1 recorded from neuromuscular junctions in the hemidiaphragm.

2 ostal regions of the stimulated and inactive hemidiaphragms.

3 a levels increased 80% compared with control hemidiaphragms.

4 an those values for the normally functioning hemidiaphragm (1.7 +/- 0.2 mm versus 2.7 +/- 0.5 mm [mea

5 m concomitant rupture of the spleen and left hemidiaphragm after a traumatic event involving the sple

6 ed with the navigator localized at the right hemidiaphragm and at the left ventricle were objectively

7                                   In a mouse hemidiaphragm assay, scFv binding epitopes 1 and 2 signi

8 dies did not protect the mouse phrenic nerve-hemidiaphragm from toxin-induced neuromuscular blockade.

9 ximum inspired volumes on the left and right hemidiaphragms increased significantly.

10 ost vertically oriented portion of the right hemidiaphragm muscle (VDML) were measured.

11 iaphragm electrodes were implanted into each hemidiaphragm near the phrenic nerve motor points via la

12 lated activity in the left phrenic nerve and hemidiaphragm of rats subjected to an ipsilateral C2 spi

13 ry connections which restore function to the hemidiaphragm paralyzed by an ipsilateral spinal cord he

14 y which mediates recovery of the ipsilateral hemidiaphragm paralyzed by C2 spinal cord hemisection.

15               Here, we treated mouse NMJs in hemidiaphragm preparations with anti-GQ1b antibodies and

16  to mice and in vitro on mouse phrenic nerve-hemidiaphragm preparations.

17 cular junctions in adult mouse phrenic nerve hemidiaphragm preparations.

18                                        Right hemidiaphragm silhouette length (PADL) and the length of

19 ACh from motor terminals in the rat isolated hemidiaphragm using an electrophysiological approach.

20 sis, t(di) and delta t(di) for the paralyzed hemidiaphragm were significantly less than those values