1 the extent of oxidant attack on the unstable hemoglobin E.

2                                              Hemoglobin E beta thalassemia is the commonest form of s

3 r, the very common subgroup of patients with hemoglobin E beta-thalassemia that makes up approximatel

4                                              Hemoglobin E/beta thalassemia is now a worldwide clinica

5 even-year-old man with transfusion dependent hemoglobin E/beta-thalassemia disease was treated with h

6  appreciation that double heterozygosity for hemoglobin E/beta-thalassemia, while causing variable an

7 llels between the heterotropic regulation of hemoglobin (e.g., the Bohr effect) and the roles of nucl

8 ily is described in which the genes for both hemoglobin E (Hb E) and pyrimidine 5' nucleotidase defic

9                                              Hemoglobin E (HbE) beta-thalassemia is the most common s

10 e have studied two comparable populations of hemoglobin E (HbE)/beta thalassemics, one regularly tran

11 ed the Hbb(th3/+) beta-thalassemia mouse and hemoglobin E (HbE)/beta-thalassemia patients to investig

12                                              Hemoglobin E (HbE; alpha2beta226glu-lys), globally the c

13 with sickle cell anemia and in subjects with hemoglobin E or beta thalassemia trait from Thailand and