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1 lost or significantly downregulated in human hepatic cancer.
2 inactivation of nrf1 in the liver developed hepatic cancer.
3 A system to treat patients with unresectable hepatic cancer.
4 d CaMKIV may represent potential targets for hepatic cancer.
5 eful therapeutic targets in the treatment of hepatic cancers.
6 alternative in the treatment of unresectable hepatic cancers.
7 tumor types including breast, prostate, and hepatic cancers, as well as the process of bone metastas
8 ly hFX increased transduction of several non-hepatic cancer cell lines and Chinese hamster ovary (CHO
9 KK2 protein is highly expressed in all eight hepatic cancer cell lines evaluated and is markedly up-r
11 n results in significant regression of bulky hepatic cancers confined to the liver in the majority of
12 r Liver transplantation (LTx), recurrence of hepatic cancer, de novo cancers, and donor-transmitted c
14 vel bacterium may cause an increased risk of hepatic cancer induction in susceptible strains of mice.
18 orting-isolated EpCAM(+) HCC cells displayed hepatic cancer stem cell-like traits including the abili
20 ochondria by autophagy, positively regulates hepatic cancer stem cells (CSCs) by suppressing the tumo
21 , only the former is expressed dominantly in hepatic cancer stem cells and correlates significantly t
22 upregulation occurs in HBV-mediated HCCs and hepatic cancer stem cells, by a mechanism not understood
23 tocellular carcinoma (FLC) is a rare primary hepatic cancer that develops in children and young adult
24 e resections for esophageal, pancreatic, and hepatic cancer were performed at high volume centers, bu
25 tality rates for esophageal, pancreatic, and hepatic cancers were inversely related to hospital volum
26 of patients with esophageal, pancreatic, and hepatic cancers were treated at high-volume centers.
27 ity is strongly associated with prostate and hepatic cancers, whereas reduced CaMKK2 activity has bee
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