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1 itor functions, such as antithrombin III and heparin cofactor II.
2 or activity and is an alternate modulator of heparin cofactor II.
3 agulant properties, with activity mainly via heparin cofactor II.
4 f the aPTT requires neither antithrombin nor heparin cofactor II.
5 n with thrombomodulin, hirudin, rhodniin and heparin cofactor II.
6 ara et al. report that haploinsufficiency of heparin cofactor II, a glycosaminoglycan-dependent throm
7 -IV, C-I and M, histidine-rich glycoprotein, heparin cofactor II and attractin, which are involved in
8 g activity both by stimulating inhibition by heparin cofactor II and directly competing with fibrinog
11 C1 inhibitor, alpha(1)-antichymotrypsin, and heparin cofactor II cause a similar conformational trans
12 ith plasma immunodepleted of antithrombin or heparin cofactor II confirm that prolongation of the thr
13 ibitor (TFPI), antithrombin-III (AT-III) and heparin cofactor-II (HC-II) on thrombin generation were
14 Inactivation of thrombin (T) by the serpins heparin cofactor II (HCII) and antithrombin (AT) is acce
16 the second acidic region (AR2) of the serpin heparin cofactor II (HCII) during formation of the throm
18 on by the serine protease inhibitor (serpin) heparin cofactor II (HCII) in the absence and presence o
27 ycan (GAG) sequences that selectively target heparin cofactor II (HCII), a key serpin present in huma
28 re known to stimulate thrombin inhibition by heparin cofactor II (HCII), a plasma proteinase inhibito
29 hibitors including antithrombin III (ATIII), heparin cofactor II (HCII), alpha2-macroglobulin (alpha2
30 ypothesis that the corresponding sequence in heparin cofactor II (HCII), FLFLI (residues 456-460), me
31 ivation of alpha-thrombin (T) by the serpin, heparin cofactor II (HCII), is accelerated by ternary co
32 Thrombin (T) inactivation by the serpin, heparin cofactor II (HCII), is accelerated by the glycos
35 aled that antithrombin III (ATIII)-thrombin, heparin cofactor II (HCII)-thrombin, and alpha1-antitryp
36 rough direct and indirect (antithrombin- and heparin cofactor II-mediated) inhibition of thrombin.
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