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1 ute over 90% of the neurons in each thoracic hemisegment.
2 establish the future muscle pattern in each hemisegment.
3 e 21 identifiable peripheral neurons in each hemisegment.
4 al columns and four anteroposterior rows per hemisegment.
5 of mitotic sister serotonin neurons in each hemisegment.
6 of PEI within the sensory field of a single hemisegment.
7 s organized in a stereotypic pattern in each hemisegment.
8 ryo results in defective hearts with missing hemisegments.
9 ce point in approximately 40% of the somitic hemisegments and an approximately 150% increase in the n
14 the absence of zygotic Toll, close to 50% of hemisegments have muscle patterning defects consisting o
15 ts delaminate from the neuroectoderm of each hemisegment in a stereotypic orthogonal array of five ro
16 activity pattern in nerves innervating each hemisegment in both wild-type and Tbetah(nM18) larvae.
17 2 protein was detected in 11 neuroblasts per hemisegment in Drosophila embryos, 9 medial and 2 interm
18 e the principal pacemakers of the CPG in the hemisegment in which they are located, they should direc
19 dendrites from homologous neurons in the two hemisegments meet at the dorsal midline in larval stages
21 roblasts initially present in each abdominal hemisegment of the embryonic ventral nerve cord, only th
22 f the six bilateral cardioblasts within each hemisegment of the heart allows these cells to adopt a c
24 ne from one hemisegment to the contralateral hemisegment, or does not migrate at al and fail to send
26 ow that each of the 15 neurons per abdominal hemisegment spread dendrites to characteristic regions o
27 s a population of four pericardial cells per hemisegment that are distinct from previously identified
28 rise 12-16 uniquely identifiable neurons per hemisegment that have homologues in other insect larvae.
29 ally migrates, crossing the midline from one hemisegment to the contralateral hemisegment, or does no
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