ライフサイエンスコーパス: hemorrhaging

1 rs of nonfunctional blood vessels and severe hemorrhaging.

2 uito vectors, can cause vascular leakage and hemorrhaging.

3 ompanied by reduced pericyte recruitment and hemorrhaging.

4 ved the infection and had little or no brain hemorrhaging.

5 progenitors also caused widespread cerebral hemorrhaging.

6 s of vascular integrity leading to localized hemorrhaging.

7 and E14.5, because of severe intraembryonic hemorrhaging.

8 extensive pulmonary vasculitis and alveolar hemorrhaging.

9 s, both of which express Fli1 at the time of hemorrhaging.

10 tributing to a microenvironment conducive to hemorrhaging.

11 om severe intra-embryonic and intra-amniotic hemorrhaging.

12 from loss of BBB integrity and intracerebral hemorrhaging.

13 ain barrier dysfunction, brain swelling, and hemorrhaging accompanied by accumulation of parasite-spe

14 obal knockouts did not demonstrate embryonic hemorrhaging, altered mural cell development, or lethali

15 disorder of humans causing hypopigmentation, hemorrhaging and early death due to lung abnormalities.

16 mphatic hypervascularization associated with hemorrhaging and edema in the jugular region; a phenotyp

17 e exhibited subcutaneous and intraperitoneal hemorrhaging and impaired viability.

18 transgenic mice leads to partial lethality, hemorrhaging, and altered cardiac morphogenesis.

19 to produce megakaryocytes, display extensive hemorrhaging, and die by embryonic day 14.5.

20 acks structural integrity, leading to edema, hemorrhaging, and late stage embryonic lethality.

21 es, such as severe emaciation, skin lesions, hemorrhaging, and more zebrafish death.

22 fferent between when the patient is actively hemorrhaging, and once bleeding has been controlled.

23 ion of miR-126 in mice causes leaky vessels, hemorrhaging, and partial embryonic lethality, due to a

24 lung tissues demonstrated severe alveolitis, hemorrhaging, and spread of the virus throughout the ent

25 tastasis; 1 was a 2.4-cm lesion with central hemorrhaging, and the remaining 3 were lesions of less t

26 a, delayed rotation of the embryo, placental hemorrhaging, and visceral arch abnormalities.

27 s with active extravasation, the size of the hemorrhaging area was measured on arterial, portal venou

28 adverse inflammatory reactions and cerebral hemorrhaging associated with the treatments.

29 Tmod3(-/-) embryos often show hemorrhaging at E14.5 with fewer and larger platelets, i

30 deficiency in the CNC resulted in enlarged, hemorrhaging branchial arch blood vessels and hydrocepha

31 These animals are spared the hemorrhaging caused by COX inhibitors in the gut, where

32 oglin in the neural crest caused pericardial hemorrhaging, correlating with altered vascular smooth m

33 licates in the brain, where severe edema and hemorrhaging follow.

34 d in a model of blast trauma with multiorgan hemorrhaging, i.v. administration of the hemostatic nano

35 We observed hemorrhaging in fga mutants and reduced survival compare

36 tion that cyclosporin A abrogated intestinal hemorrhaging in HEV-infected turkeys strongly suggested

37 stachylysin may be one agent responsible for hemorrhaging in humans.

38 Here we describe aberrant hematopoeisis and hemorrhaging in mouse embryos homozygous for a targeted

39 C-type-lectin-like receptor (CLEC-2) exhibit hemorrhaging in the brain at mid-gestation.

40 between embryonic days 11.5 and 12.5 due to hemorrhaging in the central nervous system (CNS), at the

41 nock-in mice died in midgestation because of hemorrhaging in the central nervous system and a block o

42 n embryonic lethality at midgestation due to hemorrhaging in the central nervous system and blocks fe

43 n embryonic lethality at midgestation due to hemorrhaging in the central nervous system, and severely

44 nt lesion of this type that caused recurrent hemorrhaging in the lower part of the gastrointestinal t

45 CNS damage, which was preceded by petechial hemorrhaging in the optic lobes.

46 sculature can result in vascular leak and/or hemorrhaging into the brain; similar processes can occur

47 Hemorrhaging is preceded by symmetric, bilateral necrosi

48 tial failure in TF-/- embryos appeared to be hemorrhaging, leading to the leakage of embryonic red ce

49 abulated are edema, blood vessel enlargement/hemorrhaging, macroglossia, retention of nucleated eryth

50 bladder wet weight 2-fold and caused severe hemorrhaging of the bladder.

51 lumenal fluid accumulation followed by focal hemorrhaging of the ileum and adrenal glands.

52 Hemorrhaging of these lesions can cause seizures and let

53 quire resuscitation, either because they are hemorrhaging or because they are experiencing significan

54 ice for patients with neither obstructed nor hemorrhaging primary colorectal tumors in the setting of

55 presentation of infection included petechial hemorrhaging, redness of the abdomen, and erratic swim b

56 Hemorrhaging was also seen in the ventricles of embryos

57 In addition, sub-pleural hemorrhaging was evident as early as E14.5 in HIF1a TPM

58 rentiation, partial embryonic lethality, and hemorrhaging were observed after analysis of 2 lines of

59 ed acute embryonic lethality at E11.5 due to hemorrhaging, whereas retention of a single p85alpha all

60 ing skin disorder in neonatal epidermis, and hemorrhaging within the embryonic liver.