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1 ation leading to a marked reduction in fetal hepatic blood flow.
2 hildren by inhibiting lipolysis and reducing hepatic blood flow.
3 duced by temporal and segmental occlusion of hepatic blood flow.
4  techniques were used to quantify changes in hepatic blood flow.
5 acaval shunt (HGPCS) on portal and effective hepatic blood flow.
6 possibly a result of excessive diminution of hepatic blood flow.
7 preservation, and enhanced energy charge and hepatic blood flow after reperfusion.
8 galactose (V(max)/K(m)) from measurements of hepatic blood flow and arterial and liver vein blood gal
9 er Transplantation Cohort Study was to study hepatic blood flow and effect of portal flow modulation
10 rtant in the regulation of liver metabolism, hepatic blood flow, and bile secretion.
11                               Intestinal and hepatic blood flow (assessed by laser Doppler flowmetry)
12 asive longitudinal hemodynamic monitoring of hepatic blood flow before and after TIPS placement.
13 asive longitudinal hemodynamic monitoring of hepatic blood flow before and after transjugular intrahe
14                 Epinephrine caused increased hepatic blood flow centrally (171.1% +/- 31.7) but not p
15                                    Effective hepatic blood flow decreased during the HS period from a
16 h decreased liver injury, enhanced effective hepatic blood flow, decreased cytokines, and prevention
17 ively to undergo TIPS or HGPCS had effective hepatic blood flow determined 1 day preshunt and 5 days
18                                    Effective hepatic blood flow (EHBF) by galactose clearance, wet-dr
19                                          The hepatic blood flow of male Sprague Dawley rats was subje
20                                    Effective hepatic blood flow remained constant in the sham groups
21 rved after I/R may be due to improvements in hepatic blood flow secondary to altered hepatic hemodyna
22                                        Total hepatic blood flow (THBF) and HM was measured along with
23  been proposed that this therapy may improve hepatic blood flow via the vasodilating properties of PG
24 of (11)C-CSar from hepatocytes to blood, and hepatic blood flow was 25% higher than in experiments wi
25                                              Hepatic blood flow was determined by indocyanine green i
26                                    Effective hepatic blood flow was diminished significantly after TI
27 eased slowly and peaked 20 to 30 hours after hepatic blood flow was restored.

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