ライフサイエンスコーパス: heparin lyase

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1 ding constituent disaccharides, by bacterial heparin lyases.

2 ulfated heparan sulfate that was digested by heparin lyases.

3 ated tetrasaccharide isolated from bacterial heparin lyase 1 digests of heparin that contains a Delta

4 Complete heparin digestion with heparin lyase 2 affords a mixture of disaccharides and r

5 Heparin lyases are valuable tools for generating oligosa

6 s spectrometry (LC-MS) methods for profiling heparin lyase decomposition products have been shown.

7 eparin by its partial depolymerization using heparin lyase I (EC 4.2.2.7) in an attempt to prepare ol

8 enzymatic digestion of cell surface GAGs by heparin lyase I and heparin lyase III but not chondroiti

9 Heparin oligosaccharides, prepared using heparin lyase I were SLPI-affinity fractionated.

10 t solutions to remove lipids before applying heparin lyases I, II, and III on the tissue surfaces wit

11 Treatment of SMCs with heparin lyases II and III, but not with chondroitin ABC

12 Heparin lyase III (Hep III) effectively catalyzes the he

13 the quantitative detection of heparosan with heparin lyase III and spectrophotometry that is safer an

14 of cell surface GAGs by heparin lyase I and heparin lyase III but not chondroitinase ABC resulted in

15 olysaccharide was sensitive to the action of heparin lyase III but resistant to hyaluronan lyase.

16 metry of the oligosaccharides obtained after heparin lyase III digestion of the polysaccharide indica

17 Heparin lyase III is known to cleave the linkages betwee

18 Here we investigate the specificity of heparin lyase III toward the GlcN-GlcA linkage using a r

19 apsule was removed by treating microbes with heparin lyase III.

20 from heparin and heparan sulfate (HS) using heparin lyases is described.

21 Genetic or heparin lyase-mediated alteration of endothelial heparan

22 Heparin lyases released disaccharides bearing N-, 2-O-,

23 on but dramatically alter the composition of heparin lyase-released disaccharides.

24 or- and integrin-dependent pathways, whereas heparin lyase-treated and beta1 integrin-null cells exhi

25 y induced the spreading of SMCs, whereas the heparin lyase treatment of PDGF-AA-stimulated cultures d

26 ulfate chains with nitrous acid or bacterial heparin lyases, which cut the chain at specific sequence

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