ライフサイエンスコーパス: hemiplegia

1 into adulthood, we use the term 'alternating hemiplegia'.

2 ges were seen in six people with alternating hemiplegia.

3 explained premature mortality of alternating hemiplegia.

4 ctional correlates over and above effects of hemiplegia.

5 rk in the plegic lower limbs of persons with hemiplegia.

6 Clinically, 26.2% of children had hemiplegia, 34.4% had diplegia, 18.6% had quadriplegia,

7 I was used to study 11 patients with chronic hemiplegia after unilateral stroke that spared regions o

8 Recent theories propose that anosognosia for hemiplegia (AHP) results from specific impairments in mo

9 terally during pedalling for 15 persons with hemiplegia and 12 neurologically intact age-matched cont

10 tis was stronger for diplegia (compared with hemiplegia and quadriplegia) and for cerebral palsy with

11 All patients had unilateral hemiplegia and visual field loss.

12 The study of anosognosia for hemiplegia, Anton's syndrome, and less dramatic disturba

13 Stroke and hemiplegia are frequent complications of stroke.

14 The delusional features of anosognosia for hemiplegia can be explained as a failure of this re-repr

15 le brains, which could explain the prolonged hemiplegia, coma, and seizure phenotype in this variant

16 ee clinical forms of spastic cerebral palsy (hemiplegia, diplegia, and quadriplegia) and for cerebral

17 id) or according to the body parts involved (hemiplegia, diplegia, or quadriplegia).

18 dominant disorder characterized by transient hemiplegia during the aura phase of a migraine attack.

19 litis/trauma, and one each of hemiconvulsion-hemiplegia epilepsy and perinatal ischaemic insult) and

20 In adults with hemiplegia following stroke the threshold of the homonym

21 With hemiplegia following stroke, a person's movement respons

22 he treatment of epilepsy in association with hemiplegia for over 50 years.

23 G recordings of 52 patients with alternating hemiplegia from nine countries: all had whole-exome, who

24 for hemiplegia, i.e. apparent unawareness of hemiplegia, have been clinically observed to show 'tacit

25 gram abnormalities are common in alternating hemiplegia, have characteristics reflecting those of inh

26 Some patients with anosognosia for hemiplegia, i.e. apparent unawareness of hemiplegia, hav

27 r awareness can occur called anosognosia for hemiplegia, i.e. the denial of motor deficits contralate

28 Here we successfully modeled hemiplegia-induced protection from arthritis using the K

29 Upper extremity hemiplegia is a common consequence of unilateral cortica

30 Hemiplegia, leukemia, lymphoma, severe liver disease, an

31 diseases related to episodic ataxia, such as hemiplegia, migraine, and epilepsy.

32 ts in significant nerve dysfunction, such as hemiplegia, mood disorders, cognitive and memory impairm

33 Alternating hemiplegia of childhood (AHC) is a rare, severe neurodev

34 Alternating hemiplegia of childhood (AHC) is typically distinguished

35 miplegic migraine type 2 (FHM2), alternating hemiplegia of childhood (AHC), and rapid-onset dystonia

36 d-onset dystonia parkinsonism or alternating hemiplegia of childhood causes a dramatic reduction of N

37 Alternating hemiplegia of childhood is a rare disorder caused by de

38 ac investigation is warranted in alternating hemiplegia of childhood, as cardiac arrhythmic morbidity

39 lial Hemiplegic Migraine type 2, Alternating Hemiplegia of Childhood, Rapid-onset Dystonia Parkinsoni

40 -onset dystonia parkinsonism and alternating hemiplegia of childhood.

41 tients expressing the R192Q mutation exhibit hemiplegia only.

42 Dysautonomia may occur during hemiplegia or in isolation.

43 Fourteen subjects with chronic hemiplegia participated in the study.

44 bjects with chronic (> 6 months) post-stroke hemiplegia participated in the study.

45 ks can be accompanied by seizures, coma, and hemiplegia; patients expressing the R192Q mutation exhib

46 motor performance observed when persons with hemiplegia pedal in a horizontal position is exacerbated

47 Hemiplegia remained unchanged following surgery in 22 ou

48 that, despite stroke producing contralateral hemiplegia, surviving regions of motor cortex actively p

49 antly more common in people with alternating hemiplegia than in an age-matched disease control group

50 ificantly shorter in people with alternating hemiplegia than in the disease control group.

51 r his 45th dose of natalizumab, he developed hemiplegia that evolved into quadriparesis.

52 tion following extensive lesions (pure motor hemiplegia) to incomplete basilar pontine syndrome and r

53 young man with migraine with aura including hemiplegia, we identified a novel SLC1A3 mutation that p

54 ubjects with stroke in adulthood and spastic hemiplegia were also studied.

55 Abnormalities in alternating hemiplegia were more common in those >/=16 years old, co

56 CSF rhinorrhea occurred in one patient, and hemiplegia with homonymous hemianopsia developed as a co