ライフサイエンスコーパス: hemodynamic

1 erial echocardiography and terminal invasive hemodynamics.

2 edema on imaging and more deranged cerebral hemodynamics.

3 and thus enables assessment of microvascular hemodynamics.

4 lular interactions in modeling microvascular hemodynamics.

5 l decontamination with rifaximin may improve hemodynamics.

6 terized by disturbed systemic and splanchnic hemodynamics.

7 n the coupling between electrophysiology and hemodynamics, a phenomenon referred to as "neurovascular

8 interventions that may attenuate this renal hemodynamic abnormality.

9 irectly and functional MRI (fMRI) to measure hemodynamic activity during a task-free period (the "res

10 This was reflected in the hemodynamic activity of vocal-motor cortices, even after

11 This direct measure of neural and hemodynamic activity shows that the insula, one of the m

12 tients with depression show blunted amygdala hemodynamic activity to positive stimuli, including auto

13 ations in cortex are tightly linked to local hemodynamic activity.

14 rovide structural evidence for the bilateral hemodynamic alterations that have been reported in patie

15 single-nephron level, diabetes-related renal hemodynamic alterations-as an adaptation to reduction in

16 ovided 410,534 notifications (48% arrhythmia/hemodynamic) among 61 nursing units.

17 has gained increasing recognition as having hemodynamic and clinical consequences associated with in

18 In many patients, the hemodynamic and clinical results are comparable to that

19 ultured monocytes and endothelial cells, and hemodynamic and lung studies in a rat.

20 wn of brain homeostasis promotes significant hemodynamic and metabolic alterations, which impacts on

21 to investigate whether metoprolol reduce the hemodynamic and metabolic burden imposed by AS.

22 Thus, metoprolol displays favorable hemodynamic and metabolic effects and could improve outc

23 rscoring the important synergy of addressing hemodynamic and neurohormonal targets of HF therapy.

24 plished through a sound understanding of the hemodynamic and physiological consequences of laparoscop

25 This coincides with hemodynamic and structural changes typical of HFpEF.

26 ine triphosphate nucleotidohydrolase induced hemodynamic and vascular changes of pulmonary hypertensi

27 dioxide insufflation may produce significant hemodynamic and ventilatory consequences such as increas

28 besity has adverse effects on cardiovascular hemodynamics and cardiac structure and function, and inc

29 of work has been dedicated to the effects of hemodynamics and cytokines on leukocyte adhesion and tra

30 ts to examine their relationships to cardiac hemodynamics and determine whether exertional arterial s

31 ter, propensity score-matched study compared hemodynamics and early clinical outcomes in 246 patients

32 Altered pulmonary hemodynamics and fluid flow-induced high shear stress (H

33 Baseline in vivo hemodynamics and isolated muscle studies showed that cTn

34 e demonstrated differences in postprocedural hemodynamics and long-term clinical outcomes.

35 ng of this artifact correlates with invasive hemodynamics and mechanical aortic valve artifacts.

36 n this study, we sought to determine if lung hemodynamics and pathological changes in Mtb infected ce

37 he protective proprieties of PDE5is on renal hemodynamics and the molecular mechanisms involved.

38 cotransporter 2 (SGLT2) inhibitor, on renal hemodynamics and tubular functions in anesthetized non-d

39 ural changes and has altered transcriptomic, hemodynamic, and physiologic behavior at rest and in res

40 w mortality and complication rates, improved hemodynamics, and excellent functional and quality-of-li

41 n, functional capacity, biomarkers, invasive hemodynamics, and right ventricular functional indices,

42 Correlation with invasive hemodynamic aortic pressure tracings was performed.

43 Ventricular hemodynamics are then quantified by numerically solving

44 tic approaches enables combined anatomic and hemodynamic assessment of a coronary lesion by a single

45 st MR imaging in healthy volunteers to allow hemodynamic assessment of children and young adults with

46 METHODS AND Invasive hemodynamic assessment was performed at baseline and aft

47 rcise testing using invasive and noninvasive hemodynamic assessments to definitively confirm or refut

48 a fundamental brain function that underlies hemodynamic-based functional brain imaging techniques.

49 safe and produces similar acute and chronic hemodynamic benefits in people with heart failure and pr

50 re severe intensive care medical conditions (hemodynamic, biological, renal, and liver insults) than

51 testing with invasive monitoring to measure hemodynamics, blood gases, and gas exchange during exerc

52 ast is widely used for noninvasively mapping hemodynamic brain activity in humans, its exact link to

53 F) by enhancing cGMP signaling and improving hemodynamics, but real-world data on potential efficacy

54 hours of resuscitation, datasets comprising hemodynamics, calorimetry, blood gases, cytokines, and c

55 Quantitative assessment of conjunctival hemodynamics can potentially be useful for evaluation of

56 adversely affects compensatory responses to hemodynamic challenge has not been extensively investiga

57 To compensate for these hemodynamic changes and maintain constant BP in salt res

58 tone infusion mouse model was also used with hemodynamic changes characterized by echocardiography an

59 We investigated hemodynamic changes following SVR in patients with CSPH

60 eurons while simultaneously measuring global hemodynamic changes using BOLD and cerebral blood volume

61 Hemodynamic changes, incidence of hepatic encephalopathy

62 that DMF treatment is effective in reversing hemodynamic changes, reducing inflammation, oxidative da

63 H according to underlying pathophysiologies, hemodynamic characteristics, and treatment responses as

64 T and correlate their clinical outcomes with hemodynamic characteristics.

65 te evaluation of vascular anomalies includes hemodynamic characterization.

66 Pulmonary hypertension is a common hemodynamic complication of heart failure.

67 The rate of hemodynamic compromise and cardiac erosion is rare.

68 dy was to prospectively evaluate the risk of hemodynamic compromise and obtain medium-term survival d

69 Hemodynamic compromise occurred in 6 subjects (0.65%), b

70 ual patient on the basis of presentation and hemodynamic conditions may be beneficial.

71 circulatory support devices acutely improve hemodynamic conditions.

72 valvular disease as well as determining the hemodynamic consequences and extent of ventricular remod

73 nt and Transplantation Network database with hemodynamics consistent with POPH (defined as mean pulmo

74 ubgroup was identified with resting invasive hemodynamics consistent with pulmonary vascular disease.

75 At the neural level, hemodynamic correlates for the learning rate, expected v

76 rocytes which are critical elements in neuro-hemodynamic coupling show a significant anatomical segre

77 Notably, 9 patients (11%) met hemodynamic criteria for precapillary pulmonary hyperten

78 FFRangio uses the patient's hemodynamic data and routine angiograms to generate a co

79 w how archiving and mining of intraoperative hemodynamic data in orthotopic liver transplantation (OL

80 on, sevoflurane consumption, respiratory and hemodynamic data, Simplified Acute Physiology Score II,

81 y gas exchange and noninvasive (rebreathing) hemodynamic data.

82 1) establishing a definitive consensus for a hemodynamic definition, perhaps incorporating exercise a

83 ypertension and is correlated with classical hemodynamic derangements that develop with stress.

84 Conjunctival hemodynamic descriptors, namely vessel diameter (D), blo

85 d albuminuria without any change in systemic hemodynamics, despite persistent loss of podocyte functi

86 rea, Doppler velocity index, and evidence of hemodynamic deterioration by reintervention, adverse hem

87 Hemodynamic determinants included elevated right atrial

88 The role of embolism and hemodynamic disturbances caused by carotid artery diseas

89 rterial compliance and elastance and central hemodynamics during exercise.

90 al disease have low sensitivity, and altered hemodynamics during the LT surgery can unmask latent car

91 racted demographics, clinical data, invasive hemodynamics, echocardiography, and vital status for all

92 Hemodynamic effects are accentuated in patients with car

93 anisms involved, we evaluated their regional hemodynamic effects in conscious rats.

94 , the hemodynamics of cardiogenic shock, and hemodynamic effects of percutaneous mechanical circulato

95 behaviors via agent-based modeling (ABM) and hemodynamic effects via computational fluid dynamics (CF

96 n (single 2-mg i.v. bolus), and the regional hemodynamic effects were quantified by phase contrast ma

97 roduced by cecal ligation puncture to assess hemodynamic efficacy, cardioprotection, and biomechanics

98 eal lipopolysaccharide injection (10 mg/kg), hemodynamic efficacy, cardioprotection, and biomechanics

99 sponsiveness of circulating platelets to the hemodynamic environment of cardiovascular devices, and t

100 onditions to recapitulate the morphology and hemodynamic environment of the pulmonary microvascular e

101 rol subjects and 98 HFpEF subjects underwent hemodynamic exercise testing with simultaneous expired g

102 l assessment, echocardiography, and invasive hemodynamic exercise testing.

103 SVR on the hepatic, pulmonary, and systemic hemodynamics; factors related to HVPG >/=10% reduction a

104 oid receptor expression might participate in hemodynamic failure during sepsis.

105 We conclude that inclusion of intraoperative hemodynamic features significantly improves prediction o

106 performed E-selectin adherence assays under hemodynamic flow conditions coupled with flow cytometry

107 e sleep apnea causes intermittent hypoxemia, hemodynamic fluctuations, and sleep fragmentation, all o

108 on quantitatively investigating the role of hemodynamic forces and mechanotransduction during morpho

109 We hypothesized that LV hemodynamic forces calculated from 4D flow MRI could ser

110 r of optical tweezing in measuring low range hemodynamic forces in vivo and offers an unprecedented t

111 that such a method is powerful in measuring hemodynamic forces in vivo with accuracy and confidence.

112 or thoracic aortic disease include increased hemodynamic forces on the ascending aorta, typically due

113 Hemodynamic forces play an essential epigenetic role in

114 Hemodynamic forces regulate vascular functions.

115 re gradients over the LV volume rendered the hemodynamic forces.

116 An average of hemodynamic function was quantified for each subject dep

117 When added to conventional imaging and hemodynamic, functional, and clinical markers, three-dim

118 y goal-directed therapy bundle compliance or hemodynamic goal achievement.

119 ompliance, amount of fluid administered, and hemodynamic goal achievements were not associated with s

120 mark, and -434.0 mm Hg-day after 6 months of hemodynamic guided care, which was significantly lower t

121 first 2000 general-use patients managed with hemodynamic-guided heart failure care had higher PA pres

122 anced by the use of electrophysiological and hemodynamic imaging [3-6].

123 Systemic hemodynamics improved after SVR.

124 ng mechanics, lung morphology, and pulmonary hemodynamics in 10 swine.

125 eptor tyrosine kinase inhibitors on regional hemodynamics in conscious, freely moving rats.

126 computational framework to simulate cardiac hemodynamics in developing zebrafish embryos by coupling

127 tions affect the dynamic lesion and exercise hemodynamics in general.

128 Determine whether inhaled nitrite improves hemodynamics in HFpEF.

129 cal significance of measured cardiopulmonary hemodynamics in hypertrophic cardiomyopathy patients wit

130 venous pressure gradient or improve systemic hemodynamics in patients with cirrhosis and ascites; rif

131 ventricular performance and impacts systemic hemodynamics in pediatric PAH.

132 terial stiffening with exercise and improves hemodynamics, indicating that arterial stiffening with e

133 of sepsis biomarkers in relation to systemic hemodynamics, inflammation, and renal function.

134 it secondary immune activation responding to hemodynamic injury in the target organ.

135 neonatal cardiac surgery, where pre-existing hemodynamic instability and metabolic abnormalities are

136 enced an HBAT-related serious adverse event (hemodynamic instability characterized by bradycardia, ta

137 Severe CRS was characterized by hemodynamic instability, capillary leak, and consumptive

138 ntly perceived barriers to mobilization were hemodynamic instability, hypoxemia, and dependency on ve

139 rmed in 44 cases because of complex anatomy, hemodynamic instability, or failed percutaneous coronary

140 in intrathoracic pressure can lead to severe hemodynamic instability.

141 rs of end-organ dysfunction, and profiles of hemodynamic instability.

142 Hemodynamic insults secondary to increased intraabdomina

143 hat the relationship between gamma and local hemodynamics is not fixed, but rather context dependent.

144 introduce an Eulerian-Lagrangian model where hemodynamics is solved on a fixed Eulerian grid, while p

145 osed that the response of aortic SMCs to the hemodynamic load on a structurally defective aorta is th

146 uent increase in distal arterial caliber and hemodynamic load precipitates the flow-dependent develop

147 te the relationships between aging, elevated hemodynamic load, cardiac mechanics, and LV remodeling i

148 mpound, arjunolic acid (AA), in ameliorating hemodynamic load-induced cardiac fibrosis and identified

149 105) in which treating clinicians determined hemodynamic management.

150 s and known individual echocardiographic and hemodynamic markers of RHF.

151 males and the sexual dimorphism in cerebral hemodynamics may explain why males are more vulnerable t

152 Hemodynamic measurements and calculation of the valve ef

153 t cardiac catheterization with comparison of hemodynamic measurements before and after nitroprusside.

154 eserved in CBSC-treated hearts, and invasive hemodynamic measurements documented improved cardiac str

155 Hemodynamic measurements indicated modest increases in t

156 us pressure (CVP), as well as other invasive hemodynamic measurements, in patients hospitalized with

157 proach, encompassing metabolic, electric and hemodynamic measurements, tailored but not limited to st

158 By the same hemodynamic mechanism, pharmacological heart rate loweri

159 d heart failure hospitalization, likely with hemodynamic/metabolic-driven mechanisms of action.

160 Implantable hemodynamic monitor-derived baseline ePAD and change fro

161 analysis was to examine whether implantable hemodynamic monitor-derived baseline estimated pulmonary

162 Data on the clinical use of left atrial (LA) hemodynamic monitoring during MitraClip procedure are li

163 As in clinical trials, use of ambulatory hemodynamic monitoring in clinical practice is associate

164 udy examined the effectiveness of ambulatory hemodynamic monitoring in reducing HFH outside of the cl

165 "Next-generation" implantable hemodynamic monitors are in development, and novel appro

166 Early studies using implantable hemodynamic monitors demonstrated the potential of press

167 processes have advanced our knowledge of the hemodynamic (mostly mediated through vasodilation of cap

168 Hepatic and systemic hemodynamic, nitric oxide (NO) bioavailability, LF, HSC

169 We reviewed hemodynamics of 300 consecutive adult patients undergoin

170 omes of patients with cardiogenic shock, the hemodynamics of cardiogenic shock, and hemodynamic effec

171 eedback was delayed and blurred based on the hemodynamics of fMRI, this model learned more reliably f

172 ologic function of the kidneys in regulating hemodynamics of the body and maintaining organ health.

173 R angiography provides information regarding hemodynamics of vascular anomalies, differentiating high

174 Severely abnormal hemodynamics on echocardiograms were also infrequent and

175 evels, suggesting that PUA may augment other hemodynamic or inflammatory mechanisms that control the

176 mic deterioration by reintervention, adverse hemodynamics, or transvalvular regurgitation.

177 r, relatively little is known about how slow hemodynamic oscillations measured with fMRI relate to el

178 h low mortality, significant improvements in hemodynamic outcomes, and low incidence of adverse event

179 ressing cyclin D2 would be beneficial during hemodynamic overload.

180 sociations between RA function with invasive hemodynamics (P<0.01).

181 res were confirmed by real-time recording of hemodynamic parameters [pulsatile arterial pressure (PAP

182 ows the progression of DN in mice, improving hemodynamic parameters and vessel integrity.

183 nt might be considered as it does not affect hemodynamic parameters in braindead (BD) donors.

184 r set by the treating anesthesiologist using hemodynamic parameters in theatre, within 4 hours of ret

185 Respiratory parameters, hemodynamic parameters, and blood gas values were measur

186 To determine the midterm hemodynamic performance of balloon-expandable transcathe

187 Specifically, it was shown that the hemodynamic perturbation by AF considered led to substan

188 We have previously shown that a hemodynamic perturbation by AF or reduced cardiac output

189 Hemodynamic, physiological, and behavioral parameters we

190 l afterload and diastolic dysfunction on the hemodynamic presentation of LGSAS remains unknown.

191 highest sensitivity to determine the optimal hemodynamic profile.

192 Cpc-PH, which is characterized by an adverse hemodynamic profile.

193 s these difficulties by accurately measuring hemodynamic profiles in order to calibrate the trap stif

194 on calculated DPD, PH-LHD subclassification, hemodynamic profiles, and mortality.

195 As a result, both the pH levels and the hemodynamic properties across the entire tumor can be qu

196 ow the spatial and temporal effects on brain hemodynamics provide information about pharmacologically

197 We show that heterogeneity in hemodynamic quantities, which is a hallmark of microvasc

198 tients with LVADs underwent routine invasive hemodynamic ramp testing with right heart catheterizatio

199 Hemodynamic reassessment performed in 35 patients who we

200 improving fluid homeostasis, cardiovascular hemodynamics recovery, and limiting kidney dysfunction i

201 Hemodynamics remained stable over the study period, and

202 l cell heterogeneity and its relationship to hemodynamics remains poorly understood due to a lack of

203 ial investigates the effects of rifaximin on hemodynamics, renal function, and vasoactive hormones.

204 complexity of coronary anatomy, and/or poor hemodynamics represent an understudied and potentially u

205 s in several orders-of-magnitude increase in hemodynamic resistance, and thus provides an additional

206 The results evidenced a habituation-like hemodynamic response during encoding in the left-frontal

207 10 W was able to identify abnormal pulmonary hemodynamic response during exercise (>3.0 mm Hg/L per m

208 is adequate to screen for abnormal pulmonary hemodynamic response during exercise.

209 To compare the hemodynamic response in septic shock patients receiving

210 participants in the experimental group, the hemodynamic response in the amygdala increased relative

211 function and distribution in the brain, the hemodynamic response informs us about the functionality

212 Learning to self-regulate the hemodynamic response involves a difficult temporal credi

213 we investigated the temporal dynamics of the hemodynamic response measured by fMRI as a potential cau

214 Monitoring the hemodynamic response of portal pressure (PP) to drug the

215 This occurred along with the stereotypical hemodynamic response of the SD wave.

216 normal human ventricular physiology and best hemodynamic response remains elusive.

217 riven downstream CNS drug effects; the brain hemodynamic response shows characteristic dose-related e

218 Z10419369 elicited a dose-dependent biphasic hemodynamic response that was related to the 5-HT1BR occ

219 d-masking task, which measures the amygdalar hemodynamic response to emotional faces presented for tr

220 ulations based on a biophysical model of the hemodynamic response to neuronal activity suggested that

221 training aimed at increasing the amygdala's hemodynamic response to positive memories in patients wi

222 rtfMRI-nf training to increase the amygdala hemodynamic response to positive memories significantly

223 rtfMRI-nf training to increase the amygdalar hemodynamic response to positive memories was associated

224 The authors sought to determine the acute hemodynamic response to sodium nitroprusside in LGSAS wi

225 ve strategy to screen for abnormal pulmonary hemodynamic response, but it is technically challenging,

226 defined as inter-subject correlations in the hemodynamic response, during natural verbal communicatio

227 l discharges were mapped using both EEG-fMRI hemodynamic responses and ESI.

228 ho are trained to upregulate their amygdalar hemodynamic responses during positive autobiographical m

229 The ERN, hemodynamic responses following errors, and intraindivid

230 nnectivity with the mOFC as well as stronger hemodynamic responses for ingroup versus outgroup decisi

231 correlation between task reaction times and hemodynamic responses from left LPFC during the incongru

232 We found distinctly different speech-related hemodynamic responses in the group of children who stutt

233 Thus, FGFR1 in the DT regulates systemic hemodynamic responses opposite to those predicted by the

234 r-infrared spectroscopy (fNIRS), we examined hemodynamic responses over neural regions integral to fl

235 l plasma flow (para-aminohippurate), BP, and hemodynamic responses to an infusion of angiotensin II (

236 ork in rats and C57BL/6J mice to examine the hemodynamic responses to CO2 and somatosensory stimulati

237 nction but markedly compromised compensatory hemodynamic responses to hemorrhage.

238 tagonist GR127935 did not elicit significant hemodynamic responses, even at a 5-HT1BR cerebral occupa

239 t relationship between activated neurons and hemodynamic responses, is a fundamental brain function t

240 eptor (5-HT1BR) occupancy and the associated hemodynamic responses.

241 Core temperature, hemodynamics, serum glucose and electrolytes, and P/F we

242 actional flow reserve (FFR), an index of the hemodynamic severity of coronary stenoses, is derived fr

243 calcification loads than men for the same AS hemodynamic severity.

244 ping heart valves and is dependent upon both hemodynamic shear forces and klf2a expression.

245 data also revealed the significant effect of hemodynamic shear stress on RBC-induced microvascular in

246 ributions of frequency component elements of hemodynamic shear stress waveforms encountered in cardio

247 Hemodynamic signals during periods of rest were weakly c

248 ndicating that ACh may alter the fidelity of hemodynamic signals in assessing changes in evoked neuro

249 Spontaneous fluctuations in hemodynamic signals in the absence of a task or overt st

250 we can separate voltage response signals and hemodynamic signals on live brain surfaces.

251 Spontaneous hemodynamic signals reflect a combination of behavior, l

252 nhanced neuronal responses and the resulting hemodynamic signals, chronic loss of cholinergic input r

253 records and 41 features from intraoperative hemodynamic signals.

254 The time to hemodynamic stabilization, the risk of catecholamine the

255 We assessed hemodynamic status at baseline and after a volume expans

256 Confounding factors linked to hemodynamic status can greatly influence the relationshi

257 Hemodynamic stress during hemodialysis (HD) results in r

258 ese individuals may be very sensitive to the hemodynamic stress of increased effective blood volume,

259 Invasive hemodynamic studies performed in a subset of patients de

260 ventricular delay (AVD), contributing to its hemodynamic superiority, and evaluated its applicability

261 -13 infusion delivers distinct and optimized hemodynamic support (including positive fluid balance),

262 In patients who required perioperative hemodynamic support after cardiac surgery, low-dose levo

263 It provides efficient hemodynamic support and survival rate after the implanta

264 older age, black race, chronic steroid use, hemodynamic support at HT, and being hospitalized at HT

265 ortic balloon pump, Impella provides greater hemodynamic support but no reduction in mortality.

266 tical Care Medicine "Clinical Guidelines for Hemodynamic Support of Neonates and Children with Septic

267 dicine provided 2002 and 2007 guidelines for hemodynamic support of newborn and pediatric septic shoc

268 In high-risk cases, temporary mechanical hemodynamic support tailored to the individual patient o

269 ial involving patients in whom perioperative hemodynamic support was indicated after cardiac surgery,

270 The impact of partial hemodynamic support with a microaxial percutaneous left

271 We tested the hypothesis that partial hemodynamic support with the Impella 2.5 microaxial pLVA

272 NYHA IV subjects required more hemodynamic support, were inducible for more and slower

273 ent of included patients had respiratory and hemodynamic supports at renal replacement therapy initia

274 demonstrate that general use of implantable hemodynamic technology in a nontrial setting leads to si

275 iac function as well as systemic and uterine hemodynamics that reduces uteroplacental blood flow, a m

276 erging optical method for recording cortical hemodynamics, to perform neuroimaging with this very you

277 ions can be safely extended according to the hemodynamic tolerance to ensure an adequate dose of dial

278 Population hemodynamic trends derived from nonlinear mixed-effects

279 Despite effective hemodynamic unloading and remodeling, there were no diff

280 The prognostic importance of hemodynamic values achieved after treatment initiation i

281 Similarly, TandemHeart improves hemodynamic variables but not survival.

282 te the prognostic importance of clinical and hemodynamic variables during follow-up, including pulmon

283 odel considering only baseline variables, no hemodynamic variables independently predicted prognosis.

284 Hemodynamic variables obtained by right heart catheteriz

285 Hemodynamic variables such as cardiac index and right at

286 ors of cardiovascular function, and estimate hemodynamic variables that cannot be assessed directly f

287 SVI and right atrial pressure were the hemodynamic variables that were independently associated

288 Pulmonary hemodynamic variables were collected at the time of PoPH

289 Hemodynamic variables, volume shifts, fluid balance, and

290 Hormonal, hemodynamic variations, and yet-undefined mechanisms mig

291 uded procedure-type risk category, number of hemodynamic vulnerability indicators, renal insufficienc

292 k categories and 6 independent indicators of hemodynamic vulnerability were identified.

293 data and expert opinion, as were markers of hemodynamic vulnerability.

294 lmonary function, gas exchange, and invasive hemodynamics were measured at 4-months post-banding.

295 Regional hemodynamics were measured over 4 d, before and after da

296 y, alterations in conjunctival microvascular hemodynamics were quantitatively assessed at stages of i

297 Hepatic hemodynamics were recorded after reperfusion.

298 Hemodynamics were sustained at 1-year follow-up.

299 impairs cardiovascular function and uterine hemodynamics with consequential fetal ischemia.

300 ved cardiac function as measured by invasive hemodynamics, without altering Ca(2+) spark frequency.