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1 those antibodies were employed to isolate a heparin binding protein.
2 ment leads to degranulation and secretion of heparin-binding protein.
3 e serine proteinase inhibitor (serpin) and a heparin-binding protein.
4 (PF4) is an abundant platelet alpha-granule heparin-binding protein.
5 lly related, extracellular matrix-associated heparin-binding proteins.
6 inity comparable to that of other identified heparin-binding proteins.
7 alpha-Chemokines are known heparin-binding proteins.
10 racellular matrix protein tenascin-R and two heparin-binding proteins, amphoterin and the heparin-bin
11 We have reported previously that Noggin is a heparin-binding protein and associates with the cell sur
12 recently showed that thyroglobulin (Tg) is a heparin-binding protein and that heparin inhibits bindin
13 vestigated, for example, as aptamers against heparin-binding proteins and as purine-motif triplex-for
14 bility-increasing protein, azurocidin (CAP37/heparin-binding protein), and neutrophil elastase were e
15 its target protease kallikrein 7 (KLK7) are heparin-binding proteins, and inhibition of KLK7 by vasp
19 eparin release, we demonstrated that Tg is a heparin-binding protein, as are several megalin ligands.
20 ple ionophores to model recognition sites of heparin-binding proteins at liquid/liquid interfaces.
21 peptide sequence analysis revealed that this heparin-binding protein corresponded to the extravesicul
23 phosphodiester oligonucleotides can bind to heparin-binding proteins (eg, basic fibroblast growth fa
26 udies have shown that the neutrophil-derived heparin-binding protein (HBP), also known as CAP37 or az
28 tivated polymorphonuclear leukocytes release heparin-binding protein (HBP; also known as CAP37 or azu
29 nding sites for a diverse array of mammalian heparin binding proteins, including adhesive glycoprotei
30 ents presenting at the emergency department, heparin-binding protein is an early indicator of infecti
31 sic fibroblast growth factor (bFGF), another heparin-binding protein is increased in Alzheimer's dise
33 o wounding was demonstrated by the fact that heparin-binding proteins isolated from wounded, but not
34 f the latter patients had an elevated plasma heparin-binding protein level (>30 ng/mL) prior to devel
35 n polarized myeloma cells are incubated with heparin-binding proteins, like hepatocyte growth factor
36 studies indicated that Tia, as a prokaryotic heparin binding protein, may also interact via sulfated
44 atform is used to assess binding of specific heparin-binding protein targets at very high sensitivity
45 We demonstrate that this holds true for all heparin binding proteins tested but not for epidermal gr
46 way was able to interact with four different heparin-binding proteins tested, i.e., TSG-6, chemokines
47 early gene Cyr61, a secreted, cysteine-rich, heparin binding protein that promotes endothelial cell a
48 ed Cyr61 protein is a secreted, cystein-rich heparin-binding protein that associates with the cell su
49 rly gene, CYR61 is a secreted, cysteine-rich heparin-binding protein that associates with the extrace
50 Cyr61 is an extracellular matrix-associated heparin-binding protein that can mediate cell adhesion,
51 , and subsequent studies identified a second heparin-binding protein that co-purified with FGF-1.
52 mily, is an extracellular matrix-associated, heparin-binding protein that mediates cell adhesion, pro
53 ducible midkine gene encodes a highly basic, heparin-binding protein that possesses potential functio
54 f amphoterin and HB-GAM (Kd = 0.3-8 nM), two heparin-binding proteins that are developmentally regula
56 lthough they are traditionally classified as heparin-binding proteins, under normal physiological con
58 modimer, we sought to determine whether this heparin-binding protein was involved in the release of F
60 or glypican-1 insofar as no other identified heparin-binding proteins were isolated using our affinit
63 ere designed based on consensus sequences in heparin-binding proteins: XBBXBX and XBBBXXBX, where X a
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