ライフサイエンスコーパス: hemisection

1 ould occur after unilateral axotomy via C(2) hemisection.

2 al hemidiaphragm paralyzed by C2 spinal cord hemisection.

3 ragm paralyzed by an ipsilateral spinal cord hemisection.

4 h and beyond the lesion gap of a spinal cord hemisection.

5 er, a complete spinalization below the first hemisection.

6 ays to phrenic motoneurons below a C2 spinal hemisection.

7 lowing Fluoro-Gold injections, caudal to the hemisection.

8 cending axonal projections after spinal cord hemisection.

9 rhizotomy and midthoracic dorsal spinal cord hemisection.

10 the spinal cord gray matter after unilateral hemisection.

11 vity in the phrenic nerve ipsilateral to the hemisection.

12 activity indicating a functionally complete hemisection.

13 d-phrenic pathways after left C2 spinal cord hemisection.

14 l ND from 1 to 14 days following spinal cord hemisection.

15 n neurons of the ND and RN after spinal cord hemisection.

16 ity of phrenic motoneurons ipsilateral to C2 hemisection.

17 naltered in the RN neurons after spinal cord hemisection.

18 with wild-type controls after spinal dorsal hemisection.

19 rounding dysgranular S1 contralateral to the hemisection.

20 intrathecally after midthoracic dorsal over-hemisection.

21 leus following an ipsilateral C2 spinal cord hemisection and 30-day survival period.

22 al performance after both T10 lateral spinal hemisection and a unilateral cortical stroke.

23 Two months following T8 hemisection and Bcl-2/LacZ DNA injection, there was no s

24 multiple experimental data on the effects of hemisection and selective silencing or activation of V1

25 rated motor axon regeneration beyond both C5 hemisection and T3 complete transection sites.

26 uniculus on one side, dorsal and over-dorsal hemisections, and subtotal transections that spared pred

27 n the present study, the consequences of T13 hemisection are examined acutely at 45 min in MR neurons

28 t 28 days after injury, the effects of acute hemisection are unknown and predicted to be different th

29 renic nucleus 30 days after a C2 spinal cord hemisection as compared to nonhemisected controls.

30 Cervical spinal hemisection at C2 (SH) removes premotor drive to phrenic

31 Spinal hemisection at C2 reveals caudal synaptic pathways that

32 eurons whose axons were severed by a lateral hemisection at C3 (HX) and in lateral vestibular nucleus

33 lesions at thoracic level 9 (T9) or lateral hemisection at cervical level 5 (C5), our results reveal

34 ormation with spinal neurons, or caudal to a hemisection at L2/3, to assess restoration of function b

35 capability of cats after a unilateral spinal hemisection at T10 to cope with such a demanding locomot

36 /6 mice) and MRL/MpJ mice underwent a dorsal hemisection at T9 (thoracic vertebra 9).

37 hamsters were subjected to right spinal cord hemisection at the C7/T1 vertebral level.

38 We performed, in 9 adult cats, a spinal hemisection at thoracic level 10 and then a complete spi

39 sing microarrays, 24 voxel images of coronal hemisections at the level of the hippocampus of both the

40 Following C5 hemisections, athymic rats were treated with patches loa

41 Acutely, ipsilateral to the hemisection, both spontaneous and evoked activity of MR

42 N neurons occurred at L-1 ipsilateral to T-8 hemisection by 14 days, which reached 31% at 2 months an

43 ience such as required for adaptation to the hemisection, can remarkably respond to subsequent locomo

44 e cells were grafted into a partial cervical hemisection cavity that completely interrupted one RST.

45 siRhoA, and p21) in experimental spinal cord hemisection, contusion, or transection on locomotor reco

46 oro-gold labeling experiments following a T1 hemisection established that, as in the rat, the hamster

47 imental series, animals were subjected to T1 hemisection, followed by transplantation of a predegener

48 terminals was also found to be longer in the hemisection group (0.36 +/- 0.022 microns) as compared t

49 te terminals was significantly longer in the hemisection group (0.37 +/- 0.013 microns) as compared t

50 nes per terminal, however, is greater in the hemisection group (1.45 +/- 0.03) than in the control gr

51 To test whether C(2) hemisection had induced a supraspinal change in respirat

52 Animals in which T13 was inserted caudal to hemisection had significantly less spasticity and muscle

53 l tract (CST) axons after spinal cord dorsal hemisection has yielded conflicting results.

54 icits induced by chronic midthoracic lateral hemisection (HX) injury: (1) synaptic transmission to lu

55 Chronic unilateral hemisection (HX) of the adult rat spinal cord diminishes

56 clicality in female rats after a spinal cord hemisection (HX), implantation of EMG wires into selecte

57 ur different donors into a subtotal cervical hemisection in adult female rats and found that cells in

58 investigated following unilateral C4 spinal hemisection in adult rats.

59 h the hindlimbs after a thoracic spinal cord hemisection in adult rats.

60 After a spinal hemisection in cats, locomotor plasticity occurring at t

61 After dorsal hemisection in the midthoracic cord, CST axons did not s

62 eatment with PUFAs after lateral spinal cord hemisection in the rat.

63 ion site, but not caudal to it, after dorsal hemisection in the transgenic mice.

64 injury (SCI) were evaluated by T9 cord level hemisection in wild-type mice (C57BL/6J and Bax+/+ mice)

65 spinal delivery of Ch'ase ABC, following T10 hemisections in adult cats, enhances adaptive movement f

66 After hemisection, inactivation of only V2b interneurons led t

67 paradigm, we recently demonstrated that the hemisection induces durable changes in the symmetry of l

68 titive antagonist at the time of spinal cord hemisection induces significant regeneration of corticos

69 Rats then received cervical dorsal hemisection injuries and salmon fibrin was injected into

70 Mice received dorsal hemisection injuries at thoracic level 6-7, which comple

71 gene targeting starting 8 weeks after spinal hemisection injury and monitored locomotion in the open

72 Spinal hemisection injury at T13 results in development of perm

73 Indeed, in a spinal cord dorsal hemisection injury model, we observed a significant impa

74 s can be efficiently delivered to a cervical hemisection injury site by intrathecal delivery at the l

75 After a dorsal hemisection injury, increased corticospinal and raphespi

76 unctional recovery after mid-thoracic dorsal hemisection injury.

77 Three months after C5 lateral hemisections, iPSCs survived and differentiated into neu

78 laterally in adult Sprague-Dawley rats by T8 hemisection, leaving the contralateral (left) CN as an i

79 k into the C5 spinal cord, having bypassed a hemisection lesion at C3.

80 Two weeks after a hemisection lesion at C3/4, with embryonic spinal tissue

81 ere then grafted to acute spinal cord dorsal hemisection lesion cavities.

82 otor skills, but only in animals with dorsal hemisection lesions as opposed to dorsal column lesions.

83 cell grafts placed within sites of right C7 hemisection lesions in the rhesus macaque.

84 by Basso Mouse Scale score following dorsal hemisection, likely due to developmental defects in the

85 The present study uses a rodent spinal hemisection model of SCI in which mechanical and thermal

86 old-neural stem cells unit into an adult rat hemisection model of SCI promoted long-term improvement

87 t rats or the injury site in a dorsal column hemisection model of spinal cord injury, a population of

88 to promote motor recovery after SCI in a rat hemisection model when delivered in their original stem

89 Unilateral T13 hemisection of the rat spinal cord produces a model of c

90 Contralateral and ipsilateral hemisection of the spinal cord (C5/C6) produced the same

91 g of corticospinal tract fibers after dorsal hemisection of the spinal cord did not reveal an obvious

92 In this rare condition, a hemisection of the spinal cord typically leaves one side

93 Distal and medial hemisections of the endotracheal tube were assessed to q

94 Dorsal hemisections of the mid thoracic cord did not alter the

95 roxytryptamine prevented the effects of C(2) hemisection on contralateral phrenic neurogram amplitude

96 al segments] with a contralateral transverse hemisection one segment anterior to the hindlimb enlarge

97 ipsilateral hindlimb than did animals with a hemisection only.

98 In animals with hemisections only, corticospinal, brainstem-spinal, and

99 Recovery of motor function after dorsal hemisection or complete transection of the spinal cord i

100 In contrast, animals receiving hemisections plus transplants exhibited growth of these

101 C inhibitor, Go6976, into the site of dorsal hemisection promotes regeneration of dorsal column axons

102 atterns in the spinal turtle with transverse hemisection provided support for the concept of bilatera

103 tivity and relative stability after cervical hemisection raise speculation for potentially diverse ro

104 alyzed, ventilated, and spinally injured (C2 hemisection) rats that were exposed to either normoxia o

105 Spinal hemisection reduced immunostaining of the ipsilateral do

106 EP1 40 to rats with mid-thoracic spinal cord hemisection results in significant axon growth of the co

107 al and serotonergic fibers in a rat cervical hemisection SCI model.

108 as diminished paw drag often observed after hemisection.SIGNIFICANCE STATEMENT This paper introduces

109 hite matter 3-7 mm rostral and caudal to the hemisection site 8 d after injury.

110 he normal spinal cord, or just caudal to the hemisection site.

111 Also, in rat T9-T10 hemisection spinal cord injury (SCI), we demonstrated th

112 rticospinal and serotonergic fibers in a rat hemisection spinal cord injury model.

113 acutely vulnerable in response to T8 dorsal hemisection spinal cord injury.

114 ing Ryk antibodies into the dorsal bilateral hemisectioned spinal cord either prevented the retractio

115 In animals 28 days after hemisection, spontaneous activity of MR neurons was comp

116 the intact cord but lose this function after hemisection, suggesting that they are activated by input

117 ulation in the spinal turtle with transverse hemisection supports the concept that hip extensor circu

118 After a lateral left hemisection (T10), cats recovered stepping with both hin

119 oxide (NO) production following spinal cord hemisection tends to lead to neurodegeneration in neuron

120 rainstem respiratory motor output after C(2) hemisection that coincided with the spontaneous recovery

121 in the lumbar spinal cord after T13 lateral hemisection that correlated with emergence of mechanical

122 Additional rats with hemisections that received NT-3 continuously via mini-pu

123 Lesions included subtotal dorsal hemisections that spared only the lateral half of the do

124 Therefore, after spinal hemisection the time course of changes in NF mRNA expres

125 als in the phrenic nucleus was reduced after hemisection, the number of 5-HT terminals in the hemisec

126 With a chronic left hemisection, the number of muscle nerves displaying loco

127 We used lateral hemisection to model central neuropathic pain and herpes

128 an intact spinal cord or after a unilateral hemisection to walk with a precise foot placement on the

129 cord injury, we administered C7 spinal cord hemisections to adult rhesus monkeys and analyzed behavi

130 lap debridement, apically repositioned flap, hemisection, tunneling or extraction, to regenerative th

131 s subjected to an ipsilateral C2 spinal cord hemisection was confirmed electrophysiologically 24 h af

132 Intensive LTM training after hemisection was found to change features of locomotion,

133 mmetrical state of locomotion induced by the hemisection was retained durably after the subsequent sp

134 labeling patterns following lateral cervical hemisection was then addressed.

135 ether lumbar segments of cats with a chronic hemisection were able to generate fictive locomotion-tha

136 matosensory cortex (S1) contralateral to the hemisection were determined using standard multiunit map

137 spinal networks were indeed modified after a hemisection with a clear asymmetry between left and righ

138 We report that 6 weeks after dorsal hemisection with peripheral conditioning lesion, C3(-/-)

139 f spatially and temporally separated lateral hemisections with or without the excitotoxic ablation of

140 d male hamsters were subjected to a right T1 hemisection, with half of the operated animals immediate