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1 is inhibited by FGF-1, heparan sulfate, and heparinoids.
3 Surprisingly, heparin, chemically modified heparinoids, and monoclonal antibodies to heparan sulfat
4 anticoagulation regimens, including heparin, heparinoids, antithrombins, or fibrinolytics (e.g., tiss
7 lly we showed that certain low anticoagulant heparinoids can inhibit properdin binding to tubular HS,
8 bitor (e.g., argatroban or lepirudin) or the heparinoid danaparoid (where available) reduces the risk
9 Stroke Treatment, a randomized trial of the heparinoid danaparoid, were analyzed to determine whethe
10 contrast, smaller oligomers or the synthetic heparinoid fondaparinux were not able to block the bindi
11 ed with unfractionated heparin, the modified heparinoids had inhibitory activity in this general orde
13 Interaction of anti-thrombin III (AT) with heparinoids is studied using a mixture of oligoheparin m
16 vides access to structural variants of small heparinoid oligomers as versatile building blocks for ge
17 t compared heparins (unfractionated heparin, heparinoids, or low-molecular-weight heparin) with aspir
18 n be manipulated with some low anticoagulant heparinoids, which can be important for preventing compl
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