ライフサイエンスコーパス: hemosiderin

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1 epatocytes that contained greater amounts of hemosiderin.

2 as due to ferritin; there was no evidence of hemosiderin, a ferritin decomposition product.

3 no abnormal susceptibility and contained no hemosiderin at tissue analysis.

4 ses and a striking increase in the number of hemosiderin-containing macrophages is observed associate

5 -type levels) exhibited a similar pattern of hemosiderin deposition and fibrosis in their hearts.

6 d a selective heart defect that consisted of hemosiderin deposition and fibrosis.

7 This destructive process is characterized by hemosiderin deposition in the superficial and deeper lay

8 In two of 27 cases, parenchymal hemosiderin deposition led to an increase in LAI into th

9 y those with both fatty liver and coexistent hemosiderin deposition or radiologically occult diffuse

10 ceptor expression and increased splenic iron/hemosiderin deposition.

11 racteristics of human CCM lesions, including hemosiderin deposits, immune cell infiltration, increase

12 nomenon, distended vessels, damaged vessels, hemosiderin deposits, vessel tortuosity, and microaneury

13 g concomitant Abeta immunohistochemistry and hemosiderin detection.

14 itochondrial and blood doublets and included hemosiderin features.

15 Hemosiderin foci on brain magnetic resonance imaging wer

16 The macrophages stain for hemosiderin, indicating that diffuse alveolar hemorrhage

17 models could be differentiated according to hemosiderin iron accumulation-both in tumors and systemi

18 Routine detection of hemosiderin iron aggregates in macrophages in other sett

19 rogressively bloodier return and/or over 20% hemosiderin-laden macrophages in diffuse alveolar hemorr

20 Hemosiderin-laden macrophages were abundant in the lungs

21 car and reactive changes, mainly presence of hemosiderin-laden macrophages.

22 Furthermore, hemosiderin-laden trophoblasts were more frequent in the

23 nion exchange) and Mallory's stain for iron (hemosiderin), markers of previous intraplaque hemorrhage

24 uggested that the predominant iron in PSP is hemosiderin, not ferritin.

25 r Development Index score (P=0.02) and brain hemosiderin (P=0.04) remained significantly associated w

26 the putamen consistent with accumulation of hemosiderin (posterior portion) and neuromelanin (remain

27 ges all decreased over time, suggesting that hemosiderin products undergo continued, subtle evolution

28 It lacks the "hemosiderin rim" of cavernous angioma and demonstrates i

29 The formation of hemosiderin was also observed.

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