocturia (n = 10 each; 25%).

51 At 1,000 mg, three patients developed hematuria and one had a skin reaction resembling grade 3

52 ry clinical manifestations of this disorder, hematuria and papillary necrosis, are discussed.

53 s (questions and answers) on 5 urology CPGs (hematuria and priapism [HP]; staghorn calculi, infertili

54 re age 1 year, have persistent hypertension, hematuria and proteinuria (sometimes in the nephrotic ra

55 The majority of patients had hematuria and proteinuria at time of recurrence.

56 Children with combined hematuria and proteinuria had a higher prevalence of sig

57 cal examination should indicate the cause of hematuria and proteinuria in most cases.

58 The review will discuss the major causes of hematuria and proteinuria in the pediatric population, a

59 The presence of hematuria and proteinuria together significantly increas

60 ticle describes the clinical epidemiology of hematuria and the current state of practice and science

61 classified patients as those with persistent hematuria and those with negative or minimal hematuria.

62 h sickle cell trait who presented with gross hematuria and was subsequently found to have renal papil

63 high-risk group according to the presence of hematuria and/or axial fracture on radiographs.

64 d with 86-24 Stm(r) or B2F1 Stm(r) developed hematuria and/or histological damage to glomeruli or thr

65 l pelvis with calculus increases the risk of hematuria and/or hydronephrosis, presenting with colicky

66 inal outcome of examination of children with hematuria and/or proteinuria.

67 on of this in cases of otherwise unexplained hematuria), and human immunodeficiency virus-associated

68 et of hypertension, early and frequent gross hematuria, and among women, three or more pregnancies.

69 ondary to a combination of thrombocytopenia, hematuria, and anemia that required transfusion.

70 body levels, the presence of proteinuria and hematuria, and by histopathologic analysis of kidney tis

71 Prodromal symptoms included fever, hematuria, and flank pain.

72 a history of acute urinary retention, gross hematuria, and left flank pain for 2 days.

73 sent with normal renal function, microscopic hematuria, and minimal or no proteinuria is not well des

74 uding hearing loss, intellectual disability, hematuria, and orofacial clefting.

75 e resulted in the resolution of proteinuria, hematuria, and PKD.

76 complexes resulting in kidney inflammation, hematuria, and proteinuria, mice expressing IgA1 only di

77 Therefore, the prevalence of proteinuria, hematuria, and reduced GFR in the Australian adult popul

78 ociates with impaired urinary concentration, hematuria, and renal papillary necrosis, but its prevale

79 ol level, urinalysis proteinuria, urinalysis hematuria, and serum creatinine level.

80 Leukocytes, hematuria, and urobilinogen concentrations in urine were

81 cities were one episode each of esophagitis, hematuria, and vomiting.

82 ian adult population has either proteinuria, hematuria, and/or reduced GFR, indicating the presence o

83 teinuria; age, gender, and hypertension with hematuria; and age, gender, and hypertension with reduce

84 n causes an astonishing breadth of sequelae: hematuria, anemia, dysuria, stunting, uremia, bladder ca

85 ere thrombocytopenia plus head trauma and/or hematuria appeared to be at particularly high risk of IC

86 forms of urolithiasis, acute flank pain and hematuria are the typical symptoms of indinavir urolithi

87 sappeared in all 5 patients with significant hematuria at baseline.

88 ion rate, or the presence of hypertension or hematuria at the time of diagnosis.

89 Similarly, those who had an episode of gross hematuria before age 30 had a worse renal outcome than t

90 tis experienced complete resolution of gross hematuria by week 6.

91 Hematuria (by dipstick only) occurred in 25 (41%) of 61

92 Moreover, hematuria consistently decreased after treatment.

93 72 patients who had never had nonglomerular hematuria developed bladder cancer.

94 After hematuria disappearance, which occurred in 46% of the pa

95 1 month after the last dose (P < 0.005), and hematuria disappeared in all 5 patients with significant

96 or renal disorders involving proteinuria and hematuria due to podocytopathy and/or segmental splittin

97 CE 6: Clinicians should pursue evaluation of hematuria even if the patient is receiving antiplatelet

98 urologic evaluation in all adults with gross hematuria, even if self-limited.

99 Hematuria following radiation therapy for prostate cance

100 9-year-old patient was admitted with massive hematuria following transurethral resection of prostate

101 calculi in his urine after the occurrence of hematuria, following which pain decreased in intensity.

102 laints of breathlessness, abdominal pain and hematuria for the last 6 months.

103 Recognition that glomerular hematuria frequently has a genetic basis is important fo

104 veraged proteinuria >0.75 g/d and persistent hematuria had significantly worse renal survival than th

105 unodepletion before TGN markedly exacerbated hematuria (hemorrhage), inflammation, and injury, wherea

106 merular defects), microscopic urinalysis for hematuria (hemorrhagic cystitis, bladder cancer), ECG (a

107 Its pathologic features include hematuria, high levels of circulating IgA-fibronectin (F

108 ected in 2.4% of cases (95% CI: 1.6%, 3.1%), hematuria in 4.6% (95% CI: 3.8%, 5.4%), and reduced GFR

109 arin") increased serum creatinine levels and hematuria in 5/6-nephrectomized rats but not in controls

110 sits and reduces inflammation, fibrosis, and hematuria in a mouse IgAN model, and therefore may be a

111 els in four patients (6%) and transient mild hematuria in one patient (1%).

112 ing in adults with microscopically confirmed hematuria in the absence of some demonstrable benign cau

113 moking history presented with painless gross hematuria in the fall of 2015 and was ultimately diagnos

114 RE ADVICE 1: Clinicians should include gross hematuria in their routine review of systems and specifi

115 Dose-limiting toxicities included hematuria, increased gamma-glutamyltransferase or ALT, i

116 Hematuria is a cardinal symptom in IgA nephropathy, but

117 Asymptomatic, microscopic hematuria is seen in 8-21% of the general population, ha

118 The presence of blood in the urine, or hematuria, is a common finding in clinical practice and

119 In 13 (36%) of 36 patients, hematuria lasted longer than 24 hours but resolved witho

120 mal electrocardiogram findings, proteinuria, hematuria, low hemoglobin level, elevated erythrocyte se

121 ted liver transaminases (LR range, 2.5-5.2), hematuria (LR range, 3.7-4.1), anemia (LR range, 2.2-3.3

122 In conclusion, remission of hematuria may have a significant favorable effect on IgA

123 he high NPV of CT urography in patients with hematuria may obviate cystoscopy in selected patients.

124 more prominent hypertension and microscopic hematuria may provide clues to the presence of HCV-GD, r

125 cations, including urinary tract infections, hematuria, metabolic acidosis, dehydration, and reflux p

126 lated complications defined as pain, visible hematuria, migration, fragmentation, and urinary tract i

127 of hypothetical predictors of missed injury: hematuria (n = 96) or fracture of the sixth through 12th

128 Infection and hematuria need to be ruled out.

129 maging, prior suspected stone episode, gross hematuria, nonobstructing (asymptomatic) stone on imagin

130 Nonglomerular hematuria occurred in 73 of 145 patients treated with cy

131 y fever, myalgias, pruritus, and proteinuria/hematuria, occurred in 83% vs 50% of those receiving tri

132 ling catheter drainage and two patients with hematuria, one of whom required overnight bladder irriga

133 , on further classification by time-averaged hematuria, only those patients with time-averaged protei

134 6 years, range of 18-86 years) evaluated for hematuria or a history of urothelial cancer, who had und

135 Fifty-one patients with hematuria or a suspicious renal mass underwent CT urogra

136 ing a similar protocol, no animals developed hematuria or glomerulonephritis, despite having identica

137 Most children with either isolated hematuria or isolated proteinuria had benign disease pro

138 od urea nitrogen, creatinine, bleeding time, hematuria or proteinuria, and aspartate aminotransferase

139 is indicated by the presence of proteinuria, hematuria, or reduced GFR.

140 (PCPs) for CT evaluation of abdominal pain, hematuria, or weight loss were identified.

141 In patients with abdominal pain, hematuria, or weight loss, leading diagnoses changed aft

142 continued to have asymptomatic, microscopic hematuria over 1 month.

143 ptoms: pain (P < .001), shivers, (P = .020), hematuria (P < .001), hematochezia (P < .001), and hemoe

144 iopsies, with choices limited to none, gross hematuria, perinephric hematoma, and other.

145 e Lama5(neo) allele) results in proteinuria, hematuria, polycystic kidney disease (PKD), and death 3

146 teriorate within 3-6 days, with oliguria and hematuria progressing to anuria, and the kidneys were ex

147 ome is an inherited disease characterized by hematuria, progressive renal failure, hearing loss, and

148 At presentation, hematuria, proteinuria >/=1 g/d, hypertension, and renal

149 ather, homozygous mutant mice had glomerular hematuria, proteinuria, and podocytopathy.

150 2.0 microg/kg; n = 5) to renal failure with hematuria, proteinuria, thrombocytopenia, schistocytosis

151 ignificantly associated with higher rates of hematuria-related complications (including emergency dep

152 To characterize rates of hematuria-related complications among patients taking an

153 median follow-up of 7.3 years, the rates of hematuria-related complications were 123.95 events per 1

154 unexposed to thrombotic agents, the rates of hematuria-related complications were 191.61 events per 1

155 Hematuria-related complications, defined as emergency de

156 Hematuria resolved after insertion of a 3-way urinary ca

157 In two prospective donors, hematuria resolved after treatment for urinary tract inf

158 The hematuria resolved with conservative therapy consisting

159 wn markers, including long-term follow-up of hematuria screening, recent studies in DNA methylation f

160 creatinine ratio (abnormal: >/=0.20 mg/mg); hematuria-spot urine dipstick (abnormal: 1+ or greater)

161 , increases C3 concentrations, and decreases hematuria, suggesting that the drug has immunomodulatory

162 s C), rigors, malaise, lethargy, flank pain, hematuria, suprapubic discomfort, dysuria, and urgent or

163 antly greater among patients with persistent hematuria than patients with minimal or negative hematur

164 One patient developed hematuria that required transfusion but no further inter

165 ultivariable analysis revealed time-averaged hematuria, time-averaged proteinuria, renal function at

166 , and bladder--and thus allows patients with hematuria to be evaluated comprehensively.

167 e), transaminase elevation (one course), and hematuria (two courses).

168 dominal/right upper quadrant gunshots and/or hematuria underwent mandatory CT with intravenous contra

169 the sensitivity or specificity of tests for hematuria, urinary cytology, or other urinary biomarkers

170 th a range of clinical presentations such as hematuria, voiding dysfunction, flank pain, abdominal pa

171 Nonglomerular hematuria was a frequent manifestation of cyclophosphami

172 s showed that only microscopic nonglomerular hematuria was a significant risk factor for the developm

173 Hematuria was common among both diabetics and nondiabeti

174 other relevant studies on the evaluation of hematuria was conducted, with particular emphasis on con

175 Among nondiabetics, however, hematuria was more common among women.

176 uminuria, although the degree of microscopic hematuria was reduced by this intervention.

177 Gross hematuria was reported in six patients (2.8%): two SB (2

178 of 11 placebo recipients, although no gross hematuria was reported.

179 Among diabetics, the crude prevalence of hematuria was similar among men and women.

180 Hematuria was the first tumor marker in a field that has

181 According to the magnitude of time-averaged hematuria, we classified patients as those with persiste

182 Febrile neutropenia, diarrhea, and hematuria were more frequent with C25; peripheral neurop

183 xplained renal dysfunction, proteinuria, and hematuria were retrospectively reviewed.

184 Head trauma and hematuria were the most prominent features associated wi

185 ns included renal insufficiency, microscopic hematuria with active urine sediment, hypertension, and

186 gs: reduced kidney function, proteinuria, or hematuria with other causes excluded (n = 2).

187 ar-old female with TSC presenting as massive hematuria with underlying giant bilateral renal angiomyo

188 patient with HIV who presented with AKI and hematuria without concomitant systemic manifestations.