ライフサイエンスコーパス: hepatic artery thrombosis

1 he primary graft and 1 from complications of hepatic artery thrombosis).

2 lure due to chronic rejection and late onset hepatic artery thrombosis.

3 nsplantation, was frequently associated with hepatic artery thrombosis.

4 ation is associated with a high incidence of hepatic artery thrombosis.

5 thrombosis) enable prediction of subsequent hepatic artery thrombosis.

6 No patient had evidence of hepatic artery thrombosis.

7 he most common etiologies of graft loss were hepatic artery thrombosis (33.4%), acute or chronic reje

8 days following primary transplant) included hepatic artery thrombosis (5), chronic rejection (4), se

9 and the main cause for retransplantation was hepatic artery thrombosis (6 [2%] vs 17 [6%]).

10 ow Protein S may be causally associated with hepatic artery thrombosis after OLT.

11 ecurrent cytomegalovirus activation, one has hepatic artery thrombosis and one is likely to have pers

12 In an attempt to decrease the incidence of hepatic artery thrombosis and to increase collaboration

13 function, 50% for chronic rejection, 60% for hepatic artery thrombosis, and 60% for recurrent HCV.

14 -grafts were constructed for recipients with hepatic artery thrombosis, and double donor arteries wer

15 ence of ischemic type biliary strictures and hepatic artery thrombosis, and evaluated the causes of g

16 uded primary nonfunction, chronic rejection, hepatic artery thrombosis, and recurrent disease.

17 transplants; mainly for primary nonfunction, hepatic artery thrombosis, and recurrent primary disease

18 he main indication for retransplantation was hepatic artery thrombosis, and the major cause of death

19 Although duplex US remains a good screen for hepatic artery thrombosis, angiography is strongly recom

20 ed (more than 4 weeks after transplantation) hepatic artery thrombosis are less clearly defined.

21 er recipients who smoke have higher rates of hepatic artery thrombosis, biliary complications, and ma

22 ve a significantly higher incidence of early hepatic artery thrombosis compared with non-FAP transpla

23 Early hepatic artery thrombosis (eHAT) after liver transplanta

24 The incidence of hepatic artery thrombosis has decreased from 22% to 0% w

25 ansplant recipients were found to have early hepatic artery thrombosis (HAT) after a median of 7 post

26 tation, and outcome of management of delayed hepatic artery thrombosis (HAT) after liver transplant (

27 Whilst causes of hepatic artery thrombosis (HAT) after liver transplantat

28 Hepatic artery thrombosis (HAT) after liver transplantat

29 Hepatic artery thrombosis (HAT) after liver transplantat

30 igate whether center volume impacts the rate hepatic artery thrombosis (HAT) and patient survival aft

31 Although the clinical features of early hepatic artery thrombosis (HAT) are well defined, the fe

32 Hepatic artery thrombosis (HAT) can be a devastating com

33 Hepatic artery thrombosis (HAT) did not occur in any pat

34 The incidence of hepatic artery thrombosis (HAT) following orthotopic liv

35 Hepatic artery thrombosis (HAT) increases morbidity and

36 Hepatic artery thrombosis (HAT) is a cause of morbidity

37 trahepatic biliary strictures (IHBS) without hepatic artery thrombosis (HAT) is a serious complicatio

38 Hepatic artery thrombosis (HAT) is a significant cause o

39 Hepatic artery thrombosis (HAT) remains a devastating co

40 The most common predisposing factor was hepatic artery thrombosis (HAT), which occurred in eight

41 reasons (primary graft nonfunction (PGNF) or hepatic artery thrombosis [HAT]), survival was significa

42 Other causes for re-OLT included hepatic artery thrombosis in 10 (2.6%), chronic rejectio

43 e believe that emergent revascularization of hepatic artery thrombosis in asymptomatic patients and r

44 ical implications in the prevention of early hepatic artery thrombosis in FAP patients after liver tr

45 A single vascular complication occurred (one hepatic artery thrombosis in group B).

46 nastomosis and should lower the incidence of hepatic artery thrombosis in the small-caliber left hepa

47 ibed as an independent risk factor for early hepatic artery thrombosis, more studies to understand th

48 ventional angiography or surgery: transplant hepatic artery thrombosis (n = 3) or stenosis (n = 3), p

49 acute rejection at day 7, the development of hepatic artery thrombosis, nonanastomotic biliary strict

50 Reoperations identified two cases of hepatic artery thrombosis not previously identified by d

51 In addition, we reported two cases of hepatic artery thrombosis, one case of wound dehiscence

52 titis C (P<0.0001), as well as occurrence of hepatic artery thrombosis (P=0.0018) and prolonged cold

53 The incidence of hepatic artery thrombosis, portal vein stenosis/thrombos

54 There were no significant differences in hepatic artery thrombosis, portal vein thrombosis, prima

55 ant centers in North America with the lowest hepatic artery thrombosis rate and biliary complication

56 en advocated and has resulted in a decreased hepatic artery thrombosis rate in both the adult and ped

57 Complications included one case each of hepatic artery thrombosis requiring retransplantation, b

58 reas 97 patients required retransplantation; hepatic artery thrombosis was the most common indication

59 The occurrence is, partly, attributed to hepatic artery thrombosis, which is considered to be the

60 Early detection of hepatic artery thrombosis, with subsequent correction an