ライフサイエンスコーパス: hemodynamic stress

1 sentation of light stimuli before and during hemodynamic stress.

2 imulus-evoked responses were observed during hemodynamic stress.

3 sequential protocol simulating metabolic and hemodynamic stress.

4 ardiac function in response to metabolic and hemodynamic stress.

5 egrin knockout group after acute and chronic hemodynamic stress.

6 role of calpain in the heart in response to hemodynamic stress.

7 d adaptation of the cardiovascular system to hemodynamic stress.

8 d pathway regulating the cardiac response to hemodynamic stress.

9 the risk of aortic dissection by decreasing hemodynamic stress.

10 ts undergoing hypertrophic growth induced by hemodynamic stress.

11 iomyocyte survival in mice with both age and hemodynamic stress.

12 architectural response of the vasculature to hemodynamic stress.

13 diator of cardiac hypertrophy in response to hemodynamic stress.

14 e an important cardiac signaling pathway for hemodynamic stress.

15 rently by mechanisms unique to Ang II and by hemodynamic stress.

16 gestive heart failure: changes in perfusion, hemodynamic stresses, alterations in calcium metabolism,

17 pertrophy is a common response to injury and hemodynamic stress and an important harbinger of heart f

18 preserved cardiac function when subjected to hemodynamic stress and neurohormonal excess.

19 pha, the heart cannot respond effectively to hemodynamic stress and rapidly fails.

20 yocardial phospholipid remodeling induced by hemodynamic stress and reveal novel links between this p

21 The interaction of biomarkers of hemodynamic stress and the effects of ranolazine warrant

22 -directional velocity encoding for assessing hemodynamic stresses and corresponding blood damage inde

23 and serum biomarkers of collagen metabolism, hemodynamic stress, and myocardial injury to evaluate su

24 and serum biomarkers of collagen synthesis, hemodynamic stress, and myocardial injury were also avai

25 Physical forces of gravity, hemodynamic stresses, and movement play a critical role

26 urin inhibition by MCIP1 under conditions of hemodynamic stress are unknown.

27 e in HFpEF but not controls, suggesting that hemodynamic stresses beyond passive stiffness and increa

28 bility of the elastic vessel wall to sustain hemodynamic stress by disrupting microfibrillar assembly

29 During hemodynamic stress, catecholamines and neurohumoral stim

30 Because acute hemodynamic stress caused by intravenous infusion of sod

31 that CRF-mediated increases in LC-NE due to hemodynamic stress disrupts the transmission of informat

32 The aorta is exposed to hemodynamic stress during exercise, but whether or not t

33 Hemodynamic stress during hemodialysis (HD) results in r

34 kinase expression both at baseline and after hemodynamic stress; focal adhesion kinase expression was

35 ific inhibition of SGK1 protected mice after hemodynamic stress from fibrosis, heart failure, and sod

36 ble for medial remodeling, and resolves once hemodynamic stresses have normalized without obvious int

37 tivity by physiological manipulations (i.e., hemodynamic stress, hypovolumia) remains unclear.

38 n HF through increased sympathetic activity, hemodynamic stress, hypoxemia, and oxidative stress.

39 markers of subclinical myocardial injury and hemodynamic stress identify asymptomatic individuals wit

40 tion; 3) increased mortality following acute hemodynamic stress imposed by transverse aortic constric

41 homeostatic arterial remodeling triggered by hemodynamic stress in mice and possibly in humans as wel

42 derlies the impact of sleep abnormalities on hemodynamic stress in patients with HF.

43 or by responding to its local environment of hemodynamic stresses, in particular, shear stress.

44 Hormone- and hemodynamic stress-induced GRK5 regulation may provide a

45 Intima-media thickening (IMT) in response to hemodynamic stress is a physiological process that requi

46 perative management with goals of decreasing hemodynamic stress is important in patients with ischemi

47 biomarkers of subclinical cardiac injury and hemodynamic stress modify the association of LVH with ad

48 We hypothesized that the hemodynamic stress of increased arterial pressure could

49 ese individuals may be very sensitive to the hemodynamic stress of increased effective blood volume,

50 Superimposition of the hemodynamic stress of pressure overload on G alpha q ove

51 egulation of RAS genes is a direct effect of hemodynamic stress or is secondary to neurohumoral effec

52 have evidence of ongoing myocardial injury, hemodynamic stress, or systemic inflammation.

53 at myosin heavy chain composition depends on hemodynamic stress rather than on FGF2 or hypertrophy, a

54 -CMs survival under physiologically relevant hemodynamic stress requires gradual imposition of mechan

55 at protein aggregation occurs in response to hemodynamic stress, situating pressure-overload heart di

56 ndicate that calpain protects the heart from hemodynamic stresses, such as pressure overload.

57 DM, and MR-proANP are emerging biomarkers of hemodynamic stress that have been associated with advers

58 complications, particularly with the altered hemodynamic stresses that LT patients face in the immedi

59 fferences between the RV and LV responses to hemodynamic stress, the unique stressors on the RV assoc

60 Vascular remodeling normalizes abnormal hemodynamic stresses through structural changes affectin

61 We quantified cardiac hemodynamic stress using B-type natriuretic peptide (BNP

62 Hemodynamic stress via hypotensive challenge has been sh

63 ate the effects of a physiological stressor [hemodynamic stress via sodium nitroprusside (SNP) i.v.]

64 Intradialytic hemodynamic stress was quantified using the extrema poin

65 Saphenous vein grafts are exposed to hemodynamic stress when interposed in the arterial circu

66 ta support the value of combining markers of hemodynamic stress with traditional approaches to risk a