ライフサイエンスコーパス: hemiplegic

1 ars, with spasticity of perinatal origin (11 hemiplegic, 11 quadriplegic, 16 with Rett syndrome) and

2 hophysiological explanation for why signs of hemiplegic cerebral palsy appear late and progress over

3 Subjects with severe hemiplegic cerebral palsy have increased ipsilateral cor

4 e underlies an important share of congenital hemiplegic cerebral palsy, and probably some spastic qua

5 is is the first reported association between hemiplegic cerebral palsy, placental thrombosis, and fac

6 ause of in utero cerebrovascular disease and hemiplegic cerebral palsy.

7 opmental syndrome characterized by recurrent hemiplegic episodes and distinct neurological manifestat

8 y early-onset, recurrent, often alternating, hemiplegic episodes; seizures and non-paroxysmal neurolo

9 Median nerve stimulation of the hemiplegic hand showed reproducible early-latency ipsila

10 imary sensorimotor area) for movement of the hemiplegic hand than for movement of the normal hand.

11 ask practice and behavioral shaping with the hemiplegic hand) or usual and customary care (n = 116; r

12 ning hemisphere with passive movement of the hemiplegic hand.

13 es had residual sensorimotor function in the hemiplegic hand.

14 pastic cerebral palsy, which was unilateral (hemiplegic) in 336 (24%).

15 The previously isolated hemiplegic, induction-negative, repression-positive muta

16 genetic susceptibility of both rare familial hemiplegic migraine (FHM) and more common types of migra

17 HC) is typically distinguished from familial hemiplegic migraine (FHM) by infantile onset of the char

18 Familial hemiplegic migraine (FHM) has been related to mutations

19 Familial hemiplegic migraine (FHM) is a rare subtype of migraine

20 Familial hemiplegic migraine (FHM) is an autosomal dominant disor

21 Familial hemiplegic migraine (FHM) is an autosomal dominant subty

22 emory difficulties observed in some familial hemiplegic migraine (FHM) patients, we examined hippocam

23 Familial hemiplegic migraine (FHM), a migraine subtype that also

24 thin a candidate region at 1q23 for familial hemiplegic migraine (FHM).

25 o several human diseases, including familial hemiplegic migraine (FHM).

26 venous sinus thrombosis and seizure (n = 3), hemiplegic migraine (n = 1), and hyperacute arterial inf

27 odic ataxia and one PNKD family had familial hemiplegic migraine alone.

28 med on 7 family members, 5 with a history of hemiplegic migraine and 2 without history of migraine.

29 variant was identified in two families with hemiplegic migraine and in one patient with migraine wit

30 h controls the PCr/Pi ratio in patients with hemiplegic migraine and in patients with persistent aura

31 Studies of linkage between familial hemiplegic migraine and marker 19p13 in 13 extended fami

32 a gain-of-function and associated with both hemiplegic migraine and migraine with aura in patients.

33 rare monogenic migraine syndromes, in which hemiplegic migraine and non-hemiplegic migraine with or

34 ered to provide an understanding of familial hemiplegic migraine and possibly, by extrapolation, may

35 We suggest that the hemiplegic migraine attacks and the cerebellar degenerat

36 Of these, hemiplegic migraine emerges as a novel PRRT2-associated

37 Familial hemiplegic migraine is associated with at least 13 diffe

38 Familial hemiplegic migraine is caused by mutations in the calciu

39 Familial hemiplegic migraine knock-in mice expressing the S218L o

40 aptic transmission resulting from a familial hemiplegic migraine mutation (S218L).

41 Two PRRT2 mutations were in familial hemiplegic migraine or episodic ataxia, one SLC2A1 famil

42 and was significantly lower in patients with hemiplegic migraine than in patients with non-motor aura

43 3 patients with familial episodic ataxia and hemiplegic migraine to investigate the mutation frequenc

44 Familial hemiplegic migraine type 1 (FHM1) arises from missense m

45 Familial hemiplegic migraine type 1 (FHM1) is a subtype of migrai

46 Familial hemiplegic migraine type 1 (FHM1) is an autosomal domina

47 Familial hemiplegic migraine type 1 (FHM1), a monogenic migraine

48 aV2.1 channels, are associated with familial hemiplegic migraine type 1 (FHM1), a rare monogenic subt

49 Familial hemiplegic migraine type 1 (FHM1), a severe migraine wit

50 minant form of this common disease, familial hemiplegic migraine type 1 (FHM1), arises from missense

51 mutations that are associated with familial hemiplegic migraine type 1 (FHM1).

52 In familial hemiplegic migraine type 1 mutant mice expressing human

53 into subcortical structures in both familial hemiplegic migraine type 1 mutants.

54 rization in mice carrying the human familial hemiplegic migraine type 1 R192Q missense mutation as we

55 Familial hemiplegic migraine type 1, a monogenic migraine variant

56 ttene et al. study a mouse model of familial hemiplegic migraine type 1, and provide evidence for the

57 The neurological disorders familial hemiplegic migraine type 2 (FHM2), alternating hemiplegi

58 severe human pathologies including Familial Hemiplegic Migraine type 2, Alternating Hemiplegia of Ch

59 Familial hemiplegic migraine type 3 (FHM3) is a severe autosomal

60 The neurological disorder familial hemiplegic migraine type II (FHM2) is caused by mutation

61 (rapid-onset dystonia parkinsonism, familial hemiplegic migraine type-2), as well as reduction in Na,

62 dromes, in which hemiplegic migraine and non-hemiplegic migraine with or without aura are part of a w

63 Familial hemiplegic migraine, a rare Mendelian form of MA, can be

64 with neurological disorders, such as ataxia, hemiplegic migraine, and epilepsy.

65 The recent discovery that familial hemiplegic migraine, episodic ataxia type 2, and spinoce

66 type voltage-gated calcium channel (familial hemiplegic migraine, episodic ataxia type 2, spinocerebe

67 such human neurological diseases as familial hemiplegic migraine, episodic ataxia-2, and spinocerebel

68 um channel gene are associated with familial hemiplegic migraine, episodic or progressive ataxia, com

69 ns included a high frequency of migraine and hemiplegic migraine.

70 mechanism for enhanced CSD susceptibility in hemiplegic migraine.

71 ified a novel PNKD gene deletion in familial hemiplegic migraine.

72 rders is much broader than strictly familial hemiplegic migraine.

73 human neurologic diseases including familial hemiplegic migraine.

74 myoclonic epilepsy of infancy, and familial hemiplegic migraine.

75 cium channel gene associated with ataxia and hemiplegic migraine.

76 teral sclerosis, parkinsonism, epilepsy, and hemiplegic migraine.

77 linkage and association studies of familial hemiplegic migraine.

78 These hemiplegic mutations identify amino acid residues that,

79 tation of motor and cognitive impairments in hemiplegic or paraplegic patients by offering on-line fe

80 icantly slower than a control group of aware hemiplegic patients in performing the inhibition task wi

81 A group of anosognosic hemiplegic patients was significantly slower than a cont

82 ning in awareness in patients with AHP: Four hemiplegic patients with and four without anosognosia we

83 In contrast, hemiplegic patients without neglect will reach across an

84 tients should undergo ultrasonography of the hemiplegic shoulder to define the nature and extent of s

85 rmed to sonographically evaluate post-stroke hemiplegic shoulders and explore possible relationship(s

86 Hemiplegic shoulders exhibited significantly higher numb

87 Hemiplegic shoulders have significantly higher number of

88 The most frequent pathologies in the hemiplegic shoulders were the following: tendinosis of t

89 he long head of bicep tendon was commoner in hemiplegic shoulders with poor motor status than those w

90 al abnormalities were found in all 45 (100%) hemiplegic shoulders, 25 (55.6%) unaffected shoulders of

91 pecialty-specific operation complicated by a hemiplegic stroke and respiratory failure.

92 Rehabilitation after hemiplegic stroke has typically relied on the training o

93 Hemiplegic stroke patients should undergo ultrasonograph

94 ird of patients presenting with a unilateral hemiplegic stroke, yet its neurophysiological basis rema

95 consistent and reliable clinical features of hemiplegic stroke.