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1 elices that are biologically promiscuous and hemolytic.
4 protein, a molecular mass of 2.4kDa and low hemolytic activity (<50% hemolysis of human erythrocytes
6 zole hybrids, their time-kill studies, their hemolytic activity against murine erythrocytes, as well
7 also has glycan-binding properties and that hemolytic activity against RBCs can be blocked with the
10 is responsible for the previously described hemolytic activity associated with the choA orthologue i
13 up, the corresponding decrease in complement hemolytic activity distinguished survivors from nonsurvi
15 umolysin, which is associated with increased hemolytic activity in ST3081 and may contribute to incre
16 ases in the DeltavfrB mutant did not restore hemolytic activity in the DeltavfrB mutant on blood agar
18 nding that Rv0888 accounts for half of Mtb's hemolytic activity is consistent with its sphingomyelina
24 in Cytolysin A (ClyA) is responsible for the hemolytic activity of various Escherichia coli and Salmo
25 gative and Gram-positive bacteria, while its hemolytic activity remained around 10% or less, even aft
26 osteoblast and Caco-2 epithelial cells, and hemolytic activity was assessed on rabbit erythrocytes a
28 e than 1000 phosphate units suppressed total hemolytic activity with a concentration to reduce maxima
29 e polar, uncharged subunits can diminish the hemolytic activity with minimal impact on the antibacter
30 s with eukaryotic cells, as evidenced by low hemolytic activity, and protects the polypeptide backbon
32 n of the lectin domain seriously compromises hemolytic activity, it is thought that carbohydrate-depe
41 ate that exposure of cultured macrophages to hemolytic aged red blood cells, heme, or iron causes the
43 ts reveal that VG16KRKP, a non-toxic and non-hemolytic analogue of VG16, shows significant antimicrob
46 how that the monomeric preparation displayed hemolytic and cytotoxic activities suggesting that the m
48 ium and Lactobacillus species), and were non-hemolytic and non-toxic to mammalian cell lines HepG2 an
49 y compared to W4A9, as quantified via ELISA, hemolytic, and cell-based assays, and showed improved so
50 activities, along with resistance/enzymatic, hemolytic, and cytotoxicity assays were also studied.
51 plasma as well as being hemocompatible (non-hemolytic, and poor activator of the complement system).
52 ve hemophagocytic syndrome (44%), autoimmune hemolytic anemia (33%), and lymphoma (22%) were the comm
53 ively investigated in 308 primary autoimmune hemolytic anemia (AIHA) cases and correlated with serolo
55 The development of warm-antibody autoimmune hemolytic anemia (also known as warm autoimmune hemolyti
57 zed by thrombocytopenia and microangiopathic hemolytic anemia (MAHA) without an obvious cause, and ma
59 olytic anemia (also known as warm autoimmune hemolytic anemia [WAHA]) in patients with babesiosis has
60 l pathways that can lead to microangiopathic hemolytic anemia and a procoagulant state with or withou
62 ll lymphoproliferation results in autoimmune hemolytic anemia and cold-induced circulatory symptoms.
63 ited blood disorder characterized by chronic hemolytic anemia and episodic vaso-occlusive pain crises
65 substantial improvement of sickle-associated hemolytic anemia and reticulocytosis, key pathophysiolog
67 ce was reflected in reduced pancreatitis and hemolytic anemia and this was associated with distinct c
69 phlebotomy-induced acute anemia and chronic hemolytic anemia because of 4.1R deficiency, the ratio o
70 ies to treat not only chronic nonspherocytic hemolytic anemia caused by severe G6PD variants, but als
71 is (HX; MIM 194380) is an autosomal-dominant hemolytic anemia characterized by primary erythrocyte de
72 lmonary hypertension associated with chronic hemolytic anemia has been moved from Group 1 PAH to Grou
73 eferred second-line therapy of warm antibody hemolytic anemia in adults, although no prospective stud
75 cold agglutinin disease (CAD), an autoimmune hemolytic anemia in which autoantibodies (cold agglutini
77 f red blood cells in human subjects, causing hemolytic anemia linked to impaired nicotinamide adenine
78 component of the management of warm antibody hemolytic anemia not only after relapse but as soon as t
79 Finally, we chemically induce hypoxemia via hemolytic anemia resulting in HIF stabilization within c
81 genus babesia, is characterized by nonimmune hemolytic anemia that resolves with antimicrobial treatm
82 -phosphate dehydrogenase, exhibit non-immune hemolytic anemia upon exposure to aspirin and various an
84 eficiency are at risk for the development of hemolytic anemia when given 8-aminoquinolines (8-AQs), a
85 cantly impaired erythropoietic recovery from hemolytic anemia, acute blood loss and myeloablation.
86 ll spectrum HUS, including thrombocytopenia, hemolytic anemia, and AKI with glomerular thrombotic mic
88 ing preexisting TTP signs; thrombocytopenia, hemolytic anemia, and organ damage could not be reversed
89 ection-site erythema and pyrexia, autoimmune hemolytic anemia, and suspected lack of vaccine efficacy
90 at emerge on erythrocytes lead to autoimmune hemolytic anemia, and the causative auto-IgM cross-react
91 in the liver also occurs in mouse models of hemolytic anemia, anemia of inflammation, and sickle cel
92 c disorder diagnosed by thrombocytopenia and hemolytic anemia, associated with a deficiency in von Wi
93 a gene mutated in Mendelian non-spherocytic hemolytic anemia, associated with HGB and HCT (SKAT p <
94 ong descriptive diagnoses such as autoimmune hemolytic anemia, chronic immune thrombocytopenia, Evans
95 immune thrombocytopenia and warm autoimmune hemolytic anemia, circulating immunoglobulin G (IgG)-ops
96 vere genetic blood disorder characterized by hemolytic anemia, episodic vaso-occlusion, and progressi
97 ng numerous clinical settings such as severe hemolytic anemia, infection, tissue injury, or blood tra
99 ets, which account for the thrombocytopenia, hemolytic anemia, schistocytes, and tissue injury that c
100 angiopathy characterized by microangiopathic hemolytic anemia, severe thrombocytopenia, and organ isc
101 tic link between the induction of autoimmune hemolytic anemia, the reduction in naive T cells, and po
102 remic syndrome is a disease characterized by hemolytic anemia, thrombocytopenia and acute renal failu
103 sel thrombosis resulting in microangiopathic hemolytic anemia, thrombocytopenia and renal failure.
104 surface level that leads to microangiopathic hemolytic anemia, thrombocytopenia, and acute kidney fai
107 hy that is characterized by microangiopathic hemolytic anemia, thrombocytopenia, and renal failure.
108 marrow failure disorder that manifests with hemolytic anemia, thrombosis, and peripheral blood cytop
120 human AE1 cause several types of hereditary hemolytic anemias and/or distal renal tubular acidosis.
121 ematocrit, sickle cell disease, thalassemia, hemolytic anemias, and malaria, with both arterial and v
125 otective function of FH in sheep erythrocyte hemolytic assays and increase cell-surface C3b depositio
127 gram-negative bacilli and regular, grey non-hemolytic colonies appearing the next day on blood agar.
128 l activity of C5 can be screened using a 50% hemolytic complement (CH50) assay, which is sensitive to
129 , TMPMs that were devoid of any demonstrable hemolytic, cytotoxic, and antifungal activities chemosen
131 aniofacial anomalies, chromosomal disorders, hemolytic disease, multiple gestation, third-trimester m
133 Hemopexin protects against heme toxicity in hemolytic diseases and conditions, sepsis, and sickle ce
136 he effects of free heme and free iron during hemolytic diseases such as sickle cell disease, dengue f
137 es (MPs) concentrate cell-free heme in human hemolytic diseases, and that heme-laden MPs have a physi
139 inemia cases in newborns are associated with hemolytic diseases, we emphasize here the impaired abili
140 due to malaria, sickle cell disease or other hemolytic diseases, will enable identification of high-r
142 . Dacie was known for his pioneering work on hemolytic disorders, especially paroxysmal nocturnal hem
145 d in a 2.5- to 4.8-fold increase in the anti-hemolytic effect but this was undetected in chemical ass
150 s (GBS) or Streptococcus agalactiae are beta-hemolytic gram-positive bacteria that colonize the lower
151 cci (GBS; Streptococcus agalactiae) are beta-hemolytic, Gram-positive bacteria that are common asympt
152 (GBS) or Streptococcus agalactiae is a beta-hemolytic, Gram-positive bacterium that is a leading cau
153 ity to detect Streptococcus group A and beta-hemolytic groups C/G using rapid antigen-negative pharyn
154 ta provide novel insights into mechanisms of hemolytic inflammation and further support perspectives
155 flammatory effects by inhibiting rapid-onset hemolytic inflammation via an NO-dependent mechanism, in
158 ed that, irrespective of the stress-induced (hemolytic or post-erythropoietin [Epo]) treatment, only
161 changed the PLYa of ST8 strain 6308 with the hemolytic PLY (PLYh) of ST3 A66.1 and vice versa and det
167 aim of improving antimicrobial activity and hemolytic properties, we use these reactivity difference
169 TSAR2 overexpression specifically boosted hemolytic saponin biosynthesis, whereas TSAR1 overexpres
170 ca associated with a different strongly beta-hemolytic spirochete that has been molecularly and pheno
172 tis occurs more frequently than group A beta-hemolytic streptococcal-positive pharyngitis in a studen
173 The Lyra assay correctly detected all beta-hemolytic streptococci (group A, n = 19; group C/G, n =
175 r cellulitis without purulent drainage, beta-hemolytic streptococci are presumed to be the predominan
183 st promising compounds were found to be less hemolytic than the FDA-approved antifungal agent voricon
189 antibodies against RBC non-ABO Ags can cause hemolytic transfusion reactions and limit availability o
190 s because p erythrocytes may pose a risk for hemolytic transfusion reactions due to their elevated PX
193 increase the probability of life-threatening hemolytic transfusion reactions, not all patients genera
197 inhibition therapy in patients with atypical hemolytic uremic syndrome (aHUS) are remarkable in contr
198 in, have been well characterized in atypical hemolytic uremic syndrome (aHUS) but have been less well
200 escribe an assay that could convert atypical hemolytic uremic syndrome (aHUS) from a diagnosis of exc
201 eneic HSCT shares similarities with atypical hemolytic uremic syndrome (aHUS) in the underlying patho
216 ions in complement C3 identified in atypical hemolytic uremic syndrome (aHUS) patients cause dysregul
217 he thrombotic microangiopathy (TMA) atypical hemolytic uremic syndrome (aHUS) resulted in the success
218 iseases C3 glomerulopathy (C3G) and atypical hemolytic uremic syndrome (aHUS) strongly associate with
219 ave been described in patients with atypical hemolytic uremic syndrome (aHUS), a rare condition chara
221 h was described in association with atypical hemolytic uremic syndrome (aHUS), also confers high risk
222 ed CFH, whereas R53H-CFH, linked to atypical hemolytic uremic syndrome (aHUS), was defective in C3bBb
226 hrombotic thrombocytopenic purpura (TTP) and hemolytic uremic syndrome (HUS) are appropriately at the
228 104:H4 caused an outbreak with >800 cases of hemolytic uremic syndrome (HUS) in Germany, including 90
233 e offending agent of postdiarrhea-associated hemolytic uremic syndrome (HUS), a disorder of glomerula
234 tion status, the development and severity of hemolytic uremic syndrome (HUS), and adverse outcomes in
235 e progresses to hemorrhagic colitis (HC) and hemolytic uremic syndrome (HUS), due to the expression o
236 role in the pathogenesis of postenteropathic hemolytic uremic syndrome (HUS), most commonly caused by
237 moxetumomab pasudotox for 10 doses developed hemolytic uremic syndrome (HUS), thrombotic microangiopa
244 to severe renal diseases, including atypical hemolytic uremic syndrome and C3 glomerulopathies, and a
245 associated with the renal diseases atypical hemolytic uremic syndrome and dense deposit disease and
246 lation-mediated endothelial damage: atypical hemolytic uremic syndrome and thrombotic thrombocytopeni
247 the basis of clinical presentation (atypical hemolytic uremic syndrome as thrombotic microangiopathy)
248 afety of anticomplement therapy for atypical hemolytic uremic syndrome during pregnancy, and implicat
251 FHR) genes and autoantibody-positive form of hemolytic uremic syndrome is characterized by the presen
253 011 Shiga toxin-producing E. coli-associated hemolytic uremic syndrome outbreak in Germany, critical
259 tations in FH and MCP are linked to atypical hemolytic uremic syndrome, a type of thrombotic microang
260 g age-related macular degeneration, atypical hemolytic uremic syndrome, and C3 glomerulopathies.
261 e-specific diseases such as AMD and atypical hemolytic uremic syndrome, and leads to a better underst
262 these genes have been described in atypical hemolytic uremic syndrome, arising commonly through nona
263 nocturnal hemoglobinuria (PNH) and atypical hemolytic uremic syndrome, blocks the terminal complemen
264 sent in patients with the autoimmune form of hemolytic uremic syndrome, is involved in B cell regulat
266 a spectrum of rare diseases such as atypical hemolytic uremic syndrome, thrombotic thrombocytopenic p
267 Compared with wild type FH19-20, atypical hemolytic uremic syndrome-associated mutants were less a
268 h preventive or therapeutic ends, for use in hemolytic uremic syndrome-endemic areas or during future
287 family of cytotoxic proteins that can cause hemolytic-uremic syndrome (HUS), a thrombotic microangio
289 ions, including hemorrhagic colitis (HC) and hemolytic-uremic syndrome (HUS), which is the most commo
292 ith defective regulation of the AP (atypical hemolytic-uremic syndrome) or with inadequate cleavage b
293 ding thrombotic thrombocytopenic purpura and hemolytic-uremic syndrome, have been reported to have a
299 erized member of a novel lipase family, is a hemolytic virulence factor that exhibits the most promin
300 ghly active against bacteria but also highly hemolytic, we found that replacing a small proportion of
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