ライフサイエンスコーパス: hemodynamic instability

1 s typically presenting as abdominal pain and hemodynamic instability.

2 ardial infarction and death without inducing hemodynamic instability.

3 rs of end-organ dysfunction, and profiles of hemodynamic instability.

4 y in patients with sepsis, such as fever and hemodynamic instability.

5 using severe acidosis, renal impairment, and hemodynamic instability.

6 ated archiving method as early indicators of hemodynamic instability.

7 ta are limited in children and patients with hemodynamic instability.

8 tion of severe liver disease results in more hemodynamic instability.

9 en with cessation of neurologic function and hemodynamic instability.

10 in intrathoracic pressure can lead to severe hemodynamic instability.

11 c arrest (CA) have been low, presumably from hemodynamic instability.

12 mitted to the intensive care unit because of hemodynamic instability.

13 hypercapnia was associated with significant hemodynamic instability.

14 patients required immediate exploration for hemodynamic instability.

15 e due to hypercapnia, and seven secondary to hemodynamic instability.

16 did not translate into increased cardiac or hemodynamic instability.

17 baseline values, leading to life-threatening hemodynamic instability.

18 ion has been corroborated by data indicating hemodynamic instability.

19 fewer early terminations of sustained VT for hemodynamic instability (1.0 vs. 4.0; p = 0.001).

20 l treatment included poor prognosis (33.7%), hemodynamic instability (19.8%), death before surgery (2

21 The main reason for ICU admission was hemodynamic instability (58%), predominantly related to

22 r transplant recipients with coagulopathy or hemodynamic instability after allograft reperfusion.

23 In multivariate analysis hemodynamic instability, age >/= 75 years, history of st

24 riteria were CMV+ donors to CMV- recipients, hemodynamic instability, age >50, size mismatch (donor w

25 The incidence of clinical hemodynamic instability and bleeding complications tende

26 related to suggested detrimental effects on hemodynamic instability and enhanced oxidative stress.

27 clinical evaluation of patients for pain and hemodynamic instability and evaluation of MR images for

28 neonatal cardiac surgery, where pre-existing hemodynamic instability and metabolic abnormalities are

29 ve strategy to salvage patients with extreme hemodynamic instability and multiorgan injury.

30 They are prone to hemodynamic instability and must be monitored with vario

31 and the incidence of sepsis that can lead to hemodynamic instability and organ failure.

32 Hemodynamic instability and oxygenation failure as trach

33 tion in 10 patients; only those (n = 3) with hemodynamic instability and relatively low plasma argini

34 Addition of 15 mg/kg of DEA resulted in hemodynamic instability and thus DFT was not obtained.

35 grafts from suicidal hanging donors (without hemodynamic instability and with downward trend in the d

36 salvage patients with cardiac arrest, severe hemodynamic instability, and multiorgan failure results

37 of low clinical risk included no evidence of hemodynamic instability, arrhythmias or electrocardiogra

38 uence of RV infarction is thought to produce hemodynamic instability by reducing left ventricular (LV

39 Severe CRS was characterized by hemodynamic instability, capillary leak, and consumptive

40 enced an HBAT-related serious adverse event (hemodynamic instability characterized by bradycardia, ta

41 scuing patients from tamponade and reversing hemodynamic instability complicating invasive cardiac ca

42 edictors of adverse outcome were measures of hemodynamic instability, disease severity, demographics

43 irculatory support for patients experiencing hemodynamic instability due to myocardial infarction, ca

44 ic variables for early detection of imminent hemodynamic instability during progressive central hypov

45 s in brain white matter were associated with hemodynamic instability (higher mean arterial pressure e

46 owed significantly increased mortality, more hemodynamic instability, higher nitric oxide levels, and

47 ntly perceived barriers to mobilization were hemodynamic instability, hypoxemia, and dependency on ve

48 rs; range, 4.5-102 hours), and the cause was hemodynamic instability in 10 of the 12 patients (83.3%)

49 oninferiority trial to compare perioperative hemodynamic instability in 92 steroid-treated IBD patien

50 all risk of the Norwood operation and sudden hemodynamic instability in the intensive care unit.

51 currence of GM-IVH is highly associated with hemodynamic instability in the premature brain, yet the

52 Hemodynamic instability in the trauma patient is most co

53 t studies on the use of steroids in treating hemodynamic instability in these children.

54 on models resulted in the greatest degree of hemodynamic instability (mean [SD] arterial pressure dec

55 Fifty organ donors were evaluated for hemodynamic instability, (mean arterial pressure [MAP]</

56 oncentrations of stress hormones, as well as hemodynamic instability, occurred after brain death.

57 bleeding, and detected episodes of clinical hemodynamic instability occurring as long as 4 weeks aft

58 ium, or other systemic bleeding resulting in hemodynamic instability or blood transfusions.

59 rvived with lung injury combined with either hemodynamic instability or hepatic and renal failure.

60 rameters, including duration and severity of hemodynamic instability or hypoxia might be a better pre

61 Cooling was well tolerated, with no hemodynamic instability or increase in arrhythmia.

62 Patients with hemodynamic instability or liver dysfunction were exclud

63 on is indicated when the arrhythmia leads to hemodynamic instability or myocardial ischemia.

64 racheal intubations when the child had acute hemodynamic instability or oxygen failure and when the c

65 Patients were excluded for hemodynamic instability or significant renal or hepatic

66 No complications occurred, including hemodynamic instability or uncontrollable decreases in h

67 rdial infarction (OR = 2.5), CHF (OR = 2.4), hemodynamic instability (OR = 2.8), cardiopulmonary bypa

68 rmed in 44 cases because of complex anatomy, hemodynamic instability, or failed percutaneous coronary

69 ment groups in the prevalence of barotrauma, hemodynamic instability, or mucus plugging.

70 Multiple morphologies, hemodynamic instability, or noninducibility may limit ve

71 Hemodynamic instability, organ malperfusion, increasing

72 s affected by emergent status (P < .001) and hemodynamic instability (P = .04) but not by age, sex, b

73 Patients with hemodynamic instability, peritonitis, or an unevaluable

74 e short-term support to patients with severe hemodynamic instability, permit recovery of multiorgan i

75 The hospital course was complicated by hemodynamic instability, renal failure, pneumonia, and a

76 In 1 patient, hemodynamic instability required an intra-aortic balloon

77 of CPB for those patients with preoperative hemodynamic instability requiring a salvage CABG operati

78 tion, preoperative intraaortic balloon pump, hemodynamic instability, shock, intravenous nitroglyceri

79 14 (group I) had a cardiac arrest or severe hemodynamic instability (systolic blood pressure </=75 m

80 Women had a higher incidence of shock or hemodynamic instability than men (25% versus 17%, P<0.05

81 k fluid draining from the bronchial orifice, hemodynamic instability, thrombocytopenia, and coagulopa

82 Pediatric patients meeting criteria for hemodynamic instability underwent serial echocardiograms

83 criteria included patients with peritonitis, hemodynamic instability, unreliable physical examination

84 dysfunction, especially when associated with hemodynamic instability unresponsive to conventional tre

85 well tolerated in a normal heart often cause hemodynamic instability when they occur in the immediate

86 olam in two nonsurviving patients because of hemodynamic instability, which persisted despite the cha

87 ysaccharide administration induced transient hemodynamic instability without significant impact on mo