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2 tern is consistent with data from those with hemispatial neglect after stroke and raises the possibil
4 te right subcortical infarcts using tests of hemispatial neglect and magnetic resonance diffusion-wei
5 l attention deficits in stroke patients with hemispatial neglect are correlated at 2 weeks postonset
6 udy, we test the hypothesis that recovery of hemispatial neglect correlates with a return of network
7 e hypothesized that most cases of aphasia or hemispatial neglect due to acute, subcortical infarct ca
8 ected attention and recovery from multimodal hemispatial neglect following cortical lesions of medial
10 attention-deficit hyperactivity disorder and hemispatial neglect following right-hemisphere stroke.
12 ssessments of right-hemisphere patients with hemispatial neglect have revealed significant overall de
13 schizophrenia suggest a subtle form of right hemispatial neglect in acutely ill patients but not in c
16 can cause patients to ignore half of space (hemispatial neglect syndrome), but only for right (not l
17 we present a case series of 14 patients with hemispatial neglect who, when asked to clap, repeatedly
18 ine-scale functional architecture underlying hemispatial neglect, an attentional deficit caused by pa
19 Lesions of parietal cortex often lead to hemispatial neglect, an impairment of choices of targets
20 ent prospects of neurological disorders like hemispatial neglect, hemianopia and other deficits after
22 e that sometimes follows brain injury called hemispatial neglect, in which patients exhibit attention
23 environment and, with that facility lost in hemispatial neglect, search becomes uselessly stuck with
24 wed no difference in performance on tests of hemispatial neglect, volume of infarct or sex distributi
37 main unclear, with some studies supporting a hemispatial theory emphasizing a dominant function of th
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