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1 er two important molecules: nitric oxide and heme oxygenase-1.
2 f pathogenic CD8(+) T cells and induction of heme oxygenase-1.
3 n, the release of heme, and the induction of heme oxygenase-1.
4 l abrogated by pharmacological inhibition of heme oxygenase-1.
5 d on increased expression of the Nrf2 target heme oxygenase-1.
6 ement of NF-erythroid 2-related factor 2 and heme oxygenase-1.
7 anced expression of glucose transporters and heme oxygenase-1.
8 nitric oxide synthase, cyclooxygenase-2, and heme oxygenase-1.
9 ivity of inducible nitric oxide synthase and heme oxygenase-1.
10 + monocytes expressed higher basal levels of heme oxygenase-1.
11 cyte anti-inflammatory heme-degrading enzyme heme oxygenase-1.
12 f Bcl-2 (5.5-folds), Bcl-xl (5.5-folds), and heme oxygenase-1 (4.4-folds); decreased expression of IC
13 ) mice have increased expression/activity of heme oxygenase-1, a phase II antioxidant, and NF (erythr
16 The carboxyhemoglobin level (a measure of heme oxygenase 1 activity) has not been assessed in adul
19 aimed to investigate the mechanisms by which heme oxygenase-1, an anti-inflammatory enzyme, is protec
20 howed that Bach2 transcriptionally represses heme oxygenase-1, an antiapoptotic factor up-regulated i
21 , despite significantly higher expression of heme oxygenase-1, an antioxidant and cytoprotective enzy
23 orresponded with increases in cytoprotective heme oxygenase 1 and IL-10 mRNAs, selective reductions i
24 ential for linked antioxidant protection via heme oxygenase 1 and reduced foam cell formation via liv
25 ranofin displayed synergistic lethality with heme oxygenase-1 and glutamate-cysteine ligase inhibitor
27 e discuss here new insights into the role of heme oxygenase-1 and heme on cardiovascular health, and
28 ontractility rather than passive stretch via heme oxygenase-1 and histone deacetylase signalling.
30 d4 to Nrf2-binding sites on the promoters of heme oxygenase-1 and NADPH quinone oxidoreductase 1.
31 rf2 or DJ-1 attenuated Cu((II))ATSM-mediated heme oxygenase-1 and NADPH quinone oxidoreductase-1 indu
32 into the nucleus to induce transcription of heme oxygenase-1 and other cytoprotective enzymes throug
35 )S increased the expression of antioxidants (heme oxygenase-1 and thioredoxin 1), increased the expre
37 ples for hypoxia-inducible factor 1alpha and heme oxygenase 1, and 4) immunohistochemistry of hippoca
38 ntioxidant systems such as peroxiredoxins-1, heme oxygenase-1, and anti-apoptotic factors, including
39 ed anti-inflammatory factors interleukin-10, heme oxygenase-1, and Hsp70 in macrophages stimulated or
40 ing IL-1ra, IL-10, and PGE(2), but not IL-6, heme oxygenase-1, and NO, attenuated 5-FU-MSC-induced im
42 lation of the oxidative stress response gene heme oxygenase-1, and we demonstrated that NF-kappaB inh
43 evated levels of TNFalpha, reduced levels of heme-oxygenase-1, and display apparent signs of oxidativ
45 arkable cardioprotective effects ascribed to heme oxygenase-1 are best evidenced by its ability to re
47 le to upregulate the atheroprotective enzyme heme oxygenase-1 at the RNA and protein level in respons
48 ntrol animals, and inhibiting the HIF target heme-oxygenase-1 before IR reduced GFR in STN animals.
49 nhancing expression of the IL-10 target gene heme oxygenase-1 by mechanisms dependent on p38 MAPK act
52 H(2)O(2), concomitant with up-regulation of heme oxygenase 1, COX-2, and anti-apoptotic proteins (BC
54 enhances its paracine effects on RIII via a heme oxygenase-1 dependent mechanism, which may help us
56 s, whereas treatment with carbon monoxide, a heme oxygenase-1 enzymatic product, abrogated this effec
57 als treated with tin protoporphyrin (SnPP, a heme oxygenase-1 enzyme inhibitor), even after Ad.Trx1 t
59 ion, hypoxia-inducible factor 1alpha-induced heme oxygenase 1 expression resulting in improved surviv
61 fects; JunB activated whereas JunD repressed heme oxygenase-1 expression in human renal epithelial ce
64 ncreased inducible nitric oxide synthase and heme-oxygenase 1 expression, and increased MDA and plasm
65 ncreased inducible nitric oxide synthase and heme-oxygenase 1 expression, and increased plasma creati
67 ation of the CBS inhibitor, CO, a product of heme oxygenase-1, flip the operating preference of CSE f
68 athione S-transferase (GST) genes as well as heme oxygenase 1 gene (HMOX1) encode enzymes that detoxi
69 med a systematic review and meta-analysis of heme oxygenase 1 gene (HO-1) promoter polymorphisms and
71 nosine thymidine dinucleotide repeats in the heme oxygenase-1 gene promoter in 386 patients with coro
72 nosine thymidine dinucleotide repeats in the heme oxygenase-1 gene promoter is associated with cardio
73 nosine thymidine dinucleotide repeats in the heme oxygenase-1 gene promoter is associated with higher
76 expression of Nrf2-dependent genes, such as heme oxygenase-1, glutamate-cysteine ligase catalytic su
78 f the azide complex of substrate-bound human heme oxygenase 1 (hHO) has been investigated by (1)H NMR
80 adenoassociated virus (rAAV)-encoding human heme oxygenase-1 (hHO-1) in attenuating post-ischemic in
82 the CX3CR1 receptor induced upregulation of heme-oxygenase-1 (HMOX-1), an antioxidant and anti-infla
84 We showed that variation in basal levels of heme oxygenase 1 (HMOX1) contribute to the response to O
85 ed reduced progesterone levels and placental heme oxygenase 1 (Hmox1) expression and increased methyl
87 nscriptional repressor, negatively regulates heme oxygenase 1 (HMOX1), a key cytoprotective enzyme th
88 h regulates the expression of genes encoding heme oxygenase 1 (Hmox1), glutamate-cysteine ligase cata
90 o prevent accumulation, the inducible enzyme heme oxygenase-1 (HMOX1) catalyzes degradation of heme.
92 n (HP), cluster of differentiation (CD) 163, heme oxygenase-1 (HMOX1), and biliverdin reductase A (BL
93 genes increased in the DKO liver, including heme oxygenase-1 (Hmox1), which disrupts complex III and
94 ctivated protein (MAP) kinase, expression of heme oxygenase 1 (HO-1) and cyclooxygenase 2 (COX-2), an
95 ession of heat-shock protein 70 (HSP-70) and heme oxygenase 1 (HO-1) and promoted cell survival after
96 re increases the stability of mRNAs encoding heme oxygenase 1 (HO-1) and TIEG-1 in human and mouse fi
98 ctor erythroid 2-related factor 2 (Nrf2) and heme oxygenase 1 (HO-1) gene proteins in retinal tissues
100 evidence demonstrating an important role for heme oxygenase 1 (HO-1) in mediating the proangiogenic e
102 investigated whether up-regulation of DAF by heme oxygenase 1 (HO-1) is an underlying mechanism by us
104 (hIL-10R) by cmvIL-10 led to upregulation of heme oxygenase 1 (HO-1), an enzyme linked with suppressi
105 ele contained less TNFalpha, MCP-1, and more heme oxygenase 1 (HO-1), and exhibited a higher rate of
106 overexpression of the heme-degrading enzyme, heme oxygenase 1 (HO-1), has been shown to protect mice
107 NAD(P)H:quinone oxidoreductase 1 (NQO1) and heme oxygenase 1 (HO-1), typical chemoprotective gene pr
109 on a key enzyme involved in heme catabolism, heme oxygenase 1 (HO-1), which, ironically, has been poo
114 per-zinc superoxide dismutase (CuZnSOD), and heme oxygenase-1 (HO-1) (antioxidant enzymes) were reduc
115 in 1 (P<0.05), peroxiredoxin 3 (P<0.01), and heme oxygenase-1 (HO-1) (P<0.05), which are up-regulated
118 f2, NAD(P)H quinone oxidoreductase 1 (NQO1), heme oxygenase-1 (HO-1) and a high ratio of Bcl-2/Bax.
119 production of antioxidant enzymes, including heme oxygenase-1 (HO-1) and glutathione peroxidase 1 (Gp
125 NA and protein levels of HIF-dependent genes heme oxygenase-1 (Ho-1) and manganese superoxide dismuta
126 nd to oxidative stress-induced expression of heme oxygenase-1 (HO-1) and NAD(P)H:quinone oxidoreducta
127 cells by the regulations of novel molecules heme oxygenase-1 (HO-1) and programmed death-1 ligand 1
128 An ELISA assay for the Nrf2 target gene heme oxygenase-1 (HO-1) and studies using Nrf2 small int
130 a hypoxia-inducible plasmid expressing both heme oxygenase-1 (HO-1) and the Src homology domain-2 co
132 AC and selected reaction monitoring revealed heme oxygenase-1 (HO-1) as the most significantly up-reg
133 he cytoprotective and antiapoptotic molecule heme oxygenase-1 (HO-1) at the transcriptional level.
142 ermine whether oxidative adduct formation or heme oxygenase-1 (HO-1) expression are altered in retina
143 a, urine, and tissues, which in turn induces heme oxygenase-1 (HO-1) expression in the colonic epithe
145 vitro study showed that adiponectin induced heme oxygenase-1 (HO-1) expression through the peroxisom
147 ase expression, nitric oxide production, and heme oxygenase-1 (HO-1) expression, which was associated
149 al injury, and inhibited RPTC Nrf2, Agt, and heme oxygenase-1 (HO-1) gene expression in Akita Cat tra
157 lective overexpression of the stress protein heme oxygenase-1 (HO-1) in astrocytes of novel GFAP.HMOX
158 the products of the cytoprotective molecule heme oxygenase-1 (HO-1) in cancer cells, has been implic
159 of NO induce the anti-inflammatory effector heme oxygenase-1 (HO-1) in gastric epithelial cells thro
160 of the cytoprotective, heme-degrading enzyme heme oxygenase-1 (HO-1) in neutrophil progenitors in bon
161 e antioxidative and anti-inflammatory enzyme heme oxygenase-1 (HO-1) in the brains of individuals wit
162 ed a hypothesized anti-inflammatory role for heme oxygenase-1 (HO-1) in the development of metabolic
163 uanosine (8OHG), 4-hydroxynonenal (HNE), and heme oxygenase-1 (HO-1) in the pyramidal neurons of the
181 murine models of sickle cell disease (SCD), heme oxygenase-1 (HO-1) is induced in the kidney, an org
187 ion of host cell antioxidant enzymes such as heme oxygenase-1 (HO-1) may be useful therapeutically to
188 The stress-inducible cytoprotective enzyme heme oxygenase-1 (HO-1) may play a critical role in the
191 ukin-8 (IL-8), cyclooxygenase-2 (COX-2), and heme oxygenase-1 (HO-1) mRNA was measured in BEAS-2B cel
192 Treatment with CRA at nontoxic doses induced heme oxygenase-1 (HO-1) mRNA/protein expression and HO-1
193 of inducing the activity of the host enzyme heme oxygenase-1 (HO-1) on hRSV replication and pathogen
195 overexpression of the cytoprotective enzyme heme oxygenase-1 (HO-1) play a critical role in the grow
198 Only overexpression of the gene encoding heme oxygenase-1 (HO-1) significantly correlated with in
200 y amplified in human breast cancers, induced heme oxygenase-1 (HO-1) through Nrf2 transactivation in
202 gation revealed that the cytoprotective gene heme oxygenase-1 (HO-1) was induced in NF-kappaB-inhibit
203 erythroid 2p45-related factor-2 (Nrf2), and heme oxygenase-1 (HO-1) was tested in both in vitro and
204 up-regulation of the cytoprotective protein heme oxygenase-1 (HO-1) which is capable of mitigating a
205 amplification with pNaKtide and induction of heme oxygenase-1 (HO-1) with cobalt protoporphyrin (CoPP
206 d that PRRSV downregulates the expression of heme oxygenase-1 (HO-1), a pivotal cytoprotective enzyme
209 of oxidative stress caused by low levels of heme oxygenase-1 (HO-1), an important cytoprotective mol
211 lglycerol (DAG) kinase alpha (DGKalpha), and heme oxygenase-1 (HO-1), genes with well-characterized P
212 ter antioxidant transcription factor, and of heme oxygenase-1 (HO-1), one of its main target genes, i
213 ome solid tumors and myeloid leukemia cells, heme oxygenase-1 (HO-1), the anti-oxidant, anti-inflamma
216 attention as a master protective sentinel is heme oxygenase-1 (HO-1), the rate-limiting step in the c
218 ported in a human patient and mice that lack heme oxygenase-1 (HO-1), we studied iron distribution an
219 Many cancer cells constitutively express heme oxygenase-1 (HO-1), which catabolizes heme to gener
220 is the up-regulation of the inducible enzyme heme oxygenase-1 (HO-1), which catabolizes heme to gener
222 Intracellular heme levels are regulated by heme oxygenase-1 (HO-1), which catalyzes the degradation
223 ng expression of the stress response protein heme oxygenase-1 (HO-1), which interacts with and thereb
233 is generated by the stress-responsive enzyme heme oxygenase-1 (HO-1, encoded by Hmox1), which is high
235 n, protein levels, and enzymatic activity of heme oxygenase-1 (HO-1, the enzyme that produces CO), in
238 quality control is regulated in part by the heme oxygenase-1 (HO-1; Hmox1) system through the redox-
240 Q61L) mutant induced the anti-oxidant enzyme heme-oxygenase-1 (HO-1) through activation of NRF2.
243 kers (matrix metalloproteinase 1 [MMP-1] and heme oxygenase 1 [HO-1]), and proinflammatory cytokines
244 pha, monocyte chemoattractant-1 [MCP-1], and heme oxygenase-1 [HO-1]) was assessed by chromatin immun
247 ssion of hypoxia-inducible factor 1alpha and heme oxygenase 1 in the hippocampus was increased in the
250 synthase derived) regulates the induction of heme oxygenase-1 in the lung, which in turn plays an imp
251 duced expression of the Nrf2 target protein, heme oxygenase-1 in the skin and protected against UVB-i
252 ge-like cell line RAW264.7, (b) induction of heme oxygenase-1 in these RAW cells, and (c) suppression
254 Cobalt protoporphyrin (CoPP), a well known heme oxygenase 1 inducer, has been used to promote endog
255 tor-kappaB, hypoxia-inducible factor-1alpha, heme oxygenase-1, inducible nitric oxide synthase, B-cel
259 vivo and IRAK-M(-/-) AECs in vitro with the heme oxygenase-1 inhibitor, tin protoporphyrin, substant
260 bsence of inducible nitric oxide synthase or heme oxygenase-1 inhibitors (1400W or tin protoporphyrin
263 ction, by overcoming its negative regulator, heme oxygenase-1, is a key underlying mechanism responsi
264 a 7-day doxycycline treatment sustained high heme oxygenase-1 levels during the entire period of hypo
267 itochondrial superoxide dismutase (SOD), and heme oxygenase-1, mucosal receptors such as the Toll-lik
268 e regulation of key Nrf2 target genes (i.e., heme oxygenase-1, NAD(P)H dehydrogenase, quinone 1, glut
269 ificantly upregulates Nrf2-responsive genes, heme oxygenase-1, NAD(P)H quinone oxidoreductase 1, and
270 most robustly increased genes and proteins, heme oxygenase 1, NADPH-quinone oxidoreductase 1, and gr
271 n and expression of the antioxidant proteins heme oxygenase-1, NADPH quinone oxidoreductase 1, and gl
272 such as superoxide dismutase, nitric oxide, heme oxygenase-1, neutrophil infiltration, cysteamine, m
273 -dependent gene and protein markers, such as heme oxygenase-1, occurred, whereas Nrf2-deficient fibro
275 es that impair liver regeneration, including heme oxygenase-1, programmed cell death 4, and the cycli
276 al E box abolished hemin- and cadmium-driven heme oxygenase-1 promoter activation, suggesting that th
277 4.0, -7.2, and -9.2 kb, respectively, of the heme oxygenase-1 promoter in addition to one proximal re
281 itric oxide synthase inhibitor 1400W reduced heme oxygenase-1 protein expression, and lung protection
282 related with CO in the breath were levels of heme oxygenase-1 protein in serum and HMOX1 transcripts
283 , more importantly, amplified renoprotective heme-oxygenase-1 protein and mRNA expression in injured
284 based proteomics screen, we identified HO-1 (heme oxygenase-1), the rate-limiting enzyme in the degra
285 c-Jun), and heat shock proteins (HSP-70 and heme oxygenase-1) to the HMGCR promoter and transcriptio
286 ced increases of nitric oxide synthase-2 and heme oxygenase-1 transcriptions were also inhibited by 7
287 ne oxidoreductase (Nqo1), epoxide hydrolase, heme oxygenase-1, UDP-glucuronosyl-transferase (Ugt) 1a6
288 ted bitransgenic mice that overexpress human heme oxygenase-1 under doxycycline control in an inducib
289 diac function are mediated via Akt-dependent heme oxygenase-1 up-regulation under those conditions.
292 myocardial overexpression of thioredoxin-1, heme oxygenase-1, vascular endothelial growth factor, an
295 ated protein 78), and the antioxidant enzyme heme oxygenase-1 were decreased, whereas levels of infla
296 tide phosphate:quinone oxidoreductase 1, and heme oxygenase-1 were lower in group 2 than group 3.
297 ioxidant enzymes peroxiredoxin-2 (Prdx2) and heme oxygenase-1 were upregulated in cd36-/- VSMCs.
298 pression levels of survival genes, Bcl-2 and heme oxygenase-1, were analyzed by gene array analysis a
299 ulation of H2A histone family, member X, and heme oxygenase 1, which were experimentally validated as
300 rdial nuclear factor E2-related factor 2 and heme-oxygenase 1 with a dietary supplement (Protandim) p
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