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1 ation leading to a marked reduction in fetal hepatic blood flow.
2 hildren by inhibiting lipolysis and reducing hepatic blood flow.
3 duced by temporal and segmental occlusion of hepatic blood flow.
4 techniques were used to quantify changes in hepatic blood flow.
5 acaval shunt (HGPCS) on portal and effective hepatic blood flow.
6 possibly a result of excessive diminution of hepatic blood flow.
8 galactose (V(max)/K(m)) from measurements of hepatic blood flow and arterial and liver vein blood gal
9 er Transplantation Cohort Study was to study hepatic blood flow and effect of portal flow modulation
13 asive longitudinal hemodynamic monitoring of hepatic blood flow before and after transjugular intrahe
16 h decreased liver injury, enhanced effective hepatic blood flow, decreased cytokines, and prevention
17 ively to undergo TIPS or HGPCS had effective hepatic blood flow determined 1 day preshunt and 5 days
21 rved after I/R may be due to improvements in hepatic blood flow secondary to altered hepatic hemodyna
23 been proposed that this therapy may improve hepatic blood flow via the vasodilating properties of PG
24 of (11)C-CSar from hepatocytes to blood, and hepatic blood flow was 25% higher than in experiments wi
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