1 s characterized by complement deposition and hemorrhagic necrosis.

2 lands harvested for histologic evaluation of hemorrhagic necrosis.

3 ytokines and attenuating liver apoptosis and hemorrhagic necrosis.

4 panied by microvascular injury manifested by hemorrhagic necrosis and a precipitous drop in blood pla

5 eing outbred and that it is characterized by hemorrhagic necrosis and neutrophilic vasculitis in the

6 LSECs at 24 hours, followed by centrilobular hemorrhagic necrosis at 48 hours.

7 ed T-cell-independent and TNFalpha-dependent hemorrhagic necrosis at early times, followed by later C

8 on of this extracellular trypsinogen induces hemorrhagic necrosis in a setting of mild edematous panc

9               Adrenal microscopy showed 1.5+ hemorrhagic necrosis in experimental animals compared wi

10  CA4P-induced vessel narrowing, hypoxia, and hemorrhagic necrosis in murine mammary tumors were accom

11 a pigs developed lethal lung infections with hemorrhagic necrosis, massive bacterial replication in t

12                                      Massive hemorrhagic necrosis (MHN) of the liver following orthot

13 terleukin 1 alpha (IL-1 alpha) induces acute hemorrhagic necrosis, microvascular injury, and enhanced

14 e levels (a marker of hepatocyte injury) and hemorrhagic necrosis of the liver, in dramatic contrast,

15 bolite, diminution of tumor vasculature, and hemorrhagic necrosis of the tumor.

16 nventional chemotherapeutic drugs, extensive hemorrhagic necrosis of tumors often developed within 24

17 et tumor-associated vasculature resulting in hemorrhagic necrosis of tumors without injury to surroun

18  spinal cord injury (SCI) causes progressive hemorrhagic necrosis (PHN), a poorly understood patholog

19 f these mice died from massive apoptosis and hemorrhagic necrosis, whereas all wild-type mice survive